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1. Full Text Dietary Capsaicin Reduces Obesity-induced Insulin Resistance and Hepatic Steatosis in Obese Mice Fed a High-fat Diet
by Kang, Ji‐Hye and Tsuyoshi, Goto and Han, In‐Seob and Kawada, Teruo and Kim, Young Min and Yu, Rina
Obesity (Silver Spring, Md.), ISSN 1930-7381, 04/2010, Volume 18, Issue 4, pp. 780 - 787
Endocrinology & Metabolism | Life Sciences & Biomedicine | Nutrition & Dietetics | Science & Technology | Glucose Intolerance - metabolism | Dietary Fats - metabolism | Obesity - drug therapy | Capsaicin - pharmacology | Capsaicin - therapeutic use | Male | Insulin - blood | RNA, Messenger - metabolism | Adipose Tissue - metabolism | TRPV Cation Channels - metabolism | Flow Cytometry | Leptin - blood | Glucose Intolerance - etiology | Adiponectin - genetics | Dietary Fats - administration & dosage | Anti-Inflammatory Agents - therapeutic use | Adiponectin - metabolism | Disease Models, Animal | Hypoglycemic Agents - therapeutic use | Gene Expression | Fatty Liver - metabolism | Anti-Inflammatory Agents - pharmacology | Obesity - complications | Liver - metabolism | Mice, Inbred C57BL | Insulin Resistance | PPAR alpha - genetics | Reverse Transcriptase Polymerase Chain Reaction | Fatty Liver - drug therapy | TRPV Cation Channels - genetics | Hypoglycemic Agents - pharmacology | Obesity - metabolism | Triglycerides - metabolism | Animals | Lipid Metabolism - drug effects | Inflammation - genetics | Mice, Obese | Mice | PPAR alpha - metabolism | Blood Glucose - metabolism | Dietary Supplements | PPAR alpha - chemistry | Fatty Liver - etiology | Index Medicus
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2. Full Text Activation of TRPV1 reduces vascular lipid accumulation and attenuates atherosclerosis
by Ma, Liqun and Zhong, Jian and Zhao, Zhigang and Luo, Zhidan and Ma, Shuangtao and Sun, Jing and He, Hongbo and Zhu, Tianqi and Liu, Daoyan and Zhu, Zhiming and Tepel, Martin
Cardiovascular research, ISSN 0008-6363, 12/2011, Volume 92, Issue 3, pp. 504 - 513
Transient receptor potential vanilloid type-1 | Capsaicin | Vascular smooth muscle cells | Atherosclerosis | Cardiac & Cardiovascular Systems | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Cardiology. Vascular system | Biological and medical sciences | Medical sciences | Atherosclerosis (general aspects, experimental research) | Blood and lymphatic vessels | Immunohistochemistry | Dietary Fats - metabolism | Muscle, Smooth, Vascular - metabolism | Calcium - metabolism | Capsaicin - pharmacology | Atherosclerosis - genetics | Aorta - metabolism | RNA, Messenger - metabolism | Apolipoproteins E - metabolism | Aortic Diseases - metabolism | TRPV Cation Channels - deficiency | Dose-Response Relationship, Drug | TRPV Cation Channels - metabolism | Time Factors | Dietary Fats - administration & dosage | Polymerase Chain Reaction | ATP-Binding Cassette Transporters - metabolism | Aortic Diseases - prevention & control | ATP Binding Cassette Transporter 1 | Myocytes, Smooth Muscle - drug effects | Myocytes, Smooth Muscle - metabolism | Aortic Diseases - pathology | Disease Models, Animal | Muscle, Smooth, Vascular - drug effects | Cyclic AMP-Dependent Protein Kinases - metabolism | Diterpenes - pharmacology | Atherosclerosis - pathology | Tumor Suppressor Proteins - metabolism | Aorta - drug effects | Mice, Inbred C57BL | TRPV Cation Channels - agonists | Cells, Cultured | Receptors, LDL - metabolism | Atherosclerosis - metabolism | Blotting, Western | TRPV Cation Channels - genetics | Mice, Knockout | Aortic Diseases - genetics | Animals | Apolipoproteins E - genetics | Lipid Metabolism - drug effects | Mice | Atherosclerosis - prevention & control | Calcineurin - metabolism | Index Medicus
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3. Full Text Mechanisms for the coupling of cannabinoid receptors to intracellular calcium mobilization in rat insulinoma β-cells
by De Petrocellis, Luciano and Marini, Pietro and Matias, Isabel and Moriello, Aniello Schiano and Starowicz, Katarzyna and Cristino, Luigia and Nigam, Santosh and Di Marzo, Vincenzo
Experimental cell research, ISSN 0014-4827, 2007, Volume 313, Issue 14, pp. 2993 - 3004
Bombesin | Anandamide, TRPV1 | Cannabinoid receptor | Calcium, insulinoma β-cells | Oncology | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Receptor, Cannabinoid, CB2 - agonists | Calcium - metabolism | Capsaicin - metabolism | Cell Line, Tumor - drug effects | Receptor, Cannabinoid, CB2 - antagonists & inhibitors | Receptor, Cannabinoid, CB2 - genetics | Neuroprotective Agents - metabolism | Dose-Response Relationship, Drug | TRPV Cation Channels - metabolism | Arachidonic Acids - metabolism | Receptor, Cannabinoid, CB1 - agonists | Colforsin - metabolism | TRPV Cation Channels - antagonists & inhibitors | Dronabinol - analogs & derivatives | Pyrrolidinones - metabolism | Estrenes - metabolism | Phosphoinositide Phospholipase C - metabolism | Sensory System Agents - metabolism | Cannabinoids - metabolism | Cell Line, Tumor - metabolism | Phosphoinositide Phospholipase C - antagonists & inhibitors | Enzyme Inhibitors - metabolism | TRPV Cation Channels - agonists | Neurotransmitter Agents - metabolism | Pertussis Toxin - metabolism | Rats | TRPV Cation Channels - genetics | Receptor, Cannabinoid, CB1 - metabolism | Receptor, Cannabinoid, CB2 - metabolism | Animals | Receptor, Cannabinoid, CB1 - genetics | Insulinoma - metabolism | Thapsigargin - metabolism | Signal Transduction - physiology | Receptor, Cannabinoid, CB1 - antagonists & inhibitors | Dronabinol - metabolism | Bombesin - metabolism | Index Medicus
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4. Full Text Transient receptor potential vanilloid type‐1 channel regulates diet‐induced obesity, insulin resistance, and leptin resistance
by Lee, Eunjung and Jung, Dae Young and Kim, Jong Hun and Patel, Payai R and Hu, Xiaodi and Lee, Yongjin and Azuma, Yoshihiro and Wang, Hsun‐Fan and Tsitsilianos, Nicholas and Shafiq, Umber and Kwon, Jung Yeon and Lee, Hyong Joo and Lee, Ki Won and Kim, Jason K
The FASEB journal, ISSN 0892-6638, 08/2015, Volume 29, Issue 8, pp. 3182 - 3192
hyperinsulinemic‐euglycemic clamp | aging | glucose metabolism | capsaicin | Aging | Glucose metabolism | Capsaicin | Hyperinsulinemic-euglycemic clamp | Biochemistry & Molecular Biology | Biology | Life Sciences & Biomedicine - Other Topics | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Dietary Fats - metabolism | Leptin - metabolism | Mice, Inbred C57BL | Cells, Cultured | Diet, High-Fat - adverse effects | Male | Diabetes Mellitus, Type 2 - metabolism | Obesity - metabolism | TRPV Cation Channels - metabolism | Insulin - metabolism | Animals | Insulin Resistance - physiology | Glucose - metabolism | Adipose Tissue, Brown - metabolism | Mice | Energy Metabolism - physiology | Aging - metabolism | Index Medicus | hyperinsulinemic-euglycemic clamp | Research Communication
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5. Full Text The endovanilloid/endocannabinoid system in human osteoclasts: Possible involvement in bone formation and resorption
by Rossi, F and Siniscalco, D and Luongo, L and De Petrocellis, L and Bellini, G and Petrosino, S and Torella, M and Santoro, C and Nobili, B and Perrotta, S and Di Marzo, V and Maione, S
Bone (New York, N.Y.), ISSN 8756-3282, 2008, Volume 44, Issue 3, pp. 476 - 484
Orthopedics | Osteoclasts | Bone turnover | Calcium | Vanilloids/cannabinoids receptors | Anandamide | Life Sciences & Biomedicine | Endocrinology & Metabolism | Science & Technology | Fundamental and applied biological sciences. Psychology | Biological and medical sciences | Vertebrates: anatomy and physiology, studies on body, several organs or systems | Vertebrates: osteoarticular system, musculoskeletal system | Skeleton and joints | Cannabinoid Receptor Modulators - metabolism | Osteogenesis - physiology | Calcium - metabolism | Capsaicin - metabolism | Humans | Tartrate-Resistant Acid Phosphatase | Osteoclasts - cytology | TRPV Cation Channels - metabolism | Acid Phosphatase - metabolism | Arachidonic Acids - metabolism | Isoenzymes - metabolism | Bone and Bones - metabolism | Cell Differentiation | Osteoclasts - physiology | Endocannabinoids | Bone and Bones - cytology | Amidohydrolases - metabolism | Bone Resorption | Cathepsin K | Isoenzymes - genetics | Cells, Cultured | Polyunsaturated Alkamides - metabolism | Phospholipase D - metabolism | Receptor, Cannabinoid, CB1 - metabolism | Receptor, Cannabinoid, CB2 - metabolism | Animals | Cathepsins - metabolism | Mice | Acid Phosphatase - genetics | Cathepsins | Osteoporosis | Phospholipases | Fatty acids | Index Medicus
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6. Full Text NGF rapidly increases membrane expression of TRPV1 heat-gated ion channels
by Zhang, Xuming and McNaughton, Peter A and Huang, Jiehong
The EMBO journal, ISSN 0261-4189, 12/2005, Volume 24, Issue 24, pp. 4211 - 4223
pain | sensory transduction | neurotrophic factor | membrane trafficking | tyrosine kinase | Neurotrophic factor | Tyrosine kinase | Membrane trafficking | Pain | Sensory transduction | Biochemistry & Molecular Biology | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Immunohistochemistry | Phosphorylation | Ion Channels | Calcium - metabolism | Capsaicin - pharmacology | Humans | Electrophysiology | Molecular Sequence Data | Immunoblotting | Phosphatidylinositol 3-Kinases - metabolism | Recombinant Fusion Proteins - metabolism | TRPV Cation Channels - metabolism | Transfection | Nerve Growth Factor - physiology | Time Factors | Vanadates - pharmacology | src-Family Kinases - metabolism | Neurons - metabolism | Tyrosine - chemistry | Amino Acid Sequence | Cell Line | Sodium - pharmacology | Nerve Growth Factor - metabolism | Signal Transduction | Cells, Cultured | Gene Expression Regulation | Glutathione Transferase - metabolism | DNA - metabolism | Hot Temperature | Plasmids - metabolism | Sequence Homology, Amino Acid | Animals | Receptor, trkA - metabolism | Models, Biological | Protein Binding | Mice | Mutation | DNA, Complementary - metabolism | Index Medicus
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7. Full Text Endovanilloid control of pain modulation by the rostroventromedial medulla in an animal model of diabetic neuropathy
by Silva, M and Martins, D and Charrua, A and Piscitelli, F and Tavares, I and Morgado, C and Di Marzo, V
Neuropharmacology, ISSN 0028-3908, 08/2016, Volume 107, pp. 49 - 57
RVM | Descending pain modulation | FAAH | TRPV1 | Diabetic neuropathic pain | Periaqueductal grey | Pharmacology & Pharmacy | Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Amidohydrolases - metabolism | Endocannabinoids - metabolism | Chronic Pain - drug therapy | Rats, Wistar | Capsaicin - pharmacology | TRPV Cation Channels - agonists | Analgesics, Non-Narcotic - pharmacology | Medulla Oblongata - metabolism | Male | Diabetic Neuropathies - drug therapy | Nociceptive Pain - drug therapy | Polyunsaturated Alkamides - metabolism | RNA, Messenger - metabolism | Formaldehyde | Nociceptive Pain - metabolism | Oleic Acids - metabolism | Chronic Pain - metabolism | TRPV Cation Channels - metabolism | Arachidonic Acids - metabolism | Animals | Ethanolamines - metabolism | Diabetes Mellitus, Experimental - metabolism | Diabetic Neuropathies - metabolism | Palmitic Acids - metabolism | Enzymes | Diabetic neuropathies | Care and treatment | Pain | RNA | Streptozocin | Analysis | Hydrolases | Fatty acids | Chronic pain | Index Medicus
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8. Full Text Trypsin IV or Mesotrypsin and p23 Cleave Protease-activated Receptors 1 and 2 to Induce Inflammation and Hyperalgesia
by Wolfgang Knecht and Graeme S. Cottrell and Silvia Amadesi and Johanna Mohlin and Anita Skåregärde and Karin Gedda and Anders Peterson and Kevin Chapman and Morley D. Hollenberg and Nathalie Vergnolle and Nigel W. Bunnett
The Journal of biological chemistry, ISSN 0021-9258, 09/2007, Volume 282, Issue 36, pp. 26089 - 26100
Life Sciences & Biomedicine | Biochemistry & Molecular Biology | Science & Technology | Hyperalgesia - chemically induced | Inflammation - chemically induced | Inflammation - pathology | Receptors, Proteinase-Activated - metabolism | Capsaicin - pharmacology | Edema - genetics | Receptor, PAR-1 - metabolism | Humans | Male | Receptor, PAR-1 - deficiency | Receptor, PAR-2 - deficiency | Hyperalgesia - pathology | Inflammation - metabolism | Trypsin Inhibitors - chemistry | Trypsin - pharmacology | Trypsin - genetics | Granulocytes - pathology | Granulocytes - metabolism | Nociceptors - pathology | Recombinant Proteins - metabolism | Hyperalgesia - metabolism | Rats | Recombinant Proteins - chemistry | Recombinant Proteins - genetics | Recombinant Proteins - pharmacology | Trypsin - metabolism | Rats, Sprague-Dawley | Mice, Knockout | Edema - metabolism | Animals | Hyperalgesia - genetics | Calcium Signaling - drug effects | Enteropeptidase - chemistry | Ganglia, Spinal - pathology | Inflammation - genetics | Receptor, PAR-2 - physiology | Receptors, Thrombin - metabolism | Mice | Nociceptors - metabolism | Aprotinin - chemistry | Edema - chemically induced | Pain Measurement | Edema - pathology | Trypsin - chemistry | Ganglia, Spinal - metabolism | Index Medicus | Nociceptors/metabolism | Aprotinin/chemistry | Edema/chemically induced | Recombinant Proteins/chemistry | Receptor, PAR-1/deficiency | Enteropeptidase/chemistry | Capsaicin/pharmacology | Inflammation/chemically induced | Receptors, Proteinase-Activated/metabolism | Granulocytes/metabolism | Trypsin Inhibitors/chemistry | Ganglia, Spinal/metabolism | Receptor, PAR-2/deficiency | Calcium Signaling/drug effects | Trypsin/chemistry | Hyperalgesia/chemically induced | Receptors, Thrombin/metabolism
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