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Obesity, ISSN 1930-7381, 04/2010, Volume 18, Issue 4, pp. 780 - 787
Obesity‐induced inflammation contributes to the development of obesity‐related metabolic disorders such as insulin resistance, type 2 diabetes, fatty liver... 
CRUCIAL ROLE | PPAR-ALPHA | METABOLIC SYNDROME | ACTIVATION | NUTRITION & DIETETICS | ADIPONECTIN | INFLAMMATION | ENDOCRINOLOGY & METABOLISM | MONOCYTE CHEMOATTRACTANT PROTEIN-1 | RECEPTOR | TNF-ALPHA | ADIPOSE-TISSUE | Glucose Intolerance - metabolism | Dietary Fats - metabolism | Obesity - drug therapy | Capsaicin - pharmacology | Capsaicin - therapeutic use | Male | Insulin - blood | RNA, Messenger - metabolism | Adipose Tissue - metabolism | TRPV Cation Channels - metabolism | Flow Cytometry | Leptin - blood | Glucose Intolerance - etiology | Adiponectin - genetics | Dietary Fats - administration & dosage | Anti-Inflammatory Agents - therapeutic use | Adiponectin - metabolism | Disease Models, Animal | Hypoglycemic Agents - therapeutic use | Gene Expression | Fatty Liver - metabolism | Anti-Inflammatory Agents - pharmacology | Obesity - complications | Liver - metabolism | Mice, Inbred C57BL | Insulin Resistance | PPAR alpha - genetics | Reverse Transcriptase Polymerase Chain Reaction | Fatty Liver - drug therapy | TRPV Cation Channels - genetics | Hypoglycemic Agents - pharmacology | Obesity - metabolism | Triglycerides - metabolism | Animals | Lipid Metabolism - drug effects | Inflammation - genetics | Mice, Obese | Mice | PPAR alpha - metabolism | Blood Glucose - metabolism | Dietary Supplements | PPAR alpha - chemistry | Fatty Liver - etiology
Journal Article
Cardiovascular Research, ISSN 0008-6363, 12/2011, Volume 92, Issue 3, pp. 504 - 513
Aims Activation of transient receptor potential vanilloid type-1 (TRPV1) channels may affect lipid storage and the cellular inflammatory response. Now, we... 
Transient receptor potential vanilloid type-1 | Capsaicin | Vascular smooth muscle cells | Atherosclerosis | CARDIAC & CARDIOVASCULAR SYSTEMS | DEPENDENT PROTEIN-KINASE | RECEPTOR | MECHANISMS | DEFICIENT MICE | CHOLESTEROL EFFLUX | CALCINEURIN | SMOOTH-MUSCLE-CELLS | CHANNELS | EXPRESSION | Immunohistochemistry | Dietary Fats - metabolism | Muscle, Smooth, Vascular - metabolism | Calcium - metabolism | Capsaicin - pharmacology | Atherosclerosis - genetics | Aorta - metabolism | RNA, Messenger - metabolism | Apolipoproteins E - metabolism | Aortic Diseases - metabolism | TRPV Cation Channels - deficiency | Dose-Response Relationship, Drug | TRPV Cation Channels - metabolism | Time Factors | Dietary Fats - administration & dosage | Polymerase Chain Reaction | ATP-Binding Cassette Transporters - metabolism | Aortic Diseases - prevention & control | ATP Binding Cassette Transporter 1 | Myocytes, Smooth Muscle - drug effects | Myocytes, Smooth Muscle - metabolism | Aortic Diseases - pathology | Disease Models, Animal | Muscle, Smooth, Vascular - drug effects | Cyclic AMP-Dependent Protein Kinases - metabolism | Diterpenes - pharmacology | Atherosclerosis - pathology | Tumor Suppressor Proteins - metabolism | Aorta - drug effects | Mice, Inbred C57BL | TRPV Cation Channels - agonists | Cells, Cultured | Receptors, LDL - metabolism | Atherosclerosis - metabolism | Blotting, Western | TRPV Cation Channels - genetics | Mice, Knockout | Aortic Diseases - genetics | Animals | Apolipoproteins E - genetics | Lipid Metabolism - drug effects | Mice | Atherosclerosis - prevention & control | Calcineurin - metabolism
Journal Article
Experimental Cell Research, ISSN 0014-4827, 2007, Volume 313, Issue 14, pp. 2993 - 3004
In RIN m5F rat insulinoma β-cells, agonists at cannabinoid CB receptors modulate insulin release. Here we investigated in these cells the effect of the... 
Bombesin | Anandamide, TRPV1 | Cannabinoid receptor | Calcium, insulinoma β-cells | SIGNAL-TRANSDUCTION PATHWAYS | VANILLOID VR1 RECEPTORS | BOMBESIN-LIKE PEPTIDES | PHOSPHOLIPASE-C | G-PROTEINS | ENDOCANNABINOID SYSTEM | CA2+ SIGNALS | CELL BIOLOGY | insulinoma beta-cells | ENERGY-BALANCE | ONCOLOGY | anandamide | TRPV1cannabinoid receptor bombesin calcium | CB1 RECEPTORS | Receptor, Cannabinoid, CB2 - agonists | Calcium - metabolism | Capsaicin - metabolism | Cell Line, Tumor - drug effects | Receptor, Cannabinoid, CB2 - antagonists & inhibitors | Receptor, Cannabinoid, CB2 - genetics | Neuroprotective Agents - metabolism | Dose-Response Relationship, Drug | TRPV Cation Channels - metabolism | Arachidonic Acids - metabolism | Receptor, Cannabinoid, CB1 - agonists | Colforsin - metabolism | TRPV Cation Channels - antagonists & inhibitors | Dronabinol - analogs & derivatives | Pyrrolidinones - metabolism | Estrenes - metabolism | Phosphoinositide Phospholipase C - metabolism | Sensory System Agents - metabolism | Cannabinoids - metabolism | Cell Line, Tumor - metabolism | Phosphoinositide Phospholipase C - antagonists & inhibitors | Enzyme Inhibitors - metabolism | TRPV Cation Channels - agonists | Neurotransmitter Agents - metabolism | Pertussis Toxin - metabolism | Rats | TRPV Cation Channels - genetics | Receptor, Cannabinoid, CB1 - metabolism | Receptor, Cannabinoid, CB2 - metabolism | Animals | Receptor, Cannabinoid, CB1 - genetics | Insulinoma - metabolism | Thapsigargin - metabolism | Signal Transduction - physiology | Receptor, Cannabinoid, CB1 - antagonists & inhibitors | Dronabinol - metabolism | Bombesin - metabolism
Journal Article
Bone, ISSN 8756-3282, 2008, Volume 44, Issue 3, pp. 476 - 484
Journal Article
The EMBO Journal, ISSN 0261-4189, 12/2005, Volume 24, Issue 24, pp. 4211 - 4223
Nociceptors, or pain‐sensitive receptors, are unique among sensory receptors in that their sensitivity is increased by noxious stimulation. This process,... 
pain | sensory transduction | neurotrophic factor | membrane trafficking | tyrosine kinase | Neurotrophic factor | Tyrosine kinase | Membrane trafficking | Pain | Sensory transduction | PROTEIN-KINASE-C | TYROSINE PHOSPHORYLATION | PHOSPHATIDYLINOSITOL 3-KINASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | CAPSAICIN-RECEPTOR | SENSORY NEURONS | NERVE GROWTH-FACTOR | CELL BIOLOGY | NOCICEPTIVE NEURONS | ADULT-RAT | VANILLOID RECEPTOR-1 | NOXIOUS HEAT | Immunohistochemistry | Phosphorylation | Ion Channels | Calcium - metabolism | Capsaicin - pharmacology | Humans | Electrophysiology | Molecular Sequence Data | Immunoblotting | Phosphatidylinositol 3-Kinases - metabolism | Recombinant Fusion Proteins - metabolism | TRPV Cation Channels - metabolism | Transfection | Nerve Growth Factor - physiology | Time Factors | Vanadates - pharmacology | src-Family Kinases - metabolism | Neurons - metabolism | Tyrosine - chemistry | Amino Acid Sequence | Cell Line | Sodium - pharmacology | Nerve Growth Factor - metabolism | Signal Transduction | Cells, Cultured | Gene Expression Regulation | Glutathione Transferase - metabolism | DNA - metabolism | Hot Temperature | Plasmids - metabolism | Sequence Homology, Amino Acid | Animals | Receptor, trkA - metabolism | Models, Biological | Protein Binding | Mice | Mutation | DNA, Complementary - metabolism
Journal Article
Journal Article
Neuropharmacology, ISSN 0028-3908, 08/2016, Volume 107, pp. 49 - 57
Journal Article
Journal of Diabetes and Its Complications, ISSN 1056-8727, 2013, Volume 27, Issue 3, pp. 287 - 292
Abstract With increasing lifespan, therapeutic interventions for the treatment of disorders such as type 2 diabetes mellitus are in great demand. Despite... 
Endocrinology & Metabolism | GLP-1 | TRPV1 | Glucose | Hypothalamus | Insulin | GENE-RELATED PEPTIDE | IMMUNOHISTOCHEMICAL LOCALIZATION | BROWN ADIPOSE-TISSUE | GLUCAGON-LIKE PEPTIDE-1 | PARAVENTRICULAR NUCLEUS | INSULIN-RESISTANCE | ENDOCRINOLOGY & METABOLISM | CAPSICUM-FRUTESCENS | LIVER-RELATED NEURONS | CAPSAICIN-DESENSITIZED RATS | GLUCOSE-PRODUCTION | Obesity - diet therapy | Adipose Tissue, White - immunology | Capsaicin - metabolism | Capsaicin - therapeutic use | Hypoglycemic Agents - metabolism | Obesity - immunology | Humans | Adipose Tissue, White - metabolism | Diabetes Mellitus, Type 2 - diet therapy | Glucagon-Like Peptide 1 - blood | Diabetes Mellitus, Type 2 - metabolism | Insulin - blood | TRPV Cation Channels - metabolism | Insulin-Secreting Cells - metabolism | Liver - immunology | Diabetes Mellitus, Type 2 - immunology | Autonomic Nervous System - metabolism | Insulin Secretion | Hypoglycemic Agents - therapeutic use | Hypothalamus - immunology | Glucagon-Like Peptide 1 - metabolism | Liver - metabolism | TRPV Cation Channels - agonists | Insulin Resistance | Insulin-Secreting Cells - immunology | Obesity - metabolism | Insulin - metabolism | Animals | Hypothalamus - metabolism | Energy Metabolism | Autonomic Nervous System - immunology | Dietary Supplements | Type 2 diabetes | Glucose metabolism | Glucagon | Dietary supplements | Physiological aspects | Health aspects | Dextrose | glucose | insulin | hypothalamus
Journal Article
Nature, ISSN 0028-0836, 01/2008, Volume 451, Issue 7176, pp. 330 - 334
Inflammatory diseases and neuropathic insults are frequently accompanied by severe and debilitating pain, which can become chronic and often unresponsive to... 
Inflammation - chemically induced | Analgesics - pharmacology | Spinal Cord - drug effects | Spinal Cord - metabolism | Diazepam - metabolism | Rats, Wistar | Capsaicin - pharmacology | Analgesics - metabolism | Male | Ganglia, Spinal - cytology | Pain - chemically induced | Receptors, GABA-A - chemistry | Pain - metabolism | Brain - physiology | Formaldehyde | Protein Subunits - metabolism | Diazepam - pharmacology | Fluorobenzenes - pharmacology | Protein Isoforms - metabolism | Inflammation - drug therapy | Pain - drug therapy | Protein Isoforms - chemistry | Neurons - metabolism | Spinal Cord - cytology | Neurons - drug effects | Chronic Disease - drug therapy | Disease Models, Animal | Analgesics - therapeutic use | Fluorobenzenes - metabolism | Pain - prevention & control | Diazepam - administration & dosage | Analgesics - administration & dosage | Rats | Hot Temperature | Organ Specificity | Brain - drug effects | Triazoles - pharmacology | Triazoles - metabolism | Animals | Receptors, GABA-A - genetics | Mice | Protein Subunits - chemistry | Receptors, GABA-A - metabolism | Spinal Cord - physiopathology | Ganglia, Spinal - metabolism | Care and treatment | Receptors | Analgesics | GABA | Development and progression | Dosage and administration | Research | Health aspects | Chronic pain | Neurosciences | Neurotransmitters | NMR | Rodents | Pain management | Pharmacology | Nuclear magnetic resonance | Binding sites | Inflammatory diseases | Index Medicus
Journal Article
Gastroenterology, ISSN 0016-5085, 2009, Volume 137, Issue 6, pp. 2084 - 2095.e3
Background & Aims The transient receptor potential (TRP) channel family includes transducers of mechanical and chemical stimuli for visceral sensory neurons.... 
Gastroenterology and Hepatology | SENSITIZATION | PELVIC COLONIC AFFERENTS | INFLAMMATORY PAIN | MICE | HYPERALGESIA | COLD | GASTROENTEROLOGY & HEPATOLOGY | IRRITABLE-BOWEL-SYNDROME | SENSORY NEURONS | BRADYKININ | HYPERSENSITIVITY | Hyperalgesia - chemically induced | Immunohistochemistry | Intestinal Mucosa - metabolism | Intestinal Mucosa - physiopathology | Mechanotransduction, Cellular - drug effects | Capsaicin - pharmacology | Neuroanatomical Tract-Tracing Techniques | Receptor, PAR-2 - metabolism | Colon - drug effects | Afferent Pathways - physiopathology | Colon - innervation | Male | Ganglia, Spinal - physiopathology | Nodose Ganglion - metabolism | Trinitrobenzenesulfonic Acid | Pelvis - innervation | RNA, Messenger - metabolism | Transient Receptor Potential Channels - deficiency | Action Potentials | Nodose Ganglion - physiopathology | Splanchnic Nerves - physiopathology | In Situ Hybridization | Colitis - chemically induced | Nodose Ganglion - drug effects | Colon - physiopathology | Female | Transient Receptor Potential Channels - metabolism | Bradykinin - pharmacology | Disease Models, Animal | Afferent Pathways - metabolism | Hyperalgesia - metabolism | Mice, Inbred C57BL | Reverse Transcriptase Polymerase Chain Reaction | Pressure | Stimulation, Chemical | Colon - metabolism | Hyperalgesia - physiopathology | Mice, Knockout | Animals | Colitis - physiopathology | Transient Receptor Potential Channels - genetics | TRPA1 Cation Channel | Colitis - metabolism | Splanchnic Nerves - metabolism | Mice | Ganglia, Spinal - drug effects | Pain Measurement | Ganglia, Spinal - metabolism | Index Medicus | Abridged Index Medicus
Journal Article
12.