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Journal Article
Journal of Hazardous Materials, ISSN 0304-3894, 04/2019, Volume 368, pp. 514 - 522
Journal Article
Toxicology Letters, ISSN 0378-4274, 08/2018, Volume 292, pp. 162 - 174
Journal Article
Comparative Biochemistry and Physiology, Part C, ISSN 1532-0456, 01/2016, Volume 179, pp. 125 - 136
Journal Article
Journal Article
Toxicology and Applied Pharmacology, ISSN 0041-008X, 2004, Volume 200, Issue 2, pp. 159 - 168
The cardiotoxicity associated with doxorubicin (DOX) therapy limits the total cumulative dose and therapeutic success of active anticancer chemotherapy.... 
Heart | Mitochondria | Oxidative damage | Carvedilol | Mitochondrionopathy | Permeability transition pore | Atenolol | Doxorubicin | tetraphenylphosphonium cation | DMSO | DOX | TPP | mitochondrial transmembrane potential | LIPID-PEROXIDATION | mitochondria | carvedilol | oxidative damage | CARDIOTOXICITY | RAT-HEART MITOCHONDRIA | ANTIHYPERTENSIVE DRUG | INDUCED CARDIOMYOPATHY | heart | FREE-RADICALS | atenolol | NADH DEHYDROGENASE | permeability transition pore | ADRIAMYCIN | PHARMACOLOGY & PHARMACY | TOXICOLOGY | doxorubicin | mitochondrionopathy | DYSFUNCTION | Antibiotics, Antineoplastic - toxicity | Glutathione Reductase - metabolism | Mitochondria, Heart - pathology | Cardiomyopathies - prevention & control | Male | Onium Compounds | Ion Channels - physiology | Mitochondria, Heart - drug effects | Adrenergic beta-Antagonists - pharmacology | Mitochondrial Membrane Transport Proteins | Cardiomyopathies - physiopathology | Calcium - physiology | Superoxide Dismutase - metabolism | Glutathione Peroxidase - metabolism | Organophosphorus Compounds | Rats | Antioxidants - pharmacology | Random Allocation | Microscopy, Electron | Rats, Sprague-Dawley | Doxorubicin - toxicity | Animals | Oxygen Consumption - drug effects | Cell Surface Extensions - drug effects | Carbazoles - pharmacology | Propanolamines - pharmacology | Cardiomyopathies - chemically induced
Journal Article
PLoS ONE, ISSN 1932-6203, 03/2015, Volume 10, Issue 3, p. e0119478
Formaldehyde (FA) is a common environmental contaminant that has toxic effects on the central nervous system (CNS). Our previous data demonstrated that... 
NEURONAL APOPTOSIS | INDUCED APOPTOSIS | NEUROTROPHIC FACTOR | MULTIDISCIPLINARY SCIENCES | NITRIC-OXIDE | INDUCED OXIDATIVE DAMAGE | ENDOPLASMIC-RETICULUM STRESS | INDUCED NEUROTOXICITY | UP-REGULATION | PREFRONTAL CORTEX | NERVE GROWTH-FACTOR | Hydrogen Sulfide - pharmacology | Rats | PC12 Cells | Gene Expression Regulation - drug effects | Animals | Neuroprotective Agents - pharmacology | Signal Transduction - drug effects | Indole Alkaloids - pharmacology | Receptor, trkB - antagonists & inhibitors | Brain-Derived Neurotrophic Factor - metabolism | Protein Kinase Inhibitors - pharmacology | Carbazoles - pharmacology | Formaldehyde - toxicity | Receptor, trkB - metabolism | Hydrogen sulfide | Environmental aspects | Neuroprotective agents | Health aspects | Formaldehyde | Sulfide | Brain | Oxidative stress | Neuroprotection | Reactive oxygen species | Neurosciences | Bax protein | Bcl-2 protein | Laboratories | Toxicity | Hydrogen | Central nervous system | Cytotoxicity | Intracellular signalling | Biochemistry | Kinases | Caspase-3 | Proteins | Neurotoxicity | Neurodegeneration | Protein-tyrosine kinase receptors | Physiology | Inhibition | Pretreatment | Protein-tyrosine kinase | Hydrogen ion concentration | Tyrosine | Caspase | Malondialdehyde | Studies | Brain-derived neurotrophic factor | Contaminants | Pheochromocytoma cells | TrkB receptors | Intracellular | Endoplasmic reticulum | Apoptosis | Animal cognition
Journal Article
Toxicology and Applied Pharmacology, ISSN 0041-008X, 04/2015, Volume 284, Issue 2, pp. 163 - 179
Wnt/β-catenin signaling regulates essential biological functions and acts in developmental toxicity of some chemicals. The aryl hydrocarbon receptor (AHR) is... 
Aryl hydrocarbon receptor | 6-Formylindolo[3,2-b]carbazole | 3,3′,4,4′,5-Pentachlorobiphenyl | Beta-catenin | Zebrafish embryo | 3,3',4,4',5-Pentachlorobiphenyl | DIOXIN | 6-Formylindolo[3,2-b]carbazole 3,3 ',4,4 ',5-Pentachlorobiphenyl | ACTIVATION | DEVELOPMENTAL TOXICITY | TARGET GENES | INHIBITION | ARYL-HYDROCARBON RECEPTOR | SIGNALING PATHWAY | LIVER | PHARMACOLOGY & PHARMACY | TOXICOLOGY | EXPRESSION | Zebrafish Proteins - metabolism | Embryo, Nonmammalian - metabolism | Indoles - toxicity | Zebrafish | Wnt Proteins - metabolism | Embryo, Nonmammalian - drug effects | beta Catenin - metabolism | Receptors, Aryl Hydrocarbon - agonists | Animals | Carbazoles - toxicity | Polychlorinated Biphenyls - toxicity | beta Catenin - antagonists & inhibitors | Heterocyclic Compounds, 3-Ring - toxicity | Benzazepines - toxicity | MORTALITY | FERTILIZATION | DMSO | TRANSCRIPTION | PHENOTYPE | SIGNALS | ANIMAL TISSUES | ANTIGENS | TOXICITY | INTERACTIONS | 60 APPLIED LIFE SCIENCES | OLIGONUCLEOTIDES | BIOLOGICAL FUNCTIONS | EDEMA | GENES | RECEPTORS | EFFICIENCY | EMBRYOS | Basic Medicine | Medical and Health Sciences | Medicin och hälsovetenskap | 5-Pentachlorobiphenyl | 2-b]carbazole 3 | 3 | 4 | Medicinska och farmaceutiska grundvetenskaper | Farmakologi och toxikologi | Pharmacology and Toxicology | 6-Formylindolo
Journal Article
Ecotoxicology and Environmental Safety, ISSN 0147-6513, 11/2018, Volume 163, pp. 340 - 348
Journal Article