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Circulation Journal, ISSN 1346-9843, 2015
Background:Connexin43 (Cx43) is a major connexin that forms gap junction (GJ) channels in the heart and is also present in the cell membrane as... 
Carbenoxolone | Calcium | Connexin43 | Diazoxide
Journal Article
International journal of developmental neuroscience : the official journal of the International Society for Developmental Neuroscience, ISSN 0736-5748, 04/2019
Maternal stress (MS) is associated with various neuropsychiatric disorders and cognitive impairment in the offspring. However, it is unclear how early life... 
Journal Article
PLoS ONE, ISSN 1932-6203, 07/2013, Volume 8, Issue 7, p. e69233
With the availability and ease of small molecule production and design continuing to improve, robust, high-throughput methods for screening are increasingly... 
APPARENT MINERALOCORTICOID EXCESS | 11-BETA-HYDROXYSTEROID DEHYDROGENASE | IN-VITRO | GAP-JUNCTIONS | MULTIDISCIPLINARY SCIENCES | FOCAL CEREBRAL-ISCHEMIA | NEURONS | BETA-HYDROXYSTEROID DEHYDROGENASE | BRAIN | CARBENOXOLONE | CELL-DEATH | Drug Evaluation, Preclinical - methods | Humans | Drug Discovery - methods | Hippocampus - cytology | 11-beta-Hydroxysteroid Dehydrogenase Type 1 - antagonists & inhibitors | Carbenoxolone - pharmacology | Propidium | Neuroprotective Agents - pharmacology | Analysis of Variance | Glucocorticoids - metabolism | Brain Ischemia - drug therapy | Statistics, Nonparametric | Neurons - drug effects | High-Throughput Screening Assays - methods | Stroke (Disease) | Brain | Nervous system diseases | Ischemia | Neurons | Cell death | Health aspects | Occlusion | Neuroprotection | Neurosciences | Traumatic brain injury | Dehydrogenases | Glucocorticoids | Toxicity | Brain slice preparation | Systematic review | Hormones | Glucose | Assaying | Experiments | Cerebral blood flow | Rodents | Toxicity testing | Biocompatibility | Inhibition | Pretreatment | Deoxyribonucleic acid--DNA | Cerebral infarction | Deprivation | Stroke | Neurodegenerative diseases | Cortex | Rats | Cultures | Pharmacology | Medical screening | Chemical compounds | Medicine | Screening | Brain research | Infarction | In vivo methods and tests | Laboratory animals | Endocrinology | Hippocampus | Deoxyribonucleic acid | DNA
Journal Article
PLoS ONE, ISSN 1932-6203, 2011, Volume 6, Issue 6, p. e21108
Background: Glutamate released by activated microglia induces excitotoxic neuronal death, which likely contributes to non-cell autonomous neuronal death in... 
IN-VITRO | MOTOR-NEURON DEGENERATION | GLUTAMATE RELEASE | MULTIDISCIPLINARY SCIENCES | ALS | ACCUMULATION | ACTIVATED MICROGLIA | GLIA | NEUROTOXICITY | BRAIN | BETA | Gap Junctions - metabolism | Microglia - metabolism | Superoxide Dismutase - genetics | Amyotrophic Lateral Sclerosis - physiopathology | Humans | Male | Carbenoxolone - metabolism | Glycyrrhetinic Acid - metabolism | Glycyrrhetinic Acid - therapeutic use | Anti-Inflammatory Agents - metabolism | Carbenoxolone - therapeutic use | Spinal Cord - pathology | Anti-Inflammatory Agents - therapeutic use | Female | Superoxide Dismutase - metabolism | Microglia - cytology | Alzheimer Disease - physiopathology | Glycyrrhetinic Acid - analogs & derivatives | Mice, Inbred C57BL | Behavior, Animal - physiology | Mice, Transgenic | Disease Progression | Carbenoxolone - chemistry | Animals | Anti-Inflammatory Agents - chemistry | Memory Disorders - drug therapy | Ion Channels - metabolism | Mice | Neurons | Amyloid beta-protein | Hostages | Amyotrophic lateral sclerosis | Amino acids | Superoxide | Glutamate | Analysis | Physiological aspects | Models | Genetic engineering | Health aspects | Alzheimer's disease | Neuroprotection | Spinal cord | Animal models | Toxicity | Memory | Superoxide dismutase | Activation | Glutamic acid receptors | Kinases | Receptors | Neurotoxicity | Methamphetamines | Blood-brain barrier | Neurodegeneration | Rodents | Biocompatibility | Inhibition | Antigens | Cell survival | Cytokines | Neurodegenerative diseases | Medical treatment | Mortality | Environmental health | Transgenic mice | Inflammation | Pharmacology | Vectors (Biology) | Amyloid precursor protein | Diseases | Microglia | Neurological diseases | Medicine | Brain research | Acids | Simulation | Cell death | Presenilin 1 | Excitotoxicity | Death | Interferon | Mutation | Alzheimers disease
Journal Article
Journal Article
Current Eye Research, ISSN 0271-3683, 07/2019, Volume 44, Issue 7, pp. 716 - 725
Purpose: Pannexin 1 channels are deemed to play important roles in inflammation. However, there is limited information regarding their roles in fungal... 
carbenoxolone | keratitis | human corneal epithelial cells | Aspergillus fumigatus | Pannexin 1
Journal Article
International Journal of Molecular Sciences, ISSN 1661-6596, 03/2018, Volume 19, Issue 3, p. 817
Although a plethora of signaling pathways are known to drive the activation of hepatic stellate cells in liver fibrosis, the involvement of connexin-based... 
Hepatic stellate cells | Connexin43 | Inflammation | Liver fibrosis | Hemichannel | Gap junction | hepatic stellate cells | connexin43 | CONNEXIN | RAT | hemichannel | MACROPHAGES | BIOCHEMISTRY & MOLECULAR BIOLOGY | INJURY | CHEMISTRY, MULTIDISCIPLINARY | liver fibrosis | GAP-JUNCTIONS | inflammation | INTERCELLULAR COMMUNICATION | CIRRHOSIS | KUPFFER CELLS | gap junction | CHANNELS | Carbenoxolone - administration & dosage | Liver Cirrhosis - drug therapy | Liver Cirrhosis - etiology | Connexin 43 - administration & dosage | Anti-Inflammatory Agents - pharmacology | Peptide Fragments - administration & dosage | Cells, Cultured | Hepatic Stellate Cells - metabolism | Male | Peptide Fragments - pharmacology | Connexin 43 - pharmacology | Carbenoxolone - pharmacology | Carbenoxolone - therapeutic use | Animals | Thioacetamide - toxicity | Anti-Inflammatory Agents - therapeutic use | Connexin 43 - therapeutic use | Connexin 43 - antagonists & inhibitors | Mice | Mice, Inbred BALB C | Peptide Fragments - therapeutic use | Superoxide Dismutase - metabolism | Hepatic Stellate Cells - drug effects | Oxidative stress | Intercellular signalling | Liver | Homeostasis | Fluorescence | Superoxide dismutase | Activation | Connexin 43 | Proteins | Cell activation | Fluorescence recovery after photobleaching | Rodents | Communication | Photobleaching | Stellate cells | Liver diseases | Gap junctions | Superoxide | Thioacetamide | Inhibitors | Hepatocytes | Fibrosis | ATP | Bile
Journal Article
ENDOCRINOLOGY, ISSN 0013-7227, 01/2018, Volume 159, Issue 1, pp. 547 - 556
Glucocorticoid (GC) excess drives multiple cutaneous adverse effects, including skin thinning and poor wound healing. The ubiquitously expressed enzyme 11... 
CELLS | SEVERE ASTHMA | STRATUM-CORNEUM INTEGRITY | ABNORMALITIES | INFLAMMATION | ENDOCRINOLOGY & METABOLISM | SKIN | PERMEABILITY BARRIER HOMEOSTASIS | BLOCKADE | STRESS | EXPRESSION | Carbenoxolone - administration & dosage | Granulation Tissue - pathology | Anti-Inflammatory Agents - poisoning | Drug Eruptions - pathology | Skin - metabolism | Extracellular Matrix - immunology | Extracellular Matrix - metabolism | Gene Expression Profiling | Granulation Tissue - immunology | Skin - injuries | Enzyme Inhibitors - administration & dosage | Granulation Tissue - drug effects | Carbenoxolone - therapeutic use | Drug Eruptions - drug therapy | Carbenoxolone - adverse effects | Female | Corticosterone - blood | Cytokines - genetics | Wound Healing - drug effects | Skin - pathology | Enzyme Inhibitors - adverse effects | Drug Eruptions - etiology | Epidermis - metabolism | Epidermis - pathology | Cytokines - metabolism | Glucocorticoids - poisoning | Epidermis - drug effects | Extracellular Matrix - drug effects | Glucocorticoids - blood | Mice, Hairless | 11-beta-Hydroxysteroid Dehydrogenase Type 1 - antagonists & inhibitors | Enzyme Inhibitors - therapeutic use | Granulation Tissue - metabolism | Gene Expression Regulation - drug effects | Drug Eruptions - metabolism | 11-beta-Hydroxysteroid Dehydrogenase Type 1 - metabolism | Corticosterone - pharmacokinetics | Organ Size - drug effects | Animals | Epidermis - immunology | Corticosterone - poisoning | Glucocorticoids - pharmacokinetics | Cytokines - antagonists & inhibitors | Administration, Topical | Extracellular Matrix - pathology | Skin - drug effects
Journal Article