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Diabetes, ISSN 0012-1797, 05/2016, Volume 65, Issue 5, pp. 1190 - 1196
Pharmacologically induced glycosuria elicits adaptive responses in glucose homeostasis and hormone release. In type 2 diabetes (T2D), along with decrements in... 
INSULIN | KETOACIDOSIS | SGLT2 | ENDOCRINOLOGY & METABOLISM | Glucose Intolerance - metabolism | Glycosuria - chemically induced | Carbohydrate Metabolism - drug effects | Lipolysis - drug effects | Benzhydryl Compounds - administration & dosage | Humans | Glucagon-Like Peptide 1 - blood | Diabetes Mellitus, Type 2 - metabolism | Insulin - blood | Glucagon - blood | Glucose Intolerance - blood | Glucose Intolerance - drug therapy | Benzhydryl Compounds - adverse effects | Hypoglycemic Agents - administration & dosage | Time Factors | Glucosides - therapeutic use | C-Reactive Protein - analysis | Benzhydryl Compounds - therapeutic use | Insulin Secretion | Glucosides - adverse effects | Hypoglycemic Agents - therapeutic use | 3-Hydroxybutyric Acid - blood | Sodium-Glucose Transporter 2 - metabolism | Glucose Intolerance - urine | 3-Hydroxybutyric Acid - agonists | Renal Elimination - drug effects | Sodium-Glucose Transporter 2 Inhibitors | Membrane Transport Modulators - administration & dosage | Diabetes Mellitus, Type 2 - urine | Diabetes Mellitus, Type 2 - blood | Insulin - metabolism | Algorithms | Glucosides - administration & dosage | 3-Hydroxybutyric Acid - metabolism | Lipid Metabolism - drug effects | Membrane Transport Modulators - therapeutic use | Glucagon - metabolism | Diabetes Mellitus, Type 2 - drug therapy | Membrane Transport Modulators - adverse effects | Hypoglycemic Agents - adverse effects | Energy Metabolism - drug effects | Type 2 diabetes | Care and treatment | Glucagon | Dosage and administration | Triglycerides | Research | Insulin | Homeostasis | Glucose | Hormones | Diabetes | Substrates | Index Medicus | Abridged Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 2011, Volume 6, Issue 6, pp. e20944 - e20944
Background: Alterations in the composition of gut microbiota - known as dysbiosis - has been proposed to contribute to the development of obesity, thereby... 
BACTERIAL COMMUNITY | IN-VITRO | LIPID-METABOLISM | CONJUGATED LINOLEIC-ACID | INSULIN-RESISTANCE | HEALTH-BENEFITS | MULTIDISCIPLINARY SCIENCES | GUT MICROBIOTA | GLUCOSE-TOLERANCE | INTESTINAL BACTERIA | ADIPOSE-TISSUE | Intestines - drug effects | Obesity - diet therapy | Molecular Weight | Xylans - pharmacology | Diet - adverse effects | Cholesterol - blood | Body Weight - drug effects | Male | Linoleic Acids, Conjugated - metabolism | Obesity - microbiology | Metagenome - physiology | Bacterial Load - drug effects | Subcutaneous Fat - metabolism | Prevotella - physiology | Subcutaneous Fat - drug effects | Obesity - etiology | Bifidobacterium - physiology | Biomarkers - metabolism | Dietary Fats - adverse effects | Metagenome - drug effects | Mice, Inbred C57BL | Bacteroides - physiology | Insulin Resistance | Obesity - metabolism | Prebiotics | Gene Expression Regulation - drug effects | Animals | Intestines - microbiology | Xylans - chemistry | Xylans - therapeutic use | Mice | Triticum - chemistry | Microbiota (Symbiotic organisms) | Diet | Analysis | Body weight | Physiological aspects | Insulin resistance | Wheat | Fatty acids | Dysbacteriosis | Adipose tissue | Fat metabolism | Enzyme activity | Liver | Lipids | Glucose | Molecular weight | High fat diet | Proteins | Microbiota | Enzymatic activity | Rodents | Nutrients | Bacteria | Oxidation | Bioindicators | Colon | Lipid metabolism | Supplementation | Arabinoxylans | Food | Carbohydrates | Obesity | Dietary supplements | Polymerization | Inflammation | Ecology | Metabolism | Fermentation | Gene expression | Insulin | Body weight gain | Cholesterol | Studies | Nutrition research | Correlation analysis | Laboratory animals | Index Medicus
Journal Article
Cell Metabolism, ISSN 1550-4131, 2009, Volume 9, Issue 4, pp. 327 - 338
Hepatic metabolic derangements are key components in the development of fatty liver, insulin resistance, and atherosclerosis. SIRT1, a NAD -dependent protein... 
HUMDISEASE | TRANSCRIPTION FACTORS | LIPID-METABOLISM | INSULIN-RESISTANCE | COACTIVATOR PGC-1 | ENDOCRINOLOGY & METABOLISM | PROTEIN DEACETYLASES | PROLIFERATOR-ACTIVATED-RECEPTOR | CELL-SURVIVAL | LIVER-DISEASE | CALORIE RESTRICTION | ADIPOSE-TISSUE | CELL BIOLOGY | Organ Specificity - drug effects | Transcriptional Activation - drug effects | Fatty Liver - complications | Endoplasmic Reticulum - pathology | Inflammation - complications | Dietary Fats - pharmacology | Sirtuin 1 | Endoplasmic Reticulum - drug effects | Gene Deletion | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha | Protein Binding - drug effects | Dietary Fats - administration & dosage | Fatty Liver - enzymology | Oxidation-Reduction - drug effects | Homeostasis - drug effects | Sirtuins - genetics | Fatty Acids - metabolism | Hepatocytes - drug effects | Feeding Behavior - drug effects | Animals | Signal Transduction - drug effects | Lipid Metabolism - drug effects | Ligands | Trans-Activators - metabolism | Mice | Transcription Factors | PPAR alpha - metabolism | Inflammation - enzymology | Sirtuins - metabolism | Hepatocytes - enzymology | Starvation | Liver diseases | Atherosclerosis | Environmental health | Physiological aspects | Insulin resistance | Inflammation | Fatty acids | Index Medicus | PGC-1α | PPARα | inflammation | SIRT1 | hepatic fatty acid oxidation | hepatic steatosis
Journal Article
BBA - Molecular Cell Research, ISSN 0167-4889, 02/2014, Volume 1843, Issue 2, pp. 372 - 386
(CRNDE) is a novel gene that is activated early in colorectal cancer but whose regulation and functions are unknown. CRNDE transcripts are recognized as long... 
lncRNA | IGF1 | Insulin | CRNDE | Colorectal cancer | Warburg effect | LncRNA | BIOCHEMISTRY & MOLECULAR BIOLOGY | IncRNA | CELL-PROLIFERATION | GLUCOSE-TRANSPORT | MUSCLE-CELLS | CELL BIOLOGY | COLON-CANCER | RECTAL-CANCER | COLORECTAL-CANCER | MUTATIONS | CARBOHYDRATE-METABOLISM | EXPRESSION | GROWTH-FACTOR-I | Insulin-Like Growth Factor I - pharmacology | Glucose Transporter Type 4 - metabolism | TOR Serine-Threonine Kinases - metabolism | Colorectal Neoplasms - genetics | Humans | Phosphatidylinositol 3-Kinases - metabolism | RNA, Messenger - metabolism | raf Kinases - metabolism | RNA Interference | Metabolism - drug effects | Insulin-Like Growth Factor II - pharmacology | Gene Expression Regulation, Neoplastic - drug effects | Proto-Oncogene Proteins c-akt - metabolism | Colorectal Neoplasms - metabolism | Metabolism - genetics | Insulin - pharmacology | RNA, Messenger - genetics | RNA, Long Noncoding - genetics | Signal Transduction - genetics | Transcriptome - genetics | Down-Regulation - drug effects | Down-Regulation - genetics | Insulin-Like Growth Factor II - metabolism | Gene Expression Regulation - drug effects | Insulin - metabolism | Lactates - metabolism | Signal Transduction - drug effects | Models, Biological | Cell Line, Tumor | Basic Helix-Loop-Helix Leucine Zipper Transcription Factors - metabolism | Colorectal Neoplasms - pathology | RNA, Long Noncoding - metabolism | Insulin-Like Growth Factor I - metabolism | Mitogen-Activated Protein Kinases - metabolism | RNA, Small Interfering - metabolism | RNA | Genes | Analysis | Physiological aspects | Glucose | Gene expression | Dextrose | Epigenetic inheritance
Journal Article
Science, ISSN 0036-8075, 5/2012, Volume 336, Issue 6083, pp. 918 - 922
Journal Article
The Journal of Nutrition, ISSN 0022-3166, 2013, Volume 143, Issue 3, pp. 274 - 283
Resistant starch (RS) is highly fermentable by microbiota in the colon, resulting in the production of SCFAs. RS is thought to mediate a large proportion of... 
BODY-FAT | METABOLIC SYNDROME | NUTRITION & DIETETICS | COLONIC FUNCTION | GLUCAGON-LIKE PEPTIDE-1 | CHAIN FATTY-ACIDS | PHYLOGENETIC MICROARRAY | GASTROINTESTINAL-TRACT | US ADULTS | HUMAN GUT | APPETITE REGULATION | Intestinal Mucosa - metabolism | Gene Expression - drug effects | Bacteria - drug effects | Colon - drug effects | Glucagon - genetics | Cecum - microbiology | Fatty Acids, Volatile - metabolism | Intestinal Mucosa - drug effects | Bacteria - growth & development | Intestine, Large - metabolism | Intestine, Large - microbiology | Monocarboxylic Acid Transporters - metabolism | Cecum - metabolism | Swine | Female | Lipid Metabolism - genetics | Intestine, Large - drug effects | Metagenome - drug effects | Starch - pharmacology | Dietary Carbohydrates - pharmacology | Intestinal Mucosa - microbiology | Cecum - drug effects | Colon - metabolism | Animals | Diet | Lipid Metabolism - drug effects | Glucagon - metabolism | Colon - microbiology | Monocarboxylic Acid Transporters - genetics | Control | Microbiota (Symbiotic organisms) | Starch | Physiological aspects | Food and nutrition | Satiation | Methods | Index Medicus | metabolic syndrome | human gut | body-fat | chain fatty-acids | glucagon-like peptide-1 | us adults | gastrointestinal-tract | phylogenetic microarray | colonic function | appetite regulation
Journal Article
Plant Physiology, ISSN 0032-0889, 9/2010, Volume 154, Issue 1, pp. 357 - 372
Journal Article
Nature, ISSN 0028-0836, 2016, Volume 534, Issue 7609, pp. 697 - 699
Changes in the gut microbiota may underpin many human diseases, but the mechanisms that are responsible for altering microbial communities remain poorly... 
ENTERICA SEROTYPE TYPHIMURIUM | MULTIDISCIPLINARY SCIENCES | ESCHERICHIA-COLI | GROWTH | SALMONELLA-TYPHIMURIUM | NITRIC-OXIDE | RESISTANCE | MICROBIOTA | INFECTION | Carbohydrate Metabolism - drug effects | Galactose - metabolism | Intestinal Mucosa - metabolism | Salmonella typhimurium - growth & development | Carbohydrate Metabolism - genetics | Gastroenteritis - microbiology | Sugar Acids - metabolism | Male | Host-Pathogen Interactions - drug effects | Cecum - microbiology | Cecum - enzymology | Intestinal Mucosa - drug effects | Salmonella typhimurium - metabolism | Intestinal Mucosa - enzymology | Oxidation-Reduction - drug effects | Glucaric Acid - metabolism | Female | Salmonella typhimurium - drug effects | Salmonella typhimurium - pathogenicity | Streptomycin - pharmacology | Reactive Nitrogen Species - metabolism | Intestinal Mucosa - microbiology | Operon - genetics | Cecum - drug effects | Animals | Nitric Oxide Synthase Type II - genetics | Glucose - metabolism | Anti-Bacterial Agents - pharmacology | Mice | Nitric Oxide Synthase Type II - metabolism | Host-bacteria relationships | Complications and side effects | Drug resistance in microorganisms | Glucose metabolism | Streptomycin | Microbiota (Symbiotic organisms) | Antibiotics | Growth | Physiological aspects | Research | Salmonella typhimurium | Inflammatory bowel disease | Carbohydrates | Pathology | Bacteria | Oxidation | Glucose | Index Medicus
Journal Article
Bioresource Technology, ISSN 0960-8524, 05/2013, Volume 135, pp. 191 - 198
Journal Article