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Journal Article
1975, Wiley series on personality processes., ISBN 0471347280, xvii, 278
Book
American Journal of Physiology - Gastrointestinal and Liver Physiology, ISSN 0193-1857, 06/2009, Volume 296, Issue 6, pp. 1318 - 1323
Journal Article
Plant Molecular Biology, ISSN 0167-4412, 1/2013, Volume 81, Issue 1, pp. 27 - 40
Here, α-Amy2/54 gene expression was used as a molecular probe to investigate the interrelationship among nitric oxide (NO), cyclic GMP (cGMP), and heme... 
Life Sciences | Biochemistry, general | Plant Pathology | α - Amy2/54 gene expression | Nitric oxide | Heme oxygenase-1 | cGMP | Aleurone layers | Plant Sciences | Triticum aestivum | α-Amy2/54 gene expression | ARABIDOPSIS-THALIANA | SEED-GERMINATION | ADVENTITIOUS ROOTING PROCESS | PROTEIN-KINASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | GUANYLYL CYCLASE ACTIVITY | GIBBERELLIN-RESPONSE MUTANT | BARLEY ALEURONE | PLANT SCIENCES | PROGRAMMED CELL-DEATH | INHIBITS APOPTOSIS | alpha-Amy2/54 gene expression | ABSCISIC-ACID | Cyclic GMP - pharmacology | Heme Oxygenase-1 - metabolism | Gene Expression - drug effects | Nitric Oxide - pharmacology | Protoporphyrins - pharmacology | Guanylate Cyclase - antagonists & inhibitors | Nitroprusside - pharmacology | alpha-Amylases - genetics | Heme Oxygenase-1 - genetics | Carbon Monoxide - pharmacology | Cyclic GMP - analogs & derivatives | Plant Proteins - antagonists & inhibitors | Plant Proteins - metabolism | Nitric Oxide Donors - pharmacology | Triticum - enzymology | Aminoquinolines - pharmacology | Genes, Plant - drug effects | Spermine - analogs & derivatives | Signal Transduction | Enzyme Inhibitors - pharmacology | Imidazoles - pharmacology | alpha-Amylases - metabolism | Heme Oxygenase-1 - antagonists & inhibitors | Triticum - genetics | Plant Proteins - genetics | Spermine - pharmacology | Benzoates - pharmacology | Gibberellins - pharmacology | Nitric Oxide - metabolism | Plant Growth Regulators - pharmacology | Triticum - drug effects
Journal Article
Circulation, ISSN 0009-7322, 04/2007, Volume 115, Issue 13, pp. 1789 - 1797
Background-Preeclampsia is characterized clinically by hypertension and proteinuria. Soluble Flt-1 (sFlt-1; also known as soluble vascular endothelial growth... 
Pregnancy | Angiogenesis | Endothelium-derived factors | Preeclampsia | Heme oxygenase-1 | Statins | Endothelium | VITAMIN-C | endothelium | PREGNANCY-INDUCED HYPERTENSION | CARDIAC & CARDIOVASCULAR SYSTEMS | HUMAN PLACENTA | angiogenesis | pregnancy | TISSUE-DAMAGE | heme oxygenase-1 | preeclampsia | MEDIATED ANGIOGENESIS | endothelium-derived factors | TYROSINE KINASE-1 | CYTOKINE PRODUCTION | CARBON-MONOXIDE | statins | PERIPHERAL VASCULAR DISEASE | ISCHEMIA-REPERFUSION | ENDOTHELIAL GROWTH-FACTOR | Endothelium, Vascular - cytology | Oxidative Stress | Humans | Recombinant Fusion Proteins - physiology | Vascular Endothelial Growth Factor Receptor-1 - physiology | Heme Oxygenase-1 - deficiency | Pregnancy Proteins - pharmacology | Cell Hypoxia | Endoglin | Carbon Monoxide - pharmacology | Swine | Heme Oxygenase-1 - physiology | Female | Pre-Eclampsia - metabolism | Receptors, Cell Surface - physiology | Vascular Endothelial Growth Factor Receptor-2 - physiology | Culture Media, Serum-Free - pharmacology | Heme Oxygenase (Decyclizing) - genetics | Vascular Endothelial Growth Factor A - pharmacology | Organ Culture Techniques | Cells, Cultured - drug effects | Heme Oxygenase (Decyclizing) - physiology | Endothelial Cells - metabolism | RNA, Small Interfering - pharmacology | Solubility | Rats | Antioxidants - pharmacology | Pre-Eclampsia - pathology | Mice, Knockout | Tumor Necrosis Factor-alpha - pharmacology | Animals | Hydroxymethylglutaryl-CoA Reductase Inhibitors - pharmacology | Placenta - pathology | Placenta Growth Factor | Antigens, CD - physiology | Mice | Genetic Vectors | Interferon-gamma - pharmacology | Organometallic Compounds - pharmacology | Endothelial Cells - drug effects | Oxidases | Care and treatment | Physiological aspects | Diagnosis | Research | Vascular endothelial growth factor
Journal Article
Current Medicinal Chemistry, ISSN 0929-8673, 08/2015, Volume 22, Issue 24, pp. 2819 - 2857
Heterocyclic N-oxides have emerged as potent compounds with anticancer, antibacterial, antihypertensive, antiparasitic, anti-HIV, anti-inflammatory,... 
Antichagasic agents | Heterocyclic N-oxides | Bioisosteres | Factor xa inhibitors | Antimalarials | Hypoxia | Nitric oxide mimics | Coagulation cascade direct thrombin inhibitors | hypoxia | TRANSITION-METAL CATALYSIS | BETA-AMINOESTER CLASS | CHEMISTRY, MEDICINAL | ACTIVATED PROTEIN-KINASE | antichagasic agents | K-ATP CHANNELS | BIOCHEMISTRY & MOLECULAR BIOLOGY | antimalarials | HUMAN-LEUKOCYTE ELASTASE | coagulation cascade direct thrombin inhibitors | CARBON BOND FORMATION | 1,3-DIOXIDE CORE SCAFFOLDS | HIV-1 ENTRY INHIBITORS | PHARMACOLOGY & PHARMACY | bioisosteres | factor Xa inhibitors | LONG-TERM POTENTIATION | FACTOR XA INHIBITOR | nitric oxide mimics | Anti-HIV Agents - pharmacology | Neuroprotective Agents - therapeutic use | Antihypertensive Agents - pharmacology | Humans | Antineoplastic Agents - therapeutic use | Herbicides - pharmacology | Antihypertensive Agents - therapeutic use | Anti-Bacterial Agents - therapeutic use | Fibrinolytic Agents - pharmacology | Cyclic N-Oxides - therapeutic use | Animals | Neuroprotective Agents - pharmacology | Cyclic N-Oxides - pharmacology | Protein Kinase Inhibitors - therapeutic use | Fibrinolytic Agents - therapeutic use | Anti-HIV Agents - therapeutic use | Anti-Bacterial Agents - pharmacology | Antineoplastic Agents - pharmacology | Protein Kinase Inhibitors - pharmacology
Journal Article
Cell, ISSN 0092-8674, 2007, Volume 130, Issue 5, pp. 797 - 810
Antibiotic mode-of-action classification is based upon drug-target interaction and whether the resultant inhibition of cellular function is lethal to bacteria.... 
SYSBIO | MICROBIO | OXYGEN | SUPEROXIDE | OXIDATIVE DAMAGE | BIOCHEMISTRY & MOLECULAR BIOLOGY | ESCHERICHIA-COLI | DNA-DAMAGE | HYDROGEN-PEROXIDE | SOS RESPONSE | TRANSCRIPTIONAL REGULATION | GENE ONTOLOGY | IRON-SULFUR CLUSTERS | CELL BIOLOGY | Free Radical Scavengers - pharmacology | Hydroxyurea - pharmacology | Escherichia coli - drug effects | Gene Expression Profiling | Colony Count, Microbial | DNA, Bacterial - drug effects | Norfloxacin - pharmacology | Time Factors | Carbon-Sulfur Lyases - genetics | Escherichia coli - metabolism | Ampicillin - pharmacology | Membrane Proteins - metabolism | Cell Death - drug effects | Escherichia coli - growth & development | Rec A Recombinases - genetics | NAD - metabolism | Staphylococcus aureus - metabolism | 2,2'-Dipyridyl - pharmacology | Staphylococcus aureus - genetics | Rec A Recombinases - metabolism | Microbial Viability - drug effects | Membrane Proteins - genetics | Citric Acid Cycle - drug effects | Escherichia coli Proteins - metabolism | Hydroxyl Radical - metabolism | Carbon-Sulfur Lyases - metabolism | Kanamycin - pharmacology | Hydrogen Peroxide - metabolism | Gene Expression Regulation, Bacterial - drug effects | Citric Acid Cycle - genetics | Anti-Bacterial Agents - classification | Iron Chelating Agents - pharmacology | Escherichia coli - genetics | Ferrous Compounds - metabolism | Escherichia coli Proteins - genetics | Anti-Bacterial Agents - pharmacology | DNA Damage | Mutation | Oxidative Stress - drug effects | Staphylococcus aureus - drug effects | Staphylococcus aureus - growth & development | Metabolites | Antibiotics
Journal Article
British Journal of Pharmacology, ISSN 0007-1188, 11/2006, Volume 149, Issue 5, pp. 560 - 572
Background and purpose: The present study investigated whether calcium‐activated K+ channels are involved in acetylcholine‐evoked nitric oxide (NO) release and... 
K+ channels | endothelium | superior mesenteric artery | nitric oxide | acetylcholine | CA2+ ENTRY | RESISTANCE ARTERIES | ENDOTHELIUM-DEPENDENT HYPERPOLARIZATION | PORCINE CORONARY-ARTERIES | ACTIVATED POTASSIUM CHANNELS | VASCULAR SMOOTH-MUSCLE | FACTOR-MEDIATED RESPONSES | RABBIT ARTERIES | CARBON-MONOXIDE | PHARMACOLOGY & PHARMACY | HYDROGEN-PEROXIDE | Rats, Wistar | Calcium - metabolism | Endothelium, Vascular - drug effects | Male | Penicillamine - analogs & derivatives | Apamin - pharmacology | Arginine - analogs & derivatives | Endothelium, Vascular - physiology | Dose-Response Relationship, Drug | Charybdotoxin - pharmacology | Indoles - pharmacology | Vasodilation - physiology | Potassium Channels, Calcium-Activated - physiology | Vasodilator Agents - pharmacology | Acetylcholine - pharmacology | Chlorides - pharmacology | Penicillamine - pharmacology | Rats | Imidazoles - pharmacology | Oxyhemoglobins - pharmacology | Indomethacin - pharmacology | Mesenteric Artery, Superior - drug effects | Mesenteric Artery, Superior - physiology | Barium Compounds - pharmacology | Nitric Oxide - secretion | Animals | Mesenteric Artery, Superior - metabolism | Endothelium, Vascular - metabolism | Arginine - pharmacology | Benzimidazoles - pharmacology | Potassium Channels, Calcium-Activated - antagonists & inhibitors | Vasodilation - drug effects | In Vitro Techniques | Nitric Oxide - metabolism | Research Papers
Journal Article