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CRC Critical Reviews in Toxicology, ISSN 1040-8444, 2003, Volume 33, Issue 2, pp. 105 - 136
The use of many halogenated alkanes such as carbon tetrachloride (CCl4), chloroform (CHCl3) or iodoform (CHI3), has been banned or severely restricted because... 
Haloalkylation | Reactive aldehydes | Cytokines | Lipid peroxidation | Fatty degeneration | Lipid homeostasis | Recovery | Necrosis | Antioxidants | Interleukins | Growth factors | Calcium homeostasis | Apoptosis | Hepatoprotectants | reactive aldehydes | antioxidants | lipid homeostasis | apoptosis | haloalkylation | recovery | MESSENGER-RNA LEVELS | RAT-LIVER INJURY | CHEMICAL-INDUCED HEPATOTOXICITY | MITOCHONDRIAL PERMEABILITY TRANSITION | HEPATIC STELLATE CELLS | fatty degeneration | cytokines | TOXICOLOGY | growth factors | calcium homeostasis | NECROSIS-FACTOR-ALPHA | lipid peroxidation | interleukins | GROWTH-FACTOR-BETA | NITRIC-OXIDE PRODUCTION | OXYGEN PARTIAL PRESSURES | necrosis | hepatoprotectants | FREE-RADICAL PRODUCTS | Age Factors | Humans | Hepatocytes - pathology | Carbon Tetrachloride Poisoning - prevention & control | Liver - physiopathology | Halogens - adverse effects | Alkanes - metabolism | Carbon Tetrachloride Poisoning - metabolism | Liver - drug effects | Carbon Tetrachloride - metabolism | Lipid Peroxidation - drug effects | Homeostasis - drug effects | Hepatocytes - drug effects | Cytokines - metabolism | Liver - metabolism | Carbon Tetrachloride - adverse effects | Antioxidants - pharmacology | Alkanes - adverse effects | Chemical and Drug Induced Liver Injury - physiopathology | Lipid Peroxidation - physiology | Drug Synergism | Halogens - metabolism | Methylation - drug effects | Chemical and Drug Induced Liver Injury - metabolism | Homeostasis - physiology | Carbon Tetrachloride Poisoning - physiopathology | Growth Substances - metabolism
Journal Article
Toxicology, ISSN 0300-483X, 2003, Volume 189, Issue 1, pp. 113 - 127
Hepatic injury through carbon tetrachloride (CCl ) induced lipid peroxidation is well known and has been extensively used in the experimental models to... 
Isoprostanes | Antioxidants | Oxidative stress | Prostaglandins | Free radicals | Carbon tetrachloride | Lipid peroxidation | Cyclooxygenases | Inflammation | Hepatotoxin | free radicals | flepatotoxin | OXIDANT STRESS | antioxidants | carbon tetrachloride | 8-ISO-PROSTAGLANDIN F2-ALPHA | lipid peroxidation | isoprostanes | CHEMICAL-INDUCED HEPATOTOXICITY | FREE-RADICALS | PROSTANOIDS F2-ISOPROSTANES | PERFORMANCE LIQUID-CHROMATOGRAPHY | STRESS IN-VIVO | inflammation | cyclooxygenases | prostaglandins | PHARMACOLOGY & PHARMACY | TOXICOLOGY | DIETARY VITAMIN-E | ASPIRIN-LIKE DRUGS | oxidative stress | OXIDATIVE INJURY | Lipid Peroxides - biosynthesis | Reactive Oxygen Species - metabolism | Carbon Tetrachloride - pharmacology | Antioxidants - metabolism | Humans | Oxidative Stress - physiology | Carbon Tetrachloride - adverse effects | Cytochrome P-450 Enzyme System - metabolism | Lipid Peroxides - metabolism | Antioxidants - pharmacology | Dinoprost - metabolism | Isoprostanes - biosynthesis | Dinoprost - biosynthesis | Animals | Isoprostanes - metabolism | Prostaglandin-Endoperoxide Synthases - metabolism | Lipid Peroxidation - drug effects | Chemical and Drug Induced Liver Injury | Oxidative Stress - drug effects | Liver Diseases - metabolism | Reactive Oxygen Species/metabolism | Liver Diseases/chemically induced/metabolism | Carbon Tetrachloride/adverse effects/pharmacology | Lipid Peroxides/biosynthesis/metabolism | Dinoprost/biosynthesis/metabolism | Prostaglandin-Endoperoxide Synthases/metabolism | Lipid Peroxidation/drug effects | Antioxidants/metabolism/pharmacology | Cytochrome P-450 Enzyme System/metabolism | Isoprostanes/biosynthesis/metabolism | Research Support; Non-U.S. Gov't | Oxidative Stress/drug effects/physiology
Journal Article
Journal Article
Journal Article
Journal Article
Pharmacological Research, ISSN 1043-6618, 03/2016, Volume 105, pp. 108 - 120
The liver is a central organ in detoxifying molecules and would otherwise cause molecular damage throughout the organism. Numerous toxic agents including... 
Lipid peroxidation | Oxidative stress | Sepsis | Melatonin | Chemotherapeutic | Liver toxicity | RAT-LIVER | ORGAN DYSFUNCTION SYNDROME | NITRIC-OXIDE SYNTHASE | INDUCED LIVER-INJURY | REACTIVE OXYGEN | TETRACHLORIDE-INDUCED HEPATOTOXICITY | INDUCED OXIDATIVE STRESS | CADMIUM-INDUCED HEPATOTOXICITY | PHARMACOLOGY & PHARMACY | CARBON-TETRACHLORIDE | NF-KAPPA-B | Aflatoxins - metabolism | Liver - pathology | Metals, Heavy - metabolism | Antioxidants - metabolism | Humans | Liver Diseases - pathology | Protective Agents - metabolism | Sepsis - complications | Nicotine - adverse effects | Liver - drug effects | Carbon Tetrachloride - metabolism | Protective Agents - pharmacology | Liver Diseases - etiology | Chemical and Drug Induced Liver Injury - pathology | Chemical and Drug Induced Liver Injury - prevention & control | Metals, Heavy - adverse effects | Liver - metabolism | Liver Diseases - prevention & control | Carbon Tetrachloride - adverse effects | Antioxidants - pharmacology | Carbon Tetrachloride - toxicity | Nicotine - toxicity | Animals | Nicotine - metabolism | Metals, Heavy - toxicity | Chemical and Drug Induced Liver Injury - metabolism | Melatonin - metabolism | Liver Diseases - metabolism | Melatonin - pharmacology | Aflatoxins - adverse effects | Aflatoxins - toxicity | Proteins | Chemotherapy | Cell death | Liver | Carbon tetrachloride | Physiological aspects | Lipids | Heavy metals | Cancer | Index Medicus
Journal Article
Hepatology, ISSN 0270-9139, 04/2014, Volume 59, Issue 4, pp. 1577 - 1590
Hepatocellular carcinoma (HCC) is the most rapidly increasing cause of cancer‐related mortality in the United States. Because of the lack of viable treatment... 
FUNCTIONAL POLYMORPHISM | HEPATIC STELLATE CELLS | MODELS | GENE-EXPRESSION | TARGETS | RISK | MECHANISMS | CYP2E1 | GASTROENTEROLOGY & HEPATOLOGY | ERLOTINIB | HEPATOCARCINOGENESIS | Erlotinib Hydrochloride | Prognosis | Rats, Wistar | Carcinoma, Hepatocellular - prevention & control | Humans | Transcriptome | Hepatic Stellate Cells - metabolism | Receptor, Epidermal Growth Factor - drug effects | Hepatocytes - pathology | Male | Receptor, Epidermal Growth Factor - metabolism | Diethylnitrosamine - adverse effects | Liver Neoplasms - pathology | Phosphorylation - drug effects | Liver Cirrhosis - genetics | Hepatic Stellate Cells - pathology | Hepatocytes - drug effects | Disease Models, Animal | Hepatic Stellate Cells - drug effects | Liver Neoplasms - prevention & control | Liver Cirrhosis - etiology | Liver Cirrhosis - prevention & control | Carbon Tetrachloride - adverse effects | Cells, Cultured | Bile Ducts - physiopathology | Rats | Mice, Inbred Strains | Disease Progression | Animals | Quinazolines - therapeutic use | Carcinoma, Hepatocellular - pathology | Receptor, Epidermal Growth Factor - antagonists & inhibitors | Cell Proliferation - drug effects | Mice | Ligation - adverse effects | Quinazolines - pharmacology | Liver cancer | Epidermal growth factor | Rodents | Mortality | Hepatology | Gene expression | Liver cirrhosis
Journal Article
PLoS ONE, ISSN 1932-6203, 08/2018, Volume 13, Issue 8, pp. e0202362 - e0202362
Malvaviscus arboreus Cav. is a medicinal plant belonging to family Malvaceae with both ethnomedical and culinary value; however, its phytochemical and... 
ANTIOXIDANT ACTIVITY | GLYCOSIDES | CAFFEIC ACID | MULTIDISCIPLINARY SCIENCES | INDIAN MEDICINAL-PLANTS | PHENOLICS | HEPATOTOXICITY | DAMAGE | FLOWERS | EXTRACT | Plant Extracts - chemistry | Liver - pathology | Plant Extracts - pharmacology | Male | Liver - injuries | Liver - drug effects | Protective Agents - pharmacology | Malvaceae - chemistry | Plant Stems - chemistry | Phytotherapy | Chemical and Drug Induced Liver Injury - pathology | Plant Leaves - chemistry | Plants, Medicinal - chemistry | Administration, Oral | Liver - metabolism | Carbon Tetrachloride - adverse effects | Metabolome | Rats | Random Allocation | Animals | Plant Leaves - metabolism | Plant Stems - metabolism | Chemical and Drug Induced Liver Injury - metabolism | Chemical and Drug Induced Liver Injury - drug therapy | Malvaceae - metabolism | Plants, Medicinal - metabolism | Alkaline phosphatase | Care and treatment | Liver diseases | Analysis | Rattus | Research | Chemical properties | Ethnomethodology | Medicinal plants | Flavonoids | Flavones | Liver | Carbon tetrachloride | Bioflavonoids | Medicine, Botanic | Esters | Medicine, Herbal | Oxidative stress | Chemical analysis | Nuclear magnetic resonance--NMR | Transaminase | Alcohol | Bilirubin | Phenolic acids | Antioxidants | Metabolites | Aspartate transaminase | Chemical composition | Hepatotoxicity | Injuries | Food | Solvents | Alanine | Oral administration | Mass spectroscopy | Ethyl acetate | Liquid chromatography | Olive oil | Metabolism | Chromatography | Malondialdehyde | Chemicals | Organic chemistry | Petroleum ether | Injury prevention | Phytochemicals | Acids | Silymarin | Ionization | Pharmacy | Dichloromethane | Phenols | Alanine transaminase | Methylene chloride | Fats and oils | Ramadan | Acetic acid | Mass spectrometry | Pharmaceuticals | Index Medicus | Nuclear magnetic resonance | NMR
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