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CRC Critical Reviews in Toxicology, ISSN 1040-8444, 2003, Volume 33, Issue 2, pp. 105 - 136
The use of many halogenated alkanes such as carbon tetrachloride (CCl4), chloroform (CHCl3) or iodoform (CHI3), has been banned or severely restricted because... 
Haloalkylation | Reactive aldehydes | Cytokines | Lipid peroxidation | Fatty degeneration | Lipid homeostasis | Recovery | Necrosis | Antioxidants | Interleukins | Growth factors | Calcium homeostasis | Apoptosis | Hepatoprotectants | reactive aldehydes | antioxidants | lipid homeostasis | apoptosis | haloalkylation | recovery | MESSENGER-RNA LEVELS | RAT-LIVER INJURY | CHEMICAL-INDUCED HEPATOTOXICITY | MITOCHONDRIAL PERMEABILITY TRANSITION | HEPATIC STELLATE CELLS | fatty degeneration | cytokines | TOXICOLOGY | growth factors | calcium homeostasis | NECROSIS-FACTOR-ALPHA | lipid peroxidation | interleukins | GROWTH-FACTOR-BETA | NITRIC-OXIDE PRODUCTION | OXYGEN PARTIAL PRESSURES | necrosis | hepatoprotectants | FREE-RADICAL PRODUCTS | Age Factors | Humans | Hepatocytes - pathology | Carbon Tetrachloride Poisoning - prevention & control | Liver - physiopathology | Halogens - adverse effects | Alkanes - metabolism | Carbon Tetrachloride Poisoning - metabolism | Liver - drug effects | Carbon Tetrachloride - metabolism | Lipid Peroxidation - drug effects | Homeostasis - drug effects | Hepatocytes - drug effects | Cytokines - metabolism | Liver - metabolism | Carbon Tetrachloride - adverse effects | Antioxidants - pharmacology | Alkanes - adverse effects | Chemical and Drug Induced Liver Injury - physiopathology | Lipid Peroxidation - physiology | Drug Synergism | Halogens - metabolism | Methylation - drug effects | Chemical and Drug Induced Liver Injury - metabolism | Homeostasis - physiology | Carbon Tetrachloride Poisoning - physiopathology | Growth Substances - metabolism
Journal Article
European Journal of Pharmacology, ISSN 0014-2999, 2009, Volume 623, Issue 1, pp. 119 - 124
This study examined the effects of chlorogenic acid (CGA) on liver fibrosis induced by carbon tetrachloride (CCl ) and explored the possible mechanisms of... 
Chlorogenic acid | Liver fibrosis | Carbon tetrachloride | Apoptosis | TGF-BETA | ANGIOGENESIS | FIBROGENESIS | ENDOPLASMIC-RETICULUM STRESS | HEPATIC STELLATE CELLS | CIRRHOSIS | PHARMACOLOGY & PHARMACY | EXPRESSION | ASSOCIATION | ENDOTHELIAL GROWTH-FACTOR | Liver - pathology | Transforming Growth Factor beta1 - metabolism | Actins - metabolism | Collagen Type III - metabolism | Protective Agents - metabolism | Male | Protective Agents - therapeutic use | Liver - physiopathology | RNA, Messenger - metabolism | Liver Cirrhosis - chemically induced | Proto-Oncogene Proteins c-bcl-2 - metabolism | Collagen Type I - genetics | Carbon Tetrachloride - metabolism | Chlorogenic Acid - metabolism | Liver Cirrhosis - metabolism | Membrane Proteins - metabolism | Collagen Type I - metabolism | Liver Cirrhosis - prevention & control | Heat-Shock Proteins - metabolism | Rats | Transforming Growth Factor beta1 - genetics | Collagen Type III - genetics | Carbon Tetrachloride - toxicity | Reverse Transcriptase Polymerase Chain Reaction | Rats, Sprague-Dawley | HSP70 Heat-Shock Proteins - metabolism | Gene Expression Regulation - drug effects | Animals | Chlorogenic Acid - therapeutic use | Vascular Endothelial Growth Factors - genetics | Vascular Endothelial Growth Factors - metabolism | Proto-Oncogene Proteins c-bcl-2 - genetics | Messenger RNA | Liver diseases | Collagen | Liver | Fibrosis | Transforming growth factors | Muscle proteins | Vascular endothelial growth factor
Journal Article
Pharmacological Research, ISSN 1043-6618, 03/2016, Volume 105, pp. 108 - 120
The liver is a central organ in detoxifying molecules and would otherwise cause molecular damage throughout the organism. Numerous toxic agents including... 
Lipid peroxidation | Oxidative stress | Sepsis | Melatonin | Chemotherapeutic | Liver toxicity | RAT-LIVER | ORGAN DYSFUNCTION SYNDROME | NITRIC-OXIDE SYNTHASE | INDUCED LIVER-INJURY | REACTIVE OXYGEN | TETRACHLORIDE-INDUCED HEPATOTOXICITY | INDUCED OXIDATIVE STRESS | CADMIUM-INDUCED HEPATOTOXICITY | PHARMACOLOGY & PHARMACY | CARBON-TETRACHLORIDE | NF-KAPPA-B | Aflatoxins - metabolism | Liver - pathology | Metals, Heavy - metabolism | Antioxidants - metabolism | Humans | Liver Diseases - pathology | Protective Agents - metabolism | Sepsis - complications | Nicotine - adverse effects | Liver - drug effects | Carbon Tetrachloride - metabolism | Protective Agents - pharmacology | Liver Diseases - etiology | Chemical and Drug Induced Liver Injury - pathology | Chemical and Drug Induced Liver Injury - prevention & control | Metals, Heavy - adverse effects | Liver - metabolism | Liver Diseases - prevention & control | Carbon Tetrachloride - adverse effects | Antioxidants - pharmacology | Carbon Tetrachloride - toxicity | Nicotine - toxicity | Animals | Nicotine - metabolism | Metals, Heavy - toxicity | Chemical and Drug Induced Liver Injury - metabolism | Melatonin - metabolism | Liver Diseases - metabolism | Melatonin - pharmacology | Aflatoxins - adverse effects | Aflatoxins - toxicity | Proteins | Chemotherapy | Cell death | Liver | Carbon tetrachloride | Physiological aspects | Lipids | Heavy metals | Cancer | Index Medicus
Journal Article
Journal Article