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Nature (London), ISSN 1476-4687, 2018, Volume 562, Issue 7725, pp. 69 - 75
Primary liver cancer represents a major health problem. It comprises hepatocellular carcinoma (HCC) and intrahepatic cholangiocarcinoma (ICC), which differ... 
PATHOGENESIS | HEPATOCELLULAR-CARCINOMA | INTRAHEPATIC CHOLANGIOCARCINOMA | READ ALIGNMENT | HEPATOCYTES | INFLAMMATION | MULTIDISCIPLINARY SCIENCES | SEQUENCING DATA | EXPRESSION | DISCOVERY | CELL-DEATH | Humans | Tumor Microenvironment | Apoptosis - genetics | Hepatocytes - pathology | Male | Gene Expression Profiling | Genes, myc | Hepatocytes - metabolism | Proto-Oncogene Proteins c-akt - genetics | DNA-Binding Proteins - metabolism | Carcinoma, Hepatocellular - genetics | DNA Transposable Elements - genetics | Female | Liver Neoplasms - pathology | Cell Differentiation | Cell Lineage - genetics | Disease Models, Animal | Cyclin-Dependent Kinase Inhibitor p16 - deficiency | Cytokines - metabolism | Liver Neoplasms - genetics | Carcinogenesis - genetics | Transcription Factors - genetics | DNA-Binding Proteins - genetics | T-Box Domain Proteins - genetics | T-Box Domain Proteins - metabolism | Transcription Factors - metabolism | Cholangiocarcinoma - pathology | Animals | Epigenesis, Genetic - genetics | Carcinoma, Hepatocellular - pathology | Cholangiocarcinoma - genetics | Mosaicism | Mice | Necrosis - genetics | Genes, ras | Liver cancer | Research | Carcinogenesis | Oncology, Experimental | Apoptosis | Cancer | Animal models | Cytokines | Liver | Hepatocellular carcinoma | Genomes | Metastasis | Risk analysis | Gene expression | Risk factors | Metastases | Hepatocytes | Morphology | DNA methylation | Tumorigenesis | Bioinformatics | Cholangiocarcinoma | Deoxyribonucleic acid--DNA | Tumors | T-Box Domain Proteins/metabolism | Hepatocytes/pathology | DNA-Binding Proteins/metabolism | Life Sciences | Cholangiocarcinoma/pathology | Transcription Factors/metabolism | Necrosis/genetics | Cytokines/metabolism | Epigenesis, Genetic/genetics | Cyclin-Dependent Kinase Inhibitor p16/deficiency | Carcinoma, Hepatocellular/pathology | DNA Transposable Elements/genetics | Cell Lineage/genetics | T-Box Domain Proteins/genetics | Transcription Factors/genetics | Apoptosis/genetics | Proto-Oncogene Proteins c-akt/genetics | Liver Neoplasms/genetics | Cholangiocarcinoma/genetics | Liver Neoplasms/pathology | DNA-Binding Proteins/genetics | Hepatocytes/metabolism | Carcinoma, Hepatocellular/genetics | Carcinogenesis/genetics
Journal Article
2006, 1st ed., ISBN 9780471467816, xv, 557
Understanding the play between heredity and environment, and relating it to disease causation, is the task of ecogenetics. Gene-Environment Interactions:... 
Genetic Phenomena | Ecological genetics | Toxicogenetics | Genetic toxicology | Environment | Genetics | Disease susceptibility | Genetic aspects | Environmental Illness | Variation | Environmentally induced diseases | Human genetics | Life Sciences | Genetics & Genomics | SCIENCE
Book
Journal
Nature genetics, ISSN 1061-4036, 2017, Volume 49, Issue 7, pp. 1148 - 1151
Through exome sequencing, we identified six individuals with biallelic loss-of-function mutations in TRIP13. All six developed Wilms tumor. Constitutional... 
Developmental Disabilities | M Phase Cell Cycle Checkpoints | Sertoli-Leydig Cell Tumor | Humans | Protein-Serine-Threonine Kinases | Child, Preschool | Aneuploidy | Neoplasms, Multiple Primary | Leukemia, Myeloid, Acute | Microcephaly | Journal Article | DNA, Neoplasm | Female | Ovarian Neoplasms | Carrier Proteins | Seizures | Kidney Neoplasms | ATPases Associated with Diverse Cellular Activities | Chromosome Segregation | Genetic Predisposition to Disease | Microtubule-Associated Proteins | RNA Stability | Nuclear Proteins | Mosaicism | Wilms Tumor | Mutation | Cell Cycle Proteins | CELLS | INSTABILITY | PROTEIN | GENETICS & HEREDITY | AAA-ATPASE | MITOTIC CHECKPOINT | MOSAIC VARIEGATED ANEUPLOIDY | Kidney Neoplasms - genetics | Microcephaly - genetics | Seizures - genetics | Microtubule-Associated Proteins - genetics | Wilms Tumor - genetics | M Phase Cell Cycle Checkpoints - genetics | Developmental Disabilities - genetics | Ovarian Neoplasms - genetics | Cell Cycle Proteins - genetics | Nuclear Proteins - genetics | Neoplasms, Multiple Primary - genetics | Sertoli-Leydig Cell Tumor - genetics | Protein-Serine-Threonine Kinases - genetics | RNA Stability - genetics | Carrier Proteins - genetics | DNA, Neoplasm - genetics | Chromosome Segregation - genetics | Leukemia, Myeloid, Acute - genetics | Sequences | Pipelines | Genes | Impairment | Cell division | Genomes | Carcinogenesis | Proteins | Carcinogens | Microencephaly | Chromosomes | Cancer | Tumors | Index Medicus
Journal Article
Cancer genomics & proteomics, ISSN 1109-6535, 2004
Journal
Journal Article
Nature (London), ISSN 1476-4687, 2016, Volume 538, Issue 7625, pp. 378 - 382
Journal Article
Nature (London), ISSN 1476-4687, 2009, Volume 463, Issue 7278, pp. 191 - 196
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 2017, Volume 114, Issue 8, pp. 1934 - 1939
Overwhelming studies show that dysregulation of the Hippo pathway is positively correlated with cell proliferation, growth, and tumorigenesis. Paradoxically,... 
Rox8 | Drosophila | Hippo | JNK | Migration | DROSOPHILA HOMOLOG | PATHWAY CONTROLS | APOPTOSIS | MULTIDISCIPLINARY SCIENCES | YAP | PROLIFERATION | DEATH | ONCOGENIC RAS | GROWTH | migration | LUNG ADENOCARCINOMA | TUMOR-SUPPRESSOR | RNA-Binding Proteins - genetics | Tissue Array Analysis | Humans | Gene Expression Regulation, Neoplastic | Apoptosis - genetics | JNK Mitogen-Activated Protein Kinases - metabolism | MicroRNAs - metabolism | Intracellular Signaling Peptides and Proteins - metabolism | Drosophila Proteins - metabolism | Epithelial-Mesenchymal Transition - genetics | Phosphoproteins - metabolism | Cell Movement - genetics | Gene Knockdown Techniques | Drosophila melanogaster - metabolism | Neoplasms - genetics | RNA Interference | Neoplasm Invasiveness - pathology | Trans-Activators - genetics | T-Cell Intracellular Antigen-1 - genetics | Nuclear Proteins - genetics | Intracellular Signaling Peptides and Proteins - genetics | Matrix Metalloproteinase 1 - genetics | Protein-Serine-Threonine Kinases - metabolism | Disease Models, Animal | Cell Proliferation - genetics | Carcinogenesis - genetics | Protein-Serine-Threonine Kinases - genetics | Nuclear Proteins - metabolism | Signal Transduction - genetics | Phosphoproteins - genetics | T-Cell Intracellular Antigen-1 - metabolism | Carcinogenesis - pathology | Animals | Adaptor Proteins, Signal Transducing - genetics | Cell Line, Tumor | Trans-Activators - metabolism | MicroRNAs - genetics | Drosophila Proteins - genetics | Adaptor Proteins, Signal Transducing - metabolism | Matrix Metalloproteinase 1 - metabolism | Neoplasm Invasiveness - genetics | Neoplasms - pathology | Microscopy, Fluorescence | RNA-Binding Proteins - metabolism | RNA, Small Interfering - metabolism | Biological Sciences
Journal Article
Mutation research, ISSN 0027-5107, 1964
Journal
Gastroenterology, ISSN 0016-5085, 2017, Volume 153, Issue 1, pp. 166 - 177
Journal Article
Nature communications, ISSN 2041-1723, 2019, Volume 10, Issue 1, pp. 353 - 14
Mucosal melanoma is a rare and poorly characterized subtype of human melanoma. Here we perform a cross-species analysis by sequencing tumor-germline pairs from... 
POINT MUTATIONS | CANCER PATIENTS | BREAST-CANCER | MUTATIONAL PROCESSES | ARRAY-CGH | MULTIDISCIPLINARY SCIENCES | COPY NUMBER | TUMOR | OVARIAN-CANCER | MOUSE MODELS | REVEALS | Proto-Oncogene Proteins c-mdm2 - genetics | Cadherins - metabolism | Humans | Carcinogenesis - metabolism | DNA Copy Number Variations | Germ-Line Mutation | Cadherins - genetics | Neoplasm Proteins - genetics | Mouth Neoplasms - genetics | Protein-Serine-Threonine Kinases - metabolism | PTEN Phosphohydrolase - genetics | Tumor Suppressor Proteins - metabolism | Membrane Proteins - genetics | Carcinogenesis - genetics | Cell Cycle Proteins - metabolism | Melanoma - pathology | Mucous Membrane - pathology | GTP Phosphohydrolases - metabolism | GTP Phosphohydrolases - genetics | Microtubule-Associated Proteins - genetics | Species Specificity | Microtubule-Associated Proteins - metabolism | Gene Expression Regulation, Neoplastic | Mouth Neoplasms - metabolism | Receptor-Like Protein Tyrosine Phosphatases, Class 3 - genetics | Neoplasm Proteins - metabolism | Tumor Suppressor Protein p53 - genetics | Melanoma - genetics | BRCA1 Protein - metabolism | Tumor Suppressor Proteins - genetics | Cell Cycle Proteins - genetics | Membrane Proteins - metabolism | Proto-Oncogene Proteins c-mdm2 - metabolism | Melanoma - metabolism | Skin Neoplasms - pathology | Neoplasm Recurrence, Local | Protein-Serine-Threonine Kinases - genetics | Tumor Suppressor Protein p53 - metabolism | PTEN Phosphohydrolase - metabolism | Mucous Membrane - metabolism | Skin Neoplasms - metabolism | Carcinogenesis - pathology | BRCA1 Protein - genetics | Animals | BRCA2 Protein - metabolism | Receptor-Like Protein Tyrosine Phosphatases, Class 3 - metabolism | Skin Neoplasms - genetics | Dogs | Horses | Mouth Neoplasms - pathology | BRCA2 Protein - genetics | BRCA2 protein | MDM2 protein | BRCA1 protein | Copy number | p53 Protein | Mucosa | Melanoma | Data processing | Breast cancer | Metastases | Heterogeneity | Human performance | Tumorigenesis | Mutation | Comparative analysis | Species | PTEN protein | Tumors
Journal Article