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Gastroenterology, ISSN 0016-5085, 2014, Volume 146, Issue 7, pp. 1784 - 1794.e6
Background & Aims Premalignant lesions and early stage tumors contain immunosuppressive microenvironments that create barriers for cancer vaccines. Kras G12D/+... 
Gastroenterology and Hepatology | Tumor Microenvironment | Pancreatic Cancer | Listeria Vaccine | Immunity | MONOCYTOGENES | DUCTAL ADENOCARCINOMA | ONCOGENIC KRAS | CANCER | TUMORS | ADOPTIVE TRANSFER | IMMUNOTHERAPY | MOUSE MODEL | TC17 CELLS | SUPPRESSOR-CELLS | GASTROENTEROLOGY & HEPATOLOGY | Pancreatic Neoplasms - metabolism | Proto-Oncogene Proteins p21(ras) - genetics | Homeodomain Proteins - metabolism | Humans | Carcinoma, Pancreatic Ductal - metabolism | Interferon-gamma - metabolism | Mice, 129 Strain | Carcinoma, Pancreatic Ductal - genetics | T-Lymphocytes, Regulatory - immunology | Cancer Vaccines - therapeutic use | Carcinoma in Situ - genetics | Time Factors | Forkhead Transcription Factors - metabolism | Listeria monocytogenes - metabolism | Disease Models, Animal | Carcinoma in Situ - pathology | Antibodies, Monoclonal - pharmacology | Pancreatic Neoplasms - pathology | Mice, Transgenic | Disease Progression | Interleukin-17 - metabolism | Cyclophosphamide - pharmacology | Pancreatic Neoplasms - immunology | Mice | Mutation | Carcinoma in Situ - metabolism | T-Lymphocytes, Regulatory - metabolism | Carcinoma in Situ - drug therapy | Carcinoma in Situ - immunology | Listeria monocytogenes - immunology | Tumor Suppressor Protein p53 - genetics | Pancreatic Neoplasms - drug therapy | Inflammation Mediators - metabolism | Trans-Activators - genetics | Integrases - metabolism | Carcinoma, Pancreatic Ductal - immunology | Proto-Oncogene Proteins p21(ras) - metabolism | Mice, Inbred C57BL | Receptors, Chemokine - metabolism | Tumor Suppressor Protein p53 - metabolism | Pancreatic Neoplasms - genetics | Carcinoma, Pancreatic Ductal - pathology | Homeodomain Proteins - genetics | Cancer Vaccines - immunology | Carcinoma, Pancreatic Ductal - drug therapy | Animals | Listeria monocytogenes - genetics | Trans-Activators - metabolism | CD11b Antigen - metabolism | Integrases - genetics | Medical colleges | Vaccines | Listeria | Developmental biology | Pancreatic cancer | Contamination | Index Medicus | Abridged Index Medicus
Journal Article
Nature Cell Biology, ISSN 1465-7392, 01/2014, Volume 16, Issue 10, pp. 982 - 991
Journal Article
Cancer Cell, ISSN 1535-6108, 05/2014, Volume 25, Issue 5, pp. 621 - 637
Journal Article
Cancer Research, ISSN 0008-5472, 01/2013, Volume 73, Issue 1, pp. 341 - 352
Journal Article
American Journal of Surgical Pathology, ISSN 0147-5185, 02/2007, Volume 31, Issue 2, pp. 161 - 169
Journal Article
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 7/2010, Volume 107, Issue 30, pp. 13438 - 13443
Pancreatic cancer is one of the most fatal malignancies lacking effective therapies. Notch signaling is a key regulator of cell fate specification and... 
Acinar cells | Developmental biology | Cell lines | Pancreatic cells | Mice | Lesions | Carcinogenesis | Pancreatic neoplasms | Tumors | Notch receptors | K-Ras | Genetically engineered mice | Myc | Pancreatic cancer | Notch | ACTIVATION | MULTIDISCIPLINARY SCIENCES | MOUSE | C-MYC | ADULT MICE | CANCER | EPITHELIAL-MESENCHYMAL TRANSITION | pancreatic cancer | SIGNALING PATHWAY | GROWTH | ACINAR-CELLS | EXPRESSION | genetically engineered mice | Immunohistochemistry | Adenocarcinoma - pathology | Pancreatic Neoplasms - metabolism | Proto-Oncogene Proteins p21(ras) - genetics | Humans | Carcinoma, Pancreatic Ductal - metabolism | Male | Receptor, Notch2 - genetics | Gene Expression Profiling | Carcinoma, Pancreatic Ductal - genetics | Adenocarcinoma - metabolism | Carcinoma in Situ - genetics | Female | Adenocarcinoma - genetics | Proto-Oncogene Proteins p21(ras) - metabolism | Signal Transduction | Carcinoma in Situ - pathology | Pancreatic Neoplasms - pathology | Kaplan-Meier Estimate | Receptor, Notch2 - metabolism | Pancreas - pathology | Pancreatic Neoplasms - genetics | Pancreas - metabolism | Receptor, Notch1 - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Carcinoma, Pancreatic Ductal - pathology | Proto-Oncogene Proteins c-myc - metabolism | Blotting, Western | Disease Progression | Mice, Knockout | Animals | Cell Line, Tumor | Proto-Oncogene Proteins c-myc - genetics | Carcinoma in Situ - metabolism | Receptor, Notch1 - genetics | Signal transduction | Pancreas | Rodents | Cells | Index Medicus | Biological Sciences
Journal Article
Modern Pathology, ISSN 0893-3952, 03/2018, Volume 31, Issue 3, pp. 406 - 417
Journal Article
Journal Article