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Nature Communications, ISSN 2041-1723, 12/2019, Volume 10, Issue 1, pp. 368 - 13
The molecular pathogenesis of salivary gland acinic cell carcinoma (AciCC) is poorly understood. The secretory Ca-binding phosphoprotein (SCPP) gene cluster at... 
HEAD | FUSION | GENE | ALIGNMENT | NOR1 | MULTIDISCIPLINARY SCIENCES | MYB | EXPRESSION | CANCER | TUMORS | ORPHAN NUCLEAR RECEPTOR | Chromatin - metabolism | Translocation, Genetic | Multigene Family | Salivary Glands - pathology | Salivary Proteins and Peptides - genetics | Cell Proliferation | Receptors, Steroid - metabolism | Salivary Gland Neoplasms - genetics | Epigenesis, Genetic | Humans | Carcinoma, Acinar Cell - pathology | Gene Expression Regulation, Neoplastic | Male | Genetic Loci | DNA-Binding Proteins - metabolism | Carcinoma, Acinar Cell - metabolism | Receptors, Thyroid Hormone - genetics | Salivary Gland Neoplasms - metabolism | Female | Chromatin - chemistry | Acinar Cells - metabolism | Carcinoma, Acinar Cell - genetics | DNA-Binding Proteins - genetics | Salivary Proteins and Peptides - metabolism | Chromosomes, Human, Pair 4 - metabolism | Chromosomes, Human, Pair 9 - metabolism | Receptors, Thyroid Hormone - metabolism | Chromosomes, Human, Pair 4 - chemistry | Acinar Cells - pathology | Animals | Enhancer Elements, Genetic | Receptors, Steroid - genetics | Chromosomes, Human, Pair 9 - chemistry | Mice | Primary Cell Culture | Salivary Gland Neoplasms - pathology | Salivary Glands - metabolism | Cohort Studies | Binding | Cell proliferation | Chromatin | Transcription factors | Carcinoma | Salivary glands | Pathogenesis | Salivary gland | Chromosome 4 | Gene expression | Phosphoproteins | Clusters | Saliva
Journal Article
Gastroenterology, ISSN 0016-5085, 2014, Volume 147, Issue 5, pp. 1119 - 1133.e4
Background & Aims Although smoking is a leading risk factor for pancreatic ductal adenocarcinoma (PDAC), little is known about the mechanisms by which smoking... 
Gastroenterology and Hepatology | Metastasis | Pancreas | Progenitor Cells | Mouse Model | METFORMIN | STEM-CELLS | DUCTAL ADENOCARCINOMA | PROLIFERATION | EPITHELIAL-MESENCHYMAL TRANSITION | ADULT-MOUSE PANCREAS | ACETYLCHOLINE-RECEPTORS | K-RAS | GROWTH | DIFFERENTIATION | GASTROENTEROLOGY & HEPATOLOGY | Pancreatic Neoplasms - metabolism | Neoplastic Stem Cells - drug effects | Proto-Oncogene Proteins p21(ras) - genetics | Humans | Carcinoma, Pancreatic Ductal - metabolism | Epithelial-Mesenchymal Transition - drug effects | Mice, 129 Strain | Carcinoma, Pancreatic Ductal - genetics | Neoplastic Stem Cells - metabolism | Time Factors | Cell Transformation, Neoplastic - genetics | Neoplastic Stem Cells - pathology | Pancreatic Neoplasms - chemically induced | Liver Neoplasms - secondary | Nicotinic Agonists - toxicity | Proto-Oncogene Proteins c-akt - metabolism | Neoplastic Cells, Circulating - drug effects | GATA6 Transcription Factor - deficiency | Liver Neoplasms - genetics | Pancreatic Neoplasms - pathology | Pancreas - pathology | alpha7 Nicotinic Acetylcholine Receptor - genetics | Pancreas - metabolism | Mice, Knockout | Acinar Cells - pathology | Signal Transduction - drug effects | Mice, Nude | Liver Neoplasms - metabolism | Cell Line, Tumor | alpha7 Nicotinic Acetylcholine Receptor - metabolism | Mutation | Carcinoma, Pancreatic Ductal - secondary | Carcinoma, Pancreatic Ductal - chemically induced | Neoplastic Cells, Circulating - pathology | Neoplastic Cells, Circulating - metabolism | Cell Transformation, Neoplastic - chemically induced | Extracellular Signal-Regulated MAP Kinases - metabolism | Carcinoma, Pancreatic Ductal - prevention & control | Tumor Suppressor Protein p53 - genetics | Transfection | Proto-Oncogene Proteins p21(ras) - deficiency | Gene Expression Regulation, Neoplastic - drug effects | Pancreatic Neoplasms - prevention & control | Tumor Cells, Cultured | Proto-Oncogene Proteins p21(ras) - metabolism | Acinar Cells - metabolism | Cell Dedifferentiation - drug effects | Metformin - pharmacology | Mice, Inbred C57BL | Tumor Suppressor Protein p53 - metabolism | Pancreas - drug effects | Pancreatic Neoplasms - genetics | Nicotine - toxicity | Cell Transformation, Neoplastic - metabolism | Proto-Oncogene Proteins c-myc - metabolism | GATA6 Transcription Factor - metabolism | Animals | Acinar Cells - drug effects | GATA6 Transcription Factor - genetics | Cell Transformation, Neoplastic - pathology | Pancreatic cancer | Stem cells | Nicotine
Journal Article
Journal Article
Journal Article
American Journal of Surgical Pathology, ISSN 0147-5185, 04/2010, Volume 34, Issue 4, pp. 510 - 518
Background: Pancreatic acinar cell carcinomas (ACCs) are clinically and pathologically distinct from pancreatic ductal adenocarcinomas (PDAs). Whereas... 
Ductal adenocarcinoma | Acinar cell carcinoma | Mucinous | Pancreas | Mixed | PATHWAYS | SURGERY | AUTOPSY | NEOPLASMS | ADENOCARCINOMA | TUMOR | MARKERS | mucinous | mixed | PATHOLOGY | EXOCRINE | ductal adenocarcinoma | pancreas | MUTATION | EXPRESSION | acinar cell carcinoma | TRANSGENIC MICE | ras Proteins - genetics | Proto-Oncogene Proteins p21(ras) | Pancreatic Neoplasms - metabolism | Humans | Middle Aged | Carcinoma, Acinar Cell - pathology | Carcinoma, Pancreatic Ductal - metabolism | Male | Neoplasms, Multiple Primary | Carcinoma, Islet Cell - pathology | Immunoenzyme Techniques | Carcinoma, Pancreatic Ductal - genetics | Carcinoma, Acinar Cell - metabolism | Cell Transformation, Neoplastic - genetics | DNA Mutational Analysis | Mucins - metabolism | Biomarkers, Tumor - metabolism | Carcinoma, Islet Cell - mortality | Carcinoma, Pancreatic Ductal - mortality | Female | DNA, Neoplasm - analysis | Pancreatic Neoplasms - mortality | Pancreatic Neoplasms - pathology | Pancreatectomy | Pancreatic Neoplasms - genetics | Proto-Oncogene Proteins - genetics | Survival Rate | Carcinoma, Acinar Cell - genetics | Combined Modality Therapy | Carcinoma, Pancreatic Ductal - pathology | Cell Transformation, Neoplastic - metabolism | Carcinoma, Islet Cell - metabolism | Carcinoma, Acinar Cell - mortality | Virginia - epidemiology | Carcinoma, Islet Cell - genetics | Aged | Mutation | Cell Transformation, Neoplastic - pathology | New York - epidemiology
Journal Article
Journal Article
Journal Article
Journal Article
European Archives of Oto-rhino-laryngology, ISSN 0937-4477, 2016, Volume 273, Issue 11, pp. 3511 - 3531
Journal Article