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Journal Article
Human Pathology, ISSN 0046-8177, 09/2017, Volume 67, pp. 69 - 77
Endometrial intraepithelial neoplasia (EIN) and atypical endometrial hyperplasia (AH) are histomorphologically defined precursors to endometrioid... 
Clonal evolution | Endometrial hyperplasia | Endometrial intraepithelial neoplasia | Endometrioid adenocarcinoma | Next generation sequencing | PTEN | EIN | PATHOLOGY | CANCER | GENES | MUTATIONS | CARCINOMA | EXPRESSION | Immunohistochemistry | Microsatellite Instability | Cell Proliferation | Endometrial Hyperplasia - enzymology | Humans | Middle Aged | Endometrial Hyperplasia - pathology | DNA Copy Number Variations | Carcinoma in Situ - genetics | Carcinoma, Endometrioid - surgery | Carcinoma in Situ - enzymology | Carcinoma in Situ - surgery | Endometrial Neoplasms - genetics | DNA Mismatch Repair | Endometrial Hyperplasia - surgery | Aged, 80 and over | Female | Carcinoma, Endometrioid - genetics | Genetic Predisposition to Disease | Endometrial Neoplasms - enzymology | Carcinoma, Endometrioid - enzymology | Biomarkers, Tumor - analysis | Carcinoma in Situ - pathology | Endometrial Hyperplasia - genetics | Gene Dosage | Hysterectomy | DNA Repair Enzymes - analysis | Disease Progression | Phenotype | Biopsy | Endometrial Neoplasms - surgery | Endometrial Neoplasms - pathology | Aged | Biomarkers, Tumor - genetics | High-Throughput Nucleotide Sequencing | Mutation | Carcinoma, Endometrioid - pathology | Clonal Evolution | Adenocarcinoma | Gene mutations | Analysis | Hyperplasia | Genomics | Genetic research | Medical colleges | Proteins | Genes | Gynecology | Genomes | Deoxyribonucleic acid--DNA | Cancer | Esophagus
Journal Article
Cancer Cell, ISSN 1535-6108, 12/2015, Volume 28, Issue 6, pp. 785 - 799
Polymorphonuclear neutrophils (PMNs) are largely considered to foster cancer development despite wielding an arsenal of cytotoxic agents. Using a mouse model... 
LUNG | ONCOLOGY | INFLAMMATION | IN-VIVO | CXCR2 | SENESCENCE | MICE | ANTITUMOR IMMUNITY | SUPPRESSION | TUMORIGENESIS | ENDOMETRIAL CANCER | CELL BIOLOGY | Cell Proliferation | Humans | Ovarian Neoplasms - pathology | Tumor Microenvironment | Gene Expression Profiling | Uterus - immunology | Neutrophil Infiltration | Receptors, Colony-Stimulating Factor - deficiency | Ovarian Neoplasms - genetics | Receptors, Colony-Stimulating Factor - genetics | Time Factors | Bone Marrow Transplantation | Neutrophils - metabolism | Gene Transfer Techniques | PTEN Phosphohydrolase - genetics | PTEN Phosphohydrolase - deficiency | Neutrophil Activation | Ovarian Neoplasms - prevention & control | Ovariectomy | Computational Biology | Myeloid Differentiation Factor 88 - genetics | Carcinoma, Endometrioid - immunology | Tumor Burden | Cell Adhesion | Ovarian Neoplasms - enzymology | Mice, Knockout | Carcinoma, Endometrioid - prevention & control | Myeloid Differentiation Factor 88 - deficiency | Carcinoma, Endometrioid - mortality | Receptors, Interleukin-8B - genetics | Survival Analysis | Cell Line, Tumor | Carcinoma, Endometrioid - pathology | Phagocytosis | Uterus - pathology | Databases, Genetic | Ovarian Neoplasms - mortality | Cell Hypoxia | Inflammation Mediators - metabolism | Female | Carcinoma, Endometrioid - genetics | Carcinoma, Endometrioid - enzymology | Mice, Inbred C57BL | Neutrophils - immunology | Chemotaxis | Animals | Receptors, Interleukin-8B - metabolism | Uterus - enzymology | Neoplasm Staging | Ovarian Neoplasms - immunology | Development and progression | Carcinogenesis | Analysis | Tumors | Medical colleges | Lymphocytes | Inflammation | Genetic engineering | Cancer
Journal Article
BMC Cancer, ISSN 1471-2407, 08/2012, Volume 12, Issue 1, pp. 369 - 369
Journal Article
Gynecologic Oncology, ISSN 0090-8258, 2015, Volume 138, Issue 1, pp. 165 - 173
Abstract Objectives Endometrial carcinoma (EC) is the most common gynecological cancer in the Western World. Treatment options are limited for advanced and... 
Hematology, Oncology and Palliative Medicine | Obstetrics and Gynecology | NVP-BEZ235 | Endometrioid endometrial cancer | Targeted therapy | AZD6244 | Primary cell culture | MUTATED COLORECTAL-CANCER | ADVANCED SOLID TUMORS | SELUMETINIB AZD6244 | I PI3K INHIBITOR | PHASE-II | ANTITUMOR-ACTIVITY | OBSTETRICS & GYNECOLOGY | DOSE-ESCALATION | UVEAL MELANOMA | ONCOLOGY | PI3-KINASE INHIBITOR | BUPARLISIB BKM120 | TOR Serine-Threonine Kinases - metabolism | raf Kinases - antagonists & inhibitors | Humans | Imidazoles - administration & dosage | ras Proteins - metabolism | Phosphatidylinositol 3-Kinases - metabolism | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Quinolines - administration & dosage | raf Kinases - metabolism | Quinolines - pharmacology | Antineoplastic Combined Chemotherapy Protocols - pharmacology | TOR Serine-Threonine Kinases - antagonists & inhibitors | Benzimidazoles - administration & dosage | Female | Tumor Cells, Cultured | MAP Kinase Kinase Kinases - antagonists & inhibitors | Proto-Oncogene Proteins c-akt - metabolism | Endometrial Neoplasms - enzymology | Carcinoma, Endometrioid - enzymology | ras Proteins - antagonists & inhibitors | Imidazoles - pharmacology | Carcinoma, Endometrioid - drug therapy | Drug Synergism | Xenograft Model Antitumor Assays | Animals | MAP Kinase Signaling System - drug effects | Benzimidazoles - pharmacology | Endometrial Neoplasms - drug therapy | Mice | Proto-Oncogene Proteins c-akt - antagonists & inhibitors | Carcinoma | Cancer
Journal Article
Gynecologic Oncology, ISSN 0090-8258, 2015, Volume 137, Issue 3, pp. 529 - 537
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 2011, Volume 6, Issue 6, p. e20715
Despite the fact that epithelial ovarian cancers are the leading cause of death from gynecological cancer, very little is known about the pathophysiology of... 
BREAST-CANCER | SURFACE EPITHELIUM | GRANULOSA-CELLS | REPRODUCTIVE-TRACT | FOLLICULAR DEVELOPMENT | MULLERIAN-INHIBITING SUBSTANCE | BIOLOGY | TRANSGENIC MOUSE MODEL | BETA-CATENIN | EXPRESSION | CELL TUMOR-DEVELOPMENT | Neoplasm Transplantation | Carcinoma, Endometrioid - enzymology | TOR Serine-Threonine Kinases - metabolism | Humans | Mice, Inbred C57BL | Ovarian Neoplasms - pathology | Tumor Suppressor Protein p53 - metabolism | Cellular Senescence - drug effects | PTEN Phosphohydrolase - metabolism | Wnt Proteins - metabolism | Sirolimus - pharmacology | beta Catenin - metabolism | Ovarian Neoplasms - enzymology | Animals | Signal Transduction - drug effects | Tumor Burden - drug effects | Gene Deletion | Female | Cell Proliferation - drug effects | Mice | Cell Death - drug effects | Carcinoma, Endometrioid - pathology | TOR protein | Senescence | Phosphorylation | Wnt protein | Mesenchyme | Laboratories | p53 Protein | AKT protein | Digital cameras | Biology | Gene deletion | Accumulation | Medical schools | Ovarian cancer | β-catenin | Clonal deletion | Etiology | Rodents | Deletion | Inhibition | Immunoglobulins | Tumor cells | Gynecology | Histology | Rapamycin | Breast cancer | Epithelium | Obstetrics | 1-Phosphatidylinositol 3-kinase | Signaling | Hospitals | Medical prognosis | Mutation | PTEN protein | Tumors | Cancer | Apoptosis
Journal Article
American Journal of Surgical Pathology, ISSN 0147-5185, 12/2009, Volume 33, Issue 12, pp. 1869 - 1877
Endometrial carcinomas (ECs) in Young women (<= 40 y) are Usually managed conservatively in selected patients. Whether oophorectomy with total hysterectomy is... 
HNPCC/Lynch syndrome | Endometrial carcinoma | Young patients | SURGERY | FERTILITY-PRESERVING TREATMENT | LYNCH-SYNDROME | NONPOLYPOSIS COLORECTAL-CANCER | SYNCHRONOUS PRIMARY CANCERS | PROGESTERONE-RECEPTORS | PROMOTER METHYLATION | MICROSATELLITE INSTABILITY | endometrial carcinoma | CLINICAL-FEATURES | OVARIAN-CANCER | PATHOLOGY | ESTROGEN-RECEPTOR-ALPHA | Immunohistochemistry | MutL Protein Homolog 1 | Nuclear Proteins - analysis | Age Factors | Endometrial Neoplasms - mortality | Adenocarcinoma, Clear Cell - mortality | Humans | Ovarian Neoplasms - pathology | DNA-Binding Proteins - analysis | Neoplasms, Multiple Primary | Ovarian Neoplasms - mortality | Carcinoma, Endometrioid - therapy | Patient Selection | Ovarian Neoplasms - genetics | Young Adult | Endometrial Neoplasms - genetics | Time Factors | Adenosine Triphosphatases - analysis | DNA Mismatch Repair | Adult | Female | Mismatch Repair Endonuclease PMS2 | Carcinoma, Endometrioid - genetics | Endometrial Neoplasms - enzymology | Carcinoma, Endometrioid - enzymology | Risk Assessment | Down-Regulation | Risk Factors | Treatment Outcome | DNA Repair Enzymes - analysis | Ovarian Neoplasms - enzymology | Adaptor Proteins, Signal Transducing - analysis | Biopsy | Carcinoma, Endometrioid - mortality | Adenocarcinoma, Clear Cell - therapy | Endometrial Neoplasms - therapy | Ovarian Neoplasms - therapy | Endometrial Neoplasms - pathology | Adenocarcinoma, Clear Cell - pathology | MutS Homolog 2 Protein - analysis | Neoplasm Staging | Adenocarcinoma, Clear Cell - enzymology | Adenocarcinoma, Clear Cell - genetics | Carcinoma, Endometrioid - pathology
Journal Article