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1. Full Text Mouse Model of Human Ovarian Endometrioid Adenocarcinoma Based on Somatic Defects in the Wnt/β-Catenin and PI3K/Pten Signaling Pathways
by Wu, Rong and Hendrix-Lucas, Neali and Kuick, Rork and Zhai, Yali and Schwartz, Donald R and Akyol, Aytekin and Hanash, Samir and Misek, David E and Katabuchi, Hidetaka and Williams, Bart O and Fearon, Eric R and Cho, Kathleen R
Cancer Cell, ISSN 1535-6108, 04/2007, Volume 11, Issue 4, pp. 321 - 333
One histologic subtype of ovarian carcinoma, ovarian endometrioid adenocarcinoma (OEA), frequently harbors mutations that constitutively activate... 
CELLCYCLE | EPITHELIAL-MESENCHYMAL TRANSITION | GENE-MUTATIONS | K-RAS | ONCOLOGY | MICROSATELLITE INSTABILITY | FUNCTIONAL-EVALUATION | PROSTATE-CANCER | HIGH-FREQUENCY | EXPRESSION PATTERN | BETA-CATENIN | CLINICOPATHOLOGICAL FEATURES | Adenomatous Polyposis Coli Protein - physiology | Wnt1 Protein - genetics | Oligonucleotide Array Sequence Analysis | Adenocarcinoma, Mucinous - pathology | Humans | PTEN Phosphohydrolase - physiology | Cystadenocarcinoma, Serous - metabolism | Gene Expression Regulation, Neoplastic | Ovarian Neoplasms - pathology | Gene Expression Profiling | Phosphatidylinositol 3-Kinases - metabolism | Wnt1 Protein - metabolism | Cystadenocarcinoma, Serous - genetics | Cystadenocarcinoma, Serous - pathology | Tumor Suppressor Protein p53 - genetics | Carcinoma, Endometrioid - metabolism | Ovarian Neoplasms - genetics | Female | Ovarian Neoplasms - metabolism | Disease Models, Animal | Ovary - metabolism | Adenomatous Polyposis Coli Protein - genetics | PTEN Phosphohydrolase - genetics | Carcinoma, Endometrioid - genetics | Epithelium - pathology | Ovary - pathology | Epithelium - metabolism | Signal Transduction | Adenocarcinoma, Clear Cell - metabolism | Tumor Suppressor Protein p53 - metabolism | Survival Rate | beta Catenin - metabolism | beta Catenin - genetics | Phosphatidylinositol 3-Kinases - genetics | Animals | Adenocarcinoma, Mucinous - metabolism | Adenocarcinoma, Clear Cell - pathology | Mice | Adenocarcinoma, Mucinous - genetics | Mutation | Neoplasm Staging | Adenocarcinoma, Clear Cell - genetics | Carcinoma, Endometrioid - pathology | Index Medicus
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2. Full Text A novel Wnt regulatory axis in endometrioid endometrial cancer
by Zhao, Yu and Yang, Yihua and Trovik, Jone and Sun, Kun and Zhou, Liang and Jiang, Peiyong and Lau, Tat-San and Hoivik, Erling A and Salvesen, Helga B and Sun, Hao and Wang, Huating
Cancer Research, ISSN 0008-5472, 08/2014, Volume 74, Issue 18, pp. 5103 - 5117
The Protocadherin 10 (PCDH10) is inactivated often by promoter hypermethylation in various human tumors, but its possible functional role as a tumor suppressor... 
FREQUENT | METASTASIS | HYPERMETHYLATION | LONG NONCODING RNA | GENE | ONCOLOGY | MALAT-1 | TUMOR-SUPPRESSOR | PCDH10 PROMOTER METHYLATION | PROTOCADHERIN PCDH10 | IDENTIFICATION | Cell Proliferation | Cadherins - metabolism | Humans | Endometrial Neoplasms - metabolism | Gene Expression Profiling | Carcinoma, Endometrioid - metabolism | DNA Methylation | Heterografts | Endometrial Neoplasms - genetics | Female | Cadherins - genetics | Promoter Regions, Genetic | Carcinoma, Endometrioid - genetics | Down-Regulation | Gene Silencing | RNA, Long Noncoding - genetics | beta Catenin - metabolism | beta Catenin - genetics | Animals | Wnt Signaling Pathway - genetics | Mice, Nude | Cell Line, Tumor | Endometrial Neoplasms - pathology | Apoptosis - physiology | RNA, Long Noncoding - metabolism | Carcinoma, Endometrioid - pathology | Index Medicus
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3. Full Text Should grade 3 endometrioid endometrial carcinoma be considered a type 2 cancer—A clinical and pathological evaluation
by Voss, Martin A and Ganesan, Raji and Ludeman, Linmarie and McCarthy, Keith and Gornall, Robert and Schaller, Gerhard and Wei, Wenbin and Sundar, Sudha
Gynecologic Oncology, ISSN 0090-8258, 2011, Volume 124, Issue 1, pp. 15 - 20
Abstract Objective Endometrial cancer is classified into: Type I estrogen-dependent endometrioid adenocarcinoma, with good prognosis and type 2... 
Hematology, Oncology and Palliative Medicine | Obstetrics and Gynecology | Immunohistochemistry | Endometrial cancer | Uterine papillary serous and clear cell carcinoma | Type I and II | Grade 3 endometrioid | Survival | P53 OVEREXPRESSION | RECEPTOR STATUS | OBSTETRICS & GYNECOLOGY | PATHOGENESIS | ONCOLOGY | MOLECULAR-GENETIC-PATHWAYS | IMMUNOHISTOCHEMICAL ANALYSIS | SEROUS CARCINOMA | PROGNOSTIC-SIGNIFICANCE | MUTATIONS | ASSOCIATION | EXPRESSION | Receptors, Estrogen - metabolism | Carcinoma, Endometrioid - classification | Endometrial Neoplasms - classification | Humans | Middle Aged | Endometrial Neoplasms - metabolism | Receptor, ErbB-2 - metabolism | Tumor Suppressor Protein p53 - metabolism | Survival Rate | Carcinoma, Endometrioid - metabolism | Receptors, Progesterone - metabolism | Neoplasm Grading | Aged, 80 and over | Adult | Endometrial Neoplasms - pathology | Female | Aged | Neoplasm Staging | Carcinoma, Endometrioid - pathology | Carcinoma | Research | Oncology, Experimental | Cancer | Index Medicus
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4. Full Text Mutation and loss of expression of ARID1A in uterine low-grade endometrioid carcinoma
by Guan, Bin and Mao, Tsui-Lien and Panuganti, Pradeep K and Kuhn, Elisabetta and Kurman, Robert J and Maeda, Daichi and Chen, Elizabeth and Jeng, Yung-Ming and Wang, Tian-Li and Shih, Ie-Ming
American Journal of Surgical Pathology, ISSN 0147-5185, 05/2011, Volume 35, Issue 5, pp. 625 - 632
ARID1A is a recently identified tumor suppressor gene that is mutated in approximately 50% of ovarian clear cell and 30% of ovarian endometrioid carcinomas.... 
ovarian carcinoma | BAF250 | ARID1A | uterine carcinoma | SURGERY | GENE | SWI/SNF-RELATED COMPLEXES | CYCLE ARREST | PATHOLOGY | CLEAR-CELL CARCINOMA | CANCER | Immunohistochemistry | Oligonucleotide Array Sequence Analysis | Uterine Neoplasms - pathology | Humans | Ovarian Neoplasms - pathology | Gene Expression Profiling | Carcinoma, Endometrioid - metabolism | Ovarian Neoplasms - genetics | Nuclear Proteins - biosynthesis | DNA Mutational Analysis | Polymerase Chain Reaction | Female | Ovarian Neoplasms - metabolism | Nuclear Proteins - genetics | Uterine Neoplasms - genetics | Carcinoma, Endometrioid - genetics | Gene Expression | Uterine Neoplasms - metabolism | Biomarkers, Tumor - analysis | Transcription Factors - biosynthesis | Transcription Factors - genetics | Blotting, Western | Biomarkers, Tumor - genetics | Mutation | Carcinoma, Endometrioid - pathology | Index Medicus
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5. Full Text Utility of p16 expression for distinction of uterine serous carcinomas from endometrial endometrioid and endocervical adenocarcinomas: Immunohistochemical analysis of 201 cases
by Yemelyanova, Anna and Ji, Hongxiu and Shih, Ie-Ming and Wang, Tian-Li and Wu, Lee-Shu-Fune and Ronnett, Brigitte M
American Journal of Surgical Pathology, ISSN 0147-5185, 10/2009, Volume 33, Issue 10, pp. 1504 - 1514
Uterine serous carcinomas typically have a characteristic morphology (papillary architecture, high-grade nuclei) and immunoprofile (diffuse/strong p53... 
Adenocarcinoma | Immunohistochemistry | P16 | Uterus | Endometrioid carcinoma | Serous carcinoma | Endocervical adenocarcinoma | SURGERY | DIFFERENTIAL-DIAGNOSIS | CERVICAL ADENOCARCINOMAS | FEMALE GENITAL-TRACT | MICROSATELLITE INSTABILITY | adenocarcinoma | DRUG-RESISTANCE | PATHOLOGY | HUMAN-PAPILLOMAVIRUS DNA | OVARIAN-CARCINOMA | uterus | p16 | P53 GENE-MUTATIONS | endocervical adenocarcinoma | immunohistochemistry | PROGNOSTIC-SIGNIFICANCE | serous carcinoma | endometrioid carcinoma | CLEAR-CELL CARCINOMA | Humans | Cystadenocarcinoma, Serous - metabolism | Endometrial Neoplasms - metabolism | Cystadenocarcinoma, Serous - genetics | Carcinoma, Endometrioid - diagnosis | Papillomavirus Infections - diagnosis | Tumor Suppressor Protein p53 - genetics | Carcinoma, Endometrioid - metabolism | Endometrial Neoplasms - genetics | Uterine Cervical Neoplasms - metabolism | DNA Mutational Analysis | Polymerase Chain Reaction | Cyclin-Dependent Kinase Inhibitor p16 - biosynthesis | Female | Uterine Neoplasms - diagnosis | Uterine Neoplasms - genetics | Carcinoma, Endometrioid - genetics | Diagnosis, Differential | Papillomavirus Infections - complications | Uterine Neoplasms - metabolism | Uterine Cervical Neoplasms - diagnosis | Biomarkers, Tumor - analysis | In Situ Hybridization, Fluorescence | Endometrial Neoplasms - diagnosis | Papillomavirus Infections - metabolism | Uterine Cervical Neoplasms - virology | Cystadenocarcinoma, Serous - diagnosis | Index Medicus
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6. Full Text Disrupting MALAT1/miR-200c sponge decreases invasion and migration in endometrioid endometrial carcinoma
by Li, Qiulian and Zhang, Chao and Chen, Ruichao and Xiong, Hanzhen and Qiu, Fuman and Liu, Shaoyan and Zhang, Minfen and Wang, Fang and Wang, Yu and Zhou, Xuan and Xiao, Guohong and Wang, Xudong and Jiang, Qingping
Cancer Letters, ISSN 0304-3835, 2016, Volume 383, Issue 1, pp. 28 - 40
Abstract Endometrioid endometrial carcinoma (EEC) is the most common gynecologic malignancy around the world. Epithelial-to-mesenchymal transition (EMT) is a... 
Hematology, Oncology and Palliative Medicine | MALAT1 | Endometrioid endometrial carcinoma | miR-200c | Invasion | MALATI | MICRORNAS | MIR-200 FAMILY | COMPETING ENDOGENOUS RNA | EPITHELIAL-MESENCHYMAL TRANSITION | LUNG-CANCER | LONG NONCODING RNA | ONCOLOGY | CANCER METASTASIS | SQUAMOUS-CELL CARCINOMA | PROMOTES | Humans | Endometrial Neoplasms - metabolism | Gene Expression Regulation, Neoplastic | MicroRNAs - metabolism | Carcinoma, Endometrioid - metabolism | Transfection | Endometrial Neoplasms - genetics | RNA Interference | Time Factors | Epithelial-Mesenchymal Transition | Biomarkers, Tumor - metabolism | Female | 3' Untranslated Regions | Binding Sites | Carcinoma, Endometrioid - genetics | Signal Transduction | Neoplasm Invasiveness | RNA, Long Noncoding - genetics | Tumor Burden | Cell Movement - drug effects | Transforming Growth Factor beta - pharmacology | Animals | Mice, Nude | Cell Line, Tumor | Endometrial Neoplasms - pathology | Biomarkers, Tumor - genetics | Mice, Inbred BALB C | MicroRNAs - genetics | RNA, Long Noncoding - metabolism | Carcinoma, Endometrioid - pathology | Medical colleges | Carcinoma | Melanoma | Antisense RNA | Metastasis | Transforming growth factors | Ovarian cancer | Prevention | Analysis | Stem cells | Bone morphogenetic proteins | Health aspects | Cancer | Studies | Medical research | Hospitals | Endometrial cancer | Genes | Chromosomes | Tumors | Index Medicus
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7. Full Text MicroRNA‐503 suppresses proliferation and cell‐cycle progression of endometrioid endometrial cancer by negatively regulating cyclin D1
by Xu, Yan‐Ying and Wu, Hui‐Juan and Ma, Hong‐Da and Xu, Li‐Ping and Huo, Yan and Yin, Li‐Rong
FEBS Journal, ISSN 1742-464X, 08/2013, Volume 280, Issue 16, pp. 3768 - 3779
Micro RNA s (mi RNA s) are post‐transcriptional inhibitor regulators of gene expression that act by directly binding complementary m RNA and are key... 
endometrioid endometrial cancer | cyclin | RNA | micro | miR‐503 | target gene | microRNA | cyclin D1 | miR-503 | TARGET | APOPTOSIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | TISSUE | FAMILY | BREAST-CANCER | PATHWAY | DIFFERENTIATION | EXPRESSION | PCR | Cyclin D1 - metabolism | Cell Proliferation | Humans | Middle Aged | Endometrial Neoplasms - metabolism | Gene Expression Regulation, Neoplastic | MicroRNAs - metabolism | Endometrial Hyperplasia - pathology | Endometrial Neoplasms - prevention & control | Carcinoma, Endometrioid - metabolism | Cyclin D1 - antagonists & inhibitors | Endometrial Hyperplasia - metabolism | Female | 3' Untranslated Regions | Genes, Reporter | Endometrial Hyperplasia - prevention & control | Endometrium - metabolism | Recombinant Proteins - metabolism | Gene Transfer Techniques | Recombinant Proteins - antagonists & inhibitors | Xenograft Model Antitumor Assays | Carcinoma, Endometrioid - prevention & control | Animals | Cell Cycle | Cyclin D1 - genetics | Mice, Nude | Cell Line, Tumor | Endometrial Neoplasms - pathology | Aged | Mice | MicroRNAs - genetics | Endometrium - pathology | Carcinoma, Endometrioid - pathology | Development and progression | MicroRNA | Genetic transcription | Endometrial cancer | Cancer | Cell growth | Gene expression | MicroRNAs | Cell cycle | Index Medicus
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8. Full Text Molecular profile of grade 3 endometrioid endometrial Carcinoma: Is it a type i or type ii endometrial carcinoma?
by Alvarez, Teresa and Miller, Ezra and Duska, Linda and Oliva, Esther
American Journal of Surgical Pathology, ISSN 0147-5185, 05/2012, Volume 36, Issue 5, pp. 753 - 761
Two types of endometrial carcinoma (EC) have been delineated on the basis of clinicopathologic studies. Low-grade endometrioid carcinoma (EEC) is the prototype... 
endometrial endometrioid carcinoma | grade 3 | immunohistochemistry | molecular profile | SURGERY | DIFFERENTIAL EXPRESSION | IMMUNOHISTOCHEMICAL EXPRESSION | CYCLIN D1 | MICROSATELLITE INSTABILITY | PATHOLOGY | PROGNOSTIC VALUE | TUMOR GENE WT1 | DNA MISMATCH REPAIR | CLEAR-CELL | PROTEIN EXPRESSION | SEROUS PAPILLARY CARCINOMA | Immunohistochemistry | Follow-Up Studies | Endometrial Neoplasms - mortality | Tissue Array Analysis | Humans | Middle Aged | Endometrial Neoplasms - metabolism | In Situ Hybridization, Fluorescence | Survival Rate | Carcinoma, Endometrioid - metabolism | Neoplasm Grading | Carcinoma, Endometrioid - mortality | Aged, 80 and over | Biomarkers, Tumor - metabolism | Adult | Endometrial Neoplasms - pathology | Female | Aged | Neoplasm Staging | Carcinoma, Endometrioid - pathology | Index Medicus
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9. Full Text ARID1A Mutations in Endometriosis-Associated Ovarian Carcinomas
by Wiegand, Kimberly C and Shah, Sohrab P and Al-Agha, Osama M and Zhao, Yongjun and Tse, Kane and Zeng, Thomas and Senz, Janine and McConechy, Melissa K and Anglesio, Michael S and Kalloger, Steve E and Yang, Winnie and Heravi-Moussavi, Alireza and Giuliany, Ryan and Chow, Christine and Fee, John and Zayed, Abdalnasser and Prentice, Leah and Melnyk, Nataliya and Turashvili, Gulisa and Delaney, Allen D and Madore, Jason and Yip, Stephen and McPherson, Andrew W and Ha, Gavin and Bell, Lynda and Fereday, Sian and Tam, Angela and Galletta, Laura and Tonin, Patricia N and Provencher, Diane and Miller, Dianne and Jones, Steven J.M and Moore, Richard A and Morin, Gregg B and Oloumi, Arusha and Boyd, Niki and Aparicio, Samuel A and Shih, Ie-Ming and Mes-Masson, Anne-Marie and Bowtell, David D and Hirst, Martin and Gilks, Blake and Marra, Marco A and Huntsman, David G
The New England Journal of Medicine, ISSN 0028-4793, 10/2010, Volume 363, Issue 16, pp. 1532 - 1543
In this study of ovarian clear-cell or endometrioid tumors, nearly half the samples had mutations in the ARID1A gene, which encodes a component of the SWI–SNF... 
MEDICINE, GENERAL & INTERNAL | GRADE SEROUS CARCINOMA | SWI/SNF COMPLEXES | DISTINCT-HISTOLOGIC-TYPE | POOR-PROGNOSIS | MECHANISMS | CLEAR-CELL CARCINOMA | COMPARATIVE GENOMIC HYBRIDIZATION | CANCER | TUMORS | EXPRESSION | Carcinoma, Endometrioid - genetics | Gene Expression | Adenocarcinoma, Clear Cell - metabolism | Humans | Ovarian Neoplasms - pathology | Nuclear Proteins - metabolism | Endometriosis - pathology | Gene Expression Profiling | Transcription Factors - genetics | Endometriosis - complications | Carcinoma, Endometrioid - metabolism | Ovarian Neoplasms - genetics | Transcription Factors - metabolism | Sequence Analysis, RNA | Cell Line, Tumor | Female | Adenocarcinoma, Clear Cell - pathology | Ovarian Neoplasms - metabolism | Mutation | Nuclear Proteins - genetics | Adenocarcinoma, Clear Cell - genetics | Genes, Tumor Suppressor | Carcinoma, Endometrioid - pathology | Care and treatment | Gene mutations | Causes of | Endometriosis | Tumor suppressor genes | Genetic aspects | Research | Ovarian cancer | Medical research | Cancer | Index Medicus | Abridged Index Medicus
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10. Full Text Gene Expression Profiles of Serous, Endometrioid, and Clear Cell Subtypes of Ovarian and Endometrial Cancer
by Kristin K. Zorn and Tomas Bonome and Lisa Gangi and Gadisetti V.R. Chandramouli and Christopher S. Awtrey and Ginger J. Gardner and J. Carl Barrett and Jeff Boyd and Michael J. Birrer
Clinical Cancer Research, ISSN 1078-0432, 09/2005, Volume 11, Issue 18, pp. 6422 - 6430
Purpose: The presence of similar histologic subtypes of epithelial ovarian and endometrial cancers has long been noted, although the relevance of this finding... 
Clear Cell Cancers | Gynecologic cancers: other | Gene expression profiling | Gynecologic cancers: ovarian | ONCOLOGY | DISTINCT-HISTOLOGIC-TYPE | POOR-PROGNOSIS | ADENOCARCINOMA | RESISTANCE | PLATINUM | TUMOR-SUPPRESSOR | RECEPTOR | IDENTIFICATION | CARCINOMA | LESIONS | Immunohistochemistry | Carcinoma, Endometrioid - genetics | Kidney Neoplasms - genetics | Adenocarcinoma, Clear Cell - metabolism | Oligonucleotide Array Sequence Analysis - methods | Humans | Cystadenocarcinoma, Serous - metabolism | Endometrial Neoplasms - metabolism | Gene Expression Regulation, Neoplastic | Carcinoma, Renal Cell - genetics | Gene Expression Profiling | Kidney Neoplasms - metabolism | Cystadenocarcinoma, Serous - genetics | Carcinoma, Endometrioid - metabolism | Ovarian Neoplasms - genetics | Carcinoma, Renal Cell - metabolism | Tumor Suppressor Protein p53 - analysis | Endometrial Neoplasms - genetics | Female | Ovarian Neoplasms - metabolism | Adenocarcinoma, Clear Cell - genetics | Cluster Analysis | Index Medicus
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11. Full Text BAG3 Protein Is Over‐Expressed in Endometrioid Endometrial Adenocarcinomas
by Esposito, Veronica and Baldi, Carlo and Zeppa, Pio and Festa, Michelina and Guerriero, Luana and d'Avenia, Morena and Chetta, Massimiliano and Zullo, Fulvio and De Laurenzi, Vincenzo and Turco, Maria Caterina and Rosati, Alessandra and Guida, Maurizio
Journal of Cellular Physiology, ISSN 0021-9541, 02/2017, Volume 232, Issue 2, pp. 309 - 311
BAG3 protein levels has been analyzed for the first time in human endometrial cancer tissues. BAG3 anti‐apoptotic protein is over‐expressed in endometrial... 
PHYSIOLOGY | ADIPONECTIN | CANCER | CARCINOMA | MIR-29B | CELL BIOLOGY | Humans | Middle Aged | Endometrial Neoplasms - metabolism | Apoptosis Regulatory Proteins - metabolism | Carcinoma, Endometrioid - metabolism | Staining and Labeling | Aged, 80 and over | Cell Line, Tumor | Endometrial Neoplasms - pathology | Female | Aged | Adaptor Proteins, Signal Transducing - metabolism | Carcinoma, Endometrioid - pathology | Endometrial cancer | Development and progression | Index Medicus
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12. Full Text Human epididymis protein 4 (HE4) is a secreted glycoprotein that is overexpressed by serous and endometrioid ovarian carcinomas
by Drapkin, Ronny and Von Horsten, Hans Henning and Lin, Yafang and Mok, Samuel C and Crum, Christopher P and Welch, William R and Hecht, Jonathan L
Cancer Research, ISSN 0008-5472, 03/2005, Volume 65, Issue 6, pp. 2162 - 2169
Among the genes most commonly identified in gene expression profiles of epithelial ovarian carcinomas (EOC) is the gene for human epididymis protein 4 (HE4).... 
Epididymal Secretory Proteins - biosynthesis | Carcinoma, Endometrioid - genetics | Epididymal Secretory Proteins - secretion | Epithelium - metabolism | Humans | RNA, Messenger - genetics | Cystadenocarcinoma, Serous - metabolism | beta-Defensins | Cystadenocarcinoma, Serous - genetics | Carcinoma, Endometrioid - metabolism | Ovarian Neoplasms - secretion | Ovarian Neoplasms - genetics | RNA, Messenger - biosynthesis | Cell Line, Tumor | Female | Biomarkers, Tumor - genetics | Ovarian Neoplasms - metabolism | Mullerian Ducts - metabolism | Epididymal Secretory Proteins - genetics | Biomarkers, Tumor - biosynthesis | Index Medicus
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13. Full Text Clear cell and endometrioid carcinomas: are their differences attributable to distinct cells of origin?
by Cochrane, Dawn R and Tessier‐Cloutier, Basile and Lawrence, Katherine M and Nazeran, Tayyebeh and Karnezis, Anthony N and Salamanca, Clara and Cheng, Angela S and McAlpine, Jessica N and Hoang, Lien N and Gilks, C Blake and Huntsman, David G
The Journal of Pathology, ISSN 0022-3417, 09/2017, Volume 243, Issue 1, pp. 26 - 36
Endometrial epithelium is the presumed tissue of origin for both eutopic and endometriosis‐derived clear cell and endometrioid carcinomas. We had previously... 
endometriosis | endometrioid cancer | ciliated cells | clear cell cancer | AIRWAY EPITHELIAL-CELLS | ADENOCARCINOMA | NAPSIN | FALLOPIAN-TUBE | OVARIAN-CANCER | PATHOLOGY | TISSUE ANALYSIS CONSORTIUM | HORMONE | ONCOLOGY | MUTATIONS | EXPRESSION | Cilia - pathology | Epithelial Cells - metabolism | Tissue Array Analysis | Humans | Cells, Cultured | Endometrial Neoplasms - metabolism | Cystathionine gamma-Lyase - metabolism | Epithelial Cells - pathology | Stem Cells - metabolism | Cilia - metabolism | Carcinoma, Endometrioid - metabolism | Cell Lineage | Neoplastic Stem Cells - metabolism | Fluorescent Antibody Technique | Biomarkers, Tumor - metabolism | Neoplastic Stem Cells - pathology | Stem Cells - pathology | Endometrial Neoplasms - pathology | Female | Immunophenotyping - methods | Cell Differentiation | Carcinoma, Endometrioid - pathology | Immunohistochemistry | Analysis | Stem cells | Cancer | Ovarian cancer | Cell culture | Ovarian carcinoma | Markers | Staining | Cell lineage | Endometriosis | Tissues | Epithelium | Organoids | Tubes | Mutation | Endometrium | Index Medicus
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