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1. Full Text Mouse Model of Human Ovarian Endometrioid Adenocarcinoma Based on Somatic Defects in the Wnt/β-Catenin and PI3K/Pten Signaling Pathways
by Wu, Rong and Hendrix-Lucas, Neali and Kuick, Rork and Zhai, Yali and Schwartz, Donald R and Akyol, Aytekin and Hanash, Samir and Misek, David E and Katabuchi, Hidetaka and Williams, Bart O and Fearon, Eric R and Cho, Kathleen R
Cancer cell, ISSN 1535-6108, 04/2007, Volume 11, Issue 4, pp. 321 - 333
CELLCYCLE | Oncology | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Adenomatous Polyposis Coli Protein - physiology | Wnt1 Protein - genetics | Oligonucleotide Array Sequence Analysis | Adenocarcinoma, Mucinous - pathology | Humans | PTEN Phosphohydrolase - physiology | Cystadenocarcinoma, Serous - metabolism | Gene Expression Regulation, Neoplastic | Ovarian Neoplasms - pathology | Gene Expression Profiling | Phosphatidylinositol 3-Kinases - metabolism | Wnt1 Protein - metabolism | Cystadenocarcinoma, Serous - genetics | Cystadenocarcinoma, Serous - pathology | Tumor Suppressor Protein p53 - genetics | Carcinoma, Endometrioid - metabolism | Ovarian Neoplasms - genetics | Female | Ovarian Neoplasms - metabolism | Disease Models, Animal | Ovary - metabolism | Adenomatous Polyposis Coli Protein - genetics | PTEN Phosphohydrolase - genetics | Carcinoma, Endometrioid - genetics | Epithelium - pathology | Ovary - pathology | Epithelium - metabolism | Signal Transduction | Adenocarcinoma, Clear Cell - metabolism | Tumor Suppressor Protein p53 - metabolism | Survival Rate | beta Catenin - metabolism | beta Catenin - genetics | Phosphatidylinositol 3-Kinases - genetics | Animals | Adenocarcinoma, Mucinous - metabolism | Adenocarcinoma, Clear Cell - pathology | Mice | Adenocarcinoma, Mucinous - genetics | Mutation | Neoplasm Staging | Adenocarcinoma, Clear Cell - genetics | Carcinoma, Endometrioid - pathology | Index Medicus
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2. Full Text Should grade 3 endometrioid endometrial carcinoma be considered a type 2 cancer—A clinical and pathological evaluation
by Voss, Martin A and Ganesan, Raji and Ludeman, Linmarie and McCarthy, Keith and Gornall, Robert and Schaller, Gerhard and Wei, Wenbin and Sundar, Sudha
Gynecologic oncology, ISSN 0090-8258, 2011, Volume 124, Issue 1, pp. 15 - 20
Hematology, Oncology and Palliative Medicine | Obstetrics and Gynecology | Immunohistochemistry | Endometrial cancer | Uterine papillary serous and clear cell carcinoma | Type I and II | Grade 3 endometrioid | Survival | Oncology | Life Sciences & Biomedicine | Obstetrics & Gynecology | Science & Technology | Receptors, Estrogen - metabolism | Carcinoma, Endometrioid - classification | Endometrial Neoplasms - classification | Humans | Middle Aged | Endometrial Neoplasms - metabolism | Receptor, ErbB-2 - metabolism | Tumor Suppressor Protein p53 - metabolism | Survival Rate | Carcinoma, Endometrioid - metabolism | Receptors, Progesterone - metabolism | Neoplasm Grading | Aged, 80 and over | Adult | Endometrial Neoplasms - pathology | Female | Aged | Neoplasm Staging | Carcinoma, Endometrioid - pathology | Carcinoma | Research | Oncology, Experimental | Cancer | Index Medicus
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3. Full Text A novel Wnt regulatory axis in endometrioid endometrial cancer
by Zhao, Yu and Yang, Yihua and Trovik, Jone and Sun, Kun and Zhou, Liang and Jiang, Peiyong and Lau, Tat-San and Hoivik, Erling A and Salvesen, Helga B and Sun, Hao and Wang, Huating
Cancer research (Chicago, Ill.), ISSN 0008-5472, 08/2014, Volume 74, Issue 18, pp. 5103 - 5117
Life Sciences & Biomedicine | Oncology | Science & Technology | Cell Proliferation | Cadherins - metabolism | Humans | Endometrial Neoplasms - metabolism | Gene Expression Profiling | Carcinoma, Endometrioid - metabolism | DNA Methylation | Heterografts | Endometrial Neoplasms - genetics | Female | Cadherins - genetics | Promoter Regions, Genetic | Carcinoma, Endometrioid - genetics | Down-Regulation | Gene Silencing | RNA, Long Noncoding - genetics | beta Catenin - metabolism | beta Catenin - genetics | Animals | Wnt Signaling Pathway - genetics | Mice, Nude | Cell Line, Tumor | Endometrial Neoplasms - pathology | Apoptosis - physiology | RNA, Long Noncoding - metabolism | Carcinoma, Endometrioid - pathology | Index Medicus
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4. Full Text Mismatch repair deficiency identifies patients with high‐intermediate–risk (HIR) endometrioid endometrial cancer at the highest risk of recurrence: A prognostic biomarker
by Backes, Floor J and Haag, Jennifer and Cosgrove, Casey M and Suarez, Adrian and Cohn, David E and Goodfellow, Paul J
Cancer, ISSN 0008-543X, 02/2019, Volume 125, Issue 3, pp. 398 - 405
recurrence | endometrial cancer | adjuvant treatment | mismatch repair (MMR) deficiency | high‐intermediate–risk | high-intermediate–risk | Life Sciences & Biomedicine | Oncology | Science & Technology | Immunohistochemistry | Prognosis | Endometrial Neoplasms - mortality | Humans | Endometrial Neoplasms - metabolism | Neoplastic Syndromes, Hereditary - metabolism | Carcinoma, Endometrioid - diagnosis | Brain Neoplasms - metabolism | Colorectal Neoplasms - diagnosis | Carcinoma, Endometrioid - metabolism | Neoplastic Syndromes, Hereditary - mortality | DNA Repair Enzymes - metabolism | Biomarkers, Tumor - metabolism | Female | Retrospective Studies | Brain Neoplasms - mortality | Colorectal Neoplasms - metabolism | Colorectal Neoplasms - mortality | Neoplasm Recurrence, Local - metabolism | Biomarkers, Tumor - analysis | Brain Neoplasms - diagnosis | Risk Factors | Neoplastic Syndromes, Hereditary - diagnosis | Neoplasm Recurrence, Local - diagnosis | Endometrial Neoplasms - diagnosis | DNA Repair Enzymes - analysis | Carcinoma, Endometrioid - mortality | Survival Analysis | Endometrial Neoplasms - pathology | Aged | Carcinoma, Endometrioid - pathology | Endometrial cancer | MLH1 protein | MSH2 protein | Health risks | Vagina | Homology | Regression analysis | Patients | Survival | Proteins | MSH6 protein | Survival analysis | Correlation analysis | Mismatch repair | Risk assessment | Biomarkers | Hazard assessment | Repair | Endometrium | Cancer | Index Medicus | Abridged Index Medicus
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5. Full Text Utility of p16 expression for distinction of uterine serous carcinomas from endometrial endometrioid and endocervical adenocarcinomas: Immunohistochemical analysis of 201 cases
by Yemelyanova, Anna and Ji, Hongxiu and Shih, Ie-Ming and Wang, Tian-Li and Wu, Lee-Shu-Fune and Ronnett, Brigitte M
The American journal of surgical pathology, ISSN 0147-5185, 10/2009, Volume 33, Issue 10, pp. 1504 - 1514
Adenocarcinoma | Immunohistochemistry | P16 | Uterus | Endometrioid carcinoma | Serous carcinoma | Endocervical adenocarcinoma | Pathology | Life Sciences & Biomedicine | Surgery | Science & Technology | Gynecology. Andrology. Obstetrics | Investigative techniques, diagnostic techniques (general aspects) | Pathology. Cytology. Biochemistry. Spectrometry. Miscellaneous investigative techniques | Biological and medical sciences | Medical sciences | Female genital diseases | Tumors | Humans | Cystadenocarcinoma, Serous - metabolism | Endometrial Neoplasms - metabolism | Cystadenocarcinoma, Serous - genetics | Carcinoma, Endometrioid - diagnosis | Papillomavirus Infections - diagnosis | Tumor Suppressor Protein p53 - genetics | Carcinoma, Endometrioid - metabolism | Endometrial Neoplasms - genetics | Uterine Cervical Neoplasms - metabolism | DNA Mutational Analysis | Polymerase Chain Reaction | Cyclin-Dependent Kinase Inhibitor p16 - biosynthesis | Female | Uterine Neoplasms - diagnosis | Uterine Neoplasms - genetics | Carcinoma, Endometrioid - genetics | Diagnosis, Differential | Papillomavirus Infections - complications | Uterine Neoplasms - metabolism | Uterine Cervical Neoplasms - diagnosis | Biomarkers, Tumor - analysis | In Situ Hybridization, Fluorescence | Endometrial Neoplasms - diagnosis | Papillomavirus Infections - metabolism | Uterine Cervical Neoplasms - virology | Cystadenocarcinoma, Serous - diagnosis | Index Medicus
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6. Full Text Molecular Heterogeneity of Endometrioid Ovarian Carcinoma: An Analysis of 166 Cases Using the Endometrial Cancer Subrogate Molecular Classification
by Leskela, Susanna and Romero, Ignacio and Rosa-Rosa, Juan M and Caniego-Casas, Tamara and Cristobal, Eva and Pérez-Mies, Belén and Gutierrez-Pecharroman, Ana and Santón, Almudena and Ojeda, Belén and López-Reig, Raquel and Palacios-Berraquero, María L and Andrada, Encarna and Montes, Santiago and Pastor, Francisco and Gomez, Maria C and López-Guerrero, José A and Poveda, Andrés and Palacios, José
The American journal of surgical pathology, ISSN 0147-5185, 07/2020, Volume 44, Issue 7, pp. 982 - 990
Pathology | Life Sciences & Biomedicine | Surgery | Science & Technology | Immunohistochemistry | Carcinoma, Endometrioid - genetics | Prognosis | Follow-Up Studies | Ovarian Neoplasms - diagnosis | Tissue Array Analysis | Humans | Middle Aged | Ovarian Neoplasms - pathology | Carcinoma, Endometrioid - diagnosis | Carcinoma, Endometrioid - metabolism | Ovarian Neoplasms - genetics | Biomarkers, Tumor - metabolism | Adult | Female | Aged | Biomarkers, Tumor - genetics | High-Throughput Nucleotide Sequencing | Ovarian Neoplasms - metabolism | Retrospective Studies | Mutation | Neoplasm Staging | Carcinoma, Endometrioid - pathology | Index Medicus
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7. Full Text MicroRNA‐503 suppresses proliferation and cell‐cycle progression of endometrioid endometrial cancer by negatively regulating cyclin D1
by Xu, Yan‐Ying and Wu, Hui‐Juan and Ma, Hong‐Da and Xu, Li‐Ping and Huo, Yan and Yin, Li‐Rong
The FEBS journal, ISSN 1742-464X, 08/2013, Volume 280, Issue 16, pp. 3768 - 3779
microRNA | endometrioid endometrial cancer | cyclin D1 | miR‐503 | target gene | miR-503 | Life Sciences & Biomedicine | Biochemistry & Molecular Biology | Science & Technology | Cyclin D1 - metabolism | Cell Proliferation | Humans | Middle Aged | Endometrial Neoplasms - metabolism | Gene Expression Regulation, Neoplastic | MicroRNAs - metabolism | Endometrial Hyperplasia - pathology | Endometrial Neoplasms - prevention & control | Carcinoma, Endometrioid - metabolism | Cyclin D1 - antagonists & inhibitors | Endometrial Hyperplasia - metabolism | Female | 3' Untranslated Regions | Genes, Reporter | Endometrial Hyperplasia - prevention & control | Endometrium - metabolism | Recombinant Proteins - metabolism | Gene Transfer Techniques | Recombinant Proteins - antagonists & inhibitors | Xenograft Model Antitumor Assays | Carcinoma, Endometrioid - prevention & control | Animals | Cell Cycle | Cyclin D1 - genetics | Mice, Nude | Cell Line, Tumor | Endometrial Neoplasms - pathology | Aged | Mice | MicroRNAs - genetics | Endometrium - pathology | Carcinoma, Endometrioid - pathology | Development and progression | MicroRNA | Genetic transcription | Endometrial cancer | Cancer | Cell growth | Gene expression | MicroRNAs | Cell cycle | Index Medicus
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8. Full Text Disrupting MALAT1/miR-200c sponge decreases invasion and migration in endometrioid endometrial carcinoma
by Li, Qiulian and Zhang, Chao and Chen, Ruichao and Xiong, Hanzhen and Qiu, Fuman and Liu, Shaoyan and Zhang, Minfen and Wang, Fang and Wang, Yu and Zhou, Xuan and Xiao, Guohong and Wang, Xudong and Jiang, Qingping
Cancer letters, ISSN 0304-3835, 2016, Volume 383, Issue 1, pp. 28 - 40
Hematology, Oncology and Palliative Medicine | MALAT1 | Endometrioid endometrial carcinoma | miR-200c | Invasion | Life Sciences & Biomedicine | Oncology | Science & Technology | Humans | Endometrial Neoplasms - metabolism | Gene Expression Regulation, Neoplastic | MicroRNAs - metabolism | Carcinoma, Endometrioid - metabolism | Transfection | Endometrial Neoplasms - genetics | RNA Interference | Time Factors | Epithelial-Mesenchymal Transition | Biomarkers, Tumor - metabolism | Female | 3' Untranslated Regions | Binding Sites | Carcinoma, Endometrioid - genetics | Signal Transduction | Neoplasm Invasiveness | RNA, Long Noncoding - genetics | Tumor Burden | Cell Movement - drug effects | Transforming Growth Factor beta - pharmacology | Animals | Mice, Nude | Cell Line, Tumor | Endometrial Neoplasms - pathology | Biomarkers, Tumor - genetics | Mice, Inbred BALB C | MicroRNAs - genetics | RNA, Long Noncoding - metabolism | Carcinoma, Endometrioid - pathology | Medical colleges | Carcinoma | Melanoma | Antisense RNA | Metastasis | Transforming growth factors | Ovarian cancer | Prevention | Analysis | Stem cells | Bone morphogenetic proteins | Health aspects | Cancer | Index Medicus
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9. Full Text CTNNB1 Mutations and Aberrant β-Catenin Expression in Ovarian Endometrioid Carcinoma: Correlation With Patient Outcome
by Zyla, Roman E and Olkhov-Mitsel, Ekaterina and Amemiya, Yutaka and Bassiouny, Dina and Seth, Arun and Djordjevic, Bojana and Nofech-Mozes, Sharon and Parra-Herran, Carlos
The American journal of surgical pathology, ISSN 0147-5185, 01/2021, Volume 45, Issue 1, pp. 68 - 76
Carcinoma, Endometrioid - genetics | Prognosis | Biomarkers, Tumor - analysis | beta Catenin - analysis | Humans | Middle Aged | Treatment Outcome | Ovarian Neoplasms - mortality | beta Catenin - metabolism | beta Catenin - genetics | Carcinoma, Endometrioid - metabolism | Ovarian Neoplasms - genetics | Disease-Free Survival | Carcinoma, Endometrioid - mortality | Aged, 80 and over | Adult | Female | Aged | Ovarian Neoplasms - metabolism | Mutation | Index Medicus
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10. Full Text L1CAM Expression is Related to Non-Endometrioid Histology, and Prognostic for Poor Outcome in Endometrioid Endometrial Carcinoma
by Geels, Y.P and Pijnenborg, J.M.A and Gordon, B.B and Fogel, M and Altevogt, P and Masadah, R and Bulten, J and Kempen, L.C. van and Massuger, L.F.A.G
Pathology oncology research, ISSN 1219-4956, 2016, Volume 22, Issue 4, pp. 863 - 868
Immunohistochemistry | Prognostic value | Pathology | Biomedicine | Immunology | Cancer Research | Oncology | Non-endometrioid | L1CAM | Endometrial carcinoma | Biomedicine general | Histopathological diagnosis | Life Sciences & Biomedicine | Science & Technology | Prognosis | Endometrial Neoplasms - mortality | Humans | Middle Aged | Endometrial Neoplasms - metabolism | Survival Rate | Disease Progression | Carcinoma, Endometrioid - metabolism | Young Adult | Disease-Free Survival | Carcinoma, Endometrioid - mortality | Aged, 80 and over | Biomarkers, Tumor - metabolism | Adult | Endometrial Neoplasms - pathology | Female | Aged | Neural Cell Adhesion Molecule L1 - metabolism | Carcinoma, Endometrioid - pathology | Carcinoma | Cancer | Index Medicus | Short Communication
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11. Full Text Molecular Modifiers of Hormone Receptor Action: Decreased Androgen Receptor Expression in Mismatch Repair Deficient Endometrial Endometrioid Adenocarcinoma
by Gan, Qiong and Crumley, Suzanne and Broaddus, Russell R
International journal of gynecological pathology, ISSN 0277-1691, 01/2019, Volume 38, Issue 1, pp. 44 - 51
NF-κB | Androgen receptor | Endometrial endometrioid carcinoma | Mismatch repair genes | Pathology | Life Sciences & Biomedicine | Obstetrics & Gynecology | Science & Technology | Immunohistochemistry | Neoplastic Syndromes, Hereditary - pathology | Receptors, Estrogen - metabolism | Humans | Receptors, Androgen - metabolism | Brain Neoplasms - pathology | Endometrial Neoplasms - metabolism | NF-kappa B - metabolism | Neoplastic Syndromes, Hereditary - metabolism | DNA Mismatch Repair - genetics | Brain Neoplasms - metabolism | Case-Control Studies | Carcinoma, Endometrioid - metabolism | Receptors, Progesterone - metabolism | DNA Methylation | Endometrial Neoplasms - pathology | Female | Aged | MutL Protein Homolog 1 - metabolism | Androgens - metabolism | Colorectal Neoplasms - pathology | Carcinoma, Endometrioid - pathology | Colorectal Neoplasms - metabolism | Cohort Studies | Development and progression | Genetic aspects | Endometrial cancer | Health aspects | DNA mismatch repair | Index Medicus
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