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Journal Article
Cancer Research, ISSN 0008-5472, 09/2015, Volume 75, Issue 17, pp. 3479 - 3491
Journal Article
Nature, ISSN 0028-0836, 2014, Volume 513, Issue 7516, pp. 100 - 104
Journal Article
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 04/2015, Volume 290, Issue 17, pp. 11177 - 11187
Cachexia occurs in patients with advanced cancers. Despite the adverse clinical impact of cancer-induced muscle wasting, pathways causing cachexia are... 
TRANSCRIPTION FACTORS | SKELETAL-MUSCLE | PROTEIN-DEGRADATION | MYOSTATIN GENE | PATHWAY | ATROPHY | BIOCHEMISTRY & MOLECULAR BIOLOGY | KAPPA-B | UP-REGULATION | EXPRESSION | CATABOLIC CONDITIONS | Colonic Neoplasms - genetics | SKP Cullin F-Box Protein Ligases - genetics | Caspase 3 - metabolism | Ubiquitin - metabolism | Muscle, Skeletal - metabolism | Colonic Neoplasms - metabolism | Tripartite Motif Proteins | Proteolysis | Caspase 3 - genetics | Muscle Proteins - metabolism | Myostatin - genetics | CCAAT-Enhancer-Binding Protein-delta - metabolism | Myostatin - metabolism | Cachexia - metabolism | STAT3 Transcription Factor - genetics | STAT3 Transcription Factor - metabolism | Cachexia - genetics | Ubiquitin-Protein Ligases - metabolism | SKP Cullin F-Box Protein Ligases - metabolism | Ubiquitin - genetics | Carcinoma, Lewis Lung - metabolism | Mice, Knockout | Muscle Proteins - genetics | Proteasome Endopeptidase Complex - genetics | Animals | Carcinoma, Lewis Lung - genetics | CCAAT-Enhancer-Binding Protein-delta - genetics | Cachexia - pathology | Colonic Neoplasms - pathology | Carcinoma, Lewis Lung - pathology | Cell Line, Tumor | Mice | Muscle, Skeletal - pathology | Proteasome Endopeptidase Complex - metabolism | Ubiquitin-Protein Ligases - genetics | Ubiquitin | Signal Transduction | STAT3 | Caspase | Cancer Cachexia | Cancer Biology | Muscle Atrophy | Caspase-3
Journal Article
Cancer Research, ISSN 0008-5472, 09/2017, Volume 77, Issue 18, pp. 4809 - 4822
Dissociation from epithelial sheets and invasion through the surrounding stroma are critical early events during epithelial cancer metastasis. Here we find... 
TRANSCRIPTION FACTORS | BREAST-CANCER | NUCLEAR-ORGANIZATION | PERICELLULAR PROTEOLYSIS | EPITHELIAL-CELLS | ETS PROTEIN | ONCOLOGY | COLLECTIVE INVASION | BONE-MARROW | GENE-EXPRESSION | FACTORS PU.1 | Cell Proliferation | Prognosis | Carcinoma, Squamous Cell - genetics | Carcinoma, Squamous Cell - metabolism | Humans | Lung Neoplasms - metabolism | Carcinoma, Lewis Lung - secondary | Gene Expression Regulation, Neoplastic | Lung Neoplasms - pathology | Biomarkers, Tumor | Intercellular Junctions | Small Cell Lung Carcinoma - secondary | DNA-Binding Proteins - metabolism | Small Cell Lung Carcinoma - metabolism | Adenocarcinoma - metabolism | Carcinoma, Non-Small-Cell Lung - secondary | Adenocarcinoma - genetics | Tumor Cells, Cultured | Claudin-2 - metabolism | Claudin-2 - genetics | Lung Neoplasms - genetics | Carcinoma, Non-Small-Cell Lung - genetics | Neoplasm Invasiveness | Mice, Inbred C57BL | Carcinoma, Non-Small-Cell Lung - metabolism | Small Cell Lung Carcinoma - genetics | Transcription Factors - genetics | Carcinoma, Lewis Lung - metabolism | DNA-Binding Proteins - genetics | Adenocarcinoma - secondary | Transcription Factors - metabolism | Xenograft Model Antitumor Assays | Animals | Carcinoma, Lewis Lung - genetics | Carcinoma, Squamous Cell - secondary | Mice | Neoplasm Staging | Apoptosis | Vimentin | Repressing | Chromatin | Transcription factors | Mesenchyme | Invasiveness | Lung cancer | Stroma | Metastasis | Tissues | E-cadherin | Mimicry | Metastases | Lymphocytes B | Lymphocytes | Tumors | Cancer
Journal Article
Oncogene, ISSN 0950-9232, 05/2011, Volume 30, Issue 21, pp. 2475 - 2484
Tumor-associated macrophages (TAMs) constitute a major component of the immune cell infiltrate observed in the tumor microenvironment (TME). Factors present in... 
IRAK-M | lung cancer | Toll-like receptors | TGF-β | tumorassociated macrophages | TRANSFORMING GROWTH-FACTOR-BETA-1 LEVEL | ADENOCARCINOMA | BIOCHEMISTRY & MOLECULAR BIOLOGY | EARLY-STAGE | CANCER | CHEMOTHERAPY | tumor-associated macrophages | NEGATIVE REGULATOR | CELL BIOLOGY | TGF-beta | ONCOLOGY | INFLAMMATION | GENETICS & HEREDITY | PROGRESSION | DEACTIVATION | Interleukin-1 Receptor-Associated Kinases - metabolism | Interleukin-1 Receptor-Associated Kinases - genetics | Oligonucleotide Array Sequence Analysis | Interleukin-12 - genetics | Coculture Techniques | Humans | Lung Neoplasms - metabolism | Lung Neoplasms - pathology | Male | Monocytes - metabolism | Gene Expression Profiling | Interferon-gamma - metabolism | Female | Gene Expression Regulation, Neoplastic - drug effects | Interferon-gamma - genetics | Lung Neoplasms - genetics | Cell Line | Mice, Inbred C57BL | Cells, Cultured | Interleukin-12 - metabolism | Carcinoma, Lewis Lung - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Mice, Knockout | Monocytes - drug effects | Transforming Growth Factor beta - pharmacology | Macrophages - metabolism | Animals | Carcinoma, Lewis Lung - genetics | Transforming Growth Factor beta - genetics | Survival Analysis | Carcinoma, Lewis Lung - pathology | Cell Line, Tumor | Mice | Macrophages, Peritoneal - metabolism | Transforming Growth Factor beta - metabolism | toll-like receptors
Journal Article
Journal Article
Cancer Letters, ISSN 0304-3835, 2017, Volume 402, pp. 43 - 51
Abstract Id1 promotes carcinogenesis and metastasis, and predicts prognosis of non-small cell lung cancer (NSCLC)-adenocarcionoma patients. We hypothesized... 
Hematology, Oncology and Palliative Medicine | Epithelial to mesenchymal transition | Vimentin | Liver colonization | Lung cancer | Id1 | PROSTATE | PROGNOSIS | RECEPTOR | FAMILY | TARGETED INHIBITION | THERAPY | ONCOLOGY | PROTEINS | STEREOTACTIC RADIOTHERAPY | EXPRESSION | NSCLC PATIENTS | Cell Proliferation | Vimentin - metabolism | Humans | Lung Neoplasms - metabolism | Transforming Growth Factor beta1 - metabolism | Carcinoma, Lewis Lung - secondary | Gene Expression Regulation, Neoplastic | Tumor Microenvironment | Lung Neoplasms - pathology | Carcinoma, Non-Small-Cell Lung - secondary | Inhibitor of Differentiation Protein 1 - drug effects | Snail Family Transcription Factors - genetics | Time Factors | Vimentin - genetics | Epithelial-Mesenchymal Transition | Liver Neoplasms - secondary | Lung Neoplasms - genetics | Snail Family Transcription Factors - metabolism | Liver Neoplasms - genetics | Signal Transduction | Carcinoma, Non-Small-Cell Lung - genetics | Mice, Inbred C57BL | Carcinoma, Non-Small-Cell Lung - metabolism | Transforming Growth Factor beta1 - genetics | Tumor Burden | Carcinoma, Lewis Lung - metabolism | Inhibitor of Differentiation Protein 1 - metabolism | Mice, Knockout | Integrin beta1 - metabolism | Animals | Carcinoma, Lewis Lung - genetics | Liver Neoplasms - metabolism | Cell Line, Tumor | Inhibitor of Differentiation Protein 1 - genetics | Integrin beta1 - genetics | Cell Movement | Liver cancer | Metastasis | Lung cancer, Non-small cell | Transforming growth factors | Liver | Stem cells | Cell proliferation | Cancer therapies | Carcinogenesis | Metastases | Proteins | Angiogenesis | Carcinogens | Growth factors | Cell survival | Small cell lung carcinoma | Tumor cells | Mortality | Non-small cell lung carcinoma | Breast cancer | Lung carcinoma | Patients | Esophagus | Nodules | Hepatocytes | Medical prognosis | Protein expression | Mice | Differentiation | Colonization | Cancer | Index Medicus
Journal Article
Nature, ISSN 0028-0836, 06/2010, Volume 465, Issue 7299, pp. 813 - 817
Down's syndrome (DS) is a genetic disorder caused by full or partial trisomy of human chromosome 21 and presents with many clinical phenotypes including a... 
CADHERIN | APOPTOSIS | ADAMTS-1 | ENDOTHELIAL-CELL PROLIFERATION | GENE | VEGF | MULTIDISCIPLINARY SCIENCES | GROWTH | DSCR1 | MICE | ETS2 | Transcriptional Regulator ERG | Neoplasm Transplantation | Oncogene Proteins - genetics | Cell Adhesion Molecules - genetics | Chromosomes, Mammalian - genetics | Humans | Cell Adhesion Molecules - antagonists & inhibitors | Male | Down Syndrome - physiopathology | Vascular Endothelial Growth Factor A - metabolism | Immunoglobulins - genetics | Vascular Endothelial Growth Factor A - antagonists & inhibitors | Carcinoma, Lewis Lung - blood supply | Neovascularization, Pathologic - pathology | Immunoglobulins - metabolism | Carcinoma, Lewis Lung - complications | Proto-Oncogene Protein c-ets-2 - genetics | Down Syndrome - complications | Female | Proto-Oncogene Protein c-ets-2 - metabolism | Vascular Endothelial Growth Factor A - pharmacology | ADAMTS1 Protein | Disease Models, Animal | Gene Dosage - genetics | Melanoma, Experimental - blood supply | Oncogene Proteins - metabolism | Melanoma, Experimental - complications | Melanoma, Experimental - pathology | Vascular Endothelial Growth Factor Receptor-2 - metabolism | Cell Adhesion Molecules - metabolism | Carrier Proteins - genetics | ADAM Proteins - metabolism | Animals | Carcinoma, Lewis Lung - genetics | Carrier Proteins - metabolism | Melanoma, Experimental - genetics | Trisomy - genetics | Down Syndrome - genetics | Carcinoma, Lewis Lung - pathology | Neovascularization, Pathologic - genetics | Mice | Transcription Factors | ADAM Proteins - genetics | Complications and side effects | Physiological aspects | Development and progression | Genetic aspects | Down syndrome | Neovascularization | Research | Risk factors | Tumors | Proteins | Studies | Angiogenesis | Genotype & phenotype | Rodents | Blood vessels | Genetic engineering | Gene therapy | Vascular endothelial growth factor
Journal Article