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Cancer Cell, ISSN 1535-6108, 03/2012, Volume 21, Issue 3, pp. 418 - 429
Pancreatic ductal adenocarcinomas (PDAs) are characterized by a robust fibroinflammatory response. We show here that this desmoplastic reaction generates... 
INTERSTITIAL FLUID PRESSURE | SOLID TUMORS | ONCOLOGY | COMPARING STANDARD PANCREATICODUODENECTOMY | HUMAN OSTEOSARCOMA XENOGRAFTS | RANDOMIZED-TRIAL | HEAD ADENOCARCINOMA | TUMOR VASCULATURE | EXTENDED LYMPHADENECTOMY | CANCER | BLOOD-PRESSURE | CELL BIOLOGY | Adenocarcinoma - pathology | Cell Adhesion Molecules - administration & dosage | Stromal Cells - pathology | Pancreatic Neoplasms - blood supply | Deoxycytidine - pharmacology | Hyaluronic Acid - physiology | Polyethylene Glycols - therapeutic use | Antineoplastic Combined Chemotherapy Protocols - pharmacology | Pancreatic Neoplasms - drug therapy | Deoxycytidine - therapeutic use | Stromal Cells - drug effects | Hyaluronoglucosaminidase - administration & dosage | Drug Evaluation, Preclinical | Hyaluronoglucosaminidase - therapeutic use | Tumor Microenvironment - drug effects | Animals, Genetically Modified | Deoxycytidine - administration & dosage | Pancreatic Neoplasms - pathology | Microvessels - drug effects | Adenocarcinoma - drug therapy | Carcinoma, Pancreatic Ductal - pathology | Extracellular Fluid - drug effects | Polyethylene Glycols - administration & dosage | Adenocarcinoma - blood supply | Hyaluronoglucosaminidase - pharmacology | Carcinoma, Pancreatic Ductal - drug therapy | Animals | Antineoplastic Combined Chemotherapy Protocols - therapeutic use | Hyaluronic Acid - metabolism | Mice | Carcinoma, Pancreatic Ductal - blood supply | Cell Adhesion Molecules - therapeutic use | Deoxycytidine - analogs & derivatives | Enzymes | Medical colleges | Care and treatment | Glycosaminoglycans | Oncology, Experimental | Research | Chemotherapy | Universities and colleges | Drug therapy | Hyaluronic acid | Public health | Tumors | Cancer
Journal Article
Gut, ISSN 0017-5749, 01/2013, Volume 62, Issue 1, pp. 112 - 120
Objective Pancreatic ductal adenocarcinoma (PDA) is characterised by stromal desmoplasia and vascular dysfunction, which critically impair drug delivery. This... 
INTERSTITIAL FLUID PRESSURE | CELLS | THERAPY | SOLID TUMORS | MICROENVIRONMENT | PERMEABILITY | DUCTAL ADENOCARCINOMA | GASTROENTEROLOGY & HEPATOLOGY | GEMCITABINE | CHEMOTHERAPY | ENDOTHELIAL GROWTH-FACTOR | Immunohistochemistry | Cell Adhesion Molecules - administration & dosage | Tissue Array Analysis | Pancreatic Neoplasms - blood supply | Antineoplastic Agents - administration & dosage | Hyaluronic Acid - physiology | Antineoplastic Combined Chemotherapy Protocols - pharmacology | Drug Delivery Systems | Pancreatic Neoplasms - drug therapy | Hyaluronoglucosaminidase - administration & dosage | Carcinoma, Pancreatic Ductal - mortality | Cell Adhesion Molecules - pharmacology | Pancreatic Neoplasms - mortality | Doxorubicin - administration & dosage | Deoxycytidine - administration & dosage | Kaplan-Meier Estimate | Mice, Transgenic | Treatment Outcome | Biomarkers, Tumor - physiology | Recombinant Proteins - pharmacology | Recombinant Proteins - administration & dosage | Hyaluronoglucosaminidase - pharmacology | Carcinoma, Pancreatic Ductal - drug therapy | Animals | Antineoplastic Combined Chemotherapy Protocols - therapeutic use | Carcinoma, Pancreatic Ductal - physiopathology | Mice | Carcinoma, Pancreatic Ductal - blood supply | Pancreatic Neoplasms - physiopathology | Drug Resistance, Neoplasm - physiology | Deoxycytidine - analogs & derivatives | Drug Resistance, Neoplasm - drug effects | Care and treatment | Blood-vessels | Pancreatic cancer | Patient outcomes | Physiological aspects | Models | Research | Hyaluronic acid | Tumors | Studies | Angiogenesis | Medical research | Medical prognosis | Cytotoxicity | Extracellular matrix | Permeability | Kinases | Vascular endothelial growth factor | stem cells | cell death | cancer vaccines | hyaluronan | epithelial cell growth | cancer immunobiology | drug delivery | matrix | epithelial cell adhesion | cancer genetics | carcinogenesis | pancreatic fibrosis | pharmacology | pharmacokinetics | tumour microenvironment | cancer | fibrosis | pancreatic disease | Original | 1506 | pancreatic tumours | extracellular matrix | oxidative stress
Journal Article
Science, ISSN 0036-8075, 6/2009, Volume 324, Issue 5933, pp. 1457 - 1461
Pancreatic ductal adenocarcinoma (PDA) is among the most lethal human cancers in part because it is insensitive to many chemotherapeutic drugs. Studying a... 
Patent ductus arteriosus | Half lives | Perfusion | Antineoplastics | Ultrasonography | Blood vessels | Reports | Tissue transplantation | Pancreatic neoplasms | Tumors | Apoptosis | SURVIVAL | TRIAL | DIAGNOSIS | METASTASIS | THERAPY | DUCTAL ADENOCARCINOMA | SONIC HEDGEHOG | MULTIDISCIPLINARY SCIENCES | GROWTH | TUMORS | GEMCITABINE | Veratrum Alkaloids - administration & dosage | Neoplasm Transplantation | Pancreatic Neoplasms - metabolism | Receptors, G-Protein-Coupled - metabolism | Apoptosis - drug effects | Stromal Cells - pathology | Veratrum Alkaloids - pharmacokinetics | Humans | Pancreatic Neoplasms - blood supply | Carcinoma, Pancreatic Ductal - metabolism | Neovascularization, Pathologic | Hedgehog Proteins - metabolism | Drug Resistance, Neoplasm | Antineoplastic Agents - therapeutic use | Antineoplastic Agents - administration & dosage | Antineoplastic Agents - metabolism | Pancreatic Neoplasms - drug therapy | Deoxycytidine - therapeutic use | Deoxycytidine - metabolism | Kruppel-Like Transcription Factors - metabolism | Smoothened Receptor | Stromal Cells - drug effects | Disease Models, Animal | Veratrum Alkaloids - therapeutic use | Deoxycytidine - administration & dosage | Pancreatic Neoplasms - pathology | Antineoplastic Combined Chemotherapy Protocols | Hedgehog Proteins - antagonists & inhibitors | Carcinoma, Pancreatic Ductal - pathology | Carcinoma, Pancreatic Ductal - drug therapy | Animals | Signal Transduction - drug effects | Receptors, G-Protein-Coupled - antagonists & inhibitors | Cell Line, Tumor | Cell Proliferation - drug effects | Mice | Zinc Finger Protein GLI1 | Carcinoma, Pancreatic Ductal - blood supply | Deoxycytidine - analogs & derivatives | Care and treatment | Chemotherapy | Pancreatic cancer | Physiological aspects | Cellular signal transduction | Health aspects | Cancer | Proteins | Signal transduction | Cellular biology | Rodents
Journal Article
Journal of Cellular and Molecular Medicine, ISSN 1582-1838, 01/2018, Volume 22, Issue 1, pp. 655 - 667
Pancreatic ductal adenocarcinoma (PDAC) is a lethal malignancy. Long non‐coding RNAs (lncRNAs) are important regulators in pathological processes, yet their... 
hsa‐miR‐29b‐3p | linc00511 | Competing endogenous | RNA | Pancreatic ductal adenocarcinoma | VEGFA | hsa-miR-29b-3p | Competing endogenous RNA | MEDICINE, RESEARCH & EXPERIMENTAL | METASTASIS | LONG-NONCODING-RNA | CELL BIOLOGY | CELL LUNG-CANCER | BREAST-CANCER | HEPATOCELLULAR-CARCINOMA | GASTRIC-CANCER | POOR-PROGNOSIS | GENE-EXPRESSION | TUMOR-SUPPRESSOR | ENDOTHELIAL GROWTH-FACTOR | Multivariate Analysis | Adenocarcinoma - pathology | Prognosis | Humans | Middle Aged | Pancreatic Neoplasms - blood supply | Gene Expression Regulation, Neoplastic | Male | MicroRNAs - metabolism | Vascular Endothelial Growth Factor A - metabolism | Cell Movement - genetics | Carcinoma, Pancreatic Ductal - genetics | Base Sequence | Female | Adenocarcinoma - genetics | Cell Proliferation - genetics | Neoplasm Invasiveness | Pancreatic Neoplasms - pathology | Proportional Hazards Models | Pancreatic Neoplasms - genetics | Up-Regulation - genetics | RNA, Long Noncoding - genetics | Carcinoma, Pancreatic Ductal - pathology | Disease Progression | Adenocarcinoma - blood supply | Cell Line, Tumor | Neovascularization, Pathologic - genetics | MicroRNAs - genetics | Carcinoma, Pancreatic Ductal - blood supply | RNA, Long Noncoding - metabolism | Adenocarcinoma | Immunohistochemistry | Medical research | Analysis | Pancreatic cancer | Medicine, Experimental | Health savings accounts | Health aspects | Cell proliferation | Biotechnology | Regulators | Pathogenesis | Fluorescence | Malignancy | Assaying | Western blotting | Angiogenesis | Surgery | Animal tissues | Fluorescence in situ hybridization | Bioindicators | Pancreas | Vascular endothelial growth factor | RNA-mediated interference | Gene expression | Ribonucleic acid--RNA | Cell lines | Biomarkers | In vivo methods and tests | Aberration | Cell migration | Tumors | Original
Journal Article
Journal Article
Cancer Letters, ISSN 0304-3835, 2011, Volume 317, Issue 1, pp. 16 - 23
Journal Article
Gastroenterology, ISSN 0016-5085, 02/2018, Volume 154, Issue 3, pp. 675 - 688
Cells of the monocyte lineage contribute to tumor angiogenesis. Interleukin 35 (IL35) is a member of the IL12 family produced by regulatory, but not effector,... 
Infiltration | Immune Cell Migration | Anti-tumor Immune Response | Tumor Progression | PATHWAYS | METASTASIS | MACROPHAGES | RECEPTOR | MYELOID CELLS | CANCER | TRIAL | ANTICANCER THERAPIES | ENDOTHELIAL-CELLS | GASTROENTEROLOGY & HEPATOLOGY | PLUS BEVACIZUMAB | Interleukin-12 Subunit p35 - metabolism | Pancreatic Neoplasms - metabolism | Human Umbilical Vein Endothelial Cells - metabolism | Humans | Pancreatic Neoplasms - blood supply | Carcinoma, Pancreatic Ductal - metabolism | Microvessels - metabolism | Neovascularization, Pathologic | Microvessels - pathology | Deoxycytidine - pharmacology | Monocytes - metabolism | Interleukins - metabolism | Antineoplastic Combined Chemotherapy Protocols - pharmacology | Interleukin-12 Subunit p35 - antagonists & inhibitors | Pancreatic Neoplasms - drug therapy | Interleukins - genetics | Transfection | Chemotaxis, Leukocyte - drug effects | RNA Interference | Time Factors | Minor Histocompatibility Antigens - genetics | Female | Chemokine CCL5 - metabolism | Interleukin-8 - metabolism | Chemokine CXCL1 - metabolism | Paracrine Communication | Signal Transduction | Pancreatic Neoplasms - pathology | Microvessels - drug effects | Antibodies, Neutralizing - pharmacology | Interleukin-12 Subunit p35 - genetics | Tumor Burden | Mice, SCID | Carcinoma, Pancreatic Ductal - pathology | Interleukins - antagonists & inhibitors | Monocytes - drug effects | Xenograft Model Antitumor Assays | Carcinoma, Pancreatic Ductal - drug therapy | Macrophages - metabolism | Minor Histocompatibility Antigens - metabolism | Animals | Cell Line, Tumor | Cell Proliferation - drug effects | Carcinoma, Pancreatic Ductal - blood supply | Deoxycytidine - analogs & derivatives | Adenocarcinoma | Interleukins | Endothelial growth factors | Growth | Analysis | Pancreatic cancer | T cells
Journal Article
Journal of Experimental Medicine, ISSN 0022-1007, 03/2012, Volume 209, Issue 3, pp. 437 - 444
Journal Article