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Journal Article
Gut, ISSN 0017-5749, 01/2013, Volume 62, Issue 1, pp. 112 - 120
Objective Pancreatic ductal adenocarcinoma (PDA) is characterised by stromal desmoplasia and vascular dysfunction, which critically impair drug delivery. This... 
INTERSTITIAL FLUID PRESSURE | CELLS | THERAPY | SOLID TUMORS | MICROENVIRONMENT | PERMEABILITY | DUCTAL ADENOCARCINOMA | GASTROENTEROLOGY & HEPATOLOGY | GEMCITABINE | CHEMOTHERAPY | ENDOTHELIAL GROWTH-FACTOR | Immunohistochemistry | Cell Adhesion Molecules - administration & dosage | Tissue Array Analysis | Pancreatic Neoplasms - blood supply | Antineoplastic Agents - administration & dosage | Hyaluronic Acid - physiology | Antineoplastic Combined Chemotherapy Protocols - pharmacology | Drug Delivery Systems | Pancreatic Neoplasms - drug therapy | Hyaluronoglucosaminidase - administration & dosage | Carcinoma, Pancreatic Ductal - mortality | Cell Adhesion Molecules - pharmacology | Pancreatic Neoplasms - mortality | Doxorubicin - administration & dosage | Deoxycytidine - administration & dosage | Kaplan-Meier Estimate | Mice, Transgenic | Treatment Outcome | Biomarkers, Tumor - physiology | Recombinant Proteins - pharmacology | Recombinant Proteins - administration & dosage | Hyaluronoglucosaminidase - pharmacology | Carcinoma, Pancreatic Ductal - drug therapy | Animals | Antineoplastic Combined Chemotherapy Protocols - therapeutic use | Carcinoma, Pancreatic Ductal - physiopathology | Mice | Carcinoma, Pancreatic Ductal - blood supply | Pancreatic Neoplasms - physiopathology | Drug Resistance, Neoplasm - physiology | Deoxycytidine - analogs & derivatives | Drug Resistance, Neoplasm - drug effects | Care and treatment | Blood-vessels | Pancreatic cancer | Patient outcomes | Physiological aspects | Models | Research | Hyaluronic acid | Tumors | Studies | Angiogenesis | Medical research | Medical prognosis | Cytotoxicity | Extracellular matrix | Permeability | Kinases | Vascular endothelial growth factor | Index Medicus | Abridged Index Medicus | stem cells | cell death | cancer vaccines | hyaluronan | epithelial cell growth | cancer immunobiology | drug delivery | matrix | epithelial cell adhesion | cancer genetics | carcinogenesis | pancreatic fibrosis | pharmacology | pharmacokinetics | tumour microenvironment | cancer | fibrosis | pancreatic disease | Original | 1506 | pancreatic tumours | extracellular matrix | oxidative stress
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 3/2016, Volume 113, Issue 11, pp. 3078 - 3083
Pancreatic ductal adenocarcinoma (PDAC) is characterized by an exuberant inflammatory desmoplastic response. The PDAC microenvironment is complex, containing... 
Sensory neuron | Inflammation | Tumorigenesis | Pancreatic ductal adenocarcinoma | PanIN | NEUROGENIC INFLAMMATION | RAT PANCREAS | PRIMARY AFFERENT NEURONS | PERINEURAL INVASION | MAP KINASE | MULTIDISCIPLINARY SCIENCES | NEURAL INVASION | SPINAL-CORD | pancreatic ductal adenocarcinoma | NEUROPATHIC PAIN | sensory neuron | inflammation | tumorigenesis | DORSAL COLUMN | CAPSAICIN | Precancerous Conditions - physiopathology | Spinothalamic Tracts - physiopathology | Capsaicin - pharmacology | Capsaicin - therapeutic use | Pancreatic Neoplasms - etiology | Humans | Male | Pancreatitis - complications | Carcinoma, Pancreatic Ductal - prevention & control | Capsaicin - administration & dosage | Pancreas - innervation | Sensory Receptor Cells - physiology | Female | Carcinoma, Pancreatic Ductal - etiology | Pancreatic Neoplasms - prevention & control | Pancreatitis - physiopathology | Precancerous Conditions - complications | Afferent Pathways | Animals, Newborn | Adenocarcinoma in Situ - pathology | Myelitis - genetics | Myelitis - physiopathology | Neoplasm Invasiveness | Mice, Inbred C57BL | Pancreatic Neoplasms - pathology | Ceruletide - toxicity | Mice, Transgenic | Carcinoma, Pancreatic Ductal - therapy | Pancreatitis - chemically induced | Carcinoma, Pancreatic Ductal - pathology | Disease Progression | Myelitis - complications | Animals | Ganglia, Sympathetic - physiopathology | Sensory Receptor Cells - drug effects | Thoracic Vertebrae | Adenocarcinoma in Situ - physiopathology | Carcinoma, Pancreatic Ductal - physiopathology | Mice | Spinal Cord - physiopathology | Pancreatic Neoplasms - physiopathology | Precancerous Conditions - chemically induced | Genes, ras | Denervation | Pancreatic Neoplasms - therapy | Prevention | Sensory receptors | Development and progression | Genetic aspects | Health aspects | Pancreatic cancer | Nervous system | Pancreas | Neurons | Rodents | Cells | Tumors | Index Medicus | Biological Sciences
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 10/2011, Volume 121, Issue 10, pp. 4106 - 4117
Pancreatic ductal adenocarcinoma (PDAC), one of the most lethal neoplasms, is characterized by an expanded stroma with marked fibrosis (desmoplasia). We... 
BREAST-CANCER | MEDICINE, RESEARCH & EXPERIMENTAL | SIGNALING PATHWAYS | IN-VITRO | TGF-BETA | EPITHELIAL-CELLS | K-RAS | CANCER METASTASIS | II RECEPTOR | GROWTH | KAPPA-B | Protein-Serine-Threonine Kinases - deficiency | Receptors, Transforming Growth Factor beta - genetics | Proto-Oncogene Proteins p21(ras) - genetics | Stromal Cells - pathology | Humans | Proto-Oncogene Proteins p21(ras) - physiology | Receptors, Interleukin-8B - antagonists & inhibitors | Tumor Microenvironment - physiology | Carcinoma, Pancreatic Ductal - genetics | Female | Receptors, Interleukin-8B - physiology | Gene Expression | Signal Transduction | Mice, Inbred C57BL | Pancreatic Neoplasms - pathology | Protein-Serine-Threonine Kinases - genetics | Pancreatic Neoplasms - genetics | Carcinoma, Pancreatic Ductal - therapy | Carcinoma, Pancreatic Ductal - pathology | Mice, Knockout | Animals | Connective Tissue Growth Factor - physiology | Mice, Nude | Chemokines, CXC - physiology | Carcinoma, Pancreatic Ductal - physiopathology | Connective Tissue Growth Factor - genetics | Mice | Pancreatic Neoplasms - physiopathology | Stromal Cells - physiology | Pancreatic Neoplasms - therapy | Receptors, Transforming Growth Factor beta - deficiency | Pancreatic cancer | Physiological aspects | Development and progression | Research | Transforming growth factors | Gene expression | Chemokine receptors | Index Medicus | Abridged Index Medicus
Journal Article
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 01/2017, Volume 114, Issue 5, pp. 1129 - 1134
A fibroinflammatory stromal reaction cooperates with oncogenic signaling to influence pancreatic ductal adenocarcinoma (PDAC) initiation, progression, and... 
BRD2 | Histone acetylation | Cancer metabolism | Tumor microenvironment | Pancreatic ductal adenocarcinoma | DUCTAL ADENOCARCINOMA | MULTIDISCIPLINARY SCIENCES | MACROPHAGE | STELLATE CELLS | C-MYC | BROMODOMAIN | pancreatic ductal adenocarcinoma | cancer metabolism | INHIBITION | GENE | MICROENVIRONMENT | tumor microenvironment | histone acetylation | MICROARRAY DATA | PROGRESSION | Pancreatic Neoplasms - metabolism | Fibroblasts - physiology | Humans | Neoplasm Proteins - physiology | Carcinoma, Pancreatic Ductal - metabolism | Neoplasm Proteins - antagonists & inhibitors | Tumor Microenvironment - physiology | Carcinoma, Pancreatic Ductal - genetics | Protein-Serine-Threonine Kinases - antagonists & inhibitors | Histone Code | Acetylation | Intercellular Signaling Peptides and Proteins - secretion | Tumor Cells, Cultured | Gene Expression Regulation, Neoplastic - genetics | Promoter Regions, Genetic | Protein-Serine-Threonine Kinases - physiology | Pancreatic Neoplasms - pathology | Cytokines - secretion | Metabolome | Pancreatic Neoplasms - genetics | Pancreatic Stellate Cells - physiology | Carcinoma, Pancreatic Ductal - pathology | Energy Metabolism | Enhancer Elements, Genetic | Carcinoma, Pancreatic Ductal - physiopathology | Pancreatic Neoplasms - physiopathology | Stromal Cells - physiology | Proteins | Genomes | Mutation | Pancreatic cancer | Cells | Tumors | Index Medicus | Biological Sciences
Journal Article
Journal Article
Genes and Development, ISSN 0890-9369, 12/2016, Volume 30, Issue 24, pp. 2669 - 2683
Journal Article
Nature communications, ISSN 2041-1723, 2015, Volume 6, Issue 1, pp. 8695 - 8695
Journal Article
Journal Article