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Journal Article
Cancer Research, ISSN 0008-5472, 08/2011, Volume 71, Issue 15, pp. 5346 - 5356
Journal Article
Journal of Clinical Oncology, ISSN 0732-183X, 12/2017, Volume 35, Issue 35, pp. 3916 - 3923
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Journal Article
Molecular Cancer Therapeutics, ISSN 1535-7163, 02/2015, Volume 14, Issue 2, pp. 513 - 522
Journal Article
PLoS ONE, ISSN 1932-6203, 09/2012, Volume 7, Issue 9, p. e42541
Background: Endothelial progenitor cells (EPCs) may be recruited from bone marrow to sustain tumor vascularisation and promote the metastatic switch.... 
CAPACITATIVE CALCIUM-ENTRY | SIGNALING PATHWAYS | STEM-CELLS | STROMAL INTERACTION MOLECULE-1 | STIM PROTEINS | MULTIDISCIPLINARY SCIENCES | GROWTH | PROLIFERATION | CHANNELS | TUBE FORMATION | CARCINOMA | Kidney Neoplasms - genetics | Calcium Channels - metabolism | Neoplastic Stem Cells - drug effects | Humans | Middle Aged | Neovascularization, Pathologic | Carcinoma, Renal Cell - genetics | Lanthanum - pharmacology | Male | Kidney Neoplasms - metabolism | Neoplasm Proteins - metabolism | Adenosine Triphosphate - pharmacology | Neoplastic Stem Cells - metabolism | Aged, 80 and over | Neoplastic Stem Cells - pathology | Adult | Female | Indoles - pharmacology | Membrane Proteins - metabolism | Gene Expression Regulation, Neoplastic - drug effects | Neoplasm Proteins - genetics | TRPC Cation Channels - genetics | Calcium Channels - genetics | Carcinoma, Renal Cell - blood supply | Stromal Interaction Molecule 1 | Boron Compounds - pharmacology | Endothelial Cells - metabolism | Membrane Proteins - genetics | Cadmium - pharmacology | ORAI1 Protein | Carcinoma, Renal Cell - metabolism | Signal Transduction - drug effects | TRPC Cation Channels - metabolism | Aged | Primary Cell Culture | Endothelial Cells - pathology | Kidney Neoplasms - blood supply | Endothelial Cells - drug effects | Biotechnology | Low concentrations | Genetic suppression | Laboratories | STIM1 protein | Gadolinium | Oncology | Biology | mRNA | Kinases | Epidemiology | Calcium influx | Cell adhesion & migration | Metastases | Proteins | Angiogenesis | Mitochondria | Bone marrow | Calcium buffering | Physiology | Inhibition | Pharmaceutical sciences | Recombinant | Calcium channels | Stability | Calcium (intracellular) | Tumor cells | Calcium imaging | Pharmacology | Gene expression | Orai1 protein | Patients | Cyclopiazonic acid | Polymerase chain reaction | Gene silencing | Molecular modelling | Overexpression | Stem cells | Cells (biology) | Cations | Intracellular | Endoplasmic reticulum | Calcium (reticular) | Clear cell-type renal cell carcinoma | Kidney transplantation | Cancer
Journal Article
Cancer Research, ISSN 0008-5472, 02/2010, Volume 70, Issue 3, pp. 1053 - 1062
Sunitinib is a broad-spectrum small-molecule inhibitor of receptor tyrosine kinases (RTK) that serves as the present standard of care for first-line therapy of... 
SIGNALING PATHWAYS | ANGIOGENESIS | ONCOLOGY | IN-VIVO | TYROSINE KINASE INHIBITOR | FACTOR RECEPTORS | ALPHA | ANTITUMOR-ACTIVITY | CANCER | EXPRESSION | SU11248 | Humans | Endothelium, Vascular - drug effects | Receptor, Platelet-Derived Growth Factor beta - genetics | Neovascularization, Pathologic - pathology | Vascular Endothelial Growth Factor Receptor-2 - genetics | Dose-Response Relationship, Drug | RNA Interference | Female | Indoles - pharmacology | Neovascularization, Pathologic - prevention & control | Carcinoma, Renal Cell - drug therapy | Carcinoma, Renal Cell - blood supply | Receptor, Platelet-Derived Growth Factor beta - metabolism | Cell Line | Carcinoma, Renal Cell - pathology | Endothelial Cells - metabolism | Angiogenesis Inhibitors - pharmacology | Vascular Endothelial Growth Factor Receptor-2 - metabolism | Blotting, Western | Xenograft Model Antitumor Assays | Cell Movement - drug effects | Pyrroles - pharmacology | Animals | Tumor Burden - drug effects | Endothelial Cells - cytology | Endothelium, Vascular - metabolism | Mice, Nude | Cell Line, Tumor | Endothelium, Vascular - pathology | Kidney Neoplasms - pathology | Neovascularization, Pathologic - genetics | Cell Proliferation - drug effects | Mice | Mice, Inbred BALB C | Kidney Neoplasms - drug therapy | Kidney Neoplasms - blood supply | Endothelial Cells - drug effects
Journal Article
European Journal of Cancer, ISSN 0959-8049, 2017, Volume 77, pp. 155 - 164
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