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STEM CELLS, ISSN 1066-5099, 10/2011, Volume 29, Issue 10, pp. 1485 - 1495
Cancer stem cells (CSCs) that display tumor‐initiating properties have recently been identified. CD133, a surface glycoprotein linked to organ‐specific stem... 
Cancer stem cell | Uterine carcinosarcoma | Müllerian duct | Malignant mixed Müllerian tumor | CD133 | PROGNOSIS | UTERUS | STAGE | SELF-RENEWAL | PLURIPOTENCY | Malignant mixed Mullerian tumor | CELL & TISSUE ENGINEERING | CELL BIOLOGY | SIDE-POPULATION | ONCOLOGY | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | MYC | Mullerian duct | STEM/PROGENITOR CELLS | NANOG | HEMATOLOGY | EXPRESSION | Immunohistochemistry | Neoplastic Stem Cells - cytology | Wnt4 Protein - metabolism | PAX2 Transcription Factor - metabolism | Paclitaxel - pharmacology | Neoplastic Stem Cells - drug effects | Uterine Neoplasms - pathology | Vimentin - metabolism | Homeodomain Proteins - metabolism | Humans | Gene Expression Regulation, Neoplastic | Glycoproteins - metabolism | Drug Resistance, Neoplasm | Antigens, CD - metabolism | Epithelial Cell Adhesion Molecule | SOXB1 Transcription Factors - metabolism | Estrogen Receptor beta - metabolism | Mesenchymal Stromal Cells - cytology | Flow Cytometry | Peptides - metabolism | Antigens, Neoplasm - metabolism | Neoplastic Stem Cells - pathology | Female | Cell Differentiation | Mixed Tumor, Mullerian - metabolism | Carcinosarcoma - pathology | Uterine Neoplasms - genetics | Uterine Neoplasms - metabolism | Carcinosarcoma - metabolism | Nanog Homeobox Protein | Mesenchymal Stromal Cells - metabolism | Cell Separation - methods | Cisplatin - pharmacology | AC133 Antigen | Cell Adhesion Molecules - metabolism | Cell Transformation, Neoplastic - metabolism | Mixed Tumor, Mullerian - pathology | Animals | Mice, Nude | Octamer Transcription Factor-3 - metabolism | Cell Line, Tumor | Carcinosarcoma - genetics | Mice | Mice, Inbred BALB C | Cell Transformation, Neoplastic - pathology
Journal Article
Nature Medicine, ISSN 1078-8956, 09/2017, Volume 23, Issue 9, pp. 1046 - 1054
It is generally assumed that recurrent mutations within a given cancer driver gene elicit similar drug responses. Cancer genome studies have identified... 
MEDICINE, RESEARCH & EXPERIMENTAL | BROMODOMAIN INHIBITORS | ENDOMETRIAL CARCINOMAS | UBIQUITIN LIGASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | RECEPTOR | TUMORS | CELL BIOLOGY | BREAST-CANCER | GENOMIC LANDSCAPE | PROSTATE-CANCER | RESISTANCE | MUTATIONS | Immunohistochemistry | Neoplasms, Cystic, Mucinous, and Serous - metabolism | Neoplasm Transplantation | Prostatic Neoplasms - metabolism | Immunoprecipitation | Apoptosis - drug effects | Heterocyclic Compounds, 3-Ring - pharmacology | Epigenesis, Genetic | Humans | Endometrial Neoplasms - metabolism | Drug Resistance, Neoplasm | Immunoblotting | Male | Molecular Targeted Therapy | Carcinoma, Endometrioid - metabolism | Neoplasms, Cystic, Mucinous, and Serous - genetics | Ubiquitination | Prostatic Neoplasms - genetics | Endometrial Neoplasms - genetics | Mass Spectrometry | Protein-Serine-Threonine Kinases - antagonists & inhibitors | Chromatography, Liquid | Cullin Proteins - metabolism | Female | Nuclear Proteins - genetics | Protein-Serine-Threonine Kinases - metabolism | RNA-Binding Proteins - antagonists & inhibitors | Acetanilides - pharmacology | Carcinoma, Endometrioid - genetics | Carcinosarcoma - metabolism | Adenocarcinoma, Clear Cell - metabolism | Repressor Proteins - genetics | Nuclear Proteins - metabolism | Transcription Factors - antagonists & inhibitors | Reverse Transcriptase Polymerase Chain Reaction | Azepines - pharmacology | Transcription Factors - metabolism | Triazoles - pharmacology | Animals | Mice, Nude | Nuclear Proteins - antagonists & inhibitors | Cell Line, Tumor | Carcinosarcoma - genetics | Cell Proliferation - drug effects | Mutation | Adenocarcinoma, Clear Cell - genetics | RNA-Binding Proteins - metabolism | Care and treatment | Gene mutations | Proteolysis | Physiological aspects | Genetic aspects | Research | Cancer | Ubiquitin | Biodegradation | Inhibitor drugs | Endometrial cancer | Genomes | Pharmacology | Substrates | Mutants | Degradation | Proteins | Sensitivity | Missense mutation | Inhibitors | Bet protein | Gene mapping | Prostate cancer | Prostate | Endometrium | Ubiquitin-protein ligase
Journal Article
Gynecologic Oncology, ISSN 0090-8258, 2014, Volume 133, Issue 1, pp. 43 - 47
Abstract Objective Links between obesity, with its attendant estrogen abnormalities, and the endometrial carcinoma (EC) DNA Mismatch Repair Protein (MMR)... 
Hematology, Oncology and Palliative Medicine | Obstetrics and Gynecology | Body mass index | MMR | Endometrial cancer | DNA mismatch repair protein | BMI | LYNCH-SYNDROME | NONPOLYPOSIS COLORECTAL-CANCER | MICROSATELLITE INSTABILITY | RISK | MLH1 | OBSTETRICS & GYNECOLOGY | WOMEN | OBESITY | ONCOLOGY | PATHOLOGICAL FEATURES | AGE | IMMUNOHISTOCHEMISTRY | Multivariate Analysis | MutL Protein Homolog 1 | Overweight - complications | Carcinosarcoma - complications | Humans | Middle Aged | Endometrial Neoplasms - metabolism | Carcinoma, Endometrioid - complications | MutS Homolog 2 Protein - metabolism | Carcinoma, Endometrioid - metabolism | DNA-Binding Proteins - metabolism | Overweight - metabolism | Carcinoma - complications | DNA Mismatch Repair | DNA Repair Enzymes - metabolism | Endometrial Neoplasms - complications | Aged, 80 and over | Biomarkers, Tumor - metabolism | Adult | Female | Mismatch Repair Endonuclease PMS2 | Retrospective Studies | Body Mass Index | Carcinosarcoma - metabolism | Adenocarcinoma, Clear Cell - metabolism | Obesity - complications | Adenosine Triphosphatases - metabolism | Nuclear Proteins - metabolism | Obesity - metabolism | Adenocarcinoma, Clear Cell - complications | Aged | Carcinoma - metabolism | Adaptor Proteins, Signal Transducing - metabolism | Cancer | Endometrial Cancer | DNA Mismatch Repair Protein
Journal Article
European Journal of Cancer, ISSN 0959-8049, 2017, Volume 76, pp. 52 - 59
Abstract Drug resistance to conventional anticancer therapies is almost inevitable in patients with advanced ovarian cancer (AOC), limiting their available... 
Hematology, Oncology and Palliative Medicine | RMH prognostic score | Precision medicine | Antiangiogenics | Phase I trials | Poly(ADP-ribose) Polymerase (PARP) inhibitors | Ovarian cancer | TARGETED THERAPY | BEVACIZUMAB | FALLOPIAN-TUBE | BREAST | CHEMOTHERAPY | PIK3CA | ONCOLOGY | PERSPECTIVES | MUTATIONS | RECURRENT EPITHELIAL OVARIAN | PRIMARY PERITONEAL | Neoplasms, Cystic, Mucinous, and Serous - metabolism | Prognosis | Humans | Middle Aged | Ovarian Neoplasms - pathology | England | Antineoplastic Agents - therapeutic use | Granulosa Cell Tumor - pathology | Carcinoma, Endometrioid - metabolism | Neoplasms, Cystic, Mucinous, and Serous - genetics | Ovarian Neoplasms - genetics | Neoplasm Metastasis | Genes, BRCA2 | Adult | Female | Neoplasms, Cystic, Mucinous, and Serous - drug therapy | Granulosa Cell Tumor - drug therapy | Leukocyte Count | Membrane Proteins - metabolism | Ovarian Neoplasms - metabolism | Retrospective Studies | Genes, BRCA1 | CA-125 Antigen - metabolism | Ovarian Neoplasms - drug therapy | Carcinosarcoma - pathology | Severity of Illness Index | Hereditary Breast and Ovarian Cancer Syndrome - genetics | Carcinoma, Endometrioid - genetics | Carcinosarcoma - metabolism | Neoplasms, Cystic, Mucinous, and Serous - pathology | Adenocarcinoma, Clear Cell - metabolism | Granulosa Cell Tumor - genetics | Hemoglobins - metabolism | Survival Rate | Treatment Outcome | Carcinoma, Endometrioid - drug therapy | Drug Discovery | Granulosa Cell Tumor - metabolism | Adenocarcinoma, Clear Cell - pathology | Aged | Carcinosarcoma - drug therapy | Carcinosarcoma - genetics | Adenocarcinoma, Clear Cell - genetics | Carcinoma, Endometrioid - pathology | Clinical Trials, Phase I as Topic | Adenocarcinoma, Clear Cell - drug therapy | Serum Albumin - metabolism | Chemotherapy | Cancer patients | Clinical trials | Hemoglobin | Metastasis | Drug resistance | Sugars | Cancer | Monosaccharides
Journal Article
Journal of Pathology, ISSN 0022-3417, 01/2011, Volume 223, Issue 1, pp. 72 - 80
Endometrial carcinosarcomas (ECSs) undergo a true epithelial‐mesenchymal transition (EMT). The molecular determinants of the EMT in vivo are unclear, although... 
miRNA | endometrial carcinosarcoma | differentiation | epithelial‐mesenchymal transition | E‐cadherin | epithelial-mesenchymal transition | E-cadherin | CANCER-CELLS | STEM-CELLS | PHENOTYPE | PATHOLOGY | MIR-200 FAMILY | REPRESSORS ZEB1 | EMT | BHLH FACTORS | ONCOLOGY | DISEASE | EXPRESSION | PROGRESSION | Carcinosarcoma - pathology | Carcinosarcoma - metabolism | Cadherins - metabolism | Oligonucleotide Array Sequence Analysis - methods | Homeodomain Proteins - metabolism | Humans | Endometrial Neoplasms - metabolism | Gene Expression Regulation, Neoplastic | Gene Expression Profiling - methods | Epithelial-Mesenchymal Transition - genetics | Neoplasm Proteins - metabolism | Zinc Finger E-box Binding Homeobox 2 | Transcription Factors - metabolism | Endometrial Neoplasms - genetics | Reverse Transcriptase Polymerase Chain Reaction - methods | RNA, Neoplasm - genetics | Endometrial Neoplasms - pathology | Female | Carcinosarcoma - genetics | MicroRNAs - genetics | Repressor Proteins - metabolism | Zinc Finger E-box-Binding Homeobox 1 | Immunohistochemistry | Vimentin | Transcription factors | Mesenchyme | Transcription | Data processing | Transforming growth factor- beta 1 | Cadherin | Polymerase chain reaction | caveolin-1 | Molecular modelling | N-Cadherin | ECS | Repressors | E-Cadherin | Osteonectin | Endometrium
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