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Journal of Clinical Investigation, ISSN 0021-9738, 09/2009, Volume 119, Issue 9, pp. 2772 - 2786
Journal Article
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 6/2016, Volume 113, Issue 24, pp. 6701 - 6706
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 01/2013, Volume 123, Issue 1, pp. 19 - 26
Genetic mutations account for a significant percentage of cardiomyopathies, which are a leading cause of congestive heart failure. In hypertrophic... 
MEDICINE, RESEARCH & EXPERIMENTAL | SARCOMERE PROTEIN GENES | COMPLEX | HYPERTROPHIC CARDIOMYOPATHY | POLYMORPHISMS | TITIN | DREIFUSS MUSCULAR-DYSTROPHY | LAMIN-A/C DEFICIENCY | RISK | CONGESTIVE-HEART-FAILURE | MECHANOTRANSDUCTION | Cardiomyopathy, Dilated - pathology | Humans | Heart Failure - physiopathology | Myosin Heavy Chains - genetics | Myosin Heavy Chains - metabolism | Arrhythmias, Cardiac - physiopathology | Sarcomeres - pathology | Arrhythmias, Cardiac - pathology | Mitochondria - genetics | Sarcomeres - genetics | Myocardium - metabolism | Heart Failure - etiology | Arrhythmias, Cardiac - genetics | Cardiac Myosins - metabolism | Arrhythmias, Cardiac - metabolism | Calcium-Binding Proteins - metabolism | Cardiomyopathy, Dilated - genetics | Cytoskeleton - pathology | Cardiomyopathy, Dilated - complications | Sarcomeres - metabolism | Cardiac Myosins - genetics | Cytoskeleton - genetics | Heart Failure - genetics | Myocardium - pathology | Arrhythmias, Cardiac - etiology | Heart Failure - metabolism | Mitochondria - metabolism | Heart Failure - pathology | Mitochondria - pathology | Cardiomyopathy, Dilated - metabolism | Carrier Proteins - genetics | Animals | Carrier Proteins - metabolism | Cardiomyopathy, Dilated - physiopathology | Cytoskeleton - metabolism | Mutation | Calcium-Binding Proteins - genetics | Gene mutations | Cardiomyopathy | Genetic aspects | Congestive heart failure | Diagnosis | Heart diseases | Genetic screening | Methods | Review Series
Journal Article
Science, ISSN 0036-8075, 4/2007, Volume 316, Issue 5824, pp. 575 - 579
The heart responds to diverse forms of stress by hypertrophic growth accompanied by fibrosis and eventual diminution of contractility, which results from... 
Heart | Untranslated regions | Up regulation | Messenger RNA | MicroRNA | Transgenic animals | Fibrosis | Mice | Gene expression regulation | Research Articles | Gene expression | HYPERTROPHY | MULTIDISCIPLINARY SCIENCES | CARDIOMYOPATHY | PHENOTYPE | PROLIFERATION | DIFFERENTIATION | THYROID-HORMONE RECEPTOR | HUMAN HEART | MYOSIN HEAVY-CHAIN | HYPOTHYROIDISM | TRANSGENIC MICE | Up-Regulation | Oligonucleotide Array Sequence Analysis | Hypothyroidism - physiopathology | Myosin Heavy Chains - genetics | Cardiomegaly - pathology | Triiodothyronine - metabolism | Gene Deletion | Myocardium - metabolism | Mediator Complex | Cardiac Myosins - metabolism | Heart - physiopathology | Myocardial Contraction | Signal Transduction | Heart Diseases - physiopathology | Introns | Cardiac Myosins - genetics | Gene Expression Regulation | Cardiomegaly - physiopathology | Rats | Mice, Transgenic | Myocardium - pathology | Stress, Physiological - physiopathology | Transcription Factors - genetics | Transcription Factors - metabolism | Myocytes, Cardiac - pathology | Animals | Ventricular Myosins - metabolism | MicroRNAs - genetics | MicroRNAs - physiology | Heart Diseases - genetics | Heart Diseases - pathology | Ventricular Myosins - genetics | Causes of | Research | Muscle contraction | Heart muscle | Proteins | Ribonucleic acid | Hormones | Ribonucleic acid--RNA | Stress
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 7/2013, Volume 110, Issue 31, pp. 12607 - 12612
Journal Article
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 7/2013, Volume 110, Issue 31, pp. 12655 - 12660
The positive transcription elongation factor b (P-TEFb) is involved in physiological and pathological events including inflammation, cancer, AIDS, and cardiac... 
T lymphocytes | Pathology | RNA | HEK293 cells | Genes | Cell lines | Antibodies | Small nuclear RNA | Gene expression regulation | HIV 1 | 7SK SNRNA | TRANSCRIPTION FACTOR BCL11B | ELONGATION | MULTIDISCIPLINARY SCIENCES | GENE-EXPRESSION | POLYMERASE | HIV-1 TAT | MICROGLIAL CELLS | T-CELL LINEAGE | BROMODOMAIN PROTEIN BRD4 | RNA-Binding Proteins - genetics | Humans | Myosin Heavy Chains - genetics | Cardiomegaly - pathology | Positive Transcriptional Elongation Factor B - metabolism | Positive Transcriptional Elongation Factor B - genetics | Myosin Heavy Chains - metabolism | RNA, Small Nuclear - genetics | Tumor Suppressor Proteins - genetics | HEK293 Cells | Cardiac Myosins - metabolism | Repressor Proteins - metabolism | Promoter Regions, Genetic | Tumor Suppressor Proteins - metabolism | Protein Structure, Secondary | Cyclin-Dependent Kinase 9 - genetics | Cardiac Myosins - genetics | Repressor Proteins - genetics | Transcription Factors - genetics | Transcription Factors - metabolism | Animals | Cyclin-Dependent Kinase 9 - metabolism | Mice | RNA, Small Nuclear - metabolism | Cardiomegaly - genetics | RNA-Binding Proteins - metabolism | Cardiomegaly - metabolism | Physiological aspects | Transcription factors | Genetic aspects | Heart enlargement | Health aspects | Myosin | Life Sciences | Neurons and Cognition | Biological Sciences
Journal Article
Nature, ISSN 0028-0836, 2014, Volume 514, Issue 7520, pp. 102 - 106
The role of long noncoding RNA(lncRNA) in adult hearts is unknown; also unclear is how lncRNA modulates nucleosome remodelling. An estimated 70% of mouse genes... 
MORPHOGENESIS | DOMAIN | CHROMATIN | GENE | RECOGNITION | STRUCTURAL BASIS | MULTIDISCIPLINARY SCIENCES | CARDIAC-HYPERTROPHY | IN-VIVO | TRANSCRIPTION | GROWTH | Chromatin - metabolism | Transcription Factors - chemistry | Humans | Myosin Heavy Chains - genetics | Cardiomegaly - pathology | Cardiomyopathies - prevention & control | Heart Failure - prevention & control | Cardiomyopathies - genetics | Myocardium - metabolism | Nuclear Proteins - genetics | DNA Helicases - genetics | Protein Structure, Tertiary | DNA Helicases - chemistry | Cardiac Myosins - genetics | Chromatin Assembly and Disassembly | Cardiomyopathies - pathology | Heart Failure - genetics | Histone Deacetylases - metabolism | Myocardium - pathology | Nuclear Proteins - metabolism | Transcription Factors - antagonists & inhibitors | RNA, Long Noncoding - genetics | Heart Failure - pathology | Transcription Factors - genetics | Nuclear Proteins - chemistry | Organ Specificity | Transcription Factors - metabolism | DNA Helicases - metabolism | Feedback, Physiological | Poly(ADP-ribose) Polymerases - metabolism | Animals | Cardiomegaly - prevention & control | Nuclear Proteins - antagonists & inhibitors | Protein Binding | Mice | DNA Helicases - antagonists & inhibitors | Poly (ADP-Ribose) Polymerase-1 | Cardiomegaly - genetics | RNA, Long Noncoding - antagonists & inhibitors | Chromatin - genetics | RNA, Long Noncoding - metabolism | Heart | Physiological aspects | Genetic aspects | RNA | Hypertrophy | Pathology | Cardiomyocytes | RNA polymerase | Ribonucleic acid--RNA | Cardiomyopathy | Rodents
Journal Article