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La Revue du praticien, ISSN 0035-2640, 06/2016, Volume 66, Issue 6, p. 611
The pathophysiology of cardiorenal syndromes (SCR) is becoming better understood. The traditional view was that the left ventricular systolic dysfunction leads... 
Humans | Cardio-Renal Syndrome - physiopathology
Journal Article
Journal Article
Circulation. Heart Failure, ISSN 1941-3289, 04/2016, Volume 9, Issue 4, p. e002760
BACKGROUND: The combination of cardiac and renal disease driven by metabolic risk factors, referred to as cardiorenal metabolic syndrome (CRMS), is... 
mortality | heart failure | animal model | endothelial cell | cardiorenal syndrome | CARDIAC & CARDIOVASCULAR SYSTEMS | METAANALYSIS | MYOCARDIAL-INFARCTION | RENAL-FUNCTION | RISK | MODEL | NEPHROPATHY | DAMAGE | DISEASE | COHORT | DYSFUNCTION | Microvessels - physiopathology | Vascular Remodeling | Cell Proliferation | Kidney - blood supply | Ventricular Function, Left | Coronary Vessels - physiopathology | Cardio-Renal Syndrome - metabolism | Heart Failure - physiopathology | Microvessels - metabolism | Microvessels - pathology | Rats, Inbred WKY | Male | Metabolic Syndrome - metabolism | Coronary Vessels - metabolism | Ventricular Remodeling | Time Factors | Metabolic Syndrome - physiopathology | Heart Failure - diagnosis | Glomerulonephritis - etiology | Glomerulonephritis - physiopathology | Heart Failure - etiology | Cardio-Renal Syndrome - diagnosis | Cardio-Renal Syndrome - physiopathology | Disease Models, Animal | Proteinuria - etiology | Coronary Vessels - pathology | Endothelial Cells - metabolism | Hypertrophy, Left Ventricular - etiology | Cardio-Renal Syndrome - etiology | Disease Progression | Metabolic Syndrome - complications | Proteinuria - physiopathology | Rats, Zucker | Stroke Volume | Animals | Metabolic Syndrome - diagnosis | Fibrosis | Hypertrophy, Left Ventricular - physiopathology | Endothelial Cells - pathology
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 02/2018, Volume 314, Issue 2, pp. H255 - H267
Heart failure (HF) secondary to myocardial infarction (MI) is linked to kidney complications that comprise cellular, structural, functional, and survival... 
Temporal echocardiography | Ischemia injury | Fibrosis | CELLS | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | MYOCARDIAL-INFARCTION | ischemia injury | RESOLVING RESPONSE | MOUSE | ISCHEMIA | INFLAMMATION | DISEASE | PERIPHERAL VASCULAR DISEASE | fibrosis | MICE | KIDNEY | temporal echocardiography | Spleen - physiopathology | Kidney - pathology | Ventricular Function, Left | Cardio-Renal Syndrome - metabolism | Heart Failure - physiopathology | Male | Myocardial Infarction - diagnostic imaging | Cardio-Renal Syndrome - pathology | Kidney - metabolism | Ventricular Remodeling | Time Factors | Myocardial Infarction - pathology | Inflammation Mediators - metabolism | Ventricular Dysfunction, Left - pathology | Myocardial Infarction - physiopathology | Heart Failure - diagnostic imaging | Spleen - pathology | Cardio-Renal Syndrome - diagnostic imaging | Cardio-Renal Syndrome - physiopathology | Kidney - physiopathology | Disease Models, Animal | Heart - physiopathology | Echocardiography | Mice, Inbred C57BL | Heart Failure - metabolism | Myocardial Infarction - metabolism | Disease Progression | Ventricular Dysfunction, Left - diagnostic imaging | Ventricular Dysfunction, Left - physiopathology | Heart - diagnostic imaging | Ventricular Dysfunction, Left - metabolism | Collagen - metabolism | Animals | Spleen - metabolism | Chronic Disease | Heart failure | Development and progression | Health aspects | Heart ventricle, Left
Journal Article
European Heart Journal, ISSN 0195-668X, 09/2012, Volume 33, Issue 17, pp. 2135 - +
Journal Article
Journal Article