X
Search Filters
Format Format
Subjects Subjects
Subjects Subjects
X
Sort by Item Count (A-Z)
Filter by Count
humans (6315) 6315
male (4606) 4606
cardiomegaly - etiology (3751) 3751
female (3569) 3569
adult (2620) 2620
middle aged (2546) 2546
cardiomegaly - complications (2409) 2409
animals (2225) 2225
electrocardiography (1824) 1824
hypertension - complications (1806) 1806
cardiomegaly - physiopathology (1572) 1572
aged (1519) 1519
echocardiography (1251) 1251
cardiomegaly - pathology (1210) 1210
rats (1203) 1203
cardiac & cardiovascular systems (1180) 1180
cardiomegaly - diagnosis (999) 999
adolescent (959) 959
myocardium - pathology (955) 955
cardiomegaly - diagnostic imaging (948) 948
blood pressure (779) 779
child (771) 771
hypertrophy (704) 704
hypertension - physiopathology (665) 665
heart failure - etiology (623) 623
radiography (579) 579
cardiomegaly - metabolism (573) 573
hemodynamics (572) 572
hypertension (568) 568
heart (566) 566
infant (517) 517
peripheral vascular disease (510) 510
myocardium - metabolism (507) 507
child, preschool (506) 506
cardiomegaly - drug therapy (479) 479
heart - physiopathology (450) 450
cardiac catheterization (448) 448
time factors (439) 439
disease models, animal (436) 436
cardiomegaly (434) 434
hypertension - drug therapy (428) 428
myocardial infarction - complications (422) 422
mice (415) 415
heart ventricles (402) 402
diagnosis, differential (377) 377
heart failure (368) 368
heart failure - complications (363) 363
risk factors (363) 363
organ size (357) 357
infant, newborn (356) 356
cardiomegaly - prevention & control (352) 352
prognosis (351) 351
cardiac hypertrophy (329) 329
cardiovascular system (321) 321
heart ventricles - physiopathology (320) 320
myocardial contraction (312) 312
medicine, general & internal (307) 307
heart ventricles - pathology (306) 306
age factors (304) 304
blood pressure - drug effects (303) 303
follow-up studies (302) 302
pregnancy (300) 300
chronic disease (292) 292
coronary disease - complications (292) 292
arrhythmias, cardiac - etiology (283) 283
heart-failure (278) 278
fibrosis (271) 271
cardiomegaly - genetics (268) 268
heart enlargement (267) 267
physiology (267) 267
aortic valve stenosis - complications (262) 262
rats, sprague-dawley (243) 243
cardiomyopathy (241) 241
dogs (235) 235
heart failure - physiopathology (233) 233
pharmacology & pharmacy (226) 226
heart rate (225) 225
left-ventricular hypertrophy (222) 222
antihypertensive agents - therapeutic use (220) 220
research article (219) 219
heart diseases (215) 215
angiocardiography (214) 214
expression (208) 208
rats, wistar (203) 203
cardiac output (202) 202
rats, inbred strains (202) 202
autopsy (201) 201
rats, inbred shr (199) 199
vectorcardiography (198) 198
physiological aspects (196) 196
body weight (192) 192
cardiac-hypertrophy (190) 190
disease (188) 188
cell biology (184) 184
oxidative stress (182) 182
radiography, thoracic (181) 181
cardiomyopathies - complications (180) 180
heart valve prosthesis (180) 180
coronary circulation (179) 179
mitral valve stenosis - complications (177) 177
more...
Library Location Library Location
Language Language
Language Language
X
Sort by Item Count (A-Z)
Filter by Count
English (6375) 6375
German (369) 369
Russian (346) 346
Italian (317) 317
French (316) 316
Japanese (223) 223
Spanish (170) 170
Portuguese (57) 57
Polish (52) 52
Chinese (41) 41
Czech (30) 30
Romanian (25) 25
Hungarian (12) 12
Slovak (11) 11
Dutch (10) 10
Serbian (7) 7
Croatian (6) 6
Danish (6) 6
Bulgarian (5) 5
Hebrew (5) 5
Norwegian (3) 3
Finnish (2) 2
Ukrainian (2) 2
Lithuanian (1) 1
Macedonian (1) 1
Persian (1) 1
Swedish (1) 1
Turkish (1) 1
more...
Publication Date Publication Date
Click on a bar to filter by decade
Slide to change publication date range


PloS one, ISSN 1932-6203, 03/2016, Volume 11, Issue 3, p. e0150745
... of long-term inhibition of DPP-4 on the micro- and macro-vascular complications of type... 
TRANSITION | HYPERTROPHY | GLP-1 | MYOCARDIAL-INFARCTION | PROTECTION | MULTIDISCIPLINARY SCIENCES | EXENATIDE | INJURY | DPP-4 | NEPHROPATHY | Immunohistochemistry | Albuminuria - complications | Kidney - pathology | Apoptosis - drug effects | Albuminuria - blood | Cardiovascular Diseases - drug therapy | Dipeptidyl-Peptidase IV Inhibitors - therapeutic use | Glycated Hemoglobin A - metabolism | NADPH Oxidases - metabolism | Dipeptidyl Peptidase 4 - blood | Glucagon-Like Peptide 1 - blood | Male | Piperidones - therapeutic use | Dipeptidyl-Peptidase IV Inhibitors - pharmacology | Cardiovascular Diseases - complications | Forkhead Transcription Factors - metabolism | Cardiovascular Diseases - blood | Proto-Oncogene Proteins c-akt - metabolism | Diabetes Mellitus, Type 2 - complications | Ventricular Dysfunction - physiopathology | Disease Models, Animal | Kidney - drug effects | Ventricular Dysfunction - complications | Cardiomegaly - blood | Cardiomegaly - physiopathology | Podocytes - pathology | Pyrimidines - pharmacology | Piperidones - pharmacology | Cardiomegaly - complications | Diabetes Mellitus, Type 2 - blood | Animals | Ventricular Dysfunction - blood | Podocytes - drug effects | Pyrimidines - therapeutic use | Mice | Diabetes Mellitus, Type 2 - drug therapy | Forkhead Box Protein O3 | Type 2 diabetes | Complications and side effects | Models | Research | Cardiovascular diseases | Risk factors | Heart | Nephrology | Syngeneic grafts | Cardiomyopathy | Kinases | Ischemia | Rodents | Animal tissues | Inhibition | Cardiology | Heart diseases | Drug dosages | Urine | Excretion | Echocardiography | Peptidase | Kidneys | Internal medicine | Complications | Diabetes mellitus | Permeability | Medicine | Inhibitors | Nephropathy | Microscopy | Fibrosis | Diabetes | Ventricle | Laboratory animals | Apoptosis
Journal Article
Blood Pressure, ISSN 0803-7051, 2010, Volume 19, Issue 3, pp. 169 - 175
Abstract Objective. It is unclear whether improvement of left atrial (LA) and ventricular (LV) structure results in reduction in new-onset atrial fibrillation... 
left atrial size | hypertension | prognosis | Atrial fibrillation | Hypertension | Prognosis | Left atrial size | POPULATION | RISK | LOSARTAN INTERVENTION | SYSTEMIC HYPERTENSION | CARDIOVASCULAR MORBIDITY | ECHOCARDIOGRAPHIC-ASSESSMENT | TRIAL | STROKE | END-POINT REDUCTION | REGURGITATION | PERIPHERAL VASCULAR DISEASE | Antihypertensive Agents - pharmacology | Cardiovascular Diseases - drug therapy | Humans | Middle Aged | Hypertension - drug therapy | Male | Risk | Cardiovascular Diseases - complications | Electrocardiography | Female | Blood Pressure - drug effects | Heart Atria - physiopathology | Hypertrophy, Left Ventricular - drug therapy | Atenolol - pharmacology | Heart Rate | Atrial Fibrillation - drug therapy | Cardiomegaly - drug therapy | Losartan - pharmacology | Antihypertensive Agents - therapeutic use | Cardiomegaly - complications | Hypertension - physiopathology | Arrhythmias, Cardiac - drug therapy | Atenolol - therapeutic use | Atrial Fibrillation - epidemiology | Heart Ventricles - physiopathology | Hypertrophy, Left Ventricular - complications | Losartan - therapeutic use | Hypertension - complications | Aged | Arrhythmias, Cardiac - complications | Hypertrophy, Left Ventricular - physiopathology | Atrial Fibrillation - prevention & control | Heart Atria/physiopathology | Cardiac/complications/drug therapy | Hypertension/complications/drug therapy/physiopathology | Losartan/pharmacology/therapeutic use | Heart Ventricles/physiopathology | Cardiomegaly/complications/drug therapy | Cardiovascular Diseases/complications/drug therapy | Atenolol/pharmacology/therapeutic use | Kardiologi | Cardiac and Cardiovascular Systems | Blood Pressure/drug effects | Atrial Fibrillation/drug therapy/epidemiology/prevention & control | Antihypertensive Agents/pharmacology/therapeutic use | Left Ventricular/complications/drug therapy/physiopathology | Arrhythmias | Hypertrophy
Journal Article
Journal of molecular and cellular cardiology, ISSN 0022-2828, 2015, Volume 84, pp. 24 - 35
Abstract Unresolved inflammation is a major contributor to the development of heart failure following myocardial infarction (MI). Pro-resolving lipid... 
Cardiovascular | Myocardial infarction | Splenic remodeling | Resolution of inflammation | Metabololipidomics | Resolvin D1 | Neutrophils | SURVIVAL | CHRONIC HEART-FAILURE | PROTECTIVE ACTIONS | CARDIAC & CARDIOVASCULAR SYSTEMS | RESOLUTION | TISSUE | RECEPTOR | LIPID MEDIATORS | CELL BIOLOGY | THERAPY | PATHWAY | DISEASE | Cell Count | Male | Myocardial Infarction - diagnostic imaging | Spleen - drug effects | Extracellular Matrix - genetics | Inflammation - complications | Pulmonary Edema - physiopathology | Inflammation - drug therapy | Pulmonary Edema - complications | Receptors, Formyl Peptide - metabolism | Ultrasonography | Myocardial Infarction - physiopathology | Cell Polarity - drug effects | Spleen - pathology | Neutrophil Infiltration - drug effects | Ventricular Function - drug effects | Docosahexaenoic Acids - therapeutic use | Arachidonate 5-Lipoxygenase - metabolism | Docosahexaenoic Acids - chemistry | Extracellular Matrix - drug effects | Mice, Inbred C57BL | Cardiomegaly - diagnostic imaging | Cardiomegaly - drug therapy | Cardiomegaly - physiopathology | Pulmonary Edema - drug therapy | Cardiomegaly - complications | Collagen - metabolism | Docosahexaenoic Acids - pharmacology | Myocardial Infarction - complications | Macrophages - metabolism | Animals | Myocardial Infarction - drug therapy | Heart Ventricles - physiopathology | Prostaglandin-Endoperoxide Synthases - metabolism | Macrophages - drug effects | Ventricular Remodeling - drug effects | Heart Ventricles - drug effects | Inflammation | Heart attack | metabololipidomics | liposomes | myocardial infarction | splenic remodeling | neutrophils | resolution of inflammation
Journal Article
Journal Article
The FASEB journal, ISSN 1530-6860, 2014, Volume 28, Issue 10, pp. 4408 - 4419
Journal Article
American Journal of Pathology, The, ISSN 0002-9440, 2017, Volume 187, Issue 4, pp. 700 - 712
Our understanding of the pathophysiological basis of chronic thromboembolic pulmonary hypertension (CTEPH) will be accelerated by an animal model that... 
Pathology | RIGHT-VENTRICULAR DYSFUNCTION | BRONCHIAL ARTERIES | ANGIOGENESIS | INFLAMMATION | ARTERIAL-HYPERTENSION | CLASSIFICATION | PATHOLOGY | EXPRESSION | RAT MODEL | EXERCISE | ENDARTERECTOMY | Hyperplasia | Hypertension, Pulmonary - physiopathology | Cardiomegaly - pathology | Ki-67 Antigen - metabolism | Male | Lung - diagnostic imaging | Vascular Endothelial Growth Factor A - metabolism | Hypertension, Pulmonary - blood | Vascular Endothelial Growth Factor A - antagonists & inhibitors | Microspheres | Polystyrenes | Indoles - pharmacology | Heart Function Tests | Physical Conditioning, Animal | Ventricular Dysfunction - physiopathology | Tissue Inhibitor of Metalloproteinase-1 - blood | Pulmonary Embolism - complications | Lung - pathology | Ventricular Dysfunction - complications | Cardiomegaly - blood | Cardiomegaly - physiopathology | Hypoxia - complications | Rats, Sprague-Dawley | Cardiomegaly - complications | Plasminogen Activator Inhibitor 1 - blood | Pulmonary Embolism - blood | Pyrroles - pharmacology | Animals | Ventricular Dysfunction - blood | Hypoxia - pathology | Oxygen Consumption - drug effects | Partial Pressure | P-Selectin - blood | Hypoxia - physiopathology | Cell Proliferation - drug effects | Hemodynamics - drug effects | Chronic Disease | Hypertension, Pulmonary - etiology | Pulmonary Embolism - physiopathology | Regular
Journal Article
Journal Article
Journal Article
Journal of applied physiology (1985), ISSN 1522-1601, 2012, Volume 112, Issue 9, pp. 1564 - 1575
Although the signaling pathways underlying exercise-induced cardiac adaptation have been extensively studied, little is known about the molecular mechanisms... 
Pregnancy | Cardiac hypertrophy | Mitogen activated protein kinase | Protein kinase B | Akt | Glycogen synthase kinase 3β | OVERLOAD | SPORT SCIENCES | ACTIVATION | PHYSIOLOGY | pregnancy | MOUSE | mitogen activated protein kinase | EXERCISE | HEART | HYPERTROPHY | PRESSURE | glycogen synthase kinase 3 beta | GENE-EXPRESSION | MICE | cardiac hypertrophy | RAT VENTRICULAR MYOCYTES | protein kinase B | Adaptation, Physiological | Estradiol - blood | Phosphorylation | TOR Serine-Threonine Kinases - metabolism | Pregnancy Complications, Cardiovascular - diagnostic imaging | Cardiomegaly - pathology | Glycogen Synthase Kinase 3 beta | Pregnancy Complications, Cardiovascular - genetics | Progesterone - blood | Proto-Oncogene Proteins c-akt - genetics | Myocytes, Cardiac - enzymology | MAP Kinase Signaling System - genetics | Time Factors | Ultrasonography | Female | p38 Mitogen-Activated Protein Kinases - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Progesterone - pharmacology | Estradiol - pharmacology | Pregnancy Complications, Cardiovascular - pathology | Ribosomal Protein S6 Kinases, 70-kDa - metabolism | Mice, Inbred C57BL | Cardiomegaly - diagnostic imaging | Cells, Cultured | Rats | Mice, Transgenic | Cardiomegaly - enzymology | Glycogen Synthase Kinase 3 - metabolism | Gestational Age | Myocytes, Cardiac - pathology | Animals | MAP Kinase Signaling System - drug effects | Myocytes, Cardiac - drug effects | Glycogen Synthase Kinase 3 - genetics | Mitogen-Activated Protein Kinase 3 - metabolism | Mitogen-Activated Protein Kinases - genetics | Mice | Enzyme Activation | Cardiomegaly - genetics | Pregnancy Complications, Cardiovascular - enzymology | Mitogen-Activated Protein Kinase 1 - metabolism | Mitogen-Activated Protein Kinases - metabolism | glycogen synthase kinase 3β
Journal Article
Annals of cardiac anaesthesia, ISSN 0971-9784, 10/2015, Volume 18, Issue 4, pp. 609 - 611
Journal Article