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1. Full Text Resolvin D1 activates the inflammation resolving response at splenic and ventricular site following myocardial infarction leading to improved ventricular function
by Kain, Vasundhara and Ingle, Kevin A and Colas, Romain A and Dalli, Jesmond and Prabhu, Sumanth D and Serhan, Charles N and Joshi, Medha and Halade, Ganesh V
Journal of molecular and cellular cardiology, ISSN 0022-2828, 2015, Volume 84, pp. 24 - 35
Cardiovascular | Myocardial infarction | Splenic remodeling | Resolution of inflammation | Metabololipidomics | Resolvin D1 | Neutrophils | Cardiac & Cardiovascular Systems | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Cell Biology | Cell Count | Male | Myocardial Infarction - diagnostic imaging | Spleen - drug effects | Extracellular Matrix - genetics | Inflammation - complications | Pulmonary Edema - physiopathology | Inflammation - drug therapy | Pulmonary Edema - complications | Receptors, Formyl Peptide - metabolism | Ultrasonography | Myocardial Infarction - physiopathology | Cell Polarity - drug effects | Spleen - pathology | Neutrophil Infiltration - drug effects | Ventricular Function - drug effects | Docosahexaenoic Acids - therapeutic use | Arachidonate 5-Lipoxygenase - metabolism | Docosahexaenoic Acids - chemistry | Extracellular Matrix - drug effects | Mice, Inbred C57BL | Cardiomegaly - diagnostic imaging | Cardiomegaly - drug therapy | Cardiomegaly - physiopathology | Pulmonary Edema - drug therapy | Cardiomegaly - complications | Collagen - metabolism | Docosahexaenoic Acids - pharmacology | Myocardial Infarction - complications | Macrophages - metabolism | Animals | Myocardial Infarction - drug therapy | Heart Ventricles - physiopathology | Prostaglandin-Endoperoxide Synthases - metabolism | Macrophages - drug effects | Ventricular Remodeling - drug effects | Heart Ventricles - drug effects | Inflammation | Heart attack | Index Medicus | metabololipidomics | liposomes | myocardial infarction | splenic remodeling | neutrophils | resolution of inflammation
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2. Full Text Combination of angiotensin-(1-7) with perindopril is better than single therapy in ameliorating diabetic cardiomyopathy
by Hao, Panpan and Yang, Jianmin and Liu, Yanping and Zhang, Mingxiang and Zhang, Kai and Gao, Fei and Chen, Yuguo and Zhang, Cheng and Zhang, Yun
Scientific reports, ISSN 2045-2322, 2015, Volume 5, Issue 1, pp. 8794 - 8794
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Receptors, Angiotensin - metabolism | Diabetic Cardiomyopathies - metabolism | Diabetic Cardiomyopathies - drug therapy | Apoptosis - drug effects | Transforming Growth Factor beta1 - metabolism | Cardiomegaly - etiology | Cardiomegaly - pathology | Male | Peptide Fragments - pharmacology | Perindopril - administration & dosage | Perindopril - pharmacology | Heart Ventricles - ultrastructure | Peptidyl-Dipeptidase A - metabolism | Angiotensin I - pharmacology | Collagen - biosynthesis | p38 Mitogen-Activated Protein Kinases - metabolism | Phosphorylation - drug effects | Drug Therapy, Combination | Heart Ventricles - pathology | Disease Models, Animal | Fibroblasts - metabolism | Echocardiography | Peptide Fragments - administration & dosage | Angiotensin I - administration & dosage | Cardiomegaly - drug therapy | Rats | Diabetic Cardiomyopathies - diagnosis | Ventricular Dysfunction, Left - prevention & control | Blood Glucose - drug effects | Animals | Ventricular Dysfunction, Left - drug therapy | Mitogen-Activated Protein Kinase 3 - metabolism | Cell Differentiation - drug effects | Fibroblasts - drug effects | Cell Communication - drug effects | Fibrosis | Cell Proliferation - drug effects | Fibroblasts - cytology | Hemodynamics - drug effects | Oxidative Stress - drug effects | Heart Ventricles - drug effects | Mitogen-Activated Protein Kinase 1 - metabolism | Heart | Enzymes | Cardiomyopathy | Diabetes mellitus | Rodents | Angiotensin | Diabetes | Ventricle | Angiotensin II | Hypertrophy | Apoptosis | Index Medicus
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3. Full Text In-treatment reduced left atrial diameter during antihypertensive treatment is associated with reduced new-onset atrial fibrillation in hypertensive patients with left ventricular hypertrophy: The LIFE Study
by Wachtell, Kristian and Gerdts, Eva and Aurigemma, Gerard P and Boman, Kurt and Dahlöf, Björn and Nieminen, Markku S and Olsen, Michael Hecht and Okin, Peter M and Palmieri, Vittorio and Rokkedal, Jens E and Devereux, Richard B and Sahlgrenska akademin and Institutionen för medicin, avdelningen för akut och kardiovaskulär medicin and Institute of Medicine, Department of Emergeny and Cardiovascular Medicine and Göteborgs universitet and Gothenburg University and Sahlgrenska Academy
Blood pressure, ISSN 0803-7051, 2010, Volume 19, Issue 3, pp. 169 - 175
left atrial size | hypertension | prognosis | Atrial fibrillation | Hypertension | Prognosis | Left atrial size | Peripheral Vascular Disease | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Antihypertensive Agents - pharmacology | Cardiovascular Diseases - drug therapy | Humans | Middle Aged | Hypertension - drug therapy | Male | Risk | Cardiovascular Diseases - complications | Electrocardiography | Female | Blood Pressure - drug effects | Heart Atria - physiopathology | Hypertrophy, Left Ventricular - drug therapy | Atenolol - pharmacology | Heart Rate | Atrial Fibrillation - drug therapy | Cardiomegaly - drug therapy | Losartan - pharmacology | Antihypertensive Agents - therapeutic use | Cardiomegaly - complications | Hypertension - physiopathology | Arrhythmias, Cardiac - drug therapy | Atenolol - therapeutic use | Atrial Fibrillation - epidemiology | Heart Ventricles - physiopathology | Hypertrophy, Left Ventricular - complications | Losartan - therapeutic use | Hypertension - complications | Aged | Arrhythmias, Cardiac - complications | Hypertrophy, Left Ventricular - physiopathology | Atrial Fibrillation - prevention & control | Index Medicus | Heart Atria/physiopathology | Cardiac/complications/drug therapy | Hypertension/complications/drug therapy/physiopathology | Losartan/pharmacology/therapeutic use | Heart Ventricles/physiopathology | Cardiomegaly/complications/drug therapy | Cardiovascular Diseases/complications/drug therapy | Atenolol/pharmacology/therapeutic use | Kardiologi | Cardiac and Cardiovascular Systems | Blood Pressure/drug effects | Atrial Fibrillation/drug therapy/epidemiology/prevention & control | Antihypertensive Agents/pharmacology/therapeutic use | Left Ventricular/complications/drug therapy/physiopathology | Arrhythmias | Hypertrophy
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4. Full Text Anti-androgenic therapy with finasteride improves cardiac function, attenuates remodeling and reverts pathologic gene-expression after myocardial infarction in mice
by Froese, Natali and Wang, Honghui and Zwadlo, Carolin and Wang, Yong and Grund, Andrea and Gigina, Anna and Hofmann, Melanie and Kilian, Katja and Scharf, Gesine and Korf-Klingebiel, Mortimer and Melchert, Anna and Signorini, Maria Elena Ricci and Halloin, Caroline and Zweigerdt, Robert and Martin, Ulrich and Gruh, Ina and Wollert, Kai C and Geffers, Robert and Bauersachs, Johann and Heineke, Joerg
Journal of molecular and cellular cardiology, ISSN 0022-2828, 09/2018, Volume 122, pp. 114 - 124
Cardiac hypertrophy | Testosterone | Signaling | Myocardial remodeling | Cardiac & Cardiovascular Systems | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Cell Biology | Human Umbilical Vein Endothelial Cells | Cell Line | Neovascularization, Physiologic - drug effects | Gene Expression - drug effects | Humans | Ventricular Function, Left - drug effects | Cardiomegaly - drug therapy | Rats | Male | Myocardium - pathology | Rats, Sprague-Dawley | Animals | Myocardial Infarction - drug therapy | Analysis of Variance | Muscle Contraction - drug effects | Fibroblasts - drug effects | Fibrosis | Androgen Antagonists - therapeutic use | Finasteride - therapeutic use | Mice | Induced Pluripotent Stem Cells | Ventricular Remodeling - drug effects | Dihydrotestosterone - metabolism | Endothelial Cells - drug effects | Heart | Finasteride | Analysis | Genes | Health aspects | Heart attack | Index Medicus
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5. JNK pathway signaling: A novel and smarter therapeutic targets for various biological diseases
by Kumar, Ankit and Singh, Umesh K and Kini, Suvarna G and Garg, Vikas and Agrawal, Saurabh and Tomar, Praveen K and Pathak, Prateek and Chaudhary, Anurag and Gupta, Pratibha and Malik, Amrita
Future medicinal chemistry, ISSN 1756-8919, 10/2015, Volume 7, Issue 15, pp. 2065 - 2086
Pharmacology & Pharmacy | Life Sciences & Biomedicine | Chemistry, Medicinal | Science & Technology | Neoplasms - metabolism | Small Molecule Libraries - pharmacology | Asthma - metabolism | Humans | JNK Mitogen-Activated Protein Kinases - metabolism | Molecular Targeted Therapy | Neurodegenerative Diseases - drug therapy | Protein Kinase Inhibitors - chemistry | MAP Kinase Signaling System - genetics | JNK Mitogen-Activated Protein Kinases - genetics | Autoimmune Diseases - metabolism | Autoimmune Diseases - drug therapy | JNK Mitogen-Activated Protein Kinases - antagonists & inhibitors | Cardiomegaly - drug therapy | Diabetes Mellitus - drug therapy | Small Molecule Libraries - therapeutic use | Diabetes Mellitus - metabolism | Neurodegenerative Diseases - metabolism | Asthma - drug therapy | Neoplasms - drug therapy | Small Molecule Libraries - chemistry | Animals | MAP Kinase Signaling System - drug effects | Protein Kinase Inhibitors - therapeutic use | Protein Kinase Inhibitors - pharmacology | Cardiomegaly - metabolism
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6. Full Text MicroRNAs in heart failure: from biomarker to target for therapy
by Vegter, Eline L and van der Meer, Peter and de Windt, Leon J and Pinto, Yigal M and Voors, Aiaan A
European journal of heart failure, ISSN 1388-9842, 2016, Volume 18, Issue 5, pp. 457 - 468
Heart failure | Antimir | MicroRNAs | Circulation | Cardiac & Cardiovascular Systems | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Biomarkers - metabolism | Acute Disease | Prognosis | Humans | Cardiomegaly - drug therapy | Heart Failure - physiopathology | Cardiomegaly - physiopathology | MicroRNAs - metabolism | Myocardium - pathology | Heart Failure - metabolism | Molecular Targeted Therapy | Heart Failure - drug therapy | Stroke Volume | Fibrosis | Chronic Disease | Cardiomegaly - metabolism | Index Medicus
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7. Histone deacetylase: A potential therapeutic target for fibrotic disorders
by Pang, Maoyin and Zhuang, Shougang
The Journal of pharmacology and experimental therapeutics, ISSN 0022-3565, 11/2010, Volume 335, Issue 2, pp. 266 - 272
Life Sciences & Biomedicine | Pharmacology & Pharmacy | Science & Technology | Primary Myelofibrosis - drug therapy | Cystic Fibrosis - enzymology | Kidney Diseases - pathology | Humans | Cardiomegaly - drug therapy | Cystic Fibrosis - pathology | Scleroderma, Systemic - pathology | Kidney Diseases - drug therapy | Cardiomegaly - pathology | Histone Deacetylases - metabolism | Cardiomegaly - enzymology | Kidney Diseases - enzymology | Primary Myelofibrosis - pathology | Primary Myelofibrosis - enzymology | Scleroderma, Systemic - enzymology | Fibrosis | Idiopathic Pulmonary Fibrosis - pathology | Histone Deacetylase Inhibitors - therapeutic use | Idiopathic Pulmonary Fibrosis - enzymology | Cystic Fibrosis - drug therapy | Idiopathic Pulmonary Fibrosis - drug therapy | Scleroderma, Systemic - drug therapy
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8. Full Text LCZ696, Angiotensin II Receptor-Neprilysin Inhibitor, Ameliorates High-Salt-Induced Hypertension and Cardiovascular Injury More Than Valsartan Alone
by Kusaka, Hiroaki and Sueta, Daisuke and Koibuchi, Nobutaka and Hasegawa, Yu and Nakagawa, Takashi and Lin, BoWen and Ogawa, Hisao and Kim-Mitsuyama, Shokei
American journal of hypertension, ISSN 0895-7061, 12/2015, Volume 28, Issue 12, pp. 1409 - 1417
angiotensin | blood pressure | cardiovascular protection | neprilysin | dual inhibition | hypertension | Peripheral Vascular Disease | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Fibrosis - drug therapy | Tetrazoles - pharmacology | Sodium, Dietary - adverse effects | Angiotensin Receptor Antagonists - pharmacology | Endothelium, Vascular - drug effects | Hypertension - drug therapy | Male | Aminobutyrates - pharmacology | Cyclic GMP - blood | Aminobutyrates - therapeutic use | Hypertension - etiology | Valsartan - pharmacology | Hypertension - blood | Valsartan - therapeutic use | Inflammation - drug therapy | Blood Pressure - drug effects | Vascular Remodeling - drug effects | Drug Evaluation, Preclinical | Rats, Inbred SHR | Cardiomegaly - drug therapy | Sodium, Dietary - urine | Myocardium - pathology | Random Allocation | Animals | Neprilysin - antagonists & inhibitors | Heart - drug effects | Tetrazoles - therapeutic use | Oxidative Stress - drug effects | Angiotensin Receptor Antagonists - therapeutic use | Circadian Rhythm - drug effects | Hypertension | Care and treatment | Cardiac patients | Angiotensin II receptor blockers | Dosage and administration | Cardiovascular diseases | Health aspects | Risk factors | Index Medicus
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9. Full Text Targeting Cardiac Hypertrophy: Toward a Causal Heart Failure Therapy
by Bisping, Egbert and Wakula, Paulina and Poteser, Michael and Heinzel, Frank R
Journal of cardiovascular pharmacology, ISSN 0160-2446, 10/2014, Volume 64, Issue 4, pp. 293 - 305
Heart failure | Hypertension | Camkii | Reactive oxygen species | Calcium | Calcineurin | Angiotensin receptor blockers | Biased agonists | Left ventricular | Vitamin | AKT | Remodeling | Beta blockers | NFAT | Guanylatecyclase stimulators | Signaling | Nitric oxide | CGMP | Angiotensin II | Hypertrophy | Cardiac & Cardiovascular Systems | Pharmacology & Pharmacy | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Humans | Cardiomegaly - drug therapy | Antihypertensive Agents - administration & dosage | Cardiomegaly - pathology | Clinical Trials as Topic | Heart Failure - metabolism | Heart Failure - pathology | Antihypertensive Agents - therapeutic use | Molecular Targeted Therapy | Cardiomegaly - complications | Heart Failure - prevention & control | Heart Failure - etiology | Ventricular Remodeling - drug effects | Cardiomegaly - metabolism | Index Medicus
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