X
Search Filters
Format Format
Format Format
X
Sort by Item Count (A-Z)
Filter by Count
Journal Article (3240) 3240
Publication (272) 272
Book Review (18) 18
Magazine Article (8) 8
Book / eBook (7) 7
Book Chapter (7) 7
Conference Proceeding (5) 5
Data Set (2) 2
Dissertation (2) 2
more...
Subjects Subjects
Subjects Subjects
X
Sort by Item Count (A-Z)
Filter by Count
humans (1887) 1887
animals (1860) 1860
male (1721) 1721
cardiomegaly - drug therapy (1380) 1380
rats (1156) 1156
index medicus (900) 900
female (862) 862
cardiomegaly - physiopathology (831) 831
cardiomegaly - etiology (821) 821
hypertension - drug therapy (800) 800
cardiomegaly - pathology (658) 658
cardiac & cardiovascular systems (629) 629
middle aged (599) 599
cardiomegaly - metabolism (549) 549
hypertrophy (536) 536
blood pressure - drug effects (523) 523
hypertension - complications (520) 520
adult (508) 508
cardiomegaly - prevention & control (440) 440
mice (437) 437
pharmacology & pharmacy (428) 428
myocardium - pathology (425) 425
hypertension (418) 418
heart (417) 417
echocardiography (395) 395
cardiac hypertrophy (386) 386
hypertension - physiopathology (379) 379
myocardium - metabolism (366) 366
heart failure (361) 361
disease models, animal (359) 359
cardiomegaly - complications (349) 349
antihypertensive agents - therapeutic use (345) 345
heart-failure (332) 332
peripheral vascular disease (332) 332
rats, sprague-dawley (331) 331
aged (309) 309
electrocardiography (303) 303
heart failure - drug therapy (302) 302
fibrosis (267) 267
heart - drug effects (266) 266
time factors (265) 265
myocytes, cardiac - drug effects (259) 259
cardiomegaly - chemically induced (256) 256
rats, inbred shr (247) 247
heart enlargement (238) 238
blood pressure (236) 236
cardiovascular system (229) 229
myocytes, cardiac - metabolism (227) 227
physiology (224) 224
cardiac-hypertrophy (222) 222
cardiomegaly - genetics (220) 220
left-ventricular hypertrophy (219) 219
rats, wistar (219) 219
cardiomegaly - diagnostic imaging (217) 217
hemodynamics - drug effects (212) 212
mice, inbred c57bl (201) 201
expression (195) 195
heart failure - physiopathology (190) 190
cell biology (189) 189
oxidative stress (188) 188
angiotensin-converting enzyme inhibitors - therapeutic use (186) 186
myocytes, cardiac - pathology (186) 186
cells, cultured (181) 181
myocardial-infarction (171) 171
myocardial infarction - drug therapy (166) 166
signal transduction (164) 164
signal transduction - drug effects (159) 159
apoptosis (158) 158
risk factors (157) 157
rodents (154) 154
heart rate - drug effects (153) 153
myocardial contraction - drug effects (152) 152
blood-pressure (151) 151
cardiomegaly - diagnosis (151) 151
heart - physiopathology (150) 150
heart failure - etiology (150) 150
cardiomegaly - enzymology (149) 149
heart diseases (148) 148
hemodynamics (145) 145
activation (142) 142
cardiomyopathy (142) 142
medicine, research & experimental (141) 141
cardiomegaly (140) 140
gene-expression (140) 140
cardiomyocytes (138) 138
rats, inbred wky (138) 138
ventricular remodeling - drug effects (135) 135
gene expression (131) 131
physiological aspects (131) 131
antihypertensive agents - pharmacology (130) 130
medicine, general & internal (128) 128
angiotensin (127) 127
pressure-overload (127) 127
research (127) 127
article (126) 126
angiotensin-ii (125) 125
myocardial infarction - physiopathology (125) 125
organ size - drug effects (124) 124
biochemistry & molecular biology (121) 121
health aspects (121) 121
more...
Library Location Library Location
Language Language
Language Language
X
Sort by Item Count (A-Z)
Filter by Count
English (2848) 2848
German (105) 105
French (70) 70
Russian (58) 58
Japanese (48) 48
Italian (37) 37
Spanish (31) 31
Chinese (19) 19
Portuguese (10) 10
Polish (9) 9
Czech (8) 8
Romanian (4) 4
Bulgarian (2) 2
Danish (2) 2
Norwegian (2) 2
Croatian (1) 1
Dutch (1) 1
Hebrew (1) 1
Hungarian (1) 1
Slovak (1) 1
Swedish (1) 1
Ukrainian (1) 1
more...
Publication Date Publication Date
Click on a bar to filter by decade
Slide to change publication date range


Journal of Molecular and Cellular Cardiology, ISSN 0022-2828, 2015, Volume 84, pp. 24 - 35
Abstract Unresolved inflammation is a major contributor to the development of heart failure following myocardial infarction (MI). Pro-resolving lipid... 
Cardiovascular | Myocardial infarction | Splenic remodeling | Resolution of inflammation | Metabololipidomics | Resolvin D1 | Neutrophils | SURVIVAL | CHRONIC HEART-FAILURE | PROTECTIVE ACTIONS | CARDIAC & CARDIOVASCULAR SYSTEMS | RESOLUTION | TISSUE | RECEPTOR | LIPID MEDIATORS | CELL BIOLOGY | THERAPY | PATHWAY | DISEASE | Cell Count | Male | Myocardial Infarction - diagnostic imaging | Spleen - drug effects | Extracellular Matrix - genetics | Inflammation - complications | Pulmonary Edema - physiopathology | Inflammation - drug therapy | Pulmonary Edema - complications | Receptors, Formyl Peptide - metabolism | Ultrasonography | Myocardial Infarction - physiopathology | Cell Polarity - drug effects | Spleen - pathology | Neutrophil Infiltration - drug effects | Ventricular Function - drug effects | Docosahexaenoic Acids - therapeutic use | Arachidonate 5-Lipoxygenase - metabolism | Docosahexaenoic Acids - chemistry | Extracellular Matrix - drug effects | Mice, Inbred C57BL | Cardiomegaly - diagnostic imaging | Cardiomegaly - drug therapy | Cardiomegaly - physiopathology | Pulmonary Edema - drug therapy | Cardiomegaly - complications | Collagen - metabolism | Docosahexaenoic Acids - pharmacology | Myocardial Infarction - complications | Macrophages - metabolism | Animals | Myocardial Infarction - drug therapy | Heart Ventricles - physiopathology | Prostaglandin-Endoperoxide Synthases - metabolism | Macrophages - drug effects | Ventricular Remodeling - drug effects | Heart Ventricles - drug effects | Inflammation | Heart attack | metabololipidomics | liposomes | myocardial infarction | splenic remodeling | neutrophils | resolution of inflammation
Journal Article
Circulation, ISSN 0009-7322, 07/2010, Volume 122, Issue 1, pp. 11 - 19
Journal Article
Journal Article
Journal Article
Scientific Reports, ISSN 2045-2322, 2015, Volume 5, Issue 1, p. 8794
We recently found that overexpression of angiotensin (Ang)-converting enzyme 2, which metabolizes Ang-II to Ang-(1-7) and Ang-I to Ang-(1-9), may improve left... 
ACTIVATION | MYOCARDIAL FIBROSIS | MULTIDISCIPLINARY SCIENCES | CARDIAC-HYPERTROPHY | HIGH GLUCOSE | PROXIMAL TUBULAR CELLS | RECEPTOR MAS | DEFICIENT MICE | DYSFUNCTION | RAT MODEL | FIBROBLASTS | Receptors, Angiotensin - metabolism | Diabetic Cardiomyopathies - metabolism | Diabetic Cardiomyopathies - drug therapy | Apoptosis - drug effects | Transforming Growth Factor beta1 - metabolism | Cardiomegaly - etiology | Cardiomegaly - pathology | Male | Peptide Fragments - pharmacology | Perindopril - administration & dosage | Perindopril - pharmacology | Heart Ventricles - ultrastructure | Peptidyl-Dipeptidase A - metabolism | Angiotensin I - pharmacology | Collagen - biosynthesis | p38 Mitogen-Activated Protein Kinases - metabolism | Phosphorylation - drug effects | Drug Therapy, Combination | Heart Ventricles - pathology | Disease Models, Animal | Fibroblasts - metabolism | Echocardiography | Peptide Fragments - administration & dosage | Angiotensin I - administration & dosage | Cardiomegaly - drug therapy | Rats | Diabetic Cardiomyopathies - diagnosis | Ventricular Dysfunction, Left - prevention & control | Blood Glucose - drug effects | Animals | Ventricular Dysfunction, Left - drug therapy | Mitogen-Activated Protein Kinase 3 - metabolism | Cell Differentiation - drug effects | Fibroblasts - drug effects | Cell Communication - drug effects | Fibrosis | Cell Proliferation - drug effects | Fibroblasts - cytology | Hemodynamics - drug effects | Oxidative Stress - drug effects | Heart Ventricles - drug effects | Mitogen-Activated Protein Kinase 1 - metabolism | Heart | Enzymes | Cardiomyopathy | Diabetes mellitus | Rodents | Angiotensin | Diabetes | Ventricle | Angiotensin II | Hypertrophy | Apoptosis
Journal Article
Circulation Research, ISSN 0009-7330, 08/2013, Volume 113, Issue 5, pp. 603 - 616
Journal Article
PLoS ONE, ISSN 1932-6203, 10/2013, Volume 8, Issue 10, p. e78330
Background: Myocardial fibrosis is a key process in diabetic cardiomyopathy. However, their underlying mechanisms have not been elucidated, leading to a lack... 
HEART | CELLS | ACTIVATION | DPP-4 INHIBITION | GLUCAGON-LIKE PEPTIDE-1 | MYOCARDIAL-INFARCTION | RAT | MULTIDISCIPLINARY SCIENCES | GENE-EXPRESSION | RECEPTOR | IMPROVES | Diabetic Cardiomyopathies - metabolism | Metformin - therapeutic use | Diabetic Cardiomyopathies - drug therapy | Insulin - physiology | Fibroblasts - physiology | Cardiomegaly - etiology | Glucagon-Like Peptide 1 - physiology | Male | Pyrazines - therapeutic use | Diabetes Mellitus, Type 2 - metabolism | Cardiotonic Agents - therapeutic use | Glucose Intolerance - drug therapy | Sitagliptin Phosphate | Drug Evaluation, Preclinical | Diabetes Mellitus, Type 2 - complications | Disease Models, Animal | Triazoles - therapeutic use | Hypoglycemic Agents - therapeutic use | Metformin - pharmacology | Cardiomegaly - drug therapy | Cells, Cultured | Glucagon-Like Peptide 1 - pharmacology | Rats | Myocardium - pathology | Cardiotonic Agents - pharmacology | Fibronectins - metabolism | Protein Isoforms - physiology | Hypoglycemic Agents - pharmacology | Protein Isoforms - pharmacology | Triazoles - pharmacology | Animals | Myocytes, Cardiac - drug effects | Myocytes, Cardiac - physiology | Fibrosis | PPAR delta - metabolism | Diabetes Mellitus, Type 2 - drug therapy | Pyrazines - pharmacology | Apoptosis | Cardiomegaly - metabolism | Hyperglycemia | Glucagon | Heart enlargement | Glucose | Insulin | Dextrose | Diabetes therapy | Heart | Plasma | Heart attacks | Peptides | Cardiomyopathy | Deceleration | Cardiovascular disease | Kinases | Palmitic acid | Eutrophication | Proteins | Rodents | Fibroblasts | Extracellular matrix | Heart diseases | Glucagon-like peptide 1 | Heart failure | Cell survival | Hyperlipidemia | Diabetes mellitus | Cardiomyocytes | Metabolism | Signaling | Cell death | Isoforms | Metformin | Diabetes | Laboratory animals | Hypertrophy | Structure-function relationships
Journal Article