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Proceedings of the National Academy of Sciences - PNAS, ISSN 0027-8424, 7/2013, Volume 110, Issue 31, pp. 12655 - 12660
The positive transcription elongation factor b (P-TEFb) is involved in physiological and pathological events including inflammation, cancer, AIDS, and cardiac... 
T lymphocytes | Pathology | RNA | HEK293 cells | Genes | Cell lines | Antibodies | Small nuclear RNA | Gene expression regulation | HIV 1 | 7SK SNRNA | TRANSCRIPTION FACTOR BCL11B | ELONGATION | MULTIDISCIPLINARY SCIENCES | GENE-EXPRESSION | POLYMERASE | HIV-1 TAT | MICROGLIAL CELLS | T-CELL LINEAGE | BROMODOMAIN PROTEIN BRD4 | RNA-Binding Proteins - genetics | Humans | Myosin Heavy Chains - genetics | Cardiomegaly - pathology | Positive Transcriptional Elongation Factor B - metabolism | Positive Transcriptional Elongation Factor B - genetics | Myosin Heavy Chains - metabolism | RNA, Small Nuclear - genetics | Tumor Suppressor Proteins - genetics | HEK293 Cells | Cardiac Myosins - metabolism | Repressor Proteins - metabolism | Promoter Regions, Genetic | Tumor Suppressor Proteins - metabolism | Protein Structure, Secondary | Cyclin-Dependent Kinase 9 - genetics | Cardiac Myosins - genetics | Repressor Proteins - genetics | Transcription Factors - genetics | Transcription Factors - metabolism | Animals | Cyclin-Dependent Kinase 9 - metabolism | Mice | RNA, Small Nuclear - metabolism | Cardiomegaly - genetics | RNA-Binding Proteins - metabolism | Cardiomegaly - metabolism | Physiological aspects | Transcription factors | Genetic aspects | Heart enlargement | Health aspects | Myosin | Life Sciences | Neurons and Cognition | Biological Sciences
Journal Article
American journal of physiology. Heart and circulatory physiology, ISSN 1522-1539, 2015, Volume 309, Issue 9, pp. H1375 - H1389
Modification of histones is one of the important mechanisms of epigenetics, in which genetic control is determined by factors other than an individual's DNA sequence... 
FoxO | Sirtuin-activating compounds | Longevity | Cell death/survival | cell death/survival | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | sirtuin-activating compounds | FOXO TRANSCRIPTION FACTORS | ISCHEMIA-REPERFUSION INJURY | PROMOTES CELL-SURVIVAL | CEREVISIAE LIFE-SPAN | FATTY-ACID OXIDATION | MANGANESE SUPEROXIDE-DISMUTASE | STRESS-RESPONSIVE DEACETYLASE | PANCREATIC BETA-CELLS | PERIPHERAL VASCULAR DISEASE | DEPENDENT HISTONE DEACETYLASE | SMALL-MOLECULE ACTIVATORS | Cardiovascular Diseases | Heart Diseases - metabolism | Epigenesis, Genetic | Humans | Cell Survival - genetics | Apoptosis - genetics | Aging - genetics | Autophagy - genetics | Energy Metabolism - genetics | Sirtuins - genetics | Reperfusion Injury - genetics | Reperfusion Injury - metabolism | Cellular Senescence - genetics | Myocytes, Cardiac - cytology | Oxidative Stress - genetics | Heart Failure - genetics | Heart Failure - metabolism | Longevity - genetics | Animals | Myocytes, Cardiac - metabolism | Mice | Histones - metabolism | Cardiomegaly - genetics | Sirtuins - metabolism | Heart Diseases - genetics | Cardiomegaly - metabolism | Epigenetic inheritance | Complications and side effects | Care and treatment | Analysis | Influence | Mitochondrial DNA | Research | Cardiovascular diseases | longevity | Reviews | cell death | survival
Journal Article
American journal of human genetics, ISSN 0002-9297, 2012, Volume 90, Issue 6, pp. 1094 - 1101
Journal Article
Aging cell, ISSN 1474-9718, 2017, Volume 16, Issue 5, pp. 976 - 987
Summary Aging is accompanied with unfavorable geometric and functional changes in the heart involving dysregulation of Akt and autophagy. This study examined... 
cardiac geometry | contractile function | autophagy | aging | Akt | RAPAMYCIN | PROTEIN-KINASE | DIASTOLIC DYSFUNCTION | CELL BIOLOGY | GERIATRICS & GERONTOLOGY | HYPERTROPHY | LONGEVITY | OVEREXPRESSION | INSULIN-RESISTANCE | CALORIC RESTRICTION | SENESCENCE | MITOCHONDRIAL INJURY | Adaptation, Physiological | Protein Kinases - metabolism | Sirtuin 1 - metabolism | Phosphorylation | Protein Kinases - genetics | Microtubule-Associated Proteins - genetics | Calcium - metabolism | Microtubule-Associated Proteins - metabolism | Autophagy-Related Protein 7 - metabolism | Proto-Oncogene Proteins c-akt - deficiency | Atrial Remodeling - drug effects | Myocardial Contraction - drug effects | Cardiomegaly - pathology | Male | Mitochondrial Proteins - genetics | Sirtuin 1 - genetics | Autophagy - drug effects | Heterocyclic Compounds, 4 or More Rings - pharmacology | Proto-Oncogene Proteins c-akt - genetics | Mitochondrial Proteins - metabolism | Myocardium - metabolism | Membrane Proteins - metabolism | Autophagy - genetics | Forkhead Box Protein O1 - metabolism | Sarcoplasmic Reticulum Calcium-Transporting ATPases - metabolism | Signal Transduction | Membrane Proteins - genetics | Gene Expression Regulation | Myocardium - pathology | Mitochondria - metabolism | Longevity - genetics | Mitochondria - pathology | Autophagy-Related Protein 7 - genetics | Sirolimus - pharmacology | Mice, Knockout | Animals | Cardiomegaly - prevention & control | Beclin-1 - genetics | Mice | Beclin-1 - metabolism | Cardiomegaly - genetics | Sarcoplasmic Reticulum Calcium-Transporting ATPases - genetics | Forkhead Box Protein O1 - genetics | Cardiomegaly - metabolism | Heart | Laws, regulations and rules | Ablation (Surgery) | AKT protein | Autophagy | Ca2+-transporting ATPase | Mitochondria | FOXO1 protein | Aging | Oxidation | Fura-2 | Acetylation | Heart diseases | Calcium (intracellular) | Echocardiography | AKT2 protein | Cardiomyocytes | Rapamycin | Insulin | Muscle contraction | BNIP3 protein | Life span | PTEN-induced putative kinase | Parkin protein | Intracellular | PTEN protein | Phagocytosis | Hypertrophy | Original
Journal Article
Nature, ISSN 0028-0836, 10/2011, Volume 478, Issue 7367, pp. 114 - 118
...). So far, genome-wide association studies have not identified the genetic factors that underlie LVM variation(3... 
PATHOGENESIS | OXIDATIVE STRESS | REPLICATION | BIOGENESIS | MULTIDISCIPLINARY SCIENCES | CARDIOMYOPATHY | DISEASE | DEATH | MECHANISMS | LEFT-VENTRICULAR MASS | EXPRESSION | Receptors, Estrogen - metabolism | Hypertrophy, Left Ventricular - enzymology | Reactive Oxygen Species - metabolism | Chromosomes, Mammalian - genetics | Cardiomegaly - pathology | Male | Rats, Inbred Strains | Body Weight - genetics | Hypertrophy, Left Ventricular - genetics | Cell Respiration | Mitochondria - genetics | Hypertrophy, Left Ventricular - pathology | Organ Size - genetics | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha | Endodeoxyribonucleases - metabolism | Female | Endodeoxyribonucleases - deficiency | Gene Expression Regulation | Cardiomegaly - physiopathology | Rats | Lipid Metabolism | Mitochondria - metabolism | Cardiomegaly - enzymology | Mitochondria - pathology | Transcription Factors - metabolism | Genes, Mitochondrial - genetics | Endodeoxyribonucleases - genetics | Animals | Hypertrophy, Left Ventricular - physiopathology | Cardiomegaly - genetics | RNA-Binding Proteins - metabolism | Apoptosis | Crosses, Genetic | Quantitative Trait Loci - genetics | Nucleases | Mitochondria | Heart enlargement | Physiological aspects | Genetic aspects | Research | Diagnosis | Risk factors | Studies | Rodents | Biomarkers | Genetics | Cardiomyocytes | Genomes | Blood pressure | Kinases
Journal Article
Journal Article
Molecular therapy, ISSN 1525-0016, 2018, Volume 26, Issue 3, pp. 902 - 916
Journal Article
Cell, ISSN 0092-8674, 2002, Volume 110, Issue 4, pp. 479 - 488
The heart responds to stress signals by hypertrophic growth, which is accompanied by activation of the MEF2 transcription factor and reprogramming of cardiac... 
ACTIVATION | MYOGENESIS | P300 | BIOCHEMISTRY & MOLECULAR BIOLOGY | IN-VIVO | CALCINEURIN | MEF2 TRANSCRIPTION FACTOR | DEPENDENT PROTEIN-KINASE | ASSOCIATION | BINDING | MITR | CELL BIOLOGY | Physiological aspects | Enzymes | Cellular signal transduction | Regulation | Heart enlargement | Heart muscle
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 05/2011, Volume 286, Issue 21, pp. 18465 - 18473
Resistin has been suggested to be involved in the development of diabetes and insulin resistance. We recently reported that resistin is expressed in diabetic... 
MAMMALIAN TARGET | INSULIN-RECEPTOR | OBESITY | PHOSPHORYLATION | GLUCOSE | BIOCHEMISTRY & MOLECULAR BIOLOGY | HEART-FAILURE | THERAPEUTIC TARGET | DIABETIC CARDIOMYOPATHY | P70 S6 KINASE | MTOR | AMP-Activated Protein Kinases - metabolism | Resistin - genetics | TOR Serine-Threonine Kinases - metabolism | Ventricular Myosins - biosynthesis | Apoptosis - drug effects | Apoptosis - genetics | Cardiomegaly - pathology | JNK Mitogen-Activated Protein Kinases - metabolism | Insulin Receptor Substrate Proteins - metabolism | TOR Serine-Threonine Kinases - antagonists & inhibitors | Phosphorylation - genetics | TOR Serine-Threonine Kinases - genetics | JNK Mitogen-Activated Protein Kinases - genetics | Phosphorylation - drug effects | Insulin Receptor Substrate Proteins - genetics | Biomarkers - metabolism | Natriuretic Peptide, Brain - genetics | Resistin - metabolism | JNK Mitogen-Activated Protein Kinases - antagonists & inhibitors | Ribosomal Protein S6 Kinases, 70-kDa - metabolism | Signal Transduction | Gene Expression Regulation - genetics | Ribosomal Protein S6 Kinases, 70-kDa - antagonists & inhibitors | AMP-Activated Protein Kinases - antagonists & inhibitors | Cells, Cultured | Enzyme Inhibitors - pharmacology | Natriuretic Peptide, Brain - biosynthesis | Rats | Ribosomal Protein S6 Kinases, 70-kDa - genetics | Rats, Sprague-Dawley | Gene Expression Regulation - drug effects | Myocytes, Cardiac - pathology | Animals | Insulin Receptor Substrate Proteins - antagonists & inhibitors | Insulin Resistance - genetics | Myocytes, Cardiac - metabolism | Cardiomegaly - genetics | AMP-Activated Protein Kinases - genetics | Cardiomegaly - metabolism | Ventricular Myosins - genetics | Molecular Bases of Disease | Cardiomyocyte | Insulin Resistance | AMP-activated Kinase (AMPK) | Gene Transfer | Diabetes | mTOR, JNK Signaling | Cardiac Hypertrophy | Diabetic Cardiomyopathy | Resistin
Journal Article
Journal of cellular and molecular medicine, ISSN 1582-1838, 2017, Volume 21, Issue 8, pp. 1492 - 1502
Cardiac hypertrophy is an early hallmark during the clinical course of heart failure and regulated by various signalling pathways. Recently, we observed that... 
CD38 | cardiac hypertrophy | angiotensin II | SIRT3 | NFATc4 | MEDICINE, RESEARCH & EXPERIMENTAL | CYCLIC ADP-RIBOSE | OXIDATIVE STRESS | SIGNALING PATHWAYS | CARDIOMYOCYTES | CELL BIOLOGY | HEART | NAD | INTERLEUKIN-18 | CONTRACTION | MYOCARDIAL HYPERTROPHY | GROWTH | RNA, Small Interfering - genetics | Phosphorylation | Reactive Oxygen Species - metabolism | Calcium - metabolism | Natriuretic Peptide, Brain - metabolism | Cardiomegaly - pathology | ADP-ribosyl Cyclase 1 - antagonists & inhibitors | Proto-Oncogene Proteins c-akt - genetics | Forkhead Box Protein O3 - genetics | Membrane Glycoproteins - antagonists & inhibitors | Atrial Natriuretic Factor - genetics | Mitogen-Activated Protein Kinase 1 - genetics | Myocardium - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Sirtuins - genetics | Cell Line | Angiotensin II - pharmacology | Natriuretic Peptide, Brain - genetics | ADP-ribosyl Cyclase 1 - deficiency | ADP-ribosyl Cyclase 1 - genetics | Atrial Natriuretic Factor - metabolism | Mitogen-Activated Protein Kinase 3 - genetics | Signal Transduction | NFATC Transcription Factors - metabolism | Gene Expression Regulation | Rats | Myocardium - pathology | Nerve Tissue Proteins - genetics | Forkhead Box Protein O3 - metabolism | Membrane Glycoproteins - genetics | Mice, Knockout | Nerve Tissue Proteins - metabolism | Myocytes, Cardiac - pathology | Animals | Myocytes, Cardiac - drug effects | Mitogen-Activated Protein Kinase 3 - metabolism | Cardiomegaly - chemically induced | Myocytes, Cardiac - metabolism | Mice | Cardiomegaly - genetics | Sirtuins - metabolism | Membrane Glycoproteins - deficiency | Cardiomegaly - metabolism | Mitogen-Activated Protein Kinase 1 - metabolism | NFATC Transcription Factors - genetics | RNA, Small Interfering - metabolism | Heart failure | Hypertension | Heart | Antioxidants | Heart enlargement | Analysis | Angiotensin | Gene expression | Natriuretic peptides | Brain | Oxidative stress | Reactive oxygen species | Hydrogen peroxide | AKT protein | Atrial natriuretic peptide | NF-AT protein | Calcium signalling | Infusion | Signal transduction | Oxidation resistance | Reperfusion | Ischemia | Rodents | CD38 antigen | Fibroblasts | Inhibition | Angiotensin II | Heart diseases | Injuries | FOXO3 protein | Translocation | Brain natriuretic peptide | Calcium (intracellular) | Extracellular signal-regulated kinase | Embryo fibroblasts | Embryos | Nuclear transport | Fibrosis | Hypoxia | Hypertrophy | Original
Journal Article