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Proceedings of the National Academy of Sciences - PNAS, ISSN 0027-8424, 7/2013, Volume 110, Issue 31, pp. 12655 - 12660
The positive transcription elongation factor b (P-TEFb) is involved in physiological and pathological events including inflammation, cancer, AIDS, and cardiac... 
T lymphocytes | Pathology | RNA | HEK293 cells | Genes | Cell lines | Antibodies | Small nuclear RNA | Gene expression regulation | HIV 1 | 7SK SNRNA | TRANSCRIPTION FACTOR BCL11B | ELONGATION | MULTIDISCIPLINARY SCIENCES | GENE-EXPRESSION | POLYMERASE | HIV-1 TAT | MICROGLIAL CELLS | T-CELL LINEAGE | BROMODOMAIN PROTEIN BRD4 | RNA-Binding Proteins - genetics | Humans | Myosin Heavy Chains - genetics | Cardiomegaly - pathology | Positive Transcriptional Elongation Factor B - metabolism | Positive Transcriptional Elongation Factor B - genetics | Myosin Heavy Chains - metabolism | RNA, Small Nuclear - genetics | Tumor Suppressor Proteins - genetics | HEK293 Cells | Cardiac Myosins - metabolism | Repressor Proteins - metabolism | Promoter Regions, Genetic | Tumor Suppressor Proteins - metabolism | Protein Structure, Secondary | Cyclin-Dependent Kinase 9 - genetics | Cardiac Myosins - genetics | Repressor Proteins - genetics | Transcription Factors - genetics | Transcription Factors - metabolism | Animals | Cyclin-Dependent Kinase 9 - metabolism | Mice | RNA, Small Nuclear - metabolism | Cardiomegaly - genetics | RNA-Binding Proteins - metabolism | Cardiomegaly - metabolism | Physiological aspects | Transcription factors | Genetic aspects | Heart enlargement | Health aspects | Myosin | Life Sciences | Neurons and Cognition | Biological Sciences
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 09/2012, Volume 303, Issue 5, pp. 605 - 618
Apelin is an endogenous ligand for the angiotensin-like 1 receptor (APJ) and has beneficial effects against myocardial ischemia-reperfusion injury. Little is... 
Stromal cell-derived factor-1α/C-X-C chemokine receptor-4 | Vascular progenitor cell | Myocardial repair | c-kit | Jagged1/notch3 | ISCHEMIA/REPERFUSION INJURY | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | c-Kit | ANGIOGENESIS | HEART-FAILURE | myocardial repair | vascular progenitor cell | APJ RECEPTOR | PROTEIN-COUPLED RECEPTOR | EMBRYONIC STEM-CELLS | PERIPHERAL VASCULAR DISEASE | LIGAND | stromal cell-derived factor-1 alpha/C-X-C chemokine receptor-4 | jagged1/notch3 | TOPCARE-AMI | ISCHEMIC-STROKE | PARACRINE | Apoptosis - drug effects | Receptors, Notch - metabolism | Cardiomegaly - pathology | Vascular Endothelial Growth Factor A - metabolism | Antigens, CD - metabolism | Peptides - metabolism | Time Factors | Serrate-Jagged Proteins | Antigens, Ly - metabolism | Myocardial Infarction - physiopathology | Proto-Oncogene Proteins c-akt - metabolism | Apelin | Disease Models, Animal | Jagged-1 Protein | Biomarkers - metabolism | Ventricular Function, Left - drug effects | Cardiomegaly - physiopathology | Cardiotonic Agents - pharmacology | Myocardial Infarction - metabolism | Receptors, CXCR4 - metabolism | Cell Movement - drug effects | Myocytes, Cardiac - pathology | Regeneration - drug effects | Cardiomegaly - prevention & control | Chemokine CXCL12 - metabolism | Myocytes, Cardiac - drug effects | Intercellular Signaling Peptides and Proteins - pharmacology | Fibrosis | Myocytes, Cardiac - metabolism | Stem Cells - pathology | Mice | Myocardial Infarction - genetics | Neovascularization, Physiologic - drug effects | Glycoproteins - metabolism | Proto-Oncogene Proteins c-kit - metabolism | Stem Cells - metabolism | Recovery of Function | Intercellular Signaling Peptides and Proteins - metabolism | Myocardial Infarction - pathology | Membrane Proteins - metabolism | Nitric Oxide Synthase Type III - metabolism | Calcium-Binding Proteins - metabolism | Mice, Inbred C57BL | Cells, Cultured | Adipokines | AC133 Antigen | Animals | Myocardial Infarction - drug therapy | Stem Cells - drug effects | Receptor, Notch3 | Physiological aspects | Care and treatment | Health aspects | Ligands (Biochemistry) | Heart attack | stromal cell-derived factor-1α | notch3 | Integrative Cardiovascular Physiology and Pathophysiology | jagged1 | C-X-C chemokine receptor-4
Journal Article
Kidney international, ISSN 0085-2538, 2018, Volume 93, Issue 1, pp. 147 - 158
We examined activin receptor type IIA (ActRIIA) activation in chronic kidney disease (CKD) by signal analysis and inhibition in mice with Alport syndrome using... 
cardiovascular disease | chronic kidney disease | bone | fibrosis | mineral metabolism | vascular calcification | TGF-BETA | CARDIOVASCULAR EVENTS | RENAL FIBROSIS | ARTERIAL CALCIFICATION | BONE MORPHOGENETIC PROTEIN-7 | GROWTH-FACTOR 23 | UROLOGY & NEPHROLOGY | SMOOTH-MUSCLE-CELLS | LINKED ALPORT-SYNDROME | OSTEOCLAST DIFFERENTIATION | ATHEROSCLEROTIC LESION | Kidney - pathology | Recombinant Fusion Proteins - pharmacology | Blood Vessels - metabolism | Chronic Kidney Disease-Mineral and Bone Disorder - metabolism | Core Binding Factor Alpha 1 Subunit - metabolism | Chronic Kidney Disease-Mineral and Bone Disorder - genetics | Vascular Calcification - metabolism | Kidney - metabolism | Bone Resorption - genetics | Bone and Bones - metabolism | Myocardium - metabolism | Renal Insufficiency, Chronic - genetics | Bone Resorption - metabolism | Disease Models, Animal | Bone Resorption - physiopathology | Glomerular Filtration Rate | Signal Transduction | Smad2 Protein - metabolism | Cardiomegaly - physiopathology | Nephritis, Hereditary - genetics | Renal Insufficiency, Chronic - physiopathology | Mice, Knockout | Renal Insufficiency, Chronic - prevention & control | Cardiomegaly - prevention & control | Nephritis, Hereditary - metabolism | Collagen Type IV - deficiency | Activin Receptors, Type II - genetics | Fibrosis | Sp7 Transcription Factor - metabolism | Collagen Type IV - genetics | Cardiomegaly - metabolism | Activin Receptors, Type II - metabolism | Bone and Bones - pathology | Phosphorylation | Vascular Remodeling | Blood Vessels - pathology | Vascular Calcification - genetics | Actins - metabolism | Chronic Kidney Disease-Mineral and Bone Disorder - physiopathology | Activin Receptors, Type II - antagonists & inhibitors | Blood Vessels - physiopathology | Renal Insufficiency, Chronic - metabolism | Ventricular Remodeling | Chronic Kidney Disease-Mineral and Bone Disorder - prevention & control | Muscle Proteins - metabolism | Vascular Calcification - physiopathology | Microfilament Proteins - metabolism | Nephritis, Hereditary - physiopathology | Kidney - physiopathology | Vascular Calcification - prevention & control | Myocardium - pathology | Bone Resorption - prevention & control | Bone and Bones - physiopathology | Animals | Nephritis, Hereditary - drug therapy | Cardiomegaly - genetics | Bone Remodeling | Chronic kidney disease
Journal Article
Molecular Cell, ISSN 1097-2765, 10/2008, Volume 32, Issue 2, pp. 169 - 179
Elevated catecholamines in the heart evoke transcriptional activation of the Myocyte Enhancer Factor (MEF) pathway to induce a cellular response known as... 
HUMDISEASE | SIGNALING | COMPLEX | DEPENDENT REGULATION | PROTEIN-KINASE-D | PHOSPHORYLATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | HEART-FAILURE | NUCLEAR EXPORT | LONG-QT SYNDROME | CELLULAR-LOCALIZATION | HISTONE DEACETYLASE-5 | POTASSIUM CHANNEL | CELL BIOLOGY | Guanine Nucleotide Exchange Factors - physiology | Phosphorylation | Minor Histocompatibility Antigens | Humans | Cercopithecus aethiops | A Kinase Anchor Proteins - genetics | A Kinase Anchor Proteins - physiology | Phenylephrine - pharmacology | RNA Interference | Guanine Nucleotide Exchange Factors - metabolism | Protein Kinase C - metabolism | Active Transport, Cell Nucleus | A Kinase Anchor Proteins - metabolism | MEF2 Transcription Factors | Cyclic AMP-Dependent Protein Kinases - metabolism | Proto-Oncogene Proteins - metabolism | Cell Line | Guanine Nucleotide Exchange Factors - genetics | Signal Transduction | Gene Expression Regulation | Myogenic Regulatory Factors - metabolism | Rats | Histone Deacetylases - metabolism | Proto-Oncogene Proteins - genetics | 14-3-3 Proteins - metabolism | Myocytes, Cardiac - pathology | Animals | Myocytes, Cardiac - drug effects | Models, Biological | Proto-Oncogene Proteins - physiology | Myocytes, Cardiac - metabolism | COS Cells | Cardiomegaly - metabolism | Heart Ventricles - drug effects | Proteins | Catecholamines | RNA | Cardiology | Heart enlargement | Protein kinases
Journal Article
PLoS ONE, ISSN 1932-6203, 04/2012, Volume 7, Issue 4, p. e35905
Hematopoietic progenitor CD133(+)/c-kit(+) cells have been shown to be involved in myocardial healing following myocardial infarction (MI). Previously we... 
PROGENITOR CELLS | CD31(+) CELLS | ISCHEMIA/REPERFUSION INJURY | ACTIVATION | ANGIOGENESIS | MULTIDISCIPLINARY SCIENCES | BONE-MARROW | TISSUE | APELIN | TOPCARE-AMI | EXPRESSION | Up-Regulation | Capillaries - pathology | Receptors, Notch - metabolism | Cardiomegaly - pathology | Angiopoietin-1 - metabolism | Antigens, CD - metabolism | Peptides - metabolism | Serrate-Jagged Proteins | Bone Marrow - metabolism | Myocardium - metabolism | Endomyocardial Fibrosis - complications | Myocardial Infarction - physiopathology | Capillaries - metabolism | Apelin | Jagged-1 Protein | Capillaries - physiopathology | Signal Transduction | Cardiomegaly - physiopathology | Myocardial Infarction - metabolism | Receptors, CXCR4 - metabolism | Chemokine CXCL12 - metabolism | Hematopoietic Stem Cells - cytology | Endomyocardial Fibrosis - metabolism | Mice | Diabetes Mellitus, Type 2 - pathology | Cardiomegaly - metabolism | Cell Movement | Actins - metabolism | Glycoproteins - metabolism | Diabetes Mellitus, Type 2 - metabolism | Proto-Oncogene Proteins c-kit - metabolism | Intercellular Signaling Peptides and Proteins - metabolism | Platelet Endothelial Cell Adhesion Molecule-1 - metabolism | Myocardial Infarction - pathology | Endomyocardial Fibrosis - pathology | Membrane Proteins - metabolism | Heart Function Tests | Diabetes Mellitus, Type 2 - complications | Calcium-Binding Proteins - metabolism | Myocardium - pathology | Adipokines | AC133 Antigen | Cardiomegaly - complications | Myocardial Infarction - complications | Animals | Diabetes Mellitus, Type 2 - physiopathology | Bone Marrow - pathology | Receptor, Notch3 | Endomyocardial Fibrosis - physiopathology | Apoptosis | Advertising executives | Vascular endothelial growth factor | Adenoviruses | Heart attack | Myocardial infarction | Heart | Diabetic retinopathy | Heart attacks | Angiopoietin | Recovery of function | Smooth muscle | Cardiovascular disease | Recruitment | Angiogenesis | Toxicology | Cell growth | Ischemia | Data recovery | Rodents | Bone marrow | Repair | Heart diseases | Immunoglobulins | Diabetes mellitus | Coronary artery | Muscles | Pharmacology | c-Kit protein | Hemopoiesis | Studies | Ostomy | Coronary vessels | Fibrosis | Stem cells | Myocardium | Infarction | Diabetes | Laboratory animals
Journal Article
Circulation research, ISSN 1524-4571, 2013, Volume 113, Issue 5, pp. 603 - 616
Journal Article
American journal of physiology. Heart and circulatory physiology, ISSN 1522-1539, 2016, Volume 311, Issue 4, pp. H927 - H943
Although incidence and prevalence of prediabetes are increasing, little is known about its cardiac effects. Therefore, our aim was to investigate the effect of... 
Type 2 diabetes | Diabetic cardiomyopathy | Obesity | Reactive oxygen species | High-fat diet | METABOLIC SYNDROME | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | CONTRACTILE DYSFUNCTION | diabetic cardiomyopathy | LEFT-VENTRICULAR HYPERTROPHY | OXIDASE 4 NOX4 | DIABETES-MELLITUS | type 2 diabetes | high-fat diet | reactive oxygen species | CARDIOVASCULAR-DISEASE | ANIMAL-MODELS | IMPAIRED FASTING GLUCOSE | PERIPHERAL VASCULAR DISEASE | INDUCED CARDIAC-HYPERTROPHY | obesity | Body Composition | Mitochondria, Heart - ultrastructure | Prediabetic State - metabolism | Phosphorylation | Mitochondria, Heart - metabolism | Reactive Oxygen Species - metabolism | Diastole | Oxidative Stress | TOR Serine-Threonine Kinases - metabolism | Rats, Long-Evans | Male | Mitophagy | Autophagy | Mitochondrial Proteins - metabolism | Diet, High-Fat | Myocardium - metabolism | Membrane Proteins - metabolism | Adipose Tissue | Diabetes Mellitus, Experimental - metabolism | Real-Time Polymerase Chain Reaction | Calcium-Calmodulin-Dependent Protein Kinase Type 2 - metabolism | Diabetes Mellitus, Experimental - physiopathology | Myocardium - ultrastructure | Calcium-Binding Proteins - metabolism | Sarcolemma | Echocardiography | Heat-Shock Proteins - metabolism | Hypertrophy, Left Ventricular - metabolism | Cardiomegaly - physiopathology | Rats | Adipokines - metabolism | Microscopy, Electron | Diabetic Neuropathies | Ventricular Dysfunction, Left - physiopathology | Ventricular Dysfunction, Left - metabolism | Animals | Prediabetic State - physiopathology | Ventricular Pressure | Hypertrophy, Left Ventricular - physiopathology | Apoptosis | Cardiomegaly - metabolism | Physiological aspects | Oxidative stress | Diabetes | Health aspects | Call for Papers
Journal Article
Science (American Association for the Advancement of Science), ISSN 1095-9203, 2011, Volume 332, Issue 6028, pp. 458 - 461
Journal Article
The Journal of biological chemistry, ISSN 1083-351X, 2014, Volume 289, Issue 9, pp. 5914 - 5924
Journal Article
Nucleic Acids Research, ISSN 0305-1048, 11/2015, Volume 44, Issue 6, pp. 2538 - 2553
Journal Article
Aging cell, ISSN 1474-9718, 2017, Volume 16, Issue 5, pp. 976 - 987
Summary Aging is accompanied with unfavorable geometric and functional changes in the heart involving dysregulation of Akt and autophagy. This study examined... 
cardiac geometry | contractile function | autophagy | aging | Akt | RAPAMYCIN | PROTEIN-KINASE | DIASTOLIC DYSFUNCTION | CELL BIOLOGY | GERIATRICS & GERONTOLOGY | HYPERTROPHY | LONGEVITY | OVEREXPRESSION | INSULIN-RESISTANCE | CALORIC RESTRICTION | SENESCENCE | MITOCHONDRIAL INJURY | Adaptation, Physiological | Protein Kinases - metabolism | Sirtuin 1 - metabolism | Phosphorylation | Protein Kinases - genetics | Microtubule-Associated Proteins - genetics | Calcium - metabolism | Microtubule-Associated Proteins - metabolism | Autophagy-Related Protein 7 - metabolism | Proto-Oncogene Proteins c-akt - deficiency | Atrial Remodeling - drug effects | Myocardial Contraction - drug effects | Cardiomegaly - pathology | Male | Mitochondrial Proteins - genetics | Sirtuin 1 - genetics | Autophagy - drug effects | Heterocyclic Compounds, 4 or More Rings - pharmacology | Proto-Oncogene Proteins c-akt - genetics | Mitochondrial Proteins - metabolism | Myocardium - metabolism | Membrane Proteins - metabolism | Autophagy - genetics | Forkhead Box Protein O1 - metabolism | Sarcoplasmic Reticulum Calcium-Transporting ATPases - metabolism | Signal Transduction | Membrane Proteins - genetics | Gene Expression Regulation | Myocardium - pathology | Mitochondria - metabolism | Longevity - genetics | Mitochondria - pathology | Autophagy-Related Protein 7 - genetics | Sirolimus - pharmacology | Mice, Knockout | Animals | Cardiomegaly - prevention & control | Beclin-1 - genetics | Mice | Beclin-1 - metabolism | Cardiomegaly - genetics | Sarcoplasmic Reticulum Calcium-Transporting ATPases - genetics | Forkhead Box Protein O1 - genetics | Cardiomegaly - metabolism | Heart | Laws, regulations and rules | Ablation (Surgery) | AKT protein | Autophagy | Ca2+-transporting ATPase | Mitochondria | FOXO1 protein | Aging | Oxidation | Fura-2 | Acetylation | Heart diseases | Calcium (intracellular) | Echocardiography | AKT2 protein | Cardiomyocytes | Rapamycin | Insulin | Muscle contraction | BNIP3 protein | Life span | PTEN-induced putative kinase | Parkin protein | Intracellular | PTEN protein | Phagocytosis | Hypertrophy | Original
Journal Article