X
Search Filters
Format Format
Format Format
X
Sort by Item Count (A-Z)
Filter by Count
Journal Article (8797) 8797
Book Review (878) 878
Publication (744) 744
Book Chapter (24) 24
Magazine Article (19) 19
Conference Proceeding (13) 13
Book / eBook (7) 7
Dissertation (5) 5
Data Set (1) 1
more...
Subjects Subjects
Subjects Subjects
X
Sort by Item Count (A-Z)
Filter by Count
index medicus (8129) 8129
animals (5223) 5223
humans (4688) 4688
cardiomegaly - pathology (4637) 4637
male (4423) 4423
myocardium - pathology (2987) 2987
rats (2728) 2728
female (2495) 2495
cardiomegaly - physiopathology (2007) 2007
cardiomegaly - etiology (1982) 1982
cardiomegaly - metabolism (1913) 1913
cardiac & cardiovascular systems (1792) 1792
mice (1662) 1662
hypertrophy (1657) 1657
adult (1503) 1503
abridged index medicus (1434) 1434
middle aged (1420) 1420
heart (1329) 1329
myocardium - metabolism (1282) 1282
cardiomegaly - genetics (1094) 1094
disease models, animal (953) 953
cardiac hypertrophy (949) 949
peripheral vascular disease (937) 937
myocytes, cardiac - pathology (936) 936
aged (932) 932
heart-failure (922) 922
echocardiography (911) 911
electrocardiography (874) 874
myocytes, cardiac - metabolism (846) 846
heart ventricles - pathology (843) 843
heart failure (833) 833
cell biology (800) 800
fibrosis (786) 786
organ size (778) 778
cardiomegaly - complications (771) 771
hypertension (740) 740
cells, cultured (736) 736
rats, sprague-dawley (734) 734
cardiomegaly (686) 686
physiology (682) 682
cardiomegaly - chemically induced (644) 644
signal transduction (638) 638
mice, inbred c57bl (627) 627
blood pressure (626) 626
cardiomegaly - prevention & control (621) 621
expression (608) 608
cardiomegaly - diagnosis (582) 582
heart enlargement (580) 580
mice, knockout (565) 565
adolescent (562) 562
cardiomegaly - diagnostic imaging (546) 546
time factors (544) 544
cardiac-hypertrophy (529) 529
hypertension - complications (518) 518
cardiomegaly - drug therapy (515) 515
gene expression (514) 514
rats, wistar (506) 506
myocytes, cardiac - drug effects (499) 499
pathology (493) 493
rodents (493) 493
mice, transgenic (485) 485
hypertension - physiopathology (482) 482
heart - physiopathology (479) 479
cardiomegaly - enzymology (475) 475
gene-expression (475) 475
heart failure - pathology (473) 473
apoptosis (466) 466
biochemistry & molecular biology (465) 465
child (445) 445
hypertension - pathology (445) 445
cardiovascular system (438) 438
cardiomyocytes (437) 437
physiological aspects (432) 432
hemodynamics (429) 429
activation (422) 422
pharmacology & pharmacy (421) 421
cardiomyopathy (414) 414
article (411) 411
blood pressure - drug effects (411) 411
pressure-overload (389) 389
oxidative stress (388) 388
phosphorylation (377) 377
infant (376) 376
left-ventricular hypertrophy (374) 374
heart diseases (365) 365
medicine, research & experimental (356) 356
research (356) 356
animals, newborn (351) 351
proteins (350) 350
heart failure - physiopathology (348) 348
heart - drug effects (339) 339
myocardial contraction (339) 339
rats, inbred strains (333) 333
myocardial-infarction (332) 332
hematology (330) 330
heart failure - etiology (328) 328
body weight (324) 324
myocardial infarction - pathology (324) 324
diagnosis, differential (323) 323
cardiovascular (319) 319
more...
Library Location Library Location
Language Language
Language Language
X
Sort by Item Count (A-Z)
Filter by Count
English (7765) 7765
Russian (262) 262
German (215) 215
French (139) 139
Italian (92) 92
Japanese (85) 85
Spanish (71) 71
Chinese (61) 61
Polish (36) 36
Portuguese (26) 26
Czech (21) 21
Slovak (13) 13
Romanian (9) 9
Bulgarian (7) 7
Hungarian (7) 7
Danish (4) 4
Swedish (4) 4
Ukrainian (3) 3
Dutch (2) 2
Croatian (1) 1
Finnish (1) 1
Lithuanian (1) 1
Norwegian (1) 1
Turkish (1) 1
more...
Publication Date Publication Date
Click on a bar to filter by decade
Slide to change publication date range


Circulation, ISSN 0009-7322, 02/2015, Volume 131, Issue 6, pp. 550 - 559
BACKGROUND—Characterization of myocardial structural changes in heart failure with preserved ejection fraction (HFpEF) has been hindered by the limited... 
heart failure, diastolic | endothelium | pathology | hypertrophy | fibrosis | autopsy | microvessels | PROGNOSTIC IMPACT | CARDIAC & CARDIOVASCULAR SYSTEMS | IDIOPATHIC DILATED CARDIOMYOPATHY | CARDIAC STRUCTURE | RESTING TENSION | LEFT-VENTRICLE | ACTIVE RELAXATION | DIASTOLIC DYSFUNCTION | FLOW RESERVE | HYPERTROPHIC CARDIOMYOPATHY | PERIPHERAL VASCULAR DISEASE | ARTERY-DISEASE | Immunohistochemistry | Heart - anatomy & histology | Humans | Heart Failure - physiopathology | Microvessels - pathology | Cardiomegaly - epidemiology | Cardiomegaly - pathology | Male | Reference Values | Cause of Death | Autopsy | Coronary Artery Disease - pathology | Aged, 80 and over | Electrocardiography | Female | Endomyocardial Fibrosis - pathology | Heart Failure - diagnosis | Heart Failure - epidemiology | Coronary Vessels - pathology | Echocardiography | Comorbidity | Endomyocardial Fibrosis - epidemiology | Organ Size | Myocardium - pathology | Heart Failure - pathology | Stroke Volume | Myocytes, Cardiac - pathology | Algorithms | Diabetes Mellitus - epidemiology | Aged | Coronary Artery Disease - epidemiology | Endomyocardial Fibrosis - physiopathology | Postmortem Changes | Heart failure | Analysis | Research | Cardiac output | Risk factors | Endothelium | Hypertrophy | Index Medicus | Abridged Index Medicus | coronary microvessel | diastolic heart failure
Journal Article
The American Journal of Pathology, ISSN 0002-9440, 2003, Volume 163, Issue 2, pp. 673 - 682
The platelet-derived growth factors are implicated in development of fibrotic reactions and disease in several organs. We have overexpressed platelet-derived... 
PROTEASE-ACTIVATED LIGAND | CELLS | LENS-SPECIFIC EXPRESSION | PDGF-A | ATRIAL | ALPHA-RECEPTOR | GENE-EXPRESSION | MICE | PATHOLOGY | FAILURE | BETA-RECEPTOR | Up-Regulation | Cardiomyopathy, Dilated - pathology | Cardiomegaly - pathology | Male | Platelet-Derived Growth Factor - genetics | Lymphokines | Coronary Vessels - metabolism | Myocardium - metabolism | Female | Fibroblasts - metabolism | Promoter Regions, Genetic | Coronary Vessels - pathology | Echocardiography | Receptor, Platelet-Derived Growth Factor alpha - metabolism | Mice, Inbred C57BL | Cardiomegaly - diagnostic imaging | Gene Expression Regulation | Mice, Transgenic | Myocardium - pathology | Fibroblasts - pathology | Cardiomyopathy, Dilated - metabolism | Myocytes, Cardiac - pathology | Cardiomyopathy, Dilated - diagnostic imaging | Platelet-Derived Growth Factor - metabolism | Animals | Receptor, Platelet-Derived Growth Factor alpha - genetics | Fibrosis | Biomarkers | Myocytes, Cardiac - metabolism | Mice | Cardiomegaly - metabolism | Index Medicus | Abridged Index Medicus | Regular | Promoter Regions (Genetics) | Mice; Transgenic | Medical and Health Sciences | Medicin och hälsovetenskap | Biological Markers | Myocardium/metabolism/pathology | Fibroblasts/metabolism/pathology | Mice; Inbred C57BL | Receptor; Platelet-Derived Growth Factor alpha/genetics/metabolism | Coronary Vessels/metabolism/pathology | Cardiomyopathy; Dilated/metabolism/pathology/ultrasonography | Myocytes; Cardiac/metabolism/pathology | Platelet-Derived Growth Factor/genetics/metabolism | Cardiomegaly/metabolism/pathology/ultrasonography
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 07/2014, Volume 124, Issue 7, pp. 2921 - 2934
Journal Article
Nature Communications, ISSN 2041-1723, 04/2015, Volume 6, Issue 1, pp. 6656 - 6656
Honokiol (HKL) is a natural biphenolic compound derived from the bark of magnolia trees with anti-inflammatory, anti-oxidative, anti-tumour and neuroprotective... 
METABOLISM | GENE | SIRT3-MEDIATED DEACETYLATION | PATHWAY | PROTEIN-KINASE | MULTIDISCIPLINARY SCIENCES | HOMOLOG | AKT | SIRTUINS | CALORIE RESTRICTION | CELL-DEATH | Mitochondria - enzymology | Fibroblasts - enzymology | Superoxide Dismutase - genetics | Reactive Oxygen Species - metabolism | Sirtuin 3 - metabolism | Myofibroblasts - enzymology | Superoxide Dismutase - antagonists & inhibitors | Cardiomegaly - pathology | Isoproterenol | Lignans - pharmacology | Myocytes, Cardiac - enzymology | Phenylephrine - pharmacology | Biphenyl Compounds - pharmacology | Membrane Proteins - metabolism | Superoxide Dismutase - metabolism | Myofibroblasts - pathology | Sirtuin 3 - genetics | Signal Transduction | Membrane Proteins - genetics | Carrier Proteins - antagonists & inhibitors | Gene Expression Regulation | Adenosine Triphosphatases - metabolism | Myocardium - pathology | Cardiotonic Agents - pharmacology | Mitochondria - drug effects | Mitochondria - pathology | Fibroblasts - pathology | Myofibroblasts - drug effects | Adenosine Triphosphatases - antagonists & inhibitors | Carrier Proteins - genetics | Myocytes, Cardiac - pathology | Acetylation - drug effects | Myocardium - enzymology | Reactive Oxygen Species - antagonists & inhibitors | Animals | Cardiomegaly - prevention & control | Carrier Proteins - metabolism | Myocytes, Cardiac - drug effects | Membrane Proteins - antagonists & inhibitors | Cell Differentiation - drug effects | Fibroblasts - drug effects | Cardiomegaly - chemically induced | Adenosine Triphosphatases - genetics | Cell Proliferation - drug effects | Mice | Enzyme Activation | Primary Cell Culture | Cardiomegaly - genetics | Index Medicus
Journal Article
Journal Article
Nature, ISSN 0028-0836, 2014, Volume 514, Issue 7520, pp. 102 - 106
The role of long noncoding RNA(lncRNA) in adult hearts is unknown; also unclear is how lncRNA modulates nucleosome remodelling. An estimated 70% of mouse genes... 
MORPHOGENESIS | DOMAIN | CHROMATIN | GENE | RECOGNITION | STRUCTURAL BASIS | MULTIDISCIPLINARY SCIENCES | CARDIAC-HYPERTROPHY | IN-VIVO | TRANSCRIPTION | GROWTH | Chromatin - metabolism | Transcription Factors - chemistry | Humans | Myosin Heavy Chains - genetics | Cardiomegaly - pathology | Cardiomyopathies - prevention & control | Heart Failure - prevention & control | Cardiomyopathies - genetics | Myocardium - metabolism | Nuclear Proteins - genetics | DNA Helicases - genetics | Protein Structure, Tertiary | DNA Helicases - chemistry | Cardiac Myosins - genetics | Chromatin Assembly and Disassembly | Cardiomyopathies - pathology | Heart Failure - genetics | Histone Deacetylases - metabolism | Myocardium - pathology | Nuclear Proteins - metabolism | Transcription Factors - antagonists & inhibitors | RNA, Long Noncoding - genetics | Heart Failure - pathology | Transcription Factors - genetics | Nuclear Proteins - chemistry | Organ Specificity | Transcription Factors - metabolism | DNA Helicases - metabolism | Feedback, Physiological | Poly(ADP-ribose) Polymerases - metabolism | Animals | Cardiomegaly - prevention & control | Nuclear Proteins - antagonists & inhibitors | Protein Binding | Mice | DNA Helicases - antagonists & inhibitors | Poly (ADP-Ribose) Polymerase-1 | Cardiomegaly - genetics | RNA, Long Noncoding - antagonists & inhibitors | Chromatin - genetics | RNA, Long Noncoding - metabolism | Heart | Physiological aspects | Genetic aspects | RNA | Hypertrophy | Pathology | Cardiomyocytes | RNA polymerase | Ribonucleic acid--RNA | Cardiomyopathy | Rodents | Index Medicus
Journal Article
Circulation, ISSN 0009-7322, 08/2013, Volume 128, Issue 9, pp. 970 - 981
BACKGROUND—Sarcoplasmic reticulum (SR) Ca leak through ryanodine receptor type 2 (RyR2) dysfunction is of major pathophysiological relevance in human heart... 
Heart failure | Calcium calmodulin-dependent protein kinase type 2 | Sarcoplasmic reticulum | Protein kinases | Hypertrophy | heart failure | RYANODINE RECEPTOR PHOSPHORYLATION | CARDIAC & CARDIOVASCULAR SYSTEMS | CONTRACTILITY | hypertrophy | protein kinases | VENTRICULAR MYOCYTES | RELEASE | FAILING HEARTS | calcium-calmodulin-dependent protein kinase type 2 | sarcoplasmic reticulum | ATRIAL-FIBRILLATION | CAMKII | CALCIUM SPARKS | PKA PHOSPHORYLATION | PERIPHERAL VASCULAR DISEASE | HEART-FAILURE PROGRESSION | Phosphorylation | Calcium - metabolism | Humans | Middle Aged | Homeostasis | Cardiomegaly - pathology | Male | Ryanodine Receptor Calcium Release Channel - metabolism | Calcium-Calmodulin-Dependent Protein Kinase Type 2 - drug effects | Case-Control Studies | Myocardium - metabolism | Female | Sarcoplasmic Reticulum - metabolism | Cyclic AMP-Dependent Protein Kinases - antagonists & inhibitors | Cyclic AMP-Dependent Protein Kinases - drug effects | Calcium-Calmodulin-Dependent Protein Kinase Type 2 - metabolism | Cyclic AMP-Dependent Protein Kinases - metabolism | Enzyme Inhibitors - pharmacology | Myocardium - pathology | Heart Failure - metabolism | Heart Failure - pathology | Calcium-Calmodulin-Dependent Protein Kinase Type 2 - antagonists & inhibitors | Disease Progression | Aged | Cardiomegaly - metabolism | Physiological aspects | Calcium channels | Research | Index Medicus | Abridged Index Medicus
Journal Article
Journal Article
Journal of the American College of Cardiology, ISSN 0735-1097, 06/2014, Volume 63, Issue 24, pp. 2734 - 2741
Objectives This study sought to investigate the effect of endothelial dysfunction on the development of cardiac hypertrophy and fibrosis. Background... 
Cardiovascular | Internal Medicine | endothelial-mesenchymal transition | diastolic dysfunction | endothelium | NADPH (nicotinamide adenine dinucleotide phosphate) oxidase | angiotensin II | CELLS | OXIDATIVE STRESS | CARDIAC & CARDIOVASCULAR SYSTEMS | ANGIOGENESIS | HEART-FAILURE | ATHEROSCLEROSIS | ANGIOTENSIN-II | PATHOPHYSIOLOGY | HYPERTROPHY | MICE | CONTRIBUTES | Heart Failure, Diastolic - genetics | Mesenchymal Stromal Cells - enzymology | Humans | Cardiomegaly - pathology | Male | Endothelium, Vascular - enzymology | Membrane Glycoproteins - physiology | Ventricular Dysfunction, Left - genetics | Ventricular Dysfunction, Left - pathology | NADPH Oxidases - genetics | Ventricular Dysfunction, Left - enzymology | Inflammation Mediators - physiology | Fibrosis - genetics | Fibrosis - enzymology | Cells, Cultured | Heart Failure, Diastolic - enzymology | Mice, Transgenic | Cardiomegaly - enzymology | Heart Failure, Diastolic - pathology | NADPH Oxidase 2 | Membrane Glycoproteins - genetics | Animals | Endothelium, Vascular - pathology | Mice | Fibrosis - pathology | Mesenchymal Stromal Cells - pathology | Cardiomegaly - genetics | NADPH Oxidases - physiology | Oxidases | Heart | Fibrosis | Stem cells | Endothelium | Hypertension | Heart failure | Genotype & phenotype | Stroke | Heart attacks | Pathogenesis | Rodents | Cardiomyocytes | Variance analysis | Index Medicus | Abridged Index Medicus
Journal Article
Nature, ISSN 0028-0836, 05/2012, Volume 484, Issue 7397, pp. 251 - 255
Journal Article