X
Search Filters
Format Format
Format Format
X
Sort by Item Count (A-Z)
Filter by Count
Journal Article (8682) 8682
Magazine Article (19) 19
Conference Proceeding (9) 9
Book / eBook (7) 7
Book Chapter (2) 2
Publication (2) 2
Book Review (1) 1
Web Resource (1) 1
more...
Subjects Subjects
Subjects Subjects
X
Sort by Item Count (A-Z)
Filter by Count
cardiomegaly - physiopathology (5967) 5967
humans (5225) 5225
animals (4549) 4549
male (4484) 4484
female (2701) 2701
rats (2212) 2212
cardiomegaly - etiology (2155) 2155
adult (2111) 2111
middle aged (2048) 2048
cardiac & cardiovascular systems (1826) 1826
hypertension - physiopathology (1577) 1577
cardiomegaly - pathology (1554) 1554
electrocardiography (1548) 1548
heart - physiopathology (1435) 1435
echocardiography (1392) 1392
blood pressure (1251) 1251
myocardium - pathology (1207) 1207
hypertrophy (1198) 1198
aged (1125) 1125
cardiomegaly - metabolism (1093) 1093
myocardium - metabolism (1076) 1076
heart ventricles - physiopathology (1061) 1061
peripheral vascular disease (1022) 1022
mice (1006) 1006
hemodynamics (966) 966
myocardial contraction (934) 934
hypertension - complications (885) 885
heart failure - physiopathology (872) 872
cardiomegaly - diagnosis (845) 845
disease models, animal (835) 835
cardiomegaly - complications (816) 816
heart (812) 812
adolescent (712) 712
time factors (698) 698
physiology (657) 657
hypertension (654) 654
cardiac hypertrophy (630) 630
heart failure (624) 624
cardiomegaly - diagnostic imaging (607) 607
cardiomegaly - genetics (566) 566
heart-failure (565) 565
blood pressure - drug effects (523) 523
cardiomegaly - drug therapy (514) 514
organ size (503) 503
heart rate (498) 498
dogs (494) 494
cardiovascular system (449) 449
fibrosis (444) 444
rats, sprague-dawley (436) 436
child (432) 432
myocardial infarction - physiopathology (432) 432
cardiac output (429) 429
heart - physiology (424) 424
expression (414) 414
cardiomegaly - prevention & control (408) 408
coronary circulation (404) 404
rats, wistar (401) 401
cardiac-hypertrophy (391) 391
left-ventricular hypertrophy (387) 387
pharmacology & pharmacy (387) 387
signal transduction (384) 384
coronary disease - physiopathology (383) 383
rats, inbred strains (383) 383
heart enlargement (382) 382
mice, inbred c57bl (378) 378
cell biology (373) 373
hypertension - drug therapy (370) 370
heart ventricles - pathology (362) 362
heart - drug effects (360) 360
mice, knockout (360) 360
myocytes, cardiac - metabolism (359) 359
heart ventricles (356) 356
rats, inbred shr (354) 354
physiological aspects (347) 347
stroke volume (339) 339
cardiac catheterization (338) 338
cells, cultured (333) 333
heart failure - etiology (330) 330
hematology (324) 324
cardiomegaly - chemically induced (323) 323
mice, transgenic (321) 321
myocytes, cardiac - pathology (317) 317
diastole (308) 308
pressure (304) 304
body weight (296) 296
gene-expression (292) 292
gene expression (291) 291
cardiomyopathy (286) 286
heart conduction system - physiopathology (281) 281
chronic disease (275) 275
calcium - metabolism (274) 274
myocardial contraction - drug effects (271) 271
cardiomegaly (262) 262
blood pressure - physiology (260) 260
cardiac volume (260) 260
cardiomegaly - enzymology (260) 260
aortic valve stenosis - physiopathology (257) 257
myocardial contraction - physiology (257) 257
risk factors (257) 257
activation (254) 254
more...
Library Location Library Location
Language Language
Language Language
X
Sort by Item Count (A-Z)
Filter by Count
English (7282) 7282
Russian (366) 366
German (272) 272
Japanese (249) 249
French (230) 230
Italian (196) 196
Spanish (95) 95
Chinese (57) 57
Polish (46) 46
Portuguese (46) 46
Czech (21) 21
Slovak (21) 21
Bulgarian (20) 20
Romanian (18) 18
Hungarian (6) 6
Dutch (4) 4
Serbian (4) 4
Ukrainian (4) 4
Danish (3) 3
Hebrew (3) 3
Croatian (2) 2
Norwegian (2) 2
Swedish (2) 2
Lithuanian (1) 1
Macedonian (1) 1
more...
Publication Date Publication Date
Click on a bar to filter by decade
Slide to change publication date range


European heart journal, ISSN 1522-9645, 2015, Volume 36, Issue 17, pp. 1049 - 1058
Journal Article
Kidney international, ISSN 0085-2538, 2018, Volume 93, Issue 1, pp. 147 - 158
We examined activin receptor type IIA (ActRIIA) activation in chronic kidney disease (CKD) by signal analysis and inhibition in mice with Alport syndrome using... 
cardiovascular disease | chronic kidney disease | bone | fibrosis | mineral metabolism | vascular calcification | TGF-BETA | CARDIOVASCULAR EVENTS | RENAL FIBROSIS | ARTERIAL CALCIFICATION | BONE MORPHOGENETIC PROTEIN-7 | GROWTH-FACTOR 23 | UROLOGY & NEPHROLOGY | SMOOTH-MUSCLE-CELLS | LINKED ALPORT-SYNDROME | OSTEOCLAST DIFFERENTIATION | ATHEROSCLEROTIC LESION | Kidney - pathology | Recombinant Fusion Proteins - pharmacology | Blood Vessels - metabolism | Chronic Kidney Disease-Mineral and Bone Disorder - metabolism | Core Binding Factor Alpha 1 Subunit - metabolism | Chronic Kidney Disease-Mineral and Bone Disorder - genetics | Vascular Calcification - metabolism | Kidney - metabolism | Bone Resorption - genetics | Bone and Bones - metabolism | Myocardium - metabolism | Renal Insufficiency, Chronic - genetics | Bone Resorption - metabolism | Disease Models, Animal | Bone Resorption - physiopathology | Glomerular Filtration Rate | Signal Transduction | Smad2 Protein - metabolism | Cardiomegaly - physiopathology | Nephritis, Hereditary - genetics | Renal Insufficiency, Chronic - physiopathology | Mice, Knockout | Renal Insufficiency, Chronic - prevention & control | Cardiomegaly - prevention & control | Nephritis, Hereditary - metabolism | Collagen Type IV - deficiency | Activin Receptors, Type II - genetics | Fibrosis | Sp7 Transcription Factor - metabolism | Collagen Type IV - genetics | Cardiomegaly - metabolism | Activin Receptors, Type II - metabolism | Bone and Bones - pathology | Phosphorylation | Vascular Remodeling | Blood Vessels - pathology | Vascular Calcification - genetics | Actins - metabolism | Chronic Kidney Disease-Mineral and Bone Disorder - physiopathology | Activin Receptors, Type II - antagonists & inhibitors | Blood Vessels - physiopathology | Renal Insufficiency, Chronic - metabolism | Ventricular Remodeling | Chronic Kidney Disease-Mineral and Bone Disorder - prevention & control | Muscle Proteins - metabolism | Vascular Calcification - physiopathology | Microfilament Proteins - metabolism | Nephritis, Hereditary - physiopathology | Kidney - physiopathology | Vascular Calcification - prevention & control | Myocardium - pathology | Bone Resorption - prevention & control | Bone and Bones - physiopathology | Animals | Nephritis, Hereditary - drug therapy | Cardiomegaly - genetics | Bone Remodeling | Chronic kidney disease
Journal Article
Circulation. Heart failure, ISSN 1941-3297, 2015, Volume 8, Issue 4, pp. 788 - 798
Background-Heart failure in diabetics is associated with cardiac hypertrophy, fibrosis and diastolic dysfunction. Activation of transforming growth... 
Obesity | TGF-β | Diabetic cardiomyopathies | Diabetes mellitus | Fibrosis | PRESSURE-OVERLOAD | OXIDATIVE STRESS | CARDIAC & CARDIOVASCULAR SYSTEMS | MYOCARDIAL-INFARCTION | HEART-FAILURE | CARDIOMYOPATHY | diabetes mellitus | BETA | TGF-beta | INFLAMMATION | GROWTH | diabetic cardiomyopathies | fibrosis | FIBROBLAST PHENOTYPE | ANEURYSM | obesity | Cardiomegaly - pathology | Male | Aorta - metabolism | Diabetic Cardiomyopathies - physiopathology | Time Factors | Ventricular Dysfunction, Left - genetics | Myocardium - metabolism | Aortic Aneurysm - pathology | Dilatation, Pathologic | Disease Models, Animal | Aortic Rupture - physiopathology | Signal Transduction | Obesity - complications | Cardiomegaly - physiopathology | Obesity - physiopathology | Mice, Knockout | Aorta - pathology | Ventricular Dysfunction, Left - metabolism | Aortic Aneurysm - genetics | Cardiomegaly - metabolism | Vascular Remodeling | Diabetic Cardiomyopathies - metabolism | Aortic Aneurysm - metabolism | Diabetic Cardiomyopathies - etiology | Cardiomegaly - etiology | Smad3 Protein - metabolism | Aortic Aneurysm - etiology | Aortic Rupture - metabolism | Obesity - genetics | Smad3 Protein - deficiency | Ventricular Remodeling | Ventricular Dysfunction, Left - pathology | Female | Aorta - physiopathology | Aortic Rupture - etiology | Mice, Inbred C57BL | Aortic Aneurysm - physiopathology | Ventricular Dysfunction, Left - etiology | Aortic Rupture - pathology | Myocardium - pathology | Ventricular Dysfunction, Left - physiopathology | Obesity - metabolism | Diabetic Cardiomyopathies - genetics | Animals | Diabetic Cardiomyopathies - pathology | Matrix Metalloproteinases - metabolism | Cardiomegaly - genetics | Transforming Growth Factor beta - metabolism | diabetic cardiomyopathy | cardiac remodeling
Journal Article
Hypertension (Dallas, Tex. 1979), ISSN 1524-4563, 2014, Volume 64, Issue 4, pp. 745 - 755
Journal Article
Journal Article
Science (American Association for the Advancement of Science), ISSN 1095-9203, 2007, Volume 316, Issue 5824, pp. 575 - 579
The heart responds to diverse forms of stress by hypertrophic growth accompanied by fibrosis and eventual diminution of contractility, which results from... 
Heart | Untranslated regions | Up regulation | Messenger RNA | MicroRNA | Transgenic animals | Fibrosis | Mice | Gene expression regulation | Research Articles | Gene expression | HYPERTROPHY | MULTIDISCIPLINARY SCIENCES | CARDIOMYOPATHY | PHENOTYPE | PROLIFERATION | DIFFERENTIATION | THYROID-HORMONE RECEPTOR | HUMAN HEART | MYOSIN HEAVY-CHAIN | HYPOTHYROIDISM | TRANSGENIC MICE | Up-Regulation | Oligonucleotide Array Sequence Analysis | Hypothyroidism - physiopathology | Myosin Heavy Chains - genetics | Cardiomegaly - pathology | Triiodothyronine - metabolism | Gene Deletion | Myocardium - metabolism | Mediator Complex | Cardiac Myosins - metabolism | Heart - physiopathology | Myocardial Contraction | Signal Transduction | Heart Diseases - physiopathology | Introns | Cardiac Myosins - genetics | Gene Expression Regulation | Cardiomegaly - physiopathology | Rats | Mice, Transgenic | Myocardium - pathology | Stress, Physiological - physiopathology | Transcription Factors - genetics | Transcription Factors - metabolism | Myocytes, Cardiac - pathology | Animals | Ventricular Myosins - metabolism | MicroRNAs - genetics | MicroRNAs - physiology | Heart Diseases - genetics | Heart Diseases - pathology | Ventricular Myosins - genetics | Causes of | Research | Muscle contraction | Heart muscle | Proteins | Ribonucleic acid | Hormones | Ribonucleic acid--RNA | Stress
Journal Article
American Heart Journal, ISSN 0002-8703, 2008, Volume 156, Issue 6, pp. 1056 - 1064
Journal Article
Cardiovascular Pathology, ISSN 1054-8807, 2017, Volume 30, pp. 27 - 37
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 10/2014, Volume 307, Issue 8, pp. H1204 - H1215
In pulmonary hypertension (PH), right ventricular (RV) dysfunction and failure is the main determinant of a poor prognosis. We aimed to characterize RV... 
Right ventricular remodeling | Pressure-volume relationships | Echocardiography | SERCA2a | Aldosterone | Pulmonary hypertension model | pulmonary hypertension model | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | right ventricular remodeling | aldosterone | PERFORMANCE | echocardiography | PREDICTION | PORCINE MODEL | ARTERIAL-HYPERTENSION | CHRONIC PRESSURE-OVERLOAD | MAGNETIC-RESONANCE | PERIPHERAL VASCULAR DISEASE | pressure-volume relationships | RIGHT-HEART-FAILURE | STRAIN | MOLECULAR-MECHANISMS | Myocardial Contraction | Sarcoplasmic Reticulum Calcium-Transporting ATPases - metabolism | Fibrosis - physiopathology | Cardiomegaly - physiopathology | Hypertension, Pulmonary - physiopathology | Cardiomegaly - pathology | Hypertension, Pulmonary - metabolism | Pulmonary Artery - physiopathology | Ventricular Remodeling | Animals | Heart Ventricles - physiopathology | Swine | Endoplasmic Reticulum Stress | Ventricular Dysfunction, Right - physiopathology | Heart Ventricles - metabolism | Fibrosis - pathology | Hemodynamics | Sarcoplasmic Reticulum Calcium-Transporting ATPases - genetics | Hypertension, Pulmonary - pathology | Ventricular Dysfunction, Right - metabolism | Heart Ventricles - pathology | Pulmonary Artery - pathology | Ventricular Dysfunction, Right - pathology | Physiological aspects | Medical examination | Pulmonary hypertension | Heart ventricle, Right | Integrative Cardiovascular Physiology and Pathophysiology
Journal Article
Circulation Research, ISSN 0009-7330, 03/2013, Volume 112, Issue 7, pp. 1059 - 1072
Journal Article