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1. Full Text Effects of Intracoronary CD34+ Stem Cell Transplantation in Nonischemic Dilated Cardiomyopathy Patients: 5-Year Follow-Up
by Vrtovec, Bojan and Poglajen, Gregor and Lezaic, Luka and Sever, Matjaz and Domanovic, Dragoslav and Cernelc, Peter and Socan, Aljaz and Schrepfer, Sonja and Torre-Amione, Guillermo and Haddad, François and Wu, Joseph C
Circulation Research, ISSN 0009-7330, 01/2013, Volume 112, Issue 1, pp. 165 - 173
RATIONALE:CD34 transplantation in dilated cardiomyopathy was associated with short-term improvement in left ventricular ejection fraction and exercise... 
heart failure | CYTOKINES | CARDIAC & CARDIOVASCULAR SYSTEMS | CD34(+) cells | bone marrow cells | BONE-MARROW | HEART-FAILURE | granulocyte colony-stimulating factor mobilization | TRIAL | ENDOTHELIAL PROGENITOR CELLS | DISEASES | ACUTE MYOCARDIAL-INFARCTION | IMPROVEMENT | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | dilated cardiomyopathy | Multivariate Analysis | Stem Cells - immunology | Cardiomyopathy, Dilated - pathology | Antigens, CD34 - metabolism | Myocardium - immunology | Follow-Up Studies | Ventricular Function, Left | Cardiomyopathy, Dilated - surgery | Humans | Middle Aged | Tumor Necrosis Factor-alpha - blood | Male | Cause of Death | Recovery of Function | Cell Tracking | Myocardial Perfusion Imaging | Exercise Test | Time Factors | Interleukin-6 - blood | Peptide Fragments - blood | Cardiomyopathy, Dilated - diagnosis | Female | Slovenia | Stem Cell Transplantation - mortality | Cardiomyopathy, Dilated - mortality | Natriuretic Peptide, Brain - blood | Biomarkers - metabolism | Coronary Circulation | Echocardiography | Cardiomyopathy, Dilated - blood | Kaplan-Meier Estimate | Proportional Hazards Models | Myocardium - pathology | Treatment Outcome | California | Chi-Square Distribution | Stroke Volume | Cardiomyopathy, Dilated - physiopathology | Texas | Cardiomyopathy, Dilated - immunology | Exercise Tolerance | Cell Movement | Injections, Intra-Arterial | Stem Cell Transplantation - adverse effects
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2. Full Text Cardiac fibroblasts mediate IL-17A-driven inflammatory dilated cardiomyopathy
by Wu, Lei and Ong, SuFey and Talor, Monica V and Barin, Jobert G and Baldeviano, G.Christian and Kass, David A and Bedja, Djahida and Zhang, Hao and Sheikh, Asfandyar and Margolick, Joseph B and Iwakura, Yoichiro and Rose, Noel R and Čiháková, Daniela
Journal of Experimental Medicine, ISSN 0022-1007, 2014, Volume 211, Issue 7, pp. 1449 - 1464
Inflammatory dilated cardiomyopathy (DCMi) is a major cause of heart failure in individuals below the age of 40. We recently reported that IL-17A is required... 
MEDICINE, RESEARCH & EXPERIMENTAL | EXPERIMENTAL AUTOIMMUNE MYOCARDITIS | COLONY-STIMULATING FACTOR | GM-CSF | DENDRITIC CELL | IN-VIVO | DISEASE | MONOCYTES | INJURY | IMMUNOLOGY | MACROPHAGE HETEROGENEITY | ENDOMYOCARDIAL BIOPSY | Cardiomyopathy, Dilated - genetics | Cardiomyopathy, Dilated - pathology | Myocardium - immunology | Macrophages - pathology | Interleukin-17 - immunology | Neutrophils - immunology | Interleukin-17 - genetics | Myocardium - pathology | Monocytes - immunology | Fibroblasts - pathology | Neutrophil Infiltration - genetics | Granulocyte-Macrophage Colony-Stimulating Factor - genetics | Mice, Knockout | Animals | Granulocyte-Macrophage Colony-Stimulating Factor - immunology | Cardiomyopathy, Dilated - immunology | Monocytes - pathology | Fibroblasts - immunology | Mice | Mice, Inbred BALB C | Macrophages - immunology | Neutrophil Infiltration - immunology | Neutrophils - pathology
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3. Full Text Down-regulation of microRNA-451a facilitates the activation and proliferation of CD4+ T cells by targeting Myc in patients with dilated cardiomyopathy
by Zeng, Zhipeng and Wang, Ke and Li, Yuanyuan and Xia, Ni and Nie, Shaofang and Lv, Bingjie and Zhang, Min and Tu, Xin and Li, Qianqian and Tang, Tingting and Cheng, Xiang
Journal of Biological Chemistry, ISSN 0021-9258, 04/2017, Volume 292, Issue 14, pp. 6004 - 6013
CD4(+) T cells are abnormally activated in patients with dilated cardiomyopathy (DCM) and might be associated with the immunopathogenesis of the disease.... 
AUTOANTIBODIES | INTERLEUKIN-2 | cardiovascular disease | microRNA (miRNA) | CARDIOLOGY | BIOCHEMISTRY & MOLECULAR BIOLOGY | HEART-FAILURE | C-MYC | RECEPTOR | CLASSIFICATION | inflammation | CARDIAC-HYPERTROPHY | T helper cells | TUMOR-SUPPRESSOR | EXPRESSION | Myc (c-Myc) | Antigens, CD - immunology | Cardiomyopathy, Dilated - pathology | Cell Proliferation | Lymphocyte Activation | Humans | Proto-Oncogene Proteins c-myc - immunology | Male | CD4-Positive T-Lymphocytes - pathology | MicroRNAs - immunology | 5' Untranslated Regions - immunology | CD4-Positive T-Lymphocytes - immunology | Down-Regulation - immunology | Cardiomyopathy, Dilated - immunology | Female | Molecular Bases of Disease
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4. Full Text CD4+ CD25+ GARP+ regulatory T cells display a compromised suppressive function in patients with dilated cardiomyopathy
by Wei, Yuzhen and Yu, Kunwu and Wei, Hui and Su, Xin and Zhu, Ruirui and Shi, Huairui and Sun, Haitao and Luo, Quan and Xu, Wenbin and Xiao, Junhui and Zhong, Yucheng and Zeng, Qiutang
Immunology, ISSN 0019-2805, 07/2017, Volume 151, Issue 3, pp. 291 - 303
Summary Dilated cardiomyopathy (DCM) is a lethal inflammatory heart disease and closely connected with dysfunction of the immune system. Glycoprotein A... 
regulatory T cells | glycoprotein A repetitions predominant | immune system | dilated cardiomyopathy | HEART-FAILURE | SURFACE EXPRESSION | CLASSIFICATION | MYOCARDITIS | IMMUNOLOGY | LATENT TGF-BETA | IDENTIFICATION | SCIENTIFIC STATEMENT | EUROPEAN-SOCIETY | FOXP3 | GARP | Forkhead Transcription Factors - immunology | T-Lymphocytes, Regulatory - metabolism | Cell Proliferation | Coculture Techniques | Humans | Middle Aged | Transforming Growth Factor beta1 - metabolism | Male | Th1 Cells - immunology | Case-Control Studies | T-Lymphocytes, Regulatory - immunology | Th1 Cells - metabolism | Transforming Growth Factor beta1 - immunology | Th17 Cells - metabolism | Forkhead Transcription Factors - metabolism | Adult | Cardiomyopathy, Dilated - diagnosis | Female | Membrane Proteins - metabolism | Cytokines - immunology | Interleukin-2 Receptor alpha Subunit - immunology | Adaptive Immunity | Cytokines - metabolism | Signal Transduction | Lymphocyte Activation | Cells, Cultured | Cell Communication | Membrane Proteins - immunology | CD4 Lymphocyte Count | Cardiomyopathy, Dilated - metabolism | Phenotype | Interleukin-2 Receptor alpha Subunit - metabolism | Cardiomyopathy, Dilated - immunology | Th17 Cells - immunology | Care and treatment | Interleukins | Bone morphogenetic proteins | Interferon | Biological response modifiers | T cells | Transforming growth factors | Cardiomyopathy, Dilated | Original
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5. Full Text Inflammatory Cardiomyopathic Syndromes
by Trachtenberg, Barry H and Hare, Joshua M
Circulation Research, ISSN 0009-7330, 09/2017, Volume 121, Issue 7, pp. 803 - 818
Inflammatory activation occurs in nearly all forms of myocardial injury. In contrast, inflammatory cardiomyopathies refer to a diverse group of disorders in... 
Heart failure | Myocarditis | Cytokines | Sarcoidosis | Gene expression | heart failure | SYSTEMIC-LUPUS-ERYTHEMATOSUS | CARDIAC & CARDIOVASCULAR SYSTEMS | IDIOPATHIC DILATED CARDIOMYOPATHY | myocarditis | GIANT-CELL MYOCARDITIS | VENTRICULAR ENDOMYOCARDIAL BIOPSY | sarcoidosis | NECROSIS-FACTOR-ALPHA | IMMUNOSUPPRESSIVE THERAPY | POLYMERASE-CHAIN-REACTION | VIRAL GENOMES | PERIPHERAL VASCULAR DISEASE | cytokines | HEMATOLOGY | gene expression | ANTI-HEART AUTOANTIBODIES | FULMINANT MYOCARDITIS | Autoimmunity | Chagas Cardiomyopathy - immunology | Virus Diseases - epidemiology | Myocardium - immunology | Prognosis | Humans | Immunosuppressive Agents - therapeutic use | Myocarditis - drug therapy | Hypersensitivity - immunology | Myocarditis - diagnosis | Sarcoidosis - epidemiology | Molecular Diagnostic Techniques | Myocarditis - epidemiology | Sarcoidosis - immunology | Cardiomyopathies - diagnosis | Chagas Cardiomyopathy - epidemiology | Myocarditis - immunology | Cardiomyopathies - immunology | Cardiomyopathies - drug therapy | Inflammation Mediators - immunology | Virus Diseases - immunology | Antiviral Agents - therapeutic use | Cardiomyopathy, Dilated - epidemiology | Risk Factors | Myocardium - pathology | Cardiomyopathies - epidemiology | Animals | Biopsy | Cardiac Imaging Techniques | Cardiomyopathy, Dilated - immunology | Fibrosis | Hypersensitivity - epidemiology | Immunohistochemistry | Immunosuppression | Dilated cardiomyopathy | Cardiomyopathy | Clinical trials | Inflammation | Chagas' disease | Heart diseases
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6. Full Text Cooperation of Th1 and Th17 cells determines transition from autoimmune myocarditis to dilated cardiomyopathy
by Nindl, Veronika and Maier, Reinhard and Ratering, David and De Giuli, Rita and Züst, Roland and Thiel, Volker and Scandella, Elke and Di Padova, Franco and Kopf, Manfred and Rudin, Markus and Rülicke, Thomas and Ludewig, Burkhard
European Journal of Immunology, ISSN 0014-2980, 09/2012, Volume 42, Issue 9, pp. 2311 - 2321
Myocarditis is a potentially lethal inflammatory heart disease of children and young adults that frequently leads to dilated cardiomyopathy (DCM). Since... 
Myocarditis | T helper (Th) cells | IL‐17A | Dilated cardiomyopathy | IFN‐γ | IFN-γ | IL-17A | YOUNG-ADULTS | INTERFERON-GAMMA RECEPTOR | NOD MICE | DENDRITIC CELLS | CARDIOVASCULAR MAGNETIC-RESONANCE | IFN-GAMMA | HEART-FAILURE | IMMUNOLOGY | IFN | INTERLEUKIN-6-DEFICIENT MICE | T-CELLS | TRANSGENIC MICE | Inflammation - pathology | Cardiomyopathy, Dilated - pathology | Autoimmune Diseases - immunology | Interleukin-17 - immunology | Heart Ventricles - immunology | Male | Mice, Transgenic | Inflammation - immunology | Th1 Cells - immunology | Animals | Autoantigens - immunology | Interferon-gamma - immunology | Cardiomyopathy, Dilated - immunology | Th17 Cells - immunology | Receptors, Antigen, T-Cell - immunology | Mice | Mice, Inbred BALB C | Ventricular Remodeling - immunology | Myocarditis - pathology | Autoimmune Diseases - pathology | Myocarditis - immunology | Heart Ventricles - pathology | Disease Models, Animal | Children | T cells | Cardiomyopathy, Dilated | Diseases | Transgenic animals | Cardiomyopathy | Rodents | Cooperation
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7. Full Text Autoimmune Dilated Cardiomyopathy in PD-1 Receptor-Deficient Mice
by Hiroyuki Nishimura and Taku Okazaki and Yoshimasa Tanaka and Kazuki Nakatani and Masatake Hara and Akira Matsumori and Shigetake Sasayama and Akira Mizoguchi and Hiroshi Hiai and Nagahiro Minato and Tasuku Honjo
Science, ISSN 0036-8075, 1/2001, Volume 291, Issue 5502, pp. 319 - 322
Dilated cardiomyopathy is a severe pathology of the heart with poorly understood etiology. Disruption of the gene encoding the negative immunoregulatory... 
Heart | Antigens | Autoantibodies | Dilated cardiomyopathy | Neurons | Myocardium | Antibodies | Cardiomyopathies | Reports | Autoimmune diseases | Heart diseases | ANTIBODIES | MULTIDISCIPLINARY SCIENCES | DISEASES | Complement C3 - analysis | Autoimmune Diseases - physiopathology | Cardiomyopathy, Dilated - pathology | Molecular Weight | Myocardium - immunology | Immunoglobulin G - blood | Autoantibodies - blood | Antigens, Surface - physiology | Autoantibodies - analysis | Immunoglobulin G - analysis | Autoimmune Diseases - pathology | Heart Failure - etiology | Echocardiography | Antigens, Surface - genetics | Autoimmune Diseases - immunology | Membrane Proteins - immunology | Myocardium - pathology | Autoantigens - chemistry | Animals | Apoptosis Regulatory Proteins | Cardiomyopathy, Dilated - physiopathology | Membrane Proteins - chemistry | Autoantigens - immunology | Cardiomyopathy, Dilated - immunology | Programmed Cell Death 1 Receptor | Mice | Mice, Inbred BALB C | Causes of | Research | Cardiomyopathy, Dilated | Cardiovascular disease | Rodents | Genes
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8. Full Text TNF-α-Secreting B Cells Contribute to Myocardial Fibrosis in Dilated Cardiomyopathy
by Yu, Miao and Wen, Shuang and Wang, Min and Liang, Wei and Li, Huan-Huan and Long, Qi and Guo, He-Ping and Liao, Yu-Hua and Yuan, Jing
Journal of Clinical Immunology, ISSN 0271-9142, 7/2013, Volume 33, Issue 5, pp. 1002 - 1008
Excessive inflammation responses mediated by CD4+ T cells contributes to myocardial fibrosis in dilated cardiomyopathy (DCM) resulting from viral myocarditis.... 
Medical Microbiology | Biomedicine | Immunology | TNF-α | inflammation | Infectious Diseases | Internal Medicine | B cells | myocardial fibrosis | EFFECTOR | CYTOKINES | THERAPY | TNF-alpha | HEART-FAILURE | NECROSIS-FACTOR-ALPHA | IMMUNOLOGY | Cell Growth Processes - immunology | Inflammation - pathology | Cardiomyopathy, Dilated - pathology | Myocardium - immunology | Humans | Middle Aged | Tumor Necrosis Factor-alpha - secretion | Cardiomyopathies - pathology | Male | Myocardium - pathology | Inflammation - immunology | Collagen Type III - immunology | B-Lymphocytes - immunology | B-Lymphocytes - secretion | Cardiomyopathy, Dilated - immunology | Fibrosis - immunology | Tumor Necrosis Factor-alpha - immunology | Adult | Female | B-Lymphocytes - pathology | Fibrosis - pathology | Cardiomyopathies - immunology
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9. Full Text Autoimmune Mechanisms Underlying Dilated Cardiomyopathy
by Yoshikawa, Tsutomu and Baba, Akiyasu and Nagatomo, Yuji
Circulation Journal, ISSN 1346-9843, 2009, Volume 73, Issue 4, pp. 602 - 607
Autoimmune abnormalities, as well as viral infection and genetic abnormalities, appear to be major predisposing factors for dilated cardiomyopathy (DCM).... 
Antibodies | Receptors | Adrenergic | Congestive heart failure | Beta | IMMUNOADSORPTION | ARRHYTHMIAS | CARDIAC & CARDIOVASCULAR SYSTEMS | 2ND EXTRACELLULAR LOOP | SUDDEN-DEATH | HEART-FAILURE | BETA-ADRENERGIC RECEPTOR AUTOANTIBODIES | NA-K-ATPASE | UPSTREAM TARGETS | HYPERTROPHIC CARDIOMYOPATHY | CARDIAC TROPONIN-I | Autoimmunity | Myocytes, Cardiac - immunology | Humans | Autoimmune Diseases - immunology | Troponin I - immunology | Autoantibodies - immunology | Animals | Cardiomyopathy, Dilated - immunology | Heart Failure - immunology | Atrial Fibrillation - immunology | Calcium Channels, L-Type - immunology | Immunity, Cellular | Myocarditis - immunology
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10. Full Text Intracoronary allogeneic cardiosphere‐derived stem cells are safe for use in dogs with dilated cardiomyopathy
by Hensley, Michael Taylor and Tang, Junnan and Woodruff, Kathleen and Defrancesco, Teresa and Tou, Sandra and Williams, Christina M and Breen, Mathew and Meurs, Kathryn and Keene, Bruce and Cheng, Ke
Journal of Cellular and Molecular Medicine, ISSN 1582-1838, 08/2017, Volume 21, Issue 8, pp. 1503 - 1512
Cardiosphere‐derived cells (CDCs) have been shown to reduce scar size and increase viable myocardium in human patients with mild/moderate myocardial... 
dogs | cardiosphere‐derived cells | stem cell therapy | allogeneic | dilated cardiomyopathy | cardiosphere-derived cells | MEDICINE, RESEARCH & EXPERIMENTAL | MYOCARDIAL-INFARCTION | EFFICACY | RATS | MODEL | DOBERMAN-PINSCHER | PROBES | CELL BIOLOGY | THERAPY | DISEASE | Stem Cells - immunology | Cardiomyopathy, Dilated - pathology | Myocardium - immunology | Spheroids, Cellular - transplantation | Endoglin - immunology | Humans | Male | Stem Cells - cytology | Proto-Oncogene Proteins c-kit - immunology | Spheroids, Cellular - cytology | Transplantation, Homologous | Stem Cell Transplantation | Thy-1 Antigens - genetics | Endoglin - genetics | Female | Proto-Oncogene Proteins c-kit - genetics | Biomarkers - metabolism | Gene Expression | Echocardiography | Thy-1 Antigens - immunology | Myocardium - pathology | Coronary Vessels - immunology | Spheroids, Cellular - immunology | Cardiomyopathy, Dilated - diagnostic imaging | Animals | Coronary Vessels - cytology | Cardiomyopathy, Dilated - immunology | Dogs | Cardiomyopathy, Dilated - therapy | Transplantation | Analysis | Cardiac patients | Cardiomyopathy, Dilated | Stem cells | Myocardial infarction | Heart | Animal models | Cardiomyopathy | Vessels | Blood vessels | Stem cell transplantation | Histology | Patients | c-Kit protein | Infusion | Allografts | Dilated cardiomyopathy | Rodents | Myocardium | Infarction | Heart diseases | CD105 antigen | Original
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11. Full Text Ubiquitin-like protein ISG15 in host defense against heart failure in a mouse model of virus-induced cardiomyopathy
by Rahnefeld, Anna and Klingel, Karin and Schuermann, Anett and Diny, Nicola L and Althof, Nadine and Lindner, Anika and Bleienheuft, Philipp and Savvatis, Konstantinos and Respondek, Dorota and Opitz, Elisa and Ketscher, Lars and Sauter, Martina and Seifert, Ulrike and Tschöpe, Carsten and Poller, Wolfgang and Knobeloch, Klaus-Peter and Voigt, Antje
Circulation, ISSN 0009-7322, 09/2014, Volume 130, Issue 18, pp. 1589 - 1600
Background-Common causative agents in the development of inflammatory cardiomyopathy include cardiotropic viruses such as coxsackievirus B3 (CVB3). Here, we... 
Human | infection | models, animal | viruses | ISG15 protein, human | Cardiomyopathy | myocarditis | Dilated | ISG15 protein | cardiomyopathy, dilated | inflammation | Animal | interferon type I | Models | immunology | CARDIAC & CARDIOVASCULAR SYSTEMS | COXSACKIEVIRUS | ENTEROVIRUS MYOCARDITIS | VIRAL MYOCARDITIS | BETA | E3 LIGASE | CONJUGATION | REPLICATION | LEFT-VENTRICULAR DYSFUNCTION | POLIOVIRUS | PERIPHERAL VASCULAR DISEASE | Ubiquitins - genetics | Humans | Middle Aged | Viral Proteins - immunology | Male | T-Lymphocytes - virology | Heart Failure - virology | Heart Failure - immunology | Killer Cells, Natural - immunology | Adult | Female | Cytokines - genetics | Ubiquitins - metabolism | Cytokines - immunology | Disease Models, Animal | Cardiomyopathy, Dilated - genetics | Ubiquitins - immunology | Cytokines - metabolism | Killer Cells, Natural - virology | Mice, Inbred C57BL | Heart Failure - genetics | Cardiomyopathy, Dilated - virology | Coxsackievirus Infections - complications | Immunity, Innate - immunology | Coxsackievirus Infections - genetics | Myocytes, Cardiac - virology | Animals | Virus Replication | Biopsy | Myocytes, Cardiac - physiology | Cardiomyopathy, Dilated - immunology | Enterovirus B, Human - immunology | T-Lymphocytes - immunology | Cysteine Endopeptidases - immunology | Coxsackievirus Infections - immunology | Heart failure | Ubiquitin | Care and treatment | Usage | Mortality | Heart diseases | Risk factors
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12. Viral myocarditis and dilated cardiomyopathy: Etiology and pathogenesis
by Huber, Sally A
Current Pharmaceutical Design, ISSN 1381-6128, 02/2016, Volume 22, Issue 4, pp. 408 - 426
Myocarditis is an inflammation of the myocardium which often follows microbial infections and is a significant cause of sudden unexpected death in the young (<... 
Autoimmunity | Myocarditis | Immunosuppression | Dilated cardiomyopathy | Persistent infection | Fibrosis | Endothelial-mesenchymal transition | Picornaviruses | endothelial-mesenchymal transition | TRANSCRIPTION FACTOR IRF8 | NATURAL-KILLER-CELLS | COXSACKIEVIRUS B3 INFECTION | GIANT-CELL MYOCARDITIS | immunosuppression | picornaviruses | TUMOR-NECROSIS-FACTOR | MESENCHYMAL STROMAL CELLS | autoimmunity | HEPATITIS-C VIRUS | BIOPSY-PROVEN MYOCARDITIS | DECAY-ACCELERATING FACTOR | PHARMACOLOGY & PHARMACY | fibrosis | persistent infection | dilated cardiomyopathy | LEFT-VENTRICULAR FUNCTION | Immunity, Innate - immunology | Myocarditis - virology | Animals | Myocarditis - etiology | Virus Diseases - immunology | Cardiomyopathy, Dilated - etiology | Humans | Cardiomyopathy, Dilated - immunology | Cardiomyopathy, Dilated - virology | Myocarditis - immunology | Virus Diseases - etiology
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13. Full Text Autoantibodies against cardiac troponin I are responsible for dilated cardiomyopathy in PD-1-deficient mice
by Wang, Jian and Mitsuiye, Tamotsu and Hiai, Hiroshi and Minato, Nagahiro and Matsumori, Akira and Tanaka, Yoshimasa and Mizoguchi, Akira and Okazaki, Taku and Nishio, Ryosuke and Honjo, Tasuku and Ishida, Masayoshi
Nature Medicine, ISSN 1078-8956, 12/2003, Volume 9, Issue 12, pp. 1477 - 1483
We recently reported that mice deficient in the programmed cell death- 1 (PD- 1) immunoinhibitory coreceptor develop autoimmune dilated cardiomyopathy (DCM),... 
ANTIBODIES | IMMUNOADSORPTION | MEDICINE, RESEARCH & EXPERIMENTAL | CELLS | VIRUS MYOCARDITIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | MODEL | IDENTIFICATION | CELL BIOLOGY | HYPERTROPHY | MUTATIONS | CONGESTIVE-HEART-FAILURE | PROTEINS | Autoantibodies - metabolism | Rats, Wistar | Mice, Inbred A | Myocytes, Cardiac - immunology | Humans | DNA, Complementary - genetics | Troponin I - immunology | Base Sequence | Calcium Signaling | Cardiomyopathy, Dilated - genetics | Antigens, Surface - genetics | Rats | Cardiomyopathy, Dilated - metabolism | Mice, Knockout | Animals | Apoptosis Regulatory Proteins | Cardiomyopathy, Dilated - etiology | Antibodies, Monoclonal - administration & dosage | Mice, Nude | Cardiomyopathy, Dilated - immunology | Myocytes, Cardiac - metabolism | Programmed Cell Death 1 Receptor | Mice | Mice, Inbred BALB C | Antigens, Surface - immunology | Antigens, CD | PD-1 protein | troponin I
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