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Circulation Research, ISSN 0009-7330, 03/2009, Volume 104, Issue 5, pp. 688 - 698
ABSTRACT—The migration of vascular smooth muscle cells (VSMCs) plays an essential role during the development of atherosclerosis and restenosis. Extensive... 
Extracellular matrix | Metalloproteinase | Neointima formation | Vascular smooth muscle cell migration | neointima formation | RNA INTERFERENCE | CARDIAC & CARDIOVASCULAR SYSTEMS | metalloproteinase | vascular smooth muscle cell migration | MECHANISMS | OLIGOMERIC MATRIX PROTEIN | HYPERPLASIA | INHIBITION | DISEASE | GROWTH | MATRIX-METALLOPROTEINASE-9 | DEGRADATION | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | extracellular matrix | Tumor Necrosis Factor-alpha - metabolism | Hyperplasia | Glycoproteins - metabolism | Myocytes, Smooth Muscle - pathology | Male | NF-kappa B - metabolism | Transcription Factor AP-1 - metabolism | Carotid Artery Injuries - etiology | Matrilin Proteins | RNA Interference | Time Factors | Carotid Arteries - enzymology | Extracellular Matrix Proteins - metabolism | Carotid Artery Injuries - pathology | Disease Models, Animal | Transduction, Genetic | Myocytes, Smooth Muscle - enzymology | Proto-Oncogene Proteins c-sis | Cells, Cultured | Rats | Tunica Intima - enzymology | Rats, Sprague-Dawley | ADAM Proteins - metabolism | Muscle, Smooth, Vascular - pathology | Platelet-Derived Growth Factor - metabolism | Animals | Carotid Artery Injuries - enzymology | Tunica Intima - pathology | ADAMTS7 Protein | Catheterization - adverse effects | Carotid Arteries - pathology | ADAM Proteins - genetics | Muscle, Smooth, Vascular - enzymology | Cell Movement | RNA, Small Interfering - metabolism | Index Medicus
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 06/2008, Volume 294, Issue 6, pp. 2721 - 2735
The dietary polyphenolic compound resveratrol, by activating the protein deacetylase enzyme silent information regulator 2/sirtuin 1 (SIRT1), prolongs life... 
Vascular aging | Polyphenol | Stroke | Tobacco | Sirtuin 1 | Inflammation | Apoptosis | sirtuin 1 | LIFE-SPAN | MITOCHONDRIAL SUPEROXIDE-PRODUCTION | ELECTRON-SPIN-RESONANCE | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | DNA-DAMAGE | apoptosis | NECROSIS-FACTOR-ALPHA | polyphenol | stroke | CORONARY-ARTERIES | NAD(P)H OXIDASE | ENDOTHELIUM-DEPENDENT DILATION | inflammation | vascular aging | CALORIC RESTRICTION | PERIPHERAL VASCULAR DISEASE | tobacco | NF-KAPPA-B | Carotid Arteries - drug effects | Smoking - adverse effects | Inflammation - pathology | Reactive Oxygen Species - metabolism | Rats, Wistar | Apoptosis - drug effects | Male | NF-kappa B - metabolism | Stilbenes - pharmacology | Anti-Inflammatory Agents, Non-Steroidal - pharmacology | Smoking - physiopathology | Inflammation Mediators - metabolism | Resveratrol | Carotid Arteries - enzymology | Models, Animal | Sirtuins - genetics | Cytoprotection | DNA Damage - drug effects | Coronary Vessels - drug effects | Coronary Vessels - enzymology | Vasodilator Agents - pharmacology | Acetylcholine - pharmacology | Smoke - adverse effects | Cells, Cultured | Rats | Arteries - drug effects | Mitochondria - metabolism | Antioxidants - pharmacology | Mitochondria - drug effects | Inflammation - etiology | Smoking - metabolism | Phenotype | Animals | Arteries - enzymology | Arteries - physiopathology | Smoking - pathology | Inflammation - prevention & control | Oxidative Stress - drug effects | Vasodilation - drug effects | Endothelial Cells - enzymology | Inflammation - enzymology | Sirtuins - metabolism | Endothelial Cells - drug effects | Inflammation - physiopathology | Studies | Genotype & phenotype | Cell culture | Tobacco smoke | Antibiotics | Rodents | Coronary vessels | Effects | Index Medicus
Journal Article
Thrombosis and Haemostasis, ISSN 0340-6245, 07/2015, Volume 113, Issue 4, pp. 870 - 880
Summary Factor VII (FVII) activating protease (FSAP) is a circulating protease with a putative function in blood coagulation and fibrinolysis. Genetic... 
Stroke, Systemic or Venous Thromboembolism | Haemostasis | Fibrinolysis | FSAP | Thrombosis | HABP2 | Coagulation | LIVER FIBROSIS | RISK | PROTEOLYSIS | fibrinolysis | thrombosis | haemostasis | G534E | PERIPHERAL VASCULAR DISEASE | MARBURG-I POLYMORPHISM | HEMATOLOGY | FACTOR PATHWAY INHIBITOR | Carotid Artery Diseases - prevention & control | Serine Endopeptidases - administration & dosage | Thrombosis - chemically induced | Carotid Artery Diseases - chemically induced | Mesenteric Vascular Occlusion - genetics | Venous Thromboembolism - blood | Chlorides | Serine Endopeptidases - genetics | Carotid Arteries - enzymology | Mesenteric Vascular Occlusion - enzymology | Thrombosis - blood | Thrombosis - prevention & control | Disease Models, Animal | Genetic Predisposition to Disease | Serine Endopeptidases - deficiency | Mesenteric Vascular Occlusion - prevention & control | Mice, Inbred C57BL | Thrombosis - enzymology | Hemostasis - genetics | Lipoproteins - blood | Mesenteric Vascular Occlusion - blood | Venous Thromboembolism - enzymology | Mice, Knockout | Ferric Compounds | Phenotype | Venous Thromboembolism - genetics | Animals | Carotid Artery Diseases - enzymology | Carotid Artery Diseases - blood | Collagen | Carotid Artery Diseases - genetics | Venous Thromboembolism - chemically induced | Blood Coagulation Tests | Thrombosis - genetics | Jugular Veins - enzymology | Norepinephrine | Mesenteric Vascular Occlusion - chemically induced | Mesenteric Arteries - enzymology
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 04/2013, Volume 33, Issue 4, pp. 777 - 784
OBJECTIVE—To analyze the role of toll-like receptor 4 in modulating metabolism and endothelial function. APPROACH AND RESULTS—Type 2 diabetic mice with mutated... 
systolic arterial blood pressure | endothelium-derived contracting factor | nitric oxide | proinflammatory cytokines | obesity | OXIDATIVE STRESS | ACTIVATION | MACROPHAGES | VASCULAR SMOOTH-MUSCLE | SYNTHASE | INSULIN-RESISTANCE | NITRIC-OXIDE | PERIPHERAL VASCULAR DISEASE | NADH/NADPH OXIDASE | HYPERTENSION | HEMATOLOGY | INNATE IMMUNITY | Mesenteric Arteries - physiopathology | Oxidative Stress | Receptors, Leptin - genetics | NADPH Oxidases - metabolism | Endothelium, Vascular - drug effects | RNA, Messenger - metabolism | Toll-Like Receptor 4 - deficiency | Carotid Arteries - enzymology | NADPH Oxidases - genetics | Disease Models, Animal | NADH, NADPH Oxidoreductases - genetics | Down-Regulation | NADPH Oxidases - antagonists & inhibitors | Enzyme Inhibitors - pharmacology | Toll-Like Receptor 4 - genetics | Obesity - physiopathology | Toll-Like Receptor 4 - metabolism | Vasoconstriction - drug effects | Mice, Knockout | Receptors, Leptin - deficiency | 6-Ketoprostaglandin F1 alpha - metabolism | Lipopolysaccharides - pharmacology | Mice | NADH, NADPH Oxidoreductases - antagonists & inhibitors | Mutation | Vasodilation - drug effects | Mesenteric Arteries - enzymology | Muscle, Smooth, Vascular - enzymology | Phosphorylation | Diabetes Mellitus, Type 2 - genetics | Endothelium, Vascular - enzymology | Muscle, Smooth, Vascular - physiopathology | Obesity - genetics | Dose-Response Relationship, Drug | Superoxides - metabolism | Membrane Proteins - metabolism | Aorta - physiopathology | Nitric Oxide Synthase Type III - metabolism | NADH, NADPH Oxidoreductases - metabolism | Aorta - enzymology | Vasodilator Agents - pharmacology | Mice, Inbred C57BL | Diabetes Mellitus, Type 2 - enzymology | Endothelium, Vascular - physiopathology | NADPH Oxidase 4 | Mice, Inbred C3H | NADPH Oxidase 1 | Animals | Diabetes Mellitus, Type 2 - physiopathology | Carotid Arteries - physiopathology | Obesity - enzymology | Cyclooxygenase 1 - metabolism | Nitric Oxide - metabolism | Index Medicus
Journal Article
Cardiovascular Research, ISSN 0008-6363, 2010, Volume 87, Issue 3, pp. 569 - 577
Growing evidence suggests that arginase misregulation plays a key role in the pathophysiology of essential hypertension. In the present study, we investigated... 
Remodelling | Arginase | Spontaneously hypertensive rat | Endothelial function | Arteries | CARDIAC & CARDIOVASCULAR SYSTEMS | ENDOTHELIAL DYSFUNCTION | RESISTANCE ARTERIES | BLOOD-PRESSURE REDUCTION | GENE POLYMORPHISMS | HYPERTROPHY | SMOOTH-MUSCLE | STIFFNESS | VASCULAR FUNCTION | NITRIC-OXIDE | ANTIHYPERTENSIVE TREATMENT | Carotid Arteries - drug effects | Mesenteric Arteries - physiopathology | Compliance | Arginase - metabolism | Rats, Inbred WKY | Endothelium, Vascular - drug effects | Heart Diseases - prevention & control | Hypertension - drug therapy | Male | Mesenteric Arteries - drug effects | Endothelium, Vascular - enzymology | Arginine - analogs & derivatives | Dose-Response Relationship, Drug | Time Factors | Carotid Arteries - enzymology | Blood Pressure - drug effects | Membrane Proteins - metabolism | Aorta - physiopathology | Arginase - antagonists & inhibitors | Hypertension - enzymology | Hypertension - genetics | Aorta - enzymology | Disease Models, Animal | Rats, Inbred SHR | Vasoconstrictor Agents - pharmacology | Vasodilator Agents - pharmacology | Aorta - drug effects | Endothelium, Vascular - physiopathology | Enzyme Inhibitors - pharmacology | Rats | Myocardium - pathology | Heart Diseases - enzymology | Vasoconstriction - drug effects | Hypertension - pathology | Hypertension - physiopathology | Cardiovascular Agents - pharmacology | Collagen - metabolism | Animals | Carotid Arteries - physiopathology | Arginine - pharmacology | Cyclooxygenase 2 - metabolism | Fibrosis | Nitric Oxide Synthase - metabolism | Cyclooxygenase 1 - metabolism | Vasodilation - drug effects | Mesenteric Arteries - enzymology | Heart Diseases - pathology | Index Medicus
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 03/2015, Volume 35, Issue 3, pp. 547 - 557
OBJECTIVE—Atherosclerosis, an inflammatory disease of arterial vessel walls, requires migration and matrix metalloproteinase (MMP)-9–dependent invasion of... 
Inflammation | Migration and invasion protein | Atherosclerosis | Serum-glucocorticoid regulated kinase | atherosclerosis | migration and invasion protein | APOE-NULL MICE | ATHEROSCLEROTIC PLAQUES | INCREASED EXPRESSION | PATHOGENIC T(H)17 CELLS | MONOCYTE CHEMOATTRACTANT PROTEIN-1 | NEOINTIMA FORMATION | MATRIX METALLOPROTEINASE-3 | inflammation | MUSCLE-CELL MIGRATION | PERIPHERAL VASCULAR DISEASE | serum-glucocorticoid regulated kinase | HEMATOLOGY | NF-KAPPA-B | E-DEFICIENT MICE | Inflammation - pathology | Protein-Serine-Threonine Kinases - deficiency | Apolipoproteins E - deficiency | Atherosclerosis - genetics | Humans | Male | Immediate-Early Proteins - metabolism | Matrix Metalloproteinase 9 - metabolism | Carotid Artery Diseases - pathology | Matrix Metalloproteinase 9 - genetics | Peritonitis - genetics | Carotid Arteries - enzymology | Immediate-Early Proteins - deficiency | Transcription, Genetic | Protein-Serine-Threonine Kinases - metabolism | Disease Models, Animal | Atherosclerosis - pathology | Macrophages - pathology | NF-kappa B p50 Subunit - metabolism | Signal Transduction | Matrix Metalloproteinase 2 - metabolism | Plaque, Atherosclerotic | Macrophages - enzymology | Mice, Knockout | Aorta - pathology | Aortic Diseases - genetics | Peritonitis - enzymology | Immediate-Early Proteins - genetics | Carotid Artery Diseases - enzymology | Carotid Artery Diseases - genetics | Mutation | Inflammation - enzymology | Vascular Remodeling | I-kappa B Proteins - metabolism | Atherosclerosis - enzymology | Transfection | I-kappa B Kinase - metabolism | Thioglycolates | Aortic Diseases - enzymology | Active Transport, Cell Nucleus | Aorta - enzymology | Aortic Diseases - pathology | Cell Line | Mice, Inbred C57BL | Protein-Serine-Threonine Kinases - genetics | Peritonitis - chemically induced | Chemotaxis | Gene Expression Regulation, Enzymologic | Animals | Apolipoproteins E - genetics | Inflammation - genetics | Carotid Arteries - pathology | Index Medicus
Journal Article
Arteriosclerosis, thrombosis, and vascular biology, ISSN 1079-5642, 2015, Volume 35, Issue 1, pp. 79 - 86
The protein degrading activity of cathepsin C (CatC), combined with its role in leukocyte granule activation, suggests a contribution of this cystein protease... 
protease | inflammation | human | mice | Atherosclerosis | atherosclerosis | LOCALIZATION | ACTIVATION | ATHEROGENESIS | SERINE PROTEASES | EXPERIMENTAL ARTHRITIS | CHYMASE | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | EXPRESSION | T-CELLS | DIPEPTIDYL-PEPTIDASE-I | Carotid Artery Diseases - prevention & control | Atherosclerosis - genetics | Humans | Leukocytes - enzymology | Leukocytes - immunology | Atherosclerosis - enzymology | Time Factors | Carotid Artery Diseases - pathology | T-Lymphocytes, Helper-Inducer - immunology | T-Lymphocytes, Helper-Inducer - enzymology | Receptors, LDL - deficiency | Aortic Diseases - prevention & control | Carotid Arteries - enzymology | Female | Aortic Diseases - enzymology | Cell Differentiation | Aorta - enzymology | Aortic Diseases - pathology | Disease Models, Animal | Adaptive Immunity | Carotid Artery Diseases - immunology | Receptors, LDL - genetics | Atherosclerosis - pathology | Atherosclerosis - immunology | Aortic Diseases - immunology | Down-Regulation | Mice, Inbred C57BL | Cathepsin C - genetics | Cells, Cultured | Aorta - immunology | Foam Cells - enzymology | Immunity, Innate | Plaque, Atherosclerotic | Cathepsin C - metabolism | Disease Progression | Macrophage Activation | Mice, Knockout | Aorta - pathology | Aortic Diseases - genetics | Animals | Carotid Artery Diseases - enzymology | Carotid Arteries - immunology | Carotid Artery Diseases - genetics | Carotid Arteries - pathology | Foam Cells - immunology | Atherosclerosis - prevention & control | Index Medicus
Journal Article
AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY, ISSN 0363-6135, 01/2012, Volume 302, Issue 1, pp. H159 - H166
Romero MJ, Iddings JA, Platt DH, Ali MI, Cederbaum SD, Stepp DW, Caldwell RB, Caldwell RW. Diabetes-induced vascular dysfunction involves arginase I. Am J... 
CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | ENDOTHELIAL DYSFUNCTION | MECHANISMS | vascular stiffness | HEART-DISEASE | INHIBITION | ARTERIAL STIFFNESS | NITRIC-OXIDE | PERIPHERAL VASCULAR DISEASE | fibrosis | MICE | CARDIAC FIBROSIS | CONTRIBUTES | oxidative stress | Compliance | Diabetes Mellitus, Experimental - enzymology | Coronary Vessels - physiopathology | Arginase - metabolism | Diabetes Mellitus, Experimental - genetics | Diabetic Angiopathies - etiology | Vasodilation | Dose-Response Relationship, Drug | Diabetic Angiopathies - physiopathology | Diabetic Angiopathies - pathology | Lipid Peroxidation - drug effects | Superoxides - metabolism | Carotid Arteries - enzymology | Diabetic Angiopathies - enzymology | Diabetes Mellitus, Experimental - complications | Aorta - physiopathology | Diabetic Angiopathies - genetics | Aorta - enzymology | Diabetes Mellitus, Experimental - physiopathology | Vasoconstrictor Agents - pharmacology | Coronary Vessels - enzymology | Vasodilator Agents - pharmacology | Hydroxyproline - metabolism | Mice, Inbred C57BL | Vasoconstriction | Arginase - genetics | Arteries - drug effects | Arteries - pathology | Hydrogen Peroxide - metabolism | Mice, Knockout | Animals | Arteries - enzymology | Carotid Arteries - physiopathology | Arteries - physiopathology | Fibrosis | Diabetes Mellitus, Experimental - pathology | Mice | Oxidative stress | Care and treatment | Physiological aspects | Development and progression | Arginase | Diabetes | Research | Health aspects | Blood vessels | Cardiovascular disease | Amino acids | Protein synthesis | Rodents | Index Medicus | Vascular Biology and Microcirculation
Journal Article
Journal of Vascular Surgery, ISSN 0741-5214, 2015, Volume 64, Issue 4, pp. 1074 - 1083
Objective Effective treatments against restenosis after percutaneous transluminal angioplasty and stenting are largely lacking. Human tissue kallikrein gene... 
Surgery | Carotid Arteries - drug effects | Tumor Necrosis Factor-alpha - metabolism | Phosphorylation | Carotid Stenosis - prevention & control | Humans | Hyperplasia | Transforming Growth Factor beta1 - metabolism | Tumor Necrosis Factor-alpha - genetics | Myocytes, Smooth Muscle - pathology | Carotid Artery Injuries - drug therapy | Male | Smad3 Protein - metabolism | Interleukin-1beta - genetics | Kallikreins - administration & dosage | Angioplasty, Balloon - adverse effects | Angiography, Digital Subtraction | Foam Cells - drug effects | Neointima | Time Factors | Carotid Stenosis - genetics | Interleukin-1beta - metabolism | Diet, High-Fat | Inflammation Mediators - metabolism | Carotid Arteries - enzymology | Carotid Artery Injuries - genetics | Nitric Oxide Synthase Type III - metabolism | Myocytes, Smooth Muscle - drug effects | Foam Cells - pathology | Carotid Artery Injuries - pathology | Disease Models, Animal | Muscle, Smooth, Vascular - drug effects | Rabbits | Kallikreins - pharmacology | Drug Administration Schedule | Myocytes, Smooth Muscle - enzymology | Urine - chemistry | Smad2 Protein - metabolism | Carotid Stenosis - pathology | Foam Cells - enzymology | Kallikreins - urine | Administration, Intravenous | Muscle, Smooth, Vascular - pathology | Animals | Carotid Artery Injuries - enzymology | Signal Transduction - drug effects | Carotid Arteries - pathology | Carotid Stenosis - enzymology | Muscle, Smooth, Vascular - enzymology | Index Medicus
Journal Article
Thrombosis and Haemostasis, ISSN 0340-6245, 02/2014, Volume 111, Issue 2, pp. 308 - 322
Pharmacological treatments targeting CXC chemokines and the associated neutrophil activation and recruitment into atherosclerotic plaques hold promise for... 
inflammation | carotid artery | Atherosclerosis | Cardiovascular Biology and Cell Signalling | Inflammation | Carotid artery | atherosclerosis | CELLS | TRANSCRIPTION | DOWN-REGULATION | INJURY | VULNERABILITY | ALPHA | NAMPT | PERIPHERAL VASCULAR DISEASE | ACCUMULATION | HEMATOLOGY | ISCHEMIC-STROKE | EXPRESSION | Carotid Arteries - drug effects | Apolipoproteins E - deficiency | Atherosclerosis - genetics | Humans | Human Umbilical Vein Endothelial Cells - immunology | Nicotinamide Phosphoribosyltransferase - antagonists & inhibitors | Atherosclerosis - enzymology | Matrix Metalloproteinase 9 - metabolism | Nicotinamide Phosphoribosyltransferase - metabolism | Piperidines - pharmacology | Time Factors | Carotid Artery Diseases - pathology | Diet, High-Fat | Carotid Arteries - enzymology | Chemokine CXCL1 - metabolism | Acrylamides - pharmacology | Neutrophil Infiltration - drug effects | Carotid Artery Diseases - drug therapy | Disease Models, Animal | Carotid Artery Diseases - immunology | Atherosclerosis - pathology | Atherosclerosis - drug therapy | Atherosclerosis - immunology | Human Umbilical Vein Endothelial Cells - drug effects | Cytokines - metabolism | Anti-Inflammatory Agents - pharmacology | Mice, Inbred C57BL | Cells, Cultured | Enzyme Inhibitors - pharmacology | Plaque, Atherosclerotic | Mice, Knockout | Collagen - metabolism | Animals | Human Umbilical Vein Endothelial Cells - enzymology | Signal Transduction - drug effects | Transcription Factor RelA - metabolism | Carotid Artery Diseases - enzymology | Apolipoproteins E - genetics | Carotid Arteries - immunology | Carotid Artery Diseases - genetics | Carotid Arteries - pathology | Mice | Cytokines - antagonists & inhibitors
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 08/2015, Volume 35, Issue 8, pp. 1852 - 1861
OBJECTIVE—Vascular smooth muscle cells (VSMC) proliferation is a hallmark of atherosclerosis and vascular restenosis. The intermediate conductance Ca-activated... 
atherosclerosis | neointima formation | SK4 protein | NM23B nucleoside diphosphate kinase | KCa3.1 protein | Nme2 protein | KCNN4 protein | CD4 T-CELLS | ENDOTHELIAL DYSFUNCTION | REVERSIBLE HISTIDINE PHOSPHORYLATION | PHENOTYPIC MODULATION | 1 protein | KCa3 | CA2+-ACTIVATED K+ CHANNEL | VASCULAR-DISEASE | BLOOD-PRESSURE | BETA-GAMMA DIMERS | HUMAN ERYTHROCYTES | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | CORONARY SMOOTH-MUSCLE | Cell Proliferation | Intermediate-Conductance Calcium-Activated Potassium Channels - genetics | Myocytes, Smooth Muscle - pathology | Mice, 129 Strain | Intermediate-Conductance Calcium-Activated Potassium Channels - metabolism | NM23 Nucleoside Diphosphate Kinases - genetics | NM23 Nucleoside Diphosphate Kinases - metabolism | Neointima | Carotid Arteries - enzymology | Carotid Artery Injuries - genetics | NM23 Nucleoside Diphosphate Kinases - deficiency | Carotid Artery Injuries - pathology | Disease Models, Animal | Signal Transduction | Myocytes, Smooth Muscle - enzymology | Mice, Inbred C57BL | Cells, Cultured | Mice, Knockout | Muscle, Smooth, Vascular - pathology | Animals | Carotid Artery Injuries - enzymology | Membrane Potentials | Intermediate-Conductance Calcium-Activated Potassium Channels - deficiency | Carotid Arteries - pathology | Muscle, Smooth, Vascular - enzymology | Index Medicus
Journal Article
Circulation Research, ISSN 0009-7330, 09/2009, Volume 105, Issue 6, pp. 537 - 544
RATIONALE:Endothelial progenitor cells (EPCs, defined as sca-1flk-1lin mononuclear blood cells) contribute to vascular repair. The role of hypoxia and reactive... 
Oxidative stress | NADPH oxidase | Superoxide | ACTIVATION | CARDIAC & CARDIOVASCULAR SYSTEMS | COA REDUCTASE INHIBITORS | BONE-MARROW | TYROSINE PHOSPHATASES | SIGNAL-TRANSDUCTION | SYNTHASE | NAD(P)H OXIDASE | ERYTHROPOIETIN RECEPTOR | HINDLIMB ISCHEMIA | PERIPHERAL VASCULAR DISEASE | SMOOTH-MUSCLE-CELLS | superoxide | HEMATOLOGY | oxidative stress | Protein Tyrosine Phosphatase, Non-Receptor Type 11 - metabolism | Leukocytes, Mononuclear - metabolism | Antigens, Ly | Reactive Oxygen Species - metabolism | Membrane Glycoproteins - metabolism | NADPH Oxidases - metabolism | Vascular Endothelial Growth Factor Receptor-2 | STAT5 Transcription Factor - metabolism | Stem Cells - enzymology | Bone Marrow Transplantation | Carotid Arteries - enzymology | NADPH Oxidases - genetics | Carotid Artery Injuries - genetics | Erythropoietin - metabolism | Wound Healing - genetics | NADH, NADPH Oxidoreductases - metabolism | Carotid Artery Injuries - pathology | NADH, NADPH Oxidoreductases - genetics | Hypoxia - enzymology | Gene Expression Regulation - genetics | Cells, Cultured | Signal Transduction - genetics | NADPH Oxidase 2 | NADPH Oxidase 1 | Membrane Glycoproteins - genetics | Mice, Knockout | Membrane Proteins | Leukocytes, Mononuclear - pathology | Hypoxia - genetics | Animals | Carotid Artery Injuries - enzymology | Hypoxia - pathology | Stem Cells - pathology | Leukocytes, Mononuclear - enzymology | Carotid Arteries - pathology | Mice | Endothelial Cells - pathology | Endothelial Cells - enzymology | Index Medicus
Journal Article
Atherosclerosis, ISSN 0021-9150, 2016, Volume 256, pp. 53 - 61
Journal Article
Journal Article