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Circulation research, ISSN 1524-4571, 2009, Volume 104, Issue 4, pp. 476 - 487
.... In the present study, the time course changes and cellular distribution of miR-221 and miR-222 expression were identified in rat carotid arteries after angioplasty, in which their... 
Vascular disease | Proliferation | Gene regulation | MicroRNAs | Vascular smooth muscle cells | microRNAs | CARDIAC & CARDIOVASCULAR SYSTEMS | proliferation | DOWN-REGULATION | CARDIOVASCULAR BIOLOGY | P27(KIP1) | BREAST-CANCER | INHIBITION | vascular disease | MESSENGER-RNA | gene regulation | RAT CAROTID-ARTERY | GROWTH | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | vascular smooth muscle cells | MICRORNA EXPRESSION | PROGRESSION | Carotid Artery Diseases - prevention & control | Cyclin-Dependent Kinase Inhibitor p57 - metabolism | Cell Proliferation | Muscle, Smooth, Vascular - metabolism | Hyperplasia | Myocytes, Smooth Muscle - pathology | Oligonucleotides, Antisense - metabolism | Male | MicroRNAs - metabolism | Angioplasty, Balloon - adverse effects | Gene Knockdown Techniques | Cyclin-Dependent Kinase Inhibitor p27 - metabolism | RNA Interference | Time Factors | Carotid Artery Diseases - pathology | Carotid Artery Diseases - metabolism | Myocytes, Smooth Muscle - metabolism | Disease Models, Animal | Carotid Artery, Common - metabolism | Proto-Oncogene Proteins c-sis | Cells, Cultured | Rats | Rats, Sprague-Dawley | Tunica Intima - metabolism | Muscle, Smooth, Vascular - pathology | Platelet-Derived Growth Factor - metabolism | Animals | Tunica Intima - pathology | Carotid Artery Diseases - genetics | Carotid Artery, Common - pathology | RNA, Small Interfering - metabolism
Journal Article
EMBO molecular medicine, ISSN 1757-4676, 2013, Volume 5, Issue 3, pp. 471 - 481
... and receptors may not just be important in emigration of monocytes from the blood to the arteries, but they may also control mobilization from the bone marrow (BM... 
atherosclerosis | monocyte | chemokine | mobilization | recruitment | Chemokine | Mobilization | Monocyte | Atherosclerosis | Recruitment | ATHEROSCLEROTIC LESIONS | FRACTALKINE | MEDICINE, RESEARCH & EXPERIMENTAL | MACROPHAGES | BONE-MARROW | SUBSETS | NEUTROPHILS | MICE REVEALS | INFLAMMATION | ACCUMULATION | CCR5 | Carotid Arteries - metabolism | Chemotaxis, Leukocyte | Apolipoproteins E - deficiency | Atherosclerosis - genetics | Male | Monocytes - immunology | Adoptive Transfer | Aorta - metabolism | Aortic Diseases - metabolism | Atherosclerosis - etiology | Receptors, CCR5 - metabolism | Monocytes - transplantation | Leukocyte Reduction Procedures | Time Factors | Carotid Artery Diseases - pathology | Diet, High-Fat | Leukocyte Count | Chemokine CXCL1 - metabolism | Monocytes - classification | Receptors, Chemokine - genetics | Carotid Artery Diseases - metabolism | Aortic Diseases - pathology | Disease Models, Animal | Carotid Artery Diseases - immunology | Atherosclerosis - pathology | Atherosclerosis - immunology | Carotid Artery Diseases - etiology | Signal Transduction | Aortic Diseases - immunology | Mice, Inbred C57BL | Receptors, Chemokine - metabolism | Aorta - immunology | Atherosclerosis - metabolism | Disease Progression | Mice, Knockout | Receptors, Chemokine - deficiency | Aorta - pathology | Aortic Diseases - genetics | Animals | Apolipoproteins E - genetics | Receptors, Interleukin-8B - metabolism | Carotid Arteries - immunology | Carotid Artery Diseases - genetics | Receptors, CCR1 - metabolism | Carotid Arteries - pathology | Mice | Aortic Diseases - etiology | Apoptosis | Hypercholesterolemia | Universities and colleges | CC chemokine receptors | Funding | CX3CR1 protein | Leukocytes | Macrophages | Adoptive transfer | Blood | High fat diet | CCR1 protein | CCR2 protein | Apolipoprotein E | Bone marrow | Carotid artery | CCR5 protein | Nutrient deficiency | CXCR2 protein | Emigration | Inflammation | Monocytosis | Studies | Monocytes | Microscopy | Arteriosclerosis | Software | Chemokines | Monocyte chemoattractant protein 1
Journal Article
PloS one, ISSN 1932-6203, 06/2013, Volume 8, Issue 6, p. e68197
Free fatty acids (FFAs), elevated in metabolic syndrome and diabetes, play a crucial role in the development of atherosclerotic cardiovascular disease, and... 
SUPPLEMENTATION | ARTERY ENDOTHELIAL-CELLS | PROTEIN-KINASE | MULTIDISCIPLINARY SCIENCES | DISEASE | CALCIUM | CALCIFICATION | RISK | FREE FATTY-ACID | DEPENDENT ACTIVATION | EXPRESSION | Calcinosis - genetics | Carotid Arteries - metabolism | Osteopontin - genetics | Palmitic Acid - metabolism | Coenzyme A Ligases - genetics | Muscle, Smooth, Vascular - metabolism | Calcium - metabolism | Gene Expression - genetics | Homeodomain Proteins - metabolism | Humans | Eicosapentaenoic Acid - genetics | Myocytes, Smooth Muscle - pathology | NF-kappa B - metabolism | Osteopontin - metabolism | Aorta - metabolism | Promoter Regions, Genetic - genetics | Coenzyme A Ligases - metabolism | Cell Differentiation - genetics | Caspases - metabolism | Bone Morphogenetic Protein 2 - metabolism | Calcinosis - metabolism | Plaque, Atherosclerotic - pathology | Cell Differentiation - physiology | Myocytes, Smooth Muscle - metabolism | Bone Morphogenetic Protein 2 - genetics | Macrophages - pathology | Plaque, Atherosclerotic - metabolism | Caspases - genetics | Cells, Cultured | Eicosapentaenoic Acid - metabolism | Homeodomain Proteins - genetics | Aorta - pathology | Osteoblasts - pathology | Macrophages - metabolism | Muscle, Smooth, Vascular - pathology | NF-kappa B - genetics | Carotid Arteries - pathology | Calcinosis - pathology | Osteoblasts - metabolism | Immunohistochemistry | Smooth muscle | Cardiovascular disease | Palmitic acid | Osteoblasts | Proteins | Mineralization | Biocompatibility | Aorta | Inhibition | NF-κB protein | Cytokines | Osteopontin | Metabolism | Gene expression | Fatty acids | Studies | Osteoblastogenesis | Inhibitors | Arteriosclerosis | Calcification | Cardiovascular diseases | Metabolic disorders | Calcification (ectopic) | Health sciences | Oxidative stress | Calcium | Laboratories | Chains | Lipids | Activation | Kinases | Macrophages | Carotid arteries | Arteries | Msx2 protein | Rodents | Atherosclerosis | Tumor necrosis factor-TNF | Carotid artery | Deposition | Plaques | University graduates | Bone morphogenetic protein 2 | Diabetes mellitus | Muscles | Caspase | siRNA | Medicine | Antibiotics | Kidney diseases | Diabetes | Differentiation | Eicosapentaenoic acid | Apoptosis | Pharmaceuticals
Journal Article
PloS one, ISSN 1932-6203, 2017, Volume 12, Issue 1, p. e0168914
Journal Article
American journal of physiology. Heart and circulatory physiology, ISSN 0363-6135, 11/2011, Volume 301, Issue 5, pp. H2081 - H2091
.... Heart rate-associated mechanical stress impairs carotid but not cerebral artery compliance in dyslipidemic atherosclerotic mice... 
Resting heart rate | Endothelial function | Cerebrovascular compliance | endothelial function | INTIMA-MEDIA THICKNESS | RATE REDUCTION | WALL SHEAR-STRESS | FAMILIAL HYPERCHOLESTEROLEMIA | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | PULSE-WAVE VELOCITY | RISK-FACTORS | IMPROVES ENDOTHELIAL FUNCTION | NITRIC-OXIDE SYNTHASE | MATRIX METALLOPROTEINASES | cerebrovascular compliance | resting heart rate | PERIPHERAL VASCULAR DISEASE | HYPERTENSIVE-RATS | Carotid Arteries - drug effects | Carotid Arteries - metabolism | Compliance | Dyslipidemias - complications | Metoprolol - pharmacology | Anti-Arrhythmia Agents - pharmacology | Humans | Carotid Artery Diseases - physiopathology | Endothelium, Vascular - drug effects | Male | Vasodilation | Matrix Metalloproteinase 9 - metabolism | Atherosclerosis - therapy | Cerebrovascular Disorders - prevention & control | Time Factors | Dyslipidemias - therapy | Disease Models, Animal | Cerebrovascular Disorders - physiopathology | Atherosclerosis - physiopathology | Genotype | Mice, Transgenic | Apolipoproteins B - genetics | Benzazepines - pharmacology | Atherosclerosis - metabolism | Disease Progression | Mice, Knockout | Phenotype | Cerebral Arteries - metabolism | Dyslipidemias - metabolism | Cerebrovascular Disorders - genetics | Mice | Carotid Artery Diseases - prevention & control | Dyslipidemias - genetics | Apolipoproteins B - metabolism | Stress, Mechanical | Cerebrovascular Disorders - metabolism | Exercise Therapy | Cerebral Arteries - drug effects | Atherosclerosis - etiology | Heart Rate - drug effects | Receptors, LDL - deficiency | Nitric Oxide Synthase Type III - metabolism | Carotid Artery Diseases - metabolism | Severity of Illness Index | Receptors, LDL - genetics | Carotid Artery Diseases - etiology | Mice, Inbred C57BL | Endothelium, Vascular - physiopathology | Cerebral Arteries - physiopathology | Cerebrovascular Disorders - etiology | Animals | Carotid Arteries - physiopathology | Nitric Oxide - metabolism | Dyslipidemias - physiopathology | Heart beat | Atherosclerosis | Physiological aspects | Development and progression | Stress (Physiology) | Carotid artery | Cerebral arteries | Health aspects
Journal Article
Cardiovascular research, ISSN 1755-3245, 2015, Volume 108, Issue 3, pp. 377 - 386
Neointimal hyperplasia is a common feature of fibro-proliferative vascular disease and characterizes initial stages of atherosclerosis. Neointimal lesions... 
ARTERIAL INJURY | MYOCARDIN | PULMONARY-HYPERTENSION | Neointimal hyperplasia | ATHEROSCLEROSIS | CADHERIN EXPRESSION | SMOOTH-MUSCLE-CELL | FLOW | Fibrosis | ERK5 | IN-VIVO | GROWTH | Shear stress | GENE-EXPRESSION | Endothelial-to-mesenchymal transition | ACTIVATION | CARDIAC & CARDIOVASCULAR SYSTEMS | MECHANISMS | SMOOTH-MUSCLE-CELLS | EXPRESSION | TRANSDIFFERENTIATION | Carotid Arteries - metabolism | Vascular Remodeling | Cell Proliferation | Human Umbilical Vein Endothelial Cells - metabolism | Humans | Carotid Artery Diseases - physiopathology | Stress, Mechanical | Male | Aortic Diseases - metabolism | Neointima | Transfection | RNA Interference | Time Factors | Carotid Artery Diseases - pathology | Swine | MAP Kinase Kinase 5 - metabolism | Epithelial-Mesenchymal Transition | HEK293 Cells | Carotid Artery Diseases - metabolism | Aorta, Thoracic - pathology | Aortic Diseases - pathology | Disease Models, Animal | Endothelial Cells - metabolism | Mice, Inbred C57BL | Aorta, Thoracic - metabolism | Mitogen-Activated Protein Kinase 7 - genetics | MAP Kinase Kinase 5 - genetics | Mitogen-Activated Protein Kinase 7 - metabolism | Aorta, Thoracic - physiopathology | Plaque, Atherosclerotic | Regional Blood Flow | Mechanotransduction, Cellular | Aortic Diseases - genetics | Aortic Diseases - physiopathology | Animals | Carotid Arteries - physiopathology | Carotid Artery Diseases - genetics | Human Umbilical Vein Endothelial Cells - pathology | Carotid Arteries - pathology | Endothelial Cells - pathology
Journal Article
Arteriosclerosis, thrombosis, and vascular biology, ISSN 1079-5642, 12/2016, Volume 37, Issue 2, pp. 191 - 204
... appreciation of increased disease burden and higher levels of arterial calcification in these subjects. Plaques within the coronary arteries of patients with diabetes mellitus generally... 
atherosclerosis | coronary artery disease | carotid arteries | death, sudden | diabetes mellitus | 10-YEAR FOLLOW-UP | PLAQUE MORPHOLOGY | PROTEIN-KINASE-C | RISK-FACTORS | death | ALKALINE-PHOSPHATASE | BEAM COMPUTED-TOMOGRAPHY | sudden | ACUTE MYOCARDIAL-INFARCTION | GROWTH-FACTOR 23 | CARDIOVASCULAR-DISEASE | PERIPHERAL VASCULAR DISEASE | SMOOTH-MUSCLE-CELLS | HEMATOLOGY | Carotid Arteries - metabolism | Diabetes Mellitus, Type 1 - epidemiology | Vascular Remodeling | Prognosis | Coronary Vessels - physiopathology | Humans | Carotid Artery Diseases - physiopathology | Diabetes Mellitus, Type 1 - metabolism | Diabetes Mellitus, Type 2 - metabolism | Coronary Vessels - metabolism | Diabetes Mellitus, Type 2 - epidemiology | Vascular Calcification - metabolism | Diabetic Angiopathies - physiopathology | Diabetic Angiopathies - pathology | Coronary Artery Disease - physiopathology | Carotid Artery Diseases - pathology | Coronary Artery Disease - pathology | Vascular Calcification - mortality | Vascular Calcification - physiopathology | Carotid Artery Diseases - metabolism | Diabetic Angiopathies - metabolism | Coronary Vessels - pathology | Coronary Artery Disease - metabolism | Risk Factors | Diabetic Angiopathies - mortality | Carotid Artery Diseases - mortality | Plaque, Atherosclerotic | Vascular Calcification - pathology | Disease Progression | Diabetes Mellitus, Type 1 - mortality | Animals | Carotid Arteries - physiopathology | Diabetes Mellitus, Type 2 - mortality | Death, Sudden, Cardiac - pathology | Carotid Arteries - pathology | Rupture, Spontaneous
Journal Article
Arteriosclerosis, thrombosis, and vascular biology, ISSN 1079-5642, 09/2013, Volume 33, Issue 9, pp. 2193 - 2201
...) 2 affect eicosanoids metabolism in atherosclerosis. APPROACH AND RESULTS—Analysis of human carotid plaques revealed that NOD2 was abundantly expressed at both mRNA and protein levels by endothelial cells and macrophages... 
atherosclerosis | pattern recognition receptors | eicosanoids | peptidoglycan | immunity, innate | inflammation | ACTIVATED PROTEIN-KINASE | CROHNS-DISEASE | HUMAN SYNOVIAL-CELLS | HUMAN ENDOTHELIAL-CELLS | LEUKOTRIENE B-4 | MURAMYL DIPEPTIDE | CARDIOVASCULAR-DISEASE | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | E-DEFICIENT MICE | STIMULATES COLLAGENASE | PROSTAGLANDIN E-2 | Carotid Arteries - metabolism | Tumor Necrosis Factor-alpha - metabolism | Up-Regulation | Arachidonate 12-Lipoxygenase - metabolism | Humans | Transcriptional Activation | Nod2 Signaling Adaptor Protein - genetics | Case-Control Studies | Time Factors | Carotid Artery Diseases - pathology | Interleukin-1beta - metabolism | Transcription, Genetic | p38 Mitogen-Activated Protein Kinases - metabolism | Carotid Artery Diseases - metabolism | Fibroblasts - metabolism | Carotid Artery Diseases - immunology | Arachidonate 5-Lipoxygenase - metabolism | Signal Transduction | Endothelial Cells - metabolism | Tissue Culture Techniques | Cells, Cultured | Acetylmuramyl-Alanyl-Isoglutamine - metabolism | Arachidonate 15-Lipoxygenase - metabolism | Immunity, Innate | Plaque, Atherosclerotic | Dinoprostone - metabolism | Endothelial Cells - immunology | Nod2 Signaling Adaptor Protein - metabolism | Eicosanoids - metabolism | Carotid Arteries - immunology | Carotid Artery Diseases - genetics | Cyclooxygenase 2 - metabolism | Fibroblasts - immunology | Ligands | Carotid Arteries - pathology | Kardiologi | Klinisk medicin | Medicin och hälsovetenskap | Cell- och molekylärbiologi | Medicinska och farmaceutiska grundvetenskaper
Journal Article
PloS one, ISSN 1932-6203, 2014, Volume 9, Issue 6, p. e100021
...). Reduced uterine artery blood flow (UABF) is a common cause of fetal growth restriction; abnormalities in angiogenic factors are implicated... 
CELLS | TROPHOBLAST INVASION | DEPENDENT RELAXATION | MULTIDISCIPLINARY SCIENCES | FETAL-GROWTH | NITRIC-OXIDE | PREECLAMPSIA | GENE-TRANSFER | RESTRICTION | BLOOD-FLOW | ENDOTHELIAL GROWTH-FACTOR | Cell Proliferation | Uterus - metabolism | Uterine Artery - metabolism | beta-Galactosidase - metabolism | Adenoviridae - genetics | Female | Nitric Oxide Synthase Type III - metabolism | Uterus - blood supply | Vascular Endothelial Growth Factor D - genetics | Genes, Reporter | Catheters, Indwelling | Gene Expression | Uterine Artery - cytology | Endothelial Cells - metabolism | Vasoconstriction | Nitric Oxide Synthase Type III - genetics | Pregnancy | Vascular Endothelial Growth Factor D - metabolism | Animals | Nitric Oxide Synthase Type II - genetics | Endothelial Cells - cytology | Blood Flow Velocity | Carotid Arteries - physiology | Uterus - cytology | Sheep | beta-Galactosidase - genetics | Genetic Vectors | Neovascularization, Physiologic | Placental Circulation - physiology | Nitric Oxide Synthase Type II - metabolism | Immunohistochemistry | Cell proliferation | Galactosidase | Biology | Gestation | Experiments | Blood | Arteries | Vasodilation | Angiogenesis | Ethics | Ultrasonic imaging | Uterus | Coding | Carotid artery | Vascular endothelial growth factor | Abnormalities | Fetuses | Contractility | Blood vessels | Injection | Permeability | Nitric-oxide synthase | Catheters | Endothelial cells | Blood flow | Medicine | Pets | Overexpression | Womens health | Nitric oxide | Hemodynamics | Endocrinology | Veins & arteries
Journal Article
Arteriosclerosis, thrombosis, and vascular biology, ISSN 1079-5642, 02/2013, Volume 33, Issue 2, pp. 354 - 361
.... In apolipoprotein E (ApoE)-null and ApoE/fibromodulin-null mice, a shear stress-modifying carotid artery cast induced formation of atherosclerotic plaques of different phenotypes... 
atherosclerosis | collagen | carotid arteries | fibromodulin | LDL | PROTEOGLYCANS | MACROPHAGES | FIBRILLOGENESIS | LUMICAN | ABNORMAL COLLAGEN FIBRILS | PERIPHERAL VASCULAR DISEASE | SMOOTH-MUSCLE-CELLS | EXTRACELLULAR-MATRIX | GENE-TRANSFER | HEMATOLOGY | EXPRESSION | BINDING | Carotid Arteries - metabolism | Aorta - ultrastructure | Cell Proliferation | Apolipoproteins E - deficiency | Atherosclerosis - genetics | Carotid Artery Diseases - physiopathology | Aorta - metabolism | Aortic Diseases - metabolism | Carotid Artery Diseases - pathology | Aortic Diseases - prevention & control | Lipoproteins, LDL - metabolism | Interleukin-6 - metabolism | Disease Models, Animal | Fibrillar Collagens - metabolism | Atherosclerosis - pathology | Atherosclerosis - immunology | Aortic Diseases - immunology | Atherosclerosis - physiopathology | Down-Regulation | Extracellular Matrix Proteins - genetics | Aorta - immunology | Genotype | Plaque, Atherosclerotic | Atherosclerosis - metabolism | Mice, Knockout | Aortic Diseases - genetics | Macrophages - metabolism | Phenotype | Carotid Arteries - immunology | Carotid Artery Diseases - genetics | Mice | Proteoglycans - genetics | Carotid Artery Diseases - prevention & control | Extracellular Matrix Proteins - deficiency | Stress, Mechanical | Proteoglycans - deficiency | Fibrillar Collagens - ultrastructure | Interleukin-10 - metabolism | Aorta - physiopathology | Carotid Artery Diseases - metabolism | Macrophages - immunology | Aortic Diseases - pathology | Carotid Artery Diseases - immunology | Fibromodulin | Cell Line | Microscopy, Electron, Transmission | Mice, Inbred C57BL | Regional Blood Flow | Aortic Diseases - physiopathology | Animals | Carotid Arteries - ultrastructure | Carotid Arteries - physiopathology | Apolipoproteins E - genetics | Atherosclerosis - prevention & control
Journal Article
Atherosclerosis, ISSN 0021-9150, 2012, Volume 220, Issue 2, pp. 343 - 350
...) and reduced glutathione (GSH), were detected in the injured artery. Macrophage infiltration and the production of inflammatory factors (i.e., IL-6, TNF-α and NF-κB) were also observed... 
Cardiovascular | Vascular smooth muscle cell | Reactive oxygen species | Neointima hyperplasia | Hydrogen | RATS | ATHEROSCLEROSIS | HYPERPLASIA | PROTECTS | SUCCINOBUCOL AGI-1067 | PROBUCOL | ANTIOXIDANT | PERIPHERAL VASCULAR DISEASE | INTESTINAL ISCHEMIA/REPERFUSION | STRESS | EXPRESSION | Carotid Arteries - drug effects | Carotid Arteries - metabolism | Tumor Necrosis Factor-alpha - metabolism | Hydrogen - pharmacology | Reactive Oxygen Species - metabolism | Muscle, Smooth, Vascular - metabolism | Glutathione - metabolism | Sodium Chloride - pharmacology | Carotid Artery Injuries - drug therapy | Muscle, Smooth, Vascular - immunology | NF-kappa B - metabolism | Sodium Chloride - blood | Carotid Artery Injuries - etiology | Hydrogen - blood | Time Factors | Inflammation Mediators - metabolism | Hydrogen - administration & dosage | Angioplasty, Balloon | Myocytes, Smooth Muscle - drug effects | Interleukin-6 - metabolism | Myocytes, Smooth Muscle - metabolism | Carotid Artery Injuries - metabolism | Superoxide Dismutase - metabolism | Carotid Artery Injuries - pathology | Disease Models, Animal | Muscle, Smooth, Vascular - drug effects | Malondialdehyde - metabolism | Proto-Oncogene Proteins c-sis - metabolism | Tunica Intima - immunology | Cells, Cultured | Injections, Intraperitoneal | Rats | Sodium Chloride - administration & dosage | Antioxidants - pharmacology | Rats, Sprague-Dawley | Tunica Intima - drug effects | Tunica Intima - metabolism | Muscle, Smooth, Vascular - injuries | Animals | Tunica Intima - pathology | Signal Transduction - drug effects | Antioxidants - administration & dosage | Carotid Arteries - immunology | Myocytes, Smooth Muscle - immunology | Carotid Arteries - pathology | Carotid Artery Injuries - immunology | Cell Proliferation - drug effects
Journal Article