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Circulation, ISSN 0009-7322, 10/2014, Volume 130, Issue 17, pp. 1452 - 1465
Journal Article
Atherosclerosis, ISSN 0021-9150, 2011, Volume 216, Issue 1, pp. 125 - 130
Abstract Objective To assess the influence of the TNFA rs1800629 (G > A) polymorphism in the risk of cardiovascular (CV) disease and subclinical... 
Cardiovascular | Cardiovascular disease | Genetics | rs1800629 | TNFA | Rheumatoid arthritis | Atherosclerosis | Rs1800629 | INTIMA-MEDIA THICKNESS | CORONARY-ARTERY DISEASE | MYOCARDIAL-INFARCTION | FACTOR-ALPHA GENE | PROMOTER POLYMORPHISM | ENDOTHELIAL FUNCTION | TUMOR-NECROSIS-FACTOR | HEART-DISEASE | PERIPHERAL VASCULAR DISEASE | TNF-ALPHA | Humans | Middle Aged | Tumor Necrosis Factor-alpha - genetics | Male | Cardiovascular Diseases - pathology | Arthritis, Rheumatoid - complications | Cardiovascular Diseases - genetics | Vasodilation | HLA-DR Antigens - genetics | Carotid Artery Diseases - pathology | Brachial Artery - physiopathology | Female | Odds Ratio | Carotid Artery Diseases - immunology | Cardiovascular Diseases - physiopathology | Genetic Predisposition to Disease | Risk Assessment | Gene Frequency | Risk Factors | Proportional Hazards Models | Logistic Models | Epitopes | Chi-Square Distribution | Spain | Cardiovascular Diseases - immunology | Arthritis, Rheumatoid - genetics | Phenotype | HLA-DR3 Antigen - genetics | HLA-DRB1 Chains | Carotid Artery Diseases - genetics | Aged | Carotid Arteries - pathology | Polymorphism, Single Nucleotide | Arthritis, Rheumatoid - immunology | Rheumatoid factor | Care and treatment | Histocompatibility antigens | Peptides | HLA histocompatibility antigens | Genetic aspects | Foot-and-mouth disease | Cardiovascular diseases | Risk factors | Endothelium | Index Medicus
Journal Article
Arteriosclerosis, thrombosis, and vascular biology, ISSN 1079-5642, 2014, Volume 34, Issue 8, pp. 1674 - 1680
Atherothrombosis is the main cause of myocardial infarction and ischemic stroke. Although the extrinsic (tissue factor-factor VIIa [FVIIa]) pathway is... 
Blood coagulation | Fibrin | Plaque rupture | Blood platelets | Thrombus | Tissue factor | Factor XII | blood platelets | ACTIVATION | tissue factor | blood coagulation | plaque rupture | thrombus | COAGULATION-FACTOR-XII | INHIBITION | PLASMA | HUMAN ATHEROSCLEROTIC PLAQUES | IN-VIVO | PERIPHERAL VASCULAR DISEASE | PLATELET GLYCOPROTEIN-VI | GENERATION | PROVIDES PROTECTION | fibrin | HEMATOLOGY | factor XII | Carotid Arteries - metabolism | Factor XIIa - metabolism | Apolipoproteins E - deficiency | Factor XII Deficiency - blood | Atherosclerosis - genetics | Humans | Aortic Diseases - blood | Carotid Artery Diseases - complications | Time Factors | Carotid Artery Diseases - pathology | Blood Coagulation | Factor XII Deficiency - genetics | Thrombosis - blood | Aorta, Thoracic - pathology | Aortic Diseases - pathology | Disease Models, Animal | Atherosclerosis - pathology | Factor XII - genetics | Mice, Inbred C57BL | Aorta, Thoracic - metabolism | Atherosclerosis - complications | Plaque, Atherosclerotic | Factor VIIa - metabolism | Mice, Knockout | Atherosclerosis - blood | Thrombosis - pathology | Aortic Diseases - genetics | Animals | Blood Platelets - metabolism | Thrombosis - etiology | Apolipoproteins E - genetics | Carotid Artery Diseases - blood | Factor XII - metabolism | Carotid Artery Diseases - genetics | Aortic Diseases - complications | Thrombosis - genetics | Carotid Arteries - pathology | Mice | Factor XI - metabolism | Thromboplastin - metabolism | Cholesterol, Dietary | Rupture, Spontaneous | Index Medicus
Journal Article
Cardiovascular Research, ISSN 0008-6363, 12/2015, Volume 108, Issue 3, pp. 377 - 386
Neointimal hyperplasia is a common feature of fibro-proliferative vascular disease and characterizes initial stages of atherosclerosis. Neointimal lesions... 
Shear stress | Endothelial-to-mesenchymal transition | Neointimal hyperplasia | ERK5 | Fibrosis | ARTERIAL INJURY | MYOCARDIN | ACTIVATION | CARDIAC & CARDIOVASCULAR SYSTEMS | ATHEROSCLEROSIS | MECHANISMS | FLOW | GROWTH | SMOOTH-MUSCLE-CELLS | EXPRESSION | TRANSDIFFERENTIATION | Carotid Arteries - metabolism | Vascular Remodeling | Cell Proliferation | Human Umbilical Vein Endothelial Cells - metabolism | Humans | Carotid Artery Diseases - physiopathology | Stress, Mechanical | Male | Aortic Diseases - metabolism | Neointima | Transfection | RNA Interference | Time Factors | Carotid Artery Diseases - pathology | Swine | MAP Kinase Kinase 5 - metabolism | Epithelial-Mesenchymal Transition | HEK293 Cells | Carotid Artery Diseases - metabolism | Aorta, Thoracic - pathology | Aortic Diseases - pathology | Disease Models, Animal | Endothelial Cells - metabolism | Mice, Inbred C57BL | Aorta, Thoracic - metabolism | Mitogen-Activated Protein Kinase 7 - genetics | MAP Kinase Kinase 5 - genetics | Mitogen-Activated Protein Kinase 7 - metabolism | Aorta, Thoracic - physiopathology | Plaque, Atherosclerotic | Regional Blood Flow | Mechanotransduction, Cellular | Aortic Diseases - genetics | Aortic Diseases - physiopathology | Animals | Carotid Arteries - physiopathology | Carotid Artery Diseases - genetics | Human Umbilical Vein Endothelial Cells - pathology | Carotid Arteries - pathology | Endothelial Cells - pathology | Index Medicus
Journal Article
Nature Medicine, ISSN 1078-8956, 10/2016, Volume 22, Issue 10, pp. 1140 - 1150
Adenosine-to-inosine (A-to-I) RNA editing, which is catalyzed by a family of adenosine deaminase acting on RNA (ADAR) enzymes, is important in the... 
MEDICINE, RESEARCH & EXPERIMENTAL | ANGIOGENESIS | STABILIZATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | LUNG INJURY | CELL BIOLOGY | MESSENGER-RNA | GENE-EXPRESSION | ALU ELEMENTS | HUMAN TRANSCRIPTOME | BINDING PROTEIN HUR | TUMOR-GROWTH | ENZYME ADAR1 | Human Umbilical Vein Endothelial Cells | RNA-Binding Proteins - genetics | RNA Editing - genetics | Atherosclerosis - genetics | Humans | Middle Aged | Immunoblotting | Male | RNA, Messenger - metabolism | Gene Knockdown Techniques | Aged, 80 and over | Adult | Female | 3' Untranslated Regions | Real-Time Polymerase Chain Reaction | Adenosine Deaminase - genetics | RNA Processing, Post-Transcriptional - drug effects | ELAV-Like Protein 1 - genetics | Gene Expression Regulation | Inosine - metabolism | Gene Knock-In Techniques | Tumor Necrosis Factor-alpha - pharmacology | Hypoxia - genetics | RNA Processing, Post-Transcriptional - genetics | Sequence Analysis, RNA | Cathepsins - genetics | Carotid Artery Diseases - genetics | Coronary Artery Disease - genetics | Fluorescent Antibody Technique | Adenosine - metabolism | Aged | High-Throughput Nucleotide Sequencing | Aortic Aneurysm - genetics | Interferon-gamma - pharmacology | RNA Editing - drug effects | Adenosine | Atherosclerosis | Genetic aspects | Dosage and administration | Research | Gene expression | Drug therapy | Post-transcription | Gene regulation | Cysteine proteinase | Editing | Cardiovascular disease | Proteins | Angiogenesis | Control | Vascular diseases | Aorta | Heart diseases | HuR protein | RNA editing | Cytokines | Coronary artery | Inflammation | Cathepsin S | Adenosine deaminase | Metabolism | Coronary artery disease | Substrates | Endothelial cells | RNA-binding protein | Ribonucleic acids | 3' Untranslated regions | Arteriosclerosis | Hypoxia | Control stability | Interferon | mRNA stability | Index Medicus
Journal Article
Kidney International, ISSN 0085-2538, 03/2018, Volume 93, Issue 3, pp. 727 - 732
Prior studies reported associations of nephropathy risk variants with subclinical atherosclerosis. However, these findings were limited to older individuals... 
APOL1 | cardiovascular disease | coronary artery calcification | KIDNEY-DISEASE | CORONARY-ARTERY CALCIUM | CARDIA | QUANTIFICATION | GENETIC-VARIANTS | FUNCTION DECLINE | DENSITY-LIPOPROTEIN CHOLESTEROL | CARDIOVASCULAR-DISEASE | UROLOGY & NEPHROLOGY | AFRICAN-AMERICANS | ECHOCARDIOGRAPHY | Kidney Diseases - physiopathology | United States - epidemiology | Prevalence | Prospective Studies | Humans | Kidney Diseases - diagnosis | Middle Aged | Male | Asymptomatic Diseases | Kidney Diseases - genetics | Incidence | Genetic Variation | Carotid Artery Diseases - diagnostic imaging | Hypertrophy, Left Ventricular - genetics | Adult | Female | Kidney - physiopathology | Genetic Predisposition to Disease | Glomerular Filtration Rate | Genetic Association Studies | Risk Assessment | Risk Factors | African Americans - genetics | Apolipoprotein L1 - genetics | Carotid Artery Diseases - ethnology | Kidney Diseases - ethnology | Coronary Artery Disease - ethnology | Coronary Artery Disease - diagnostic imaging | Phenotype | Age of Onset | Carotid Artery Diseases - genetics | Coronary Artery Disease - genetics | Hypertrophy, Left Ventricular - ethnology | Hypertrophy, Left Ventricular - diagnostic imaging | Heart | Calcification (ectopic) | Nephropathy | Kidneys | Risk groups | Arteriosclerosis | Coronary artery | Ventricle | Regression analysis | Hypertrophy | Glomerular filtration rate | Index Medicus
Journal Article
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 10/2009, Volume 297, Issue 4, pp. 1535 - 1543
Atherosclerosis is closely associated with disturbed flow characterized by low and oscillatory shear stress, but studies directly linking disturbed flow to... 
Endothelial dysfunction | Shear stress | Inflammation | Atherosclerosis | Endothelium | atherosclerosis | endothelium | CELLS | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | FLUID SHEAR-STRESS | NITRIC-OXIDE SYNTHASE | BONE MORPHOGENIC PROTEIN-4 | MOUSE AORTAS | endothelial dysfunction | IN-VITRO | shear stress | inflammation | PERIPHERAL VASCULAR DISEASE | E-DEFICIENT MICE | EXPRESSION | NADPH OXIDASES | Carotid Arteries - drug effects | Carotid Arteries - metabolism | Apolipoproteins E - deficiency | Tunica Intima - physiopathology | Carotid Artery Diseases - physiopathology | Stress, Mechanical | Endothelium, Vascular - drug effects | Male | Vasodilation | Dose-Response Relationship, Drug | NADPH Oxidases - deficiency | RNA - isolation & purification | Ligation | Time Factors | Carotid Artery Diseases - pathology | Computer Simulation | Dietary Fats - administration & dosage | Superoxides - metabolism | NADPH Oxidases - genetics | Female | Disease Models, Animal | Carotid Artery Diseases - etiology | Vasodilator Agents - pharmacology | Models, Cardiovascular | Mice, Inbred C57BL | Endothelium, Vascular - physiopathology | Gene Expression Regulation | Regional Blood Flow | Disease Progression | Mice, Knockout | Tunica Intima - metabolism | Animals | Tunica Intima - pathology | Carotid Arteries - physiopathology | Apolipoproteins E - genetics | Carotid Artery Diseases - genetics | Carotid Arteries - surgery | Endothelium, Vascular - pathology | Carotid Arteries - pathology | Mice | Physiological aspects | Research | RNA | Risk factors | Arteries | Models | Ribonucleic acid--RNA | Arteriosclerosis | Genes | Veins & arteries | Index Medicus | Innovative Methodology
Journal Article
Stroke, ISSN 0039-2499, 01/2014, Volume 45, Issue 1, pp. 18 - 23
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 07/2011, Volume 31, Issue 7, pp. 1565 - 1572
OBJECTIVE—Interleukin 17A (IL17A) is involved in many inflammatory processes, but its role in atherosclerosis remains controversial. We examined the role of... 
atherosclerosis | nitric oxide | cytokines | vascular biology | superoxide | IL-17A | INTACT | FOAM CELLS | PLAQUE | MONOCYTE MIGRATION | PERIPHERAL VASCULAR DISEASE | DYSFUNCTION | TISSUES | HEMATOLOGY | SUPEROXIDE-DISMUTASE | T-CELLS | Antibodies, Neutralizing - administration & dosage | Inflammation - pathology | Spleen - immunology | Apolipoproteins E - deficiency | Atherosclerosis - genetics | Dendritic Cells - immunology | Humans | Middle Aged | Interleukin-17 - immunology | Male | Aorta - metabolism | Interferon-gamma - metabolism | Aortic Diseases - metabolism | Inflammation - metabolism | Ligation | Lymphocytes - immunology | Time Factors | Carotid Artery Diseases - pathology | Dietary Fats | Superoxides - metabolism | Female | Angiotensin II | Carotid Artery Diseases - metabolism | Aortic Diseases - pathology | Disease Models, Animal | Carotid Artery Diseases - immunology | Interleukin-17 - blood | Atherosclerosis - pathology | Atherosclerosis - immunology | Aortic Diseases - immunology | Mice, Inbred C57BL | Cells, Cultured | Interleukin-17 - genetics | Aorta - immunology | Inflammation - immunology | Interleukin-17 - deficiency | Atherosclerosis - metabolism | Mice, Knockout | Interleukin-17 - metabolism | Aortic Diseases - genetics | Animals | Apolipoproteins E - genetics | Carotid Artery Diseases - genetics | Inflammation - genetics | Carotid Arteries - surgery | Aged | Mice | Nitric Oxide - metabolism | Index Medicus | Interferon-gamma | Reactive oxygen species | Apolipoprotein E | Interleukin 17 | Atherosclerosis
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 08/2017, Volume 37, Issue 8, pp. 1457 - 1461
Objective-Inflammasomes are multiprotein complexes, and their activation has been associated with cardiovascular disease. Inflammasome activation leads to... 
atherosclerosis | macrophages | inflammasomes | interleukin-1beta | CELLS | PROTEIN | INTERLEUKIN-1 RECEPTOR ANTAGONIST | CANAKINUMAB | CRYSTALS | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | EXPRESSION | Carotid Arteries - drug effects | Carotid Arteries - metabolism | Carotid Artery Diseases - prevention & control | Inflammasomes - metabolism | Apolipoproteins E - deficiency | Atherosclerosis - genetics | RNA, Messenger - metabolism | Sulfones - pharmacology | Heterocyclic Compounds, 4 or More Rings - pharmacology | Necrosis | Inflammasomes - antagonists & inhibitors | Time Factors | Carotid Artery Diseases - pathology | Interleukin-1beta - metabolism | Dendritic Cells - drug effects | Female | Vascular Cell Adhesion Molecule-1 - genetics | Carotid Artery Diseases - metabolism | Diet, Western | Dendritic Cells - metabolism | Disease Models, Animal | Atherosclerosis - pathology | Genetic Predisposition to Disease | Anti-Inflammatory Agents - pharmacology | NLR Family, Pyrin Domain-Containing 3 Protein - metabolism | Mice, Inbred C57BL | RNA, Messenger - genetics | Cells, Cultured | Crystallization | NLR Family, Pyrin Domain-Containing 3 Protein - antagonists & inhibitors | Plaque, Atherosclerotic | Atherosclerosis - metabolism | Mice, Knockout | Intercellular Adhesion Molecule-1 - metabolism | Macrophages - metabolism | Phenotype | Animals | Signal Transduction - drug effects | Apolipoproteins E - genetics | Intercellular Adhesion Molecule-1 - genetics | Carotid Artery Diseases - genetics | Lipopolysaccharides - pharmacology | Cholesterol - pharmacology | Macrophages - drug effects | Carotid Arteries - pathology | Vascular Cell Adhesion Molecule-1 - metabolism | Atherosclerosis - prevention & control | Index Medicus
Journal Article