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Circulation Research, ISSN 0009-7330, 02/2009, Volume 104, Issue 4, pp. 476 - 487
MicroRNAs (miRNAs) comprise a novel class of endogenous, small, noncoding RNAs that negatively regulate gene expression. Functionally, an individual miRNA is... 
Vascular disease | Proliferation | Gene regulation | MicroRNAs | Vascular smooth muscle cells | microRNAs | CARDIAC & CARDIOVASCULAR SYSTEMS | proliferation | DOWN-REGULATION | CARDIOVASCULAR BIOLOGY | P27(KIP1) | BREAST-CANCER | INHIBITION | vascular disease | MESSENGER-RNA | gene regulation | RAT CAROTID-ARTERY | GROWTH | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | vascular smooth muscle cells | MICRORNA EXPRESSION | PROGRESSION | Carotid Artery Diseases - prevention & control | Cyclin-Dependent Kinase Inhibitor p57 - metabolism | Cell Proliferation | Muscle, Smooth, Vascular - metabolism | Hyperplasia | Myocytes, Smooth Muscle - pathology | Oligonucleotides, Antisense - metabolism | Male | MicroRNAs - metabolism | Angioplasty, Balloon - adverse effects | Gene Knockdown Techniques | Cyclin-Dependent Kinase Inhibitor p27 - metabolism | RNA Interference | Time Factors | Carotid Artery Diseases - pathology | Carotid Artery Diseases - metabolism | Myocytes, Smooth Muscle - metabolism | Disease Models, Animal | Carotid Artery, Common - metabolism | Proto-Oncogene Proteins c-sis | Cells, Cultured | Rats | Rats, Sprague-Dawley | Tunica Intima - metabolism | Muscle, Smooth, Vascular - pathology | Platelet-Derived Growth Factor - metabolism | Animals | Tunica Intima - pathology | Carotid Artery Diseases - genetics | Carotid Artery, Common - pathology | RNA, Small Interfering - metabolism | Index Medicus
Journal Article
Cardiovascular Research, ISSN 0008-6363, 12/2015, Volume 108, Issue 3, pp. 377 - 386
Neointimal hyperplasia is a common feature of fibro-proliferative vascular disease and characterizes initial stages of atherosclerosis. Neointimal lesions... 
Shear stress | Endothelial-to-mesenchymal transition | Neointimal hyperplasia | ERK5 | Fibrosis | ARTERIAL INJURY | MYOCARDIN | ACTIVATION | CARDIAC & CARDIOVASCULAR SYSTEMS | ATHEROSCLEROSIS | MECHANISMS | FLOW | GROWTH | SMOOTH-MUSCLE-CELLS | EXPRESSION | TRANSDIFFERENTIATION | Carotid Arteries - metabolism | Vascular Remodeling | Cell Proliferation | Human Umbilical Vein Endothelial Cells - metabolism | Humans | Carotid Artery Diseases - physiopathology | Stress, Mechanical | Male | Aortic Diseases - metabolism | Neointima | Transfection | RNA Interference | Time Factors | Carotid Artery Diseases - pathology | Swine | MAP Kinase Kinase 5 - metabolism | Epithelial-Mesenchymal Transition | HEK293 Cells | Carotid Artery Diseases - metabolism | Aorta, Thoracic - pathology | Aortic Diseases - pathology | Disease Models, Animal | Endothelial Cells - metabolism | Mice, Inbred C57BL | Aorta, Thoracic - metabolism | Mitogen-Activated Protein Kinase 7 - genetics | MAP Kinase Kinase 5 - genetics | Mitogen-Activated Protein Kinase 7 - metabolism | Aorta, Thoracic - physiopathology | Plaque, Atherosclerotic | Regional Blood Flow | Mechanotransduction, Cellular | Aortic Diseases - genetics | Aortic Diseases - physiopathology | Animals | Carotid Arteries - physiopathology | Carotid Artery Diseases - genetics | Human Umbilical Vein Endothelial Cells - pathology | Carotid Arteries - pathology | Endothelial Cells - pathology | Index Medicus
Journal Article
Circulation, ISSN 0009-7322, 10/2013, Volume 128, Issue 18, pp. 2026 - 2038
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 08/2017, Volume 37, Issue 8, pp. 1457 - 1461
Objective-Inflammasomes are multiprotein complexes, and their activation has been associated with cardiovascular disease. Inflammasome activation leads to... 
atherosclerosis | macrophages | inflammasomes | interleukin-1beta | CELLS | PROTEIN | INTERLEUKIN-1 RECEPTOR ANTAGONIST | CANAKINUMAB | CRYSTALS | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | EXPRESSION | Carotid Arteries - drug effects | Carotid Arteries - metabolism | Carotid Artery Diseases - prevention & control | Inflammasomes - metabolism | Apolipoproteins E - deficiency | Atherosclerosis - genetics | RNA, Messenger - metabolism | Sulfones - pharmacology | Heterocyclic Compounds, 4 or More Rings - pharmacology | Necrosis | Inflammasomes - antagonists & inhibitors | Time Factors | Carotid Artery Diseases - pathology | Interleukin-1beta - metabolism | Dendritic Cells - drug effects | Female | Vascular Cell Adhesion Molecule-1 - genetics | Carotid Artery Diseases - metabolism | Diet, Western | Dendritic Cells - metabolism | Disease Models, Animal | Atherosclerosis - pathology | Genetic Predisposition to Disease | Anti-Inflammatory Agents - pharmacology | NLR Family, Pyrin Domain-Containing 3 Protein - metabolism | Mice, Inbred C57BL | RNA, Messenger - genetics | Cells, Cultured | Crystallization | NLR Family, Pyrin Domain-Containing 3 Protein - antagonists & inhibitors | Plaque, Atherosclerotic | Atherosclerosis - metabolism | Mice, Knockout | Intercellular Adhesion Molecule-1 - metabolism | Macrophages - metabolism | Phenotype | Animals | Signal Transduction - drug effects | Apolipoproteins E - genetics | Intercellular Adhesion Molecule-1 - genetics | Carotid Artery Diseases - genetics | Lipopolysaccharides - pharmacology | Cholesterol - pharmacology | Macrophages - drug effects | Carotid Arteries - pathology | Vascular Cell Adhesion Molecule-1 - metabolism | Atherosclerosis - prevention & control | Index Medicus
Journal Article
Circulation Research, ISSN 0009-7330, 06/2013, Volume 113, Issue 1, pp. 72 - 77
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 07/2011, Volume 31, Issue 7, pp. 1565 - 1572
OBJECTIVE—Interleukin 17A (IL17A) is involved in many inflammatory processes, but its role in atherosclerosis remains controversial. We examined the role of... 
atherosclerosis | nitric oxide | cytokines | vascular biology | superoxide | IL-17A | INTACT | FOAM CELLS | PLAQUE | MONOCYTE MIGRATION | PERIPHERAL VASCULAR DISEASE | DYSFUNCTION | TISSUES | HEMATOLOGY | SUPEROXIDE-DISMUTASE | T-CELLS | Antibodies, Neutralizing - administration & dosage | Inflammation - pathology | Spleen - immunology | Apolipoproteins E - deficiency | Atherosclerosis - genetics | Dendritic Cells - immunology | Humans | Middle Aged | Interleukin-17 - immunology | Male | Aorta - metabolism | Interferon-gamma - metabolism | Aortic Diseases - metabolism | Inflammation - metabolism | Ligation | Lymphocytes - immunology | Time Factors | Carotid Artery Diseases - pathology | Dietary Fats | Superoxides - metabolism | Female | Angiotensin II | Carotid Artery Diseases - metabolism | Aortic Diseases - pathology | Disease Models, Animal | Carotid Artery Diseases - immunology | Interleukin-17 - blood | Atherosclerosis - pathology | Atherosclerosis - immunology | Aortic Diseases - immunology | Mice, Inbred C57BL | Cells, Cultured | Interleukin-17 - genetics | Aorta - immunology | Inflammation - immunology | Interleukin-17 - deficiency | Atherosclerosis - metabolism | Mice, Knockout | Interleukin-17 - metabolism | Aortic Diseases - genetics | Animals | Apolipoproteins E - genetics | Carotid Artery Diseases - genetics | Inflammation - genetics | Carotid Arteries - surgery | Aged | Mice | Nitric Oxide - metabolism | Index Medicus | Interferon-gamma | Reactive oxygen species | Apolipoprotein E | Interleukin 17 | Atherosclerosis
Journal Article
Cardiovascular Research, ISSN 0008-6363, 11/2017, Volume 113, Issue 13, pp. 1627 - 1638
Journal Article
Journal of the American College of Cardiology, ISSN 0735-1097, 03/2016, Volume 67, Issue 11, pp. 1263 - 1274
Journal Article
Circulation, ISSN 0009-7322, 10/2014, Volume 130, Issue 17, pp. 1452 - 1465
Journal Article
The New England Journal of Medicine, ISSN 0028-4793, 01/2011, Volume 364, Issue 2, pp. 127 - 135
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 09/2016, Volume 36, Issue 9, pp. 1947 - 1961
OBJECTIVE—Key augmented processes in atherosclerosis have been identified, whereas less is known about downregulated pathways. Here, we applied a systems... 
Actin cytoskeleton | Downregulation | Smooth muscle cells | Hyperplasia | Atherosclerosis | atherosclerosis | VASCULAR DEVELOPMENT | ALPHA-ACTIN | PROTEIN | downregulation | NETWORKS | PLAQUE | actin cytoskeleton | smooth muscle cells | RAT CAROTID-ARTERY | DISEASE | GENE-EXPRESSION | SYNAPTOPODIN | hyperplasia | PERIPHERAL VASCULAR DISEASE | DIFFERENTIATION | HEMATOLOGY | Carotid Arteries - metabolism | Apolipoproteins E - deficiency | Cytoskeletal Proteins - genetics | Atherosclerosis - genetics | Humans | Middle Aged | Carotid Artery Diseases - physiopathology | Cell Dedifferentiation | Male | LIM Domain Proteins - metabolism | Autoantigens - genetics | Neointima | RNA Interference | Time Factors | Carotid Artery Diseases - pathology | Carotid Artery Injuries - genetics | Disease Models, Animal | Atherosclerosis - pathology | Actin Cytoskeleton - metabolism | Signal Transduction | Down-Regulation | Plaque, Atherosclerotic | Rats, Sprague-Dawley | Atherosclerosis - metabolism | Mice, Knockout | Phenotype | Carotid Artery Diseases - genetics | Calcium-Binding Proteins - genetics | Autoantigens - metabolism | Muscle, Smooth, Vascular - metabolism | Myocytes, Smooth Muscle - pathology | Muscle, Smooth, Vascular - physiopathology | Case-Control Studies | Transfection | Intermediate Filament Proteins - genetics | Cytoskeletal Proteins - metabolism | Microfilament Proteins - metabolism | Carotid Artery Diseases - metabolism | Microfilament Proteins - genetics | Myocytes, Smooth Muscle - metabolism | Carotid Artery Injuries - metabolism | Calcium-Binding Proteins - metabolism | Genetic Association Studies | Cells, Cultured | Vasoconstriction | Muscle, Smooth, Vascular - pathology | Animals | Carotid Arteries - physiopathology | Apolipoproteins E - genetics | Adaptor Proteins, Signal Transducing - genetics | LIM Domain Proteins - genetics | Carotid Arteries - pathology | Adaptor Proteins, Signal Transducing - metabolism | Intermediate Filament Proteins - metabolism | Index Medicus
Journal Article
Circulation, ISSN 0009-7322, 04/2009, Volume 119, Issue 13, pp. 1785 - 1794
Background-Clinical studies have demonstrated that 50% of individuals with chronic renal disease (CRD) die of cardiovascular causes, including advanced... 
Calcification | Chronic | Kidney failure | Aortic valve | Elastin | Atherosclerosis | atherosclerosis | kidney failure, chronic | aortic valve | ELASTIN PEPTIDES | CARDIAC & CARDIOVASCULAR SYSTEMS | ATHEROSCLEROTIC PLAQUES | VASCULAR CALCIFICATION | elastin | POTENTIAL MECHANISM | FAILURE | IN-VIVO | CYSTATIN-C | PERIPHERAL VASCULAR DISEASE | SMOOTH-MUSCLE-CELLS | calcification | CHRONIC KIDNEY-DISEASE | VULNERABLE PLAQUE | Tunica Media - pathology | Osteogenesis - physiology | Cystatin C - blood | Humans | Carotid Artery Diseases - physiopathology | Aorta - metabolism | Aortic Valve - pathology | Nephrectomy | Carotid Artery Diseases - pathology | Mice, Mutant Strains | Tunica Media - metabolism | Phosphates - blood | Calcinosis - metabolism | Carotid Artery Diseases - metabolism | Phosphates - pharmacology | Muscle, Smooth, Vascular - physiology | Kidney Failure, Chronic - physiopathology | Elastin - metabolism | Mice, Inbred C57BL | Osteogenesis - drug effects | Aortic Valve Stenosis - physiopathology | Cells, Cultured | Elastin - pharmacology | Muscle, Smooth, Vascular - cytology | Aortic Valve - metabolism | Kidney Failure, Chronic - metabolism | Aorta - pathology | Tunica Intima - metabolism | Animals | Aortic Valve Stenosis - pathology | Tunica Intima - pathology | Cathepsins - genetics | Cathepsins - metabolism | Apolipoproteins E - genetics | Creatinine - blood | Aortic Valve Stenosis - metabolism | Mice | Calcinosis - pathology | Cathepsins - deficiency | Calcinosis - physiopathology | Cathepsins | Physiological aspects | Research | Chronic kidney failure | Diseases | Index Medicus | Abridged Index Medicus
Journal Article