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EMBO molecular medicine, ISSN 1757-4676, 2013, Volume 5, Issue 3, pp. 471 - 481
... and receptors may not just be important in emigration of monocytes from the blood to the arteries, but they may also control mobilization from the bone marrow (BM... 
atherosclerosis | monocyte | chemokine | mobilization | recruitment | Chemokine | Mobilization | Monocyte | Atherosclerosis | Recruitment | ATHEROSCLEROTIC LESIONS | FRACTALKINE | MEDICINE, RESEARCH & EXPERIMENTAL | MACROPHAGES | BONE-MARROW | SUBSETS | NEUTROPHILS | MICE REVEALS | INFLAMMATION | ACCUMULATION | CCR5 | Carotid Arteries - metabolism | Chemotaxis, Leukocyte | Apolipoproteins E - deficiency | Atherosclerosis - genetics | Male | Monocytes - immunology | Adoptive Transfer | Aorta - metabolism | Aortic Diseases - metabolism | Atherosclerosis - etiology | Receptors, CCR5 - metabolism | Monocytes - transplantation | Leukocyte Reduction Procedures | Time Factors | Carotid Artery Diseases - pathology | Diet, High-Fat | Leukocyte Count | Chemokine CXCL1 - metabolism | Monocytes - classification | Receptors, Chemokine - genetics | Carotid Artery Diseases - metabolism | Aortic Diseases - pathology | Disease Models, Animal | Carotid Artery Diseases - immunology | Atherosclerosis - pathology | Atherosclerosis - immunology | Carotid Artery Diseases - etiology | Signal Transduction | Aortic Diseases - immunology | Mice, Inbred C57BL | Receptors, Chemokine - metabolism | Aorta - immunology | Atherosclerosis - metabolism | Disease Progression | Mice, Knockout | Receptors, Chemokine - deficiency | Aorta - pathology | Aortic Diseases - genetics | Animals | Apolipoproteins E - genetics | Receptors, Interleukin-8B - metabolism | Carotid Arteries - immunology | Carotid Artery Diseases - genetics | Receptors, CCR1 - metabolism | Carotid Arteries - pathology | Mice | Aortic Diseases - etiology | Apoptosis | Hypercholesterolemia | Universities and colleges | CC chemokine receptors | Funding | CX3CR1 protein | Leukocytes | Macrophages | Adoptive transfer | Blood | High fat diet | CCR1 protein | CCR2 protein | Apolipoprotein E | Bone marrow | Carotid artery | CCR5 protein | Nutrient deficiency | CXCR2 protein | Emigration | Inflammation | Monocytosis | Studies | Monocytes | Microscopy | Arteriosclerosis | Software | Chemokines | Monocyte chemoattractant protein 1
Journal Article
Cardiovascular research, ISSN 1755-3245, 2015, Volume 108, Issue 3, pp. 377 - 386
Neointimal hyperplasia is a common feature of fibro-proliferative vascular disease and characterizes initial stages of atherosclerosis... 
ARTERIAL INJURY | MYOCARDIN | PULMONARY-HYPERTENSION | Neointimal hyperplasia | ATHEROSCLEROSIS | CADHERIN EXPRESSION | SMOOTH-MUSCLE-CELL | FLOW | Fibrosis | ERK5 | IN-VIVO | GROWTH | Shear stress | GENE-EXPRESSION | Endothelial-to-mesenchymal transition | ACTIVATION | CARDIAC & CARDIOVASCULAR SYSTEMS | MECHANISMS | SMOOTH-MUSCLE-CELLS | EXPRESSION | TRANSDIFFERENTIATION | Carotid Arteries - metabolism | Vascular Remodeling | Cell Proliferation | Human Umbilical Vein Endothelial Cells - metabolism | Humans | Carotid Artery Diseases - physiopathology | Stress, Mechanical | Male | Aortic Diseases - metabolism | Neointima | Transfection | RNA Interference | Time Factors | Carotid Artery Diseases - pathology | Swine | MAP Kinase Kinase 5 - metabolism | Epithelial-Mesenchymal Transition | HEK293 Cells | Carotid Artery Diseases - metabolism | Aorta, Thoracic - pathology | Aortic Diseases - pathology | Disease Models, Animal | Endothelial Cells - metabolism | Mice, Inbred C57BL | Aorta, Thoracic - metabolism | Mitogen-Activated Protein Kinase 7 - genetics | MAP Kinase Kinase 5 - genetics | Mitogen-Activated Protein Kinase 7 - metabolism | Aorta, Thoracic - physiopathology | Plaque, Atherosclerotic | Regional Blood Flow | Mechanotransduction, Cellular | Aortic Diseases - genetics | Aortic Diseases - physiopathology | Animals | Carotid Arteries - physiopathology | Carotid Artery Diseases - genetics | Human Umbilical Vein Endothelial Cells - pathology | Carotid Arteries - pathology | Endothelial Cells - pathology
Journal Article
Arteriosclerosis, thrombosis, and vascular biology, ISSN 1079-5642, 12/2016, Volume 37, Issue 2, pp. 191 - 204
... appreciation of increased disease burden and higher levels of arterial calcification in these subjects. Plaques within the coronary arteries of patients with diabetes mellitus generally... 
atherosclerosis | coronary artery disease | carotid arteries | death, sudden | diabetes mellitus | 10-YEAR FOLLOW-UP | PLAQUE MORPHOLOGY | PROTEIN-KINASE-C | RISK-FACTORS | death | ALKALINE-PHOSPHATASE | BEAM COMPUTED-TOMOGRAPHY | sudden | ACUTE MYOCARDIAL-INFARCTION | GROWTH-FACTOR 23 | CARDIOVASCULAR-DISEASE | PERIPHERAL VASCULAR DISEASE | SMOOTH-MUSCLE-CELLS | HEMATOLOGY | Carotid Arteries - metabolism | Diabetes Mellitus, Type 1 - epidemiology | Vascular Remodeling | Prognosis | Coronary Vessels - physiopathology | Humans | Carotid Artery Diseases - physiopathology | Diabetes Mellitus, Type 1 - metabolism | Diabetes Mellitus, Type 2 - metabolism | Coronary Vessels - metabolism | Diabetes Mellitus, Type 2 - epidemiology | Vascular Calcification - metabolism | Diabetic Angiopathies - physiopathology | Diabetic Angiopathies - pathology | Coronary Artery Disease - physiopathology | Carotid Artery Diseases - pathology | Coronary Artery Disease - pathology | Vascular Calcification - mortality | Vascular Calcification - physiopathology | Carotid Artery Diseases - metabolism | Diabetic Angiopathies - metabolism | Coronary Vessels - pathology | Coronary Artery Disease - metabolism | Risk Factors | Diabetic Angiopathies - mortality | Carotid Artery Diseases - mortality | Plaque, Atherosclerotic | Vascular Calcification - pathology | Disease Progression | Diabetes Mellitus, Type 1 - mortality | Animals | Carotid Arteries - physiopathology | Diabetes Mellitus, Type 2 - mortality | Death, Sudden, Cardiac - pathology | Carotid Arteries - pathology | Rupture, Spontaneous
Journal Article
Arteriosclerosis, thrombosis, and vascular biology, ISSN 1079-5642, 02/2013, Volume 33, Issue 2, pp. 354 - 361
.... In apolipoprotein E (ApoE)-null and ApoE/fibromodulin-null mice, a shear stress-modifying carotid artery cast induced formation of atherosclerotic plaques of different phenotypes... 
atherosclerosis | collagen | carotid arteries | fibromodulin | LDL | PROTEOGLYCANS | MACROPHAGES | FIBRILLOGENESIS | LUMICAN | ABNORMAL COLLAGEN FIBRILS | PERIPHERAL VASCULAR DISEASE | SMOOTH-MUSCLE-CELLS | EXTRACELLULAR-MATRIX | GENE-TRANSFER | HEMATOLOGY | EXPRESSION | BINDING | Carotid Arteries - metabolism | Aorta - ultrastructure | Cell Proliferation | Apolipoproteins E - deficiency | Atherosclerosis - genetics | Carotid Artery Diseases - physiopathology | Aorta - metabolism | Aortic Diseases - metabolism | Carotid Artery Diseases - pathology | Aortic Diseases - prevention & control | Lipoproteins, LDL - metabolism | Interleukin-6 - metabolism | Disease Models, Animal | Fibrillar Collagens - metabolism | Atherosclerosis - pathology | Atherosclerosis - immunology | Aortic Diseases - immunology | Atherosclerosis - physiopathology | Down-Regulation | Extracellular Matrix Proteins - genetics | Aorta - immunology | Genotype | Plaque, Atherosclerotic | Atherosclerosis - metabolism | Mice, Knockout | Aortic Diseases - genetics | Macrophages - metabolism | Phenotype | Carotid Arteries - immunology | Carotid Artery Diseases - genetics | Mice | Proteoglycans - genetics | Carotid Artery Diseases - prevention & control | Extracellular Matrix Proteins - deficiency | Stress, Mechanical | Proteoglycans - deficiency | Fibrillar Collagens - ultrastructure | Interleukin-10 - metabolism | Aorta - physiopathology | Carotid Artery Diseases - metabolism | Macrophages - immunology | Aortic Diseases - pathology | Carotid Artery Diseases - immunology | Fibromodulin | Cell Line | Microscopy, Electron, Transmission | Mice, Inbred C57BL | Regional Blood Flow | Aortic Diseases - physiopathology | Animals | Carotid Arteries - ultrastructure | Carotid Arteries - physiopathology | Apolipoproteins E - genetics | Atherosclerosis - prevention & control
Journal Article
PloS one, ISSN 1932-6203, 06/2013, Volume 8, Issue 6, p. e68197
Free fatty acids (FFAs), elevated in metabolic syndrome and diabetes, play a crucial role in the development of atherosclerotic cardiovascular disease, and eicosapentaenoic acid (EPA... 
SUPPLEMENTATION | ARTERY ENDOTHELIAL-CELLS | PROTEIN-KINASE | MULTIDISCIPLINARY SCIENCES | DISEASE | CALCIUM | CALCIFICATION | RISK | FREE FATTY-ACID | DEPENDENT ACTIVATION | EXPRESSION | Calcinosis - genetics | Carotid Arteries - metabolism | Osteopontin - genetics | Palmitic Acid - metabolism | Coenzyme A Ligases - genetics | Muscle, Smooth, Vascular - metabolism | Calcium - metabolism | Gene Expression - genetics | Homeodomain Proteins - metabolism | Humans | Eicosapentaenoic Acid - genetics | Myocytes, Smooth Muscle - pathology | NF-kappa B - metabolism | Osteopontin - metabolism | Aorta - metabolism | Promoter Regions, Genetic - genetics | Coenzyme A Ligases - metabolism | Cell Differentiation - genetics | Caspases - metabolism | Bone Morphogenetic Protein 2 - metabolism | Calcinosis - metabolism | Plaque, Atherosclerotic - pathology | Cell Differentiation - physiology | Myocytes, Smooth Muscle - metabolism | Bone Morphogenetic Protein 2 - genetics | Macrophages - pathology | Plaque, Atherosclerotic - metabolism | Caspases - genetics | Cells, Cultured | Eicosapentaenoic Acid - metabolism | Homeodomain Proteins - genetics | Aorta - pathology | Osteoblasts - pathology | Macrophages - metabolism | Muscle, Smooth, Vascular - pathology | NF-kappa B - genetics | Carotid Arteries - pathology | Calcinosis - pathology | Osteoblasts - metabolism | Immunohistochemistry | Smooth muscle | Cardiovascular disease | Palmitic acid | Osteoblasts | Proteins | Mineralization | Biocompatibility | Aorta | Inhibition | NF-κB protein | Cytokines | Osteopontin | Metabolism | Gene expression | Fatty acids | Studies | Osteoblastogenesis | Inhibitors | Arteriosclerosis | Calcification | Cardiovascular diseases | Metabolic disorders | Calcification (ectopic) | Health sciences | Oxidative stress | Calcium | Laboratories | Chains | Lipids | Activation | Kinases | Macrophages | Carotid arteries | Arteries | Msx2 protein | Rodents | Atherosclerosis | Tumor necrosis factor-TNF | Carotid artery | Deposition | Plaques | University graduates | Bone morphogenetic protein 2 | Diabetes mellitus | Muscles | Caspase | siRNA | Medicine | Antibiotics | Kidney diseases | Diabetes | Differentiation | Eicosapentaenoic acid | Apoptosis | Pharmaceuticals
Journal Article
Journal of the American College of Cardiology, ISSN 0735-1097, 03/2016, Volume 67, Issue 11, pp. 1263 - 1274
Journal Article
Journal of magnetic resonance imaging, ISSN 1053-1807, 04/2017, Volume 45, Issue 4, pp. 1144 - 1153
...) of vascular inflammation and angiogenesis in experimental carotid disease and abdominal aortic aneurysm (AAA... 
atherosclerosis | iron | nanoparticles | angiogenesis | RGD | aneurysms | LIMITS | CELLS | NEOVASCULARIZATION | PLAQUE | RADIOLOGY, NUCLEAR MEDICINE & MEDICAL IMAGING | Carotid Arteries - metabolism | Carotid Artery Diseases - physiopathology | Magnetic Resonance Imaging - methods | Aortic Aneurysm, Abdominal - diagnostic imaging | Male | Neovascularization, Pathologic - diagnostic imaging | Aortic Aneurysm, Abdominal - metabolism | Nanoparticles | Inflammation - metabolism | Carotid Artery Diseases - diagnostic imaging | Ferritins - metabolism | Neovascularization, Pathologic - physiopathology | Inflammation - diagnostic imaging | Carotid Artery Diseases - metabolism | Disease Models, Animal | Ferric Compounds - metabolism | Vascular Diseases - physiopathology | Oligopeptides - metabolism | Vascular Diseases - diagnostic imaging | Animals | Carotid Arteries - diagnostic imaging | Aortic Aneurysm, Abdominal - physiopathology | Carotid Arteries - physiopathology | Mice | Neovascularization, Pathologic - metabolism | Vascular Diseases - metabolism | Inflammation - physiopathology | Ferritin | Inflammation | Glycine | Abdominal aneurysm | Macrophages | Apolipoproteins | Magnetic resonance imaging | Ferric oxide | Genetically modified organisms | Atherosclerosis | Angiotensin | Genetic engineering | Nuclear magnetic resonance--NMR | Intravenous administration | Aneurysm | Aneurysms | Iron | Accumulation | Angiogenesis | Vascular diseases | Apolipoprotein E | Aorta | Carotid artery | Lesions | Angiotensin II | Diabetes mellitus | Health risks | Histology | Endothelial cells | Arteriosclerosis | Iron oxides | Original Research
Journal Article
PloS one, ISSN 1932-6203, 2017, Volume 12, Issue 4, p. e0175762
.... However, the prognosis of this subpopulation remains controversial. We aimed to examine whether MHO phenotype is associated with progression of atherosclerotic activity, reflected as the changes in coronary artery calcification (CAC) over time... 
FRAMINGHAM | MORTALITY | DEFINITION | ALL-CAUSE | MULTIDISCIPLINARY SCIENCES | CALCIUM | CARDIOVASCULAR-DISEASE | ATHEROSCLEROSIS | RISK | ASSOCIATION | PREDICTION | Follow-Up Studies | Humans | Middle Aged | Fatty Liver - complications | Male | Coronary Vessels - metabolism | Atherosclerosis - etiology | Vascular Calcification - metabolism | Vascular Calcification - etiology | Coronary Artery Disease - pathology | Atherosclerosis - diagnosis | Female | Vascular Calcification - diagnosis | Atherosclerosis - pathology | Coronary Vessels - pathology | Coronary Artery Disease - etiology | Fatty Liver - metabolism | Obesity - complications | Coronary Artery Disease - metabolism | Risk Factors | Vascular Calcification - pathology | Atherosclerosis - metabolism | Disease Progression | Obesity - metabolism | Coronary Artery Disease - diagnosis | Obesity | Complications and side effects | Fatty liver | Calcification | Development and progression | Research | Risk factors | Myocardial infarction | Heart | Inventories | Spectroscopic analysis | Cardiovascular disease | Incidence | Genotype & phenotype | Computed tomography | Surgery | Hemoglobin | Blood pressure | Cross sections | Spectroscopy | Liver diseases | Internal medicine | Drinking behavior | Data acquisition | Mortality | Coronary artery | Benign | Metabolism | Studies | Cirrhosis | Browning | Computation | Arteriosclerosis | Cardiovascular diseases | Circulation | Kidney transplantation | Calcification (ectopic) | Populations | Angiography | Risk | Lipids | Population studies | Angina | Hepatology | Atherosclerosis | Reliability analysis | Population | Carotid artery | Ultrasound | Heart diseases | Fasting | Medical imaging | Health risks | Medical screening | Coronary artery disease | Ethnic factors | Meta-analysis | Medicine | Coronary vessels | Diabetes | Veins & arteries | Accidents
Journal Article
American journal of physiology. Heart and circulatory physiology, ISSN 1522-1539, 2009, Volume 297, Issue 4, pp. H1535 - H1543
.... We also describe a method of isolating intimal RNA in sufficient quantity from mouse carotid arteries... 
Endothelial dysfunction | Shear stress | Inflammation | Atherosclerosis | Endothelium | atherosclerosis | endothelium | CELLS | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | FLUID SHEAR-STRESS | NITRIC-OXIDE SYNTHASE | BONE MORPHOGENIC PROTEIN-4 | MOUSE AORTAS | endothelial dysfunction | IN-VITRO | shear stress | inflammation | PERIPHERAL VASCULAR DISEASE | E-DEFICIENT MICE | EXPRESSION | NADPH OXIDASES | Carotid Arteries - drug effects | Carotid Arteries - metabolism | Apolipoproteins E - deficiency | Tunica Intima - physiopathology | Carotid Artery Diseases - physiopathology | Stress, Mechanical | Endothelium, Vascular - drug effects | Male | Vasodilation | Dose-Response Relationship, Drug | NADPH Oxidases - deficiency | RNA - isolation & purification | Ligation | Time Factors | Carotid Artery Diseases - pathology | Computer Simulation | Dietary Fats - administration & dosage | Superoxides - metabolism | NADPH Oxidases - genetics | Female | Disease Models, Animal | Carotid Artery Diseases - etiology | Vasodilator Agents - pharmacology | Models, Cardiovascular | Mice, Inbred C57BL | Endothelium, Vascular - physiopathology | Gene Expression Regulation | Regional Blood Flow | Disease Progression | Mice, Knockout | Tunica Intima - metabolism | Animals | Tunica Intima - pathology | Carotid Arteries - physiopathology | Apolipoproteins E - genetics | Carotid Artery Diseases - genetics | Carotid Arteries - surgery | Endothelium, Vascular - pathology | Carotid Arteries - pathology | Mice | Physiological aspects | Research | RNA | Risk factors | Arteries | Innovative Methodology
Journal Article
American journal of physiology. Heart and circulatory physiology, ISSN 0363-6135, 11/2011, Volume 301, Issue 5, pp. H2081 - H2091
.... Heart rate-associated mechanical stress impairs carotid but not cerebral artery compliance in dyslipidemic atherosclerotic mice... 
Resting heart rate | Endothelial function | Cerebrovascular compliance | endothelial function | INTIMA-MEDIA THICKNESS | RATE REDUCTION | WALL SHEAR-STRESS | FAMILIAL HYPERCHOLESTEROLEMIA | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | PULSE-WAVE VELOCITY | RISK-FACTORS | IMPROVES ENDOTHELIAL FUNCTION | NITRIC-OXIDE SYNTHASE | MATRIX METALLOPROTEINASES | cerebrovascular compliance | resting heart rate | PERIPHERAL VASCULAR DISEASE | HYPERTENSIVE-RATS | Carotid Arteries - drug effects | Carotid Arteries - metabolism | Compliance | Dyslipidemias - complications | Metoprolol - pharmacology | Anti-Arrhythmia Agents - pharmacology | Humans | Carotid Artery Diseases - physiopathology | Endothelium, Vascular - drug effects | Male | Vasodilation | Matrix Metalloproteinase 9 - metabolism | Atherosclerosis - therapy | Cerebrovascular Disorders - prevention & control | Time Factors | Dyslipidemias - therapy | Disease Models, Animal | Cerebrovascular Disorders - physiopathology | Atherosclerosis - physiopathology | Genotype | Mice, Transgenic | Apolipoproteins B - genetics | Benzazepines - pharmacology | Atherosclerosis - metabolism | Disease Progression | Mice, Knockout | Phenotype | Cerebral Arteries - metabolism | Dyslipidemias - metabolism | Cerebrovascular Disorders - genetics | Mice | Carotid Artery Diseases - prevention & control | Dyslipidemias - genetics | Apolipoproteins B - metabolism | Stress, Mechanical | Cerebrovascular Disorders - metabolism | Exercise Therapy | Cerebral Arteries - drug effects | Atherosclerosis - etiology | Heart Rate - drug effects | Receptors, LDL - deficiency | Nitric Oxide Synthase Type III - metabolism | Carotid Artery Diseases - metabolism | Severity of Illness Index | Receptors, LDL - genetics | Carotid Artery Diseases - etiology | Mice, Inbred C57BL | Endothelium, Vascular - physiopathology | Cerebral Arteries - physiopathology | Cerebrovascular Disorders - etiology | Animals | Carotid Arteries - physiopathology | Nitric Oxide - metabolism | Dyslipidemias - physiopathology | Heart beat | Atherosclerosis | Physiological aspects | Development and progression | Stress (Physiology) | Carotid artery | Cerebral arteries | Health aspects
Journal Article
Arteriosclerosis, thrombosis, and vascular biology, ISSN 1079-5642, 04/2015, Volume 35, Issue 4, pp. 817 - 828
Objective-Atherosclerosis, the cause of 50% of deaths in westernized societies, is widely regarded as a chronic vascular inflammatory disease... 
atherosclerosis | smooth muscle | myocardin | inflammation | APOLIPOPROTEIN E-DEFICIENT | MONOCYTE CHEMOATTRACTANT PROTEIN-1 | BINDING PROTEIN | SERUM RESPONSE FACTOR | CARDIOVASCULAR-DISEASE | GENE-EXPRESSION | PERIPHERAL VASCULAR DISEASE | MICE | HEMATOLOGY | NF-KAPPA-B | MOLECULAR-MECHANISMS | Inflammation - pathology | Apolipoproteins E - deficiency | Human Umbilical Vein Endothelial Cells - metabolism | Atherosclerosis - genetics | Humans | Muscle, Smooth, Vascular - immunology | Male | Monocytes - immunology | Mice, 129 Strain | Inflammation - metabolism | Neointima | RNA Interference | Time Factors | Nuclear Proteins - deficiency | Carotid Artery Injuries - genetics | Disease Models, Animal | Atherosclerosis - pathology | Atherosclerosis - immunology | Genotype | Lipid Metabolism | Cell Adhesion | Cell Adhesion Molecules - metabolism | Atherosclerosis - metabolism | Mice, Knockout | Macrophages - metabolism | Phenotype | Trans-Activators - deficiency | Carotid Artery Injuries - immunology | Chemokines - metabolism | Muscle, Smooth, Vascular - metabolism | Rats, Wistar | Myocytes, Smooth Muscle - pathology | Human Umbilical Vein Endothelial Cells - immunology | Monocytes - metabolism | Transfection | Trans-Activators - genetics | Nuclear Proteins - genetics | Myocytes, Smooth Muscle - metabolism | Carotid Artery Injuries - metabolism | Macrophages - immunology | Carotid Artery Injuries - pathology | Cytokines - metabolism | Mice, Inbred C57BL | Cells, Cultured | Nuclear Proteins - metabolism | Inflammation - immunology | Chemotaxis | Muscle, Smooth, Vascular - pathology | Animals | Apolipoproteins E - genetics | Inflammation - genetics | Myocytes, Smooth Muscle - immunology | Trans-Activators - metabolism | Smooth muscle | Inflammation | Myocardin | Atherosclerosis
Journal Article