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Journal of the American College of Cardiology, ISSN 0735-1097, 2008, Volume 52, Issue 25, pp. 2198 - 2205
Journal Article
Journal Article
Epilepsia, ISSN 0013-9580, 01/2012, Volume 53, Issue 1, pp. 120 - 128
Purpose:  Long‐term therapy with antiepileptic drugs (AEDs) has been associated with metabolic consequences that lead to an increase in risk of atherosclerosis... 
Antiepileptic drugs | Monotherapy | Vascular risk factors | Atherosclerosis | Intima media thickness | INTIMA-MEDIA THICKNESS | MORTALITY | HOMOCYSTEINE | MYOCARDIAL-INFARCTION | CARBAMAZEPINE | FOLIC-ACID | CLINICAL NEUROLOGY | SODIUM VALPROATE | URIC-ACID | CARDIOVASCULAR RISK | EPILEPTIC CHILDREN | Follow-Up Studies | Humans | Middle Aged | Anticonvulsants - administration & dosage | Male | Young Adult | Anticonvulsants - adverse effects | Carbamazepine - administration & dosage | Time Factors | Adult | Female | Uric Acid - blood | Valproic Acid - adverse effects | Cross-Sectional Studies | Atherosclerosis - chemically induced | Risk Factors | Homocysteine - blood | Phenytoin - administration & dosage | Blood Glucose - drug effects | Carbamazepine - adverse effects | Valproic Acid - administration & dosage | Phenytoin - adverse effects | Carotid Artery, Common - drug effects | Adolescent | Folic Acid - blood | Carotid Artery, Common - pathology | Epilepsy - drug therapy | Aged | Medical colleges | Anticonvulsants | Blood sugar | Epilepsy | Uric acid | Homocysteine | Risk factors | Cholesterol | Folic acid | Enzymes | Acids | Drugs | C-reactive protein | Fasting | lamotrigine | thiobarbituric acid | Antiepileptic agents | Metabolism | Valproic acid | Carbamazepine | Body mass index | Arteriosclerosis | Carotid artery | Phenytoin | Sugar | Long-term effects
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 05/2004, Volume 24, Issue 5, pp. 930 - 934
Objectives-Thiazolidinediones, such as rosiglitazone, have been shown to retard atherosclerosis disease progression in diabetic subjects. These agents may have... 
Agonists | Coronary artery disease | Thiazolidinediones | PPAR-γ | Carotid intima-media thickness | coronary artery disease | C-REACTIVE PROTEIN | WALL THICKNESS | thiazolidinediones | POPULATION | PROLIFERATOR-ACTIVATED RECEPTORS | ATHEROSCLEROSIS | DEFICIENT MICE | carotid intima-media thickness | PPAR-gamma | HEART-DISEASE | agonists | IMPROVES GLYCEMIC CONTROL | INSULIN-RESISTANCE | PERIPHERAL VASCULAR DISEASE | ULTRASONOGRAPHY | HEMATOLOGY | Carotid Artery, Common - diagnostic imaging | Humans | Hyperplasia | Middle Aged | Male | Anti-Inflammatory Agents, Non-Steroidal - pharmacology | Adrenergic beta-Antagonists - pharmacology | Thiazolidinediones - therapeutic use | Carotid Artery Diseases - diagnostic imaging | Carotid Artery Diseases - pathology | Lipids - blood | Coronary Artery Disease - pathology | Ultrasonography | Female | Weight Gain - drug effects | Thiazolidinediones - pharmacology | Adrenergic beta-Antagonists - therapeutic use | Carotid Artery Diseases - drug therapy | Double-Blind Method | Insulin Resistance | Receptors, Cytoplasmic and Nuclear - agonists | Coronary Artery Disease - drug therapy | Disease Progression | Blood Glucose - drug effects | Drug Synergism | Hydroxymethylglutaryl-CoA Reductase Inhibitors - pharmacology | Anti-Inflammatory Agents, Non-Steroidal - therapeutic use | Carotid Artery, Common - drug effects | Hydroxymethylglutaryl-CoA Reductase Inhibitors - therapeutic use | Carotid Artery, Common - pathology | Aged | Transcription Factors - agonists | Cohort Studies
Journal Article
Atherosclerosis, ISSN 0021-9150, 2015, Volume 241, Issue 1, pp. 111 - 120
Abstract In vitro , insulin has mitogenic effects on vascular smooth muscle cells (VSMC) but also has protective effects on endothelial cells by stimulating... 
Cardiovascular | Endothelial nitric oxide synthase | Neointima | Angioplasty | Insulin | Re-endothelialization | MIGRATION | ACTIVATION | PHOSPHORYLATION | MECHANISMS | RAT CAROTID-ARTERY | INTIMAL HYPERPLASIA | BALLOON INJURY | PERIPHERAL VASCULAR DISEASE | SMOOTH-MUSCLE-CELLS | EXPRESSION | IN-STENT RESTENOSIS | Femoral Artery - pathology | Phosphorylation | Carotid Artery Injuries - drug therapy | Drug Implants | Vascular System Injuries - enzymology | Male | Vascular System Injuries - drug therapy | Nitric Oxide Synthase Type III - deficiency | Dose-Response Relationship, Drug | Femoral Artery - enzymology | Vascular System Injuries - pathology | Femoral Artery - injuries | Time Factors | Aorta, Thoracic - drug effects | Nitric Oxide Synthase Type III - metabolism | Aorta, Thoracic - pathology | Carotid Artery Injuries - pathology | Disease Models, Animal | Vascular System Injuries - physiopathology | Vasodilator Agents - pharmacology | Mice, Inbred C57BL | Enzyme Inhibitors - pharmacology | Aorta, Thoracic - injuries | Insulin - administration & dosage | Re-Epithelialization - drug effects | Femoral Artery - drug effects | Nitric Oxide Synthase Type III - genetics | Rats, Sprague-Dawley | Mice, Knockout | Nitric Oxide Synthase Type III - antagonists & inhibitors | Aorta, Thoracic - enzymology | Cell Movement - drug effects | Animals | Carotid Artery Injuries - enzymology | Signal Transduction - drug effects | Carotid Artery Injuries - physiopathology | Carotid Artery, Common - enzymology | Carotid Artery, Common - drug effects | Carotid Artery, Common - pathology | Vasodilation - drug effects | Growth | Nitric oxide | Analysis | Physiological aspects | Statistics | Endothelium
Journal Article
Journal of Cellular Physiology, ISSN 0021-9541, 11/2017, Volume 232, Issue 11, pp. 3088 - 3102
The Pyk2 inhibitor PF‐4594755 potently inhibits Pyk2 phosphorylation of vascular smooth muscle cells stimulated by FBS or PDGF‐BB. This is accompanied by... 
phenotype | calcium | SERCA | vascular remodeling | PF‐459475 | PF-459475 | ACTIVATION | PHYSIOLOGY | PHENOTYPIC MODULATION | HEART-FAILURE | TRANSCRIPTION | TYROSINE KINASE PYK2 | CELL BIOLOGY | REGULATES SERCA2 | CA2 | GROWTH | GENE-EXPRESSION | ION-CHANNEL | Large-Conductance Calcium-Activated Potassium Channel beta Subunits - metabolism | RNA, Messenger - metabolism | Focal Adhesion Kinase 2 - antagonists & inhibitors | Muscle, Smooth, Vascular - physiopathology | Dose-Response Relationship, Drug | Focal Adhesion Kinase 2 - genetics | Transfection | Focal Adhesion Kinase 2 - metabolism | RNA Interference | Time Factors | Kruppel-Like Transcription Factors - metabolism | Carotid Artery Injuries - genetics | Myocytes, Smooth Muscle - drug effects | Proto-Oncogene Proteins c-sis - pharmacology | Organ Culture Techniques | Large-Conductance Calcium-Activated Potassium Channel beta Subunits - genetics | Muscle, Smooth, Vascular - drug effects | ORAI1 Protein - metabolism | Sarcoplasmic Reticulum Calcium-Transporting ATPases - metabolism | Carotid Artery, Common - physiopathology | ORAI1 Protein - genetics | Myocytes, Smooth Muscle - enzymology | RNA, Messenger - genetics | Gene Expression Regulation | Becaplermin | Rats, Sprague-Dawley | Vasoconstriction - drug effects | Cell Movement - drug effects | Phenotype | Animals | Calcium Channels, L-Type - genetics | Carotid Artery Injuries - enzymology | Calcium Signaling - drug effects | Carotid Artery Injuries - physiopathology | Carotid Artery, Common - enzymology | Cell Differentiation - drug effects | Carotid Artery, Common - drug effects | Cell Proliferation - drug effects | Protein Kinase Inhibitors - pharmacology | Calcium Channels, L-Type - metabolism | Sarcoplasmic Reticulum Calcium-Transporting ATPases - genetics | Kruppel-Like Transcription Factors - genetics | Muscle, Smooth, Vascular - enzymology | Smooth muscle | Calcium channels | Platelet-derived growth factor | RNA | Protein kinases | Cell proliferation | Cell culture | Electric potential | Calcium | Disorders | Stimulation | Segments | mRNA | Kinases | Carotid arteries | Calcium influx | Arteries | Potassium channels (calcium-gated) | Ca2+-transporting ATPase | Calcium signalling | Genotype & phenotype | KLF4 protein | Rodents | Modulation | Inhibition | Carotid artery | Calcium (intracellular) | Calcium channels (voltage-gated) | Markers | Contractility | Gene expression | Orai1 protein | Muscle contraction | Platelet-derived growth factor BB | Resistance | Inhibitors | Protein kinase | Calcium conductance | Calcium channels (L-type) | Organ culture | Conductance | Intracellular | Focal adhesion kinase | Differentiation | Potassium | Veins & arteries | Integrity | Index Medicus | Kardiologi | Clinical Medicine | Cardiac and Cardiovascular Systems | Medical and Health Sciences | Medicin och hälsovetenskap | Klinisk medicin
Journal Article
Journal of Internal Medicine, ISSN 0954-6820, 05/2007, Volume 261, Issue 5, pp. 472 - 479
Background and purpose.  Angiotensin II promotes cell growth and has been implicated in the development and maintenance of left ventricular (LV) hypertrophy... 
arteriosclerosis | left ventricular hypertrophy | carotid intima‐media thickness | hypertension | Hypertension | Arteriosclerosis | Carotid intima-media thickness | Left ventricular hypertrophy | WALL THICKNESS | CONTROLLED TRIAL | MYOCARDIAL-INFARCTION | ANGIOTENSIN-II | carotid intima-media thickness | BLOOD-PRESSURE | ATHEROSCLEROSIS ELSA | STROKE | MEDICINE, GENERAL & INTERNAL | EUROPEAN LACIDIPINE | TERM ANTIHYPERTENSIVE TREATMENT | DOUBLE-BLIND | Angiotensin II Type 1 Receptor Blockers - adverse effects | Angiotensin II Type 1 Receptor Blockers - therapeutic use | Humans | Middle Aged | Biphenyl Compounds - adverse effects | Hypertension - drug therapy | Male | Biphenyl Compounds - therapeutic use | Heart Rate - drug effects | Hypertrophy, Left Ventricular - pathology | Adult | Female | Blood Pressure - drug effects | Hypertrophy, Left Ventricular - drug therapy | Double-Blind Method | Treatment Outcome | Antihypertensive Agents - therapeutic use | Hypertension - pathology | Antihypertensive Agents - adverse effects | Tunica Intima - drug effects | Atenolol - therapeutic use | Tunica Intima - pathology | Carotid Artery, Common - drug effects | Atenolol - adverse effects | Carotid Artery, Common - pathology | Tetrazoles - therapeutic use | Aged | Tetrazoles - adverse effects | Medical research | Irbesartan | Heart enlargement | Medicine, Experimental | Comparative analysis | Atenolol | Biphenyl Compounds/adverse effects/therapeutic use | Hypertrophy; Left Ventricular/drug therapy/pathology | Hypertension/drug therapy/pathology | Medical and Health Sciences | Medicin och hälsovetenskap | Angiotensin II Type 1 Receptor Blockers/adverse effects/therapeutic use | Tunica Intima/drug effects/pathology | Atenolol/adverse effects/therapeutic use | MEDICINE | Antihypertensive Agents/adverse effects/therapeutic use | Carotid Artery; Common/drug effects/pathology | Blood Pressure/drug effects | MEDICIN | Heart Rate/drug effects | Tetrazoles/adverse effects/therapeutic use
Journal Article
Biochemical and Biophysical Research Communications, ISSN 0006-291X, 02/2018, Volume 496, Issue 2, pp. 415 - 421
Vascular smooth muscle cell (VSMC) proliferation and migration are crucial events in the pathological course of restenosis after percutaneous coronary... 
N-oleoylethanolamide | Proliferation | Intimal hyperplasia | Migration | Vascular smooth muscle cells | INHIBITION | BIOPHYSICS | FENOFIBRATE | BIOCHEMISTRY & MOLECULAR BIOLOGY | MUSCLE-CELL PROLIFERATION | Cyclin D1 - metabolism | AMP-Activated Protein Kinases - metabolism | Phosphorylation | Endocannabinoids - pharmacology | Muscle, Smooth, Vascular - metabolism | Carotid Artery Injuries - drug therapy | Male | Neointima - genetics | Hyperplasia - genetics | Proliferating Cell Nuclear Antigen - genetics | Carotid Artery Injuries - genetics | Oleic Acids - pharmacology | Carotid Artery Injuries - metabolism | Hyperplasia - metabolism | Carotid Artery Injuries - pathology | Hyperplasia - prevention & control | Muscle, Smooth, Vascular - drug effects | Cell Survival - drug effects | Carotid Artery, Common - metabolism | Endothelial Cells - metabolism | Rats | PPAR alpha - genetics | Platelet-Derived Growth Factor - pharmacology | Neointima - metabolism | Rats, Sprague-Dawley | Cardiovascular Agents - pharmacology | Hyperplasia - pathology | Tunica Intima - drug effects | Tunica Intima - metabolism | Cell Movement - drug effects | Neointima - prevention & control | Muscle, Smooth, Vascular - pathology | Animals | Tunica Intima - pathology | Cyclin D1 - genetics | Carotid Artery, Common - drug effects | Neointima - pathology | Carotid Artery, Common - pathology | Cell Proliferation - drug effects | PPAR alpha - metabolism | Primary Cell Culture | Endothelial Cells - pathology | Proliferating Cell Nuclear Antigen - metabolism | Platelet-Derived Growth Factor - antagonists & inhibitors | AMP-Activated Protein Kinases - genetics | Endothelial Cells - drug effects
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 03/2017, Volume 37, Issue 3, pp. 476 - 484
Journal Article
Diabetes Care, ISSN 0149-5992, 03/2016, Volume 39, Issue 3, pp. 455 - 464
Journal Article
European Journal of Pharmacology, ISSN 0014-2999, 04/2012, Volume 680, Issue 1-3, pp. 41 - 48
Chronic cerebral hypoperfusion, induced by permanent occlusion of bilateral common carotid arteries (2VO), is related to neurological disorders and contributes... 
Oxidative stress | Chronic cerebral hypoperfusion | Inflammation | Chrysin | Spatial learning | White matter lesion | APOPTOSIS | ALZHEIMERS-DISEASE | MITOCHONDRIA | FLAVONOIDS | MODEL | BLOOD-FLOW | NEURODEGENERATIVE DISEASES | IMPAIRMENT | PATHOGENESIS | PHARMACOLOGY & PHARMACY | Neurons - pathology | Rats, Wistar | Apoptosis - drug effects | Astrocytes - pathology | Brain Damage, Chronic - drug therapy | Brain Ischemia - metabolism | Memory Disorders - metabolism | Cognition Disorders - metabolism | Male | Lipid Peroxides - metabolism | Glial Fibrillary Acidic Protein - metabolism | Brain - metabolism | Learning Disorders - pathology | Dementia - metabolism | Neuroprotective Agents - pharmacology | Flavonoids - pharmacology | Neurons - metabolism | Brain Damage, Chronic - metabolism | Neurons - drug effects | Brain Damage, Chronic - pathology | Memory Disorders - pathology | Superoxide Dismutase - metabolism | Brain Ischemia - complications | Astrocytes - drug effects | Glutathione Peroxidase - metabolism | Dementia - pathology | Carotid Artery, Common - metabolism | Cerebrovascular Circulation - drug effects | Dementia - drug therapy | Cognition Disorders - pathology | Rats | Learning Disorders - drug therapy | Antioxidants - pharmacology | Cognition Disorders - drug therapy | Maze Learning - drug effects | Brain - drug effects | Animals | Memory Disorders - drug therapy | Carotid Artery, Common - drug effects | Brain Ischemia - pathology | Brain - pathology | Carotid Artery, Common - pathology | Learning Disorders - metabolism | Astrocytes - metabolism | Brain damage | Intermediate filament proteins | Glutathione peroxidase | Glial fibrillary acidic protein | Cognitive ability | Lipid peroxidation | Superoxide dismutase | Substantia alba | Antioxidants | Neurological diseases | peroxide | Flavonoids | Ischemia | Cerebral blood flow | Neurodegeneration | Dementia disorders | Carotid artery | Immunoreactivity | Brain injury | Apoptosis | Index Medicus
Journal Article
Science, ISSN 0036-8075, 4/2002, Volume 296, Issue 5567, pp. 539 - 541
Thromboxane (Tx) A is a vasoconstrictor and platelet agonist. Aspirin affords cardioprotection through inhibition of TxA formation by platelet cyclooxygenase... 
Vascular system injuries | Receptors | Platelet activation | Microscopy | Pathologic constriction | Cyclooxygenase inhibitors | Agonists | Reports | Biosynthesis | Physical trauma | Platelets | Endothelium, Vascular - cytology | Epoprostenol - metabolism | Receptors, Epoprostenol | Cyclooxygenase 2 | Humans | Receptors, Prostaglandin - physiology | Endothelium, Vascular - drug effects | Male | Receptors, Thromboxane - genetics | Endothelium, Vascular - physiology | Epoprostenol - physiology | Carotid Artery, Common - physiology | Prostaglandin-Endoperoxide Synthases | Cell Division | Receptors, Thromboxane - antagonists & inhibitors | Platelet Aggregation - drug effects | Muscle, Smooth, Vascular - physiology | Sulfones | Carotid Artery Injuries - pathology | Muscle, Smooth, Vascular - drug effects | Carotid Artery, Common - cytology | Mice, Inbred C57BL | Mice, Transgenic | Lactones - adverse effects | Muscle, Smooth, Vascular - cytology | Lactones - therapeutic use | Mice, Knockout | Naphthalenes | Membrane Proteins | Animals | Cyclooxygenase 2 Inhibitors | Tunica Intima - cytology | Tetrahydronaphthalenes - pharmacology | Thromboxane A2 - physiology | Carotid Artery, Common - drug effects | Propionates | 15-Hydroxy-11 alpha,9 alpha-(epoxymethano)prosta-5,13-dienoic Acid - pharmacology | Mice | Platelet Activation - drug effects | Receptors, Thromboxane - physiology | Cyclooxygenase Inhibitors - adverse effects | Cyclooxygenase Inhibitors - therapeutic use | Isoenzymes - antagonists & inhibitors | Proteins | Heart | Aspirin | Rodents | Cells
Journal Article