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Circulation research, ISSN 0009-7330, 2014, Volume 114, Issue 5, pp. 770 - 779
Journal Article
by He, Yi and He, Yi and Xu, Xin and Xu, Xin and Zhu, Ting and Zhu, Ting and Tang, Min and Tang, Min and Mei, Ju and Mei, Ju and Si, Yi and Si, Yi
Cellular Physiology and Biochemistry, ISSN 1015-8987, 2017, Volume 42, Issue 4, pp. 1303 - 1312
Background/Aims: Neointimal formation following vascular injury remains a major mechanism of restenosis, whereas the precise sources of neointimal cells are... 
Carotid transplantation | Bone marrow cells | Allograft | Intimal hyperplasia | PROGENITOR CELLS | ATHEROSCLEROTIC LESIONS | PHYSIOLOGY | DIVERSE CONTRIBUTION | PROLIFERATION | DEFICIENT MICE | VEIN GRAFTS | CELL BIOLOGY | VASCULAR INJURY | THERAPY | SMOOTH-MUSCLE-CELLS | NEOINTIMAL FORMATION | Receptors, CCR2 - genetics | Antigens, CD - immunology | Carotid Artery Injuries - surgery | Chemokine CCL2 - immunology | Receptors, CCR2 - immunology | Carotid Artery, Common - surgery | Rats, Inbred Lew | Green Fluorescent Proteins - genetics | Actins - immunology | Antigens, CD - genetics | Transplantation, Homologous | Actins - genetics | Cell Tracking | Neointima - genetics | Chemokine CXCL12 - genetics | Bone Marrow Cells - immunology | Tissue Transplantation | Carotid Artery, Common - immunology | Receptors, CXCR4 - genetics | Carotid Artery Injuries - genetics | Antigens, Differentiation, Myelomonocytic - immunology | Chemokine CXCL12 - immunology | Genes, Reporter | Carotid Artery Injuries - pathology | Neointima - surgery | Rats, Transgenic | Signal Transduction | Tunica Intima - immunology | Tunica Intima - surgery | Bone Marrow Cells - pathology | Gene Expression Regulation | Neointima - immunology | Rats | Chemokine CCL2 - genetics | Antigens, Differentiation, Myelomonocytic - genetics | Tunica Intima - injuries | Endothelial Cells - immunology | Animals | Green Fluorescent Proteins - immunology | Receptors, CXCR4 - immunology | Neointima - pathology | Carotid Artery, Common - pathology | Carotid Artery Injuries - immunology | Endothelial Cells - pathology | Cell Lineage - immunology
Journal Article
Arteriosclerosis Thrombosis and Vascular Biology, ISSN 1079-5642, 03/2013, Volume 33, Issue 3, pp. E75 - +
Journal Article
Journal Article
Cardiovascular Research, ISSN 0008-6363, 11/2017, Volume 113, Issue 13, pp. 1551 - 1559
Aims Chronic kidney disease (CKD) is a powerful independent risk factor for cardiovascular events, including vein graft failure. Because CKD impairs the... 
Mast cells | Cytokines | Smooth muscle cells | Neointimal hyperplasia | Endothelial cells | ATHEROGENESIS | CARDIAC & CARDIOVASCULAR SYSTEMS | STENOSIS | ALBUMINURIA | ARTERIOVENOUS-FISTULA FAILURE | ATHEROSCLEROSIS | IL-9 | RENAL DYSFUNCTION | INHIBITION | NECROSIS-FACTOR RECEPTOR-1 | Tumor Necrosis Factor-alpha - metabolism | Phosphorylation | Vascular Diseases - pathology | Vascular Diseases - complications | Humans | Hyperplasia | Carotid Artery, Common - surgery | Renal Insufficiency, Chronic - complications | Renal Insufficiency, Chronic - metabolism | Neointima | Time Factors | Mast Cells - metabolism | Vena Cava, Inferior - pathology | Carotid Artery, Common - immunology | Interleukin-9 - metabolism | Disease Models, Animal | Mast Cells - immunology | Vascular Diseases - immunology | Renal Insufficiency, Chronic - immunology | Signal Transduction | Arteriovenous Shunt, Surgical | Carotid Artery, Common - metabolism | Endothelial Cells - metabolism | Mice, Inbred C57BL | Cells, Cultured | Vena Cava, Inferior - metabolism | Vena Cava, Inferior - immunology | Animals | Transcription Factor RelA - metabolism | Vena Cava, Inferior - transplantation | Carotid Artery, Common - pathology | Endothelial Cells - pathology | Vascular Cell Adhesion Molecule-1 - metabolism | Vascular Diseases - metabolism | Interleukin-9 - immunology | Apoptosis | Editor's Choice | Original
Journal Article
Molecular and Cellular Biochemistry, ISSN 0300-8177, 1/2013, Volume 372, Issue 1, pp. 267 - 273
Journal Article
Arthritis Care & Research, ISSN 2151-464X, 12/2011, Volume 63, Issue 12, pp. 1736 - 1744
Objective Chronic inflammatory diseases in adults have been associated with increased cardiovascular risk and impaired vascular function. We aimed to assess... 
INTIMA-MEDIA THICKNESS | RHEUMATOID-ARTHRITIS | YOUNG-ADULTS | SYSTEMIC-LUPUS-ERYTHEMATOSUS | PULSE-WAVE VELOCITY | CARDIOVASCULAR EVENTS | ARTERIAL COMPLIANCE | FORM ANALYSIS | RHEUMATOLOGY | ENDOTHELIAL FUNCTION | BLOOD-PRESSURE | Carotid Artery, Common - diagnostic imaging | Tunica Media - pathology | Age Factors | Vascular Diseases - etiology | Humans | Arthritis, Juvenile - diagnosis | Male | Case-Control Studies | Vasodilation | Greece | Brachial Artery - diagnostic imaging | Carotid Artery, Common - immunology | Brachial Artery - physiopathology | Female | Intercellular Adhesion Molecule-1 - blood | Tunica Intima - diagnostic imaging | C-Reactive Protein - analysis | Child | Endothelium, Vascular - immunology | Manometry | Tunica Media - immunology | Vascular Diseases - immunology | Cross-Sectional Studies | Risk Assessment | Tunica Intima - immunology | Ultrasonography, Doppler | Risk Factors | Endothelium, Vascular - physiopathology | Inflammation Mediators - blood | Linear Models | Vascular Diseases - physiopathology | Biomarkers - blood | Vascular Diseases - diagnosis | Brachial Artery - immunology | Arthritis, Juvenile - physiopathology | Tunica Media - diagnostic imaging | Tunica Intima - pathology | Analysis of Variance | Arthritis, Juvenile - immunology | Endothelium, Vascular - diagnostic imaging | Adolescent | Arthritis, Juvenile - complications | P-Selectin - blood | Carotid Artery, Common - pathology
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 09/2012, Volume 32, Issue 9, pp. 2250 - 2258
OBJECTIVE—Adventitia acts as an active participant in vascular inflammation but the precise mechanism underlying adventitia–mediated vascular inflammation is... 
neointima formation | adventitial fibroblasts | vascular inflammation | macrophage chemotaxis | vascular biology | ANGIOGENESIS | MONOCYTE CHEMOATTRACTANT PROTEIN-1 | FACTOR RECEPTOR-1 | FACTOR-KAPPA-B | ANGIOTENSIN-II | DEFICIENT MICE | IN-VITRO | BALLOON ANGIOPLASTY | PERIPHERAL VASCULAR DISEASE | SMOOTH-MUSCLE-CELLS | HEMATOLOGY | HYPERTENSIVE-RATS | Inflammation - pathology | Up-Regulation | Vascular Endothelial Growth Factor Receptor-1 - antagonists & inhibitors | Cell Proliferation | Mitogen-Activated Protein Kinase 3 - antagonists & inhibitors | Connective Tissue - drug effects | Male | Osteopontin - metabolism | Vascular Endothelial Growth Factor A - metabolism | Inflammation - metabolism | Neointima | Time Factors | Carotid Artery, Common - immunology | Aorta, Thoracic - pathology | Carotid Artery Injuries - metabolism | Macrophages - immunology | Carotid Artery Injuries - pathology | Disease Models, Animal | Fibroblasts - metabolism | Cell Line | Aorta, Thoracic - immunology | Connective Tissue - pathology | Signal Transduction | Carotid Artery, Common - metabolism | Mitogen-Activated Protein Kinase 1 - antagonists & inhibitors | Aorta, Thoracic - metabolism | Rats | Connective Tissue - immunology | Vascular Endothelial Growth Factor Receptor-1 - metabolism | Inflammation - immunology | Fibroblasts - pathology | Chemotaxis | Rats, Sprague-Dawley | Connective Tissue - metabolism | Macrophages - metabolism | Animals | Mitogen-Activated Protein Kinase 3 - metabolism | Carotid Artery, Common - pathology | Fibroblasts - immunology | Carotid Artery Injuries - immunology | Mice | Protein Kinase Inhibitors - pharmacology | Oligopeptides - pharmacology | Mitogen-Activated Protein Kinase 1 - metabolism
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 09/2013, Volume 33, Issue 9, pp. 2097 - 2104
OBJECTIVE—Reendothelialization after vascular injury (ie, balloon angioplasty or stent implantation) is clinically extremely relevant to promote vascular... 
neointima formation | cytokines | endothelial growth factors | vascular system injuries | toll-like receptors | MALP-2 | CELLS | ATHEROSCLEROSIS | ADAPTER MOLECULES | PANCREATIC-CARCINOMA | ANGIOPLASTY | SIGNALING PATHWAY | MOUSE MODEL | PERIPHERAL VASCULAR DISEASE | MICE | HEMATOLOGY | EXPRESSION | Toll-Like Receptor 2 - agonists | Vascular System Injuries - immunology | Lipopeptides - pharmacology | Humans | Myocytes, Smooth Muscle - pathology | Carotid Artery Injuries - drug therapy | Male | Vascular System Injuries - drug therapy | Vascular System Injuries - pathology | Neointima | Time Factors | Carotid Artery, Common - immunology | Platelet Aggregation - drug effects | Myocytes, Smooth Muscle - drug effects | Myocytes, Smooth Muscle - metabolism | Wound Healing - drug effects | Carotid Artery Injuries - metabolism | Carotid Artery Injuries - pathology | Disease Models, Animal | Toll-Like Receptor 6 - metabolism | Cytokines - metabolism | Enzyme-Linked Immunosorbent Assay | Carotid Artery, Common - metabolism | Endothelial Cells - metabolism | Mice, Inbred C57BL | Cells, Cultured | Toll-Like Receptor 2 - metabolism | Endothelial Cells - immunology | Vascular System Injuries - metabolism | Animals | MAP Kinase Signaling System - drug effects | Carotid Artery, Common - drug effects | Myocytes, Smooth Muscle - immunology | Protein Array Analysis | Carotid Artery, Common - pathology | Carotid Artery Injuries - immunology | Cell Proliferation - drug effects | Mice | Endothelial Cells - pathology | Toll-Like Receptor 6 - agonists | Endothelial Cells - drug effects | Mitogen-Activated Protein Kinases - metabolism
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 07/2011, Volume 31, Issue 7, pp. 1547 - 1554
OBJECTIVE—Tetrahydrobiopterin (BH4) is a critical cofactor for nitric oxide (NO) synthesis by NO synthase (NOS). Recently, we demonstrated that disturbed flow... 
atherosclerosis | immune system | nitric oxide synthase | superoxide | reactive oxygen species | CELLS | GTP | ENDOTHELIAL DYSFUNCTION | ANGIOTENSIN-II | MECHANISMS | VASCULAR SMOOTH-MUSCLE | E-KNOCKOUT MICE | INFLAMMATION | SHEAR-STRESS | DISEASE | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | Apolipoproteins E - deficiency | Biopterin - deficiency | Carotid Artery, Common - surgery | Endothelium, Vascular - drug effects | Endothelium, Vascular - enzymology | Biopterin - analogs & derivatives | Biopterin - administration & dosage | Dose-Response Relationship, Drug | Atherosclerosis - etiology | Atherosclerosis - enzymology | Ligation | Inflammation - drug therapy | Inflammation Mediators - metabolism | Superoxides - metabolism | Carotid Artery, Common - immunology | Nitric Oxide Synthase Type III - metabolism | Macrophages - immunology | Disease Models, Animal | Endothelium, Vascular - immunology | Atherosclerosis - drug therapy | Atherosclerosis - immunology | Carotid Artery, Common - physiopathology | Vasodilator Agents - pharmacology | Cytokines - metabolism | Atherosclerosis - physiopathology | Mice, Inbred C57BL | Inflammation - immunology | Inflammation - etiology | Regional Blood Flow | Mice, Knockout | Animals | Carotid Artery, Common - enzymology | Apolipoproteins E - genetics | Carotid Artery, Common - drug effects | Endothelium, Vascular - pathology | T-Lymphocytes - immunology | Mice | Oxidative Stress - drug effects | Vasodilation - drug effects | Inflammation - enzymology | Inflammation - physiopathology | disturbed flow | NOS uncoupling | tetrahydrobiopterin | inflammation
Journal Article
Journal of Endocrinology, ISSN 0022-0795, 2015, Volume 227, Issue 1, pp. 61 - 69
Complications of atherosclerotic vascular disease, such as myocardial infarction and stroke, are the most common causes of death in postmenopausal women.... 
atherosclerosis | estrogen | menopause | inflammation | GPER | GPR30 | cyclooxygenase | vasoconstriction | acetylcholine | ovariectomy | ENDOTHELIAL DYSFUNCTION | SPONTANEOUSLY HYPERTENSIVE-RATS | CORONARY-ARTERIES | AGONIST G-1 | IN-VIVO | ENDOCRINOLOGY & METABOLISM | MICE | POSTMENOPAUSAL WOMEN | EXPRESSION | Tumor Necrosis Factor-alpha - metabolism | Receptors, Estrogen - metabolism | Receptors, G-Protein-Coupled - metabolism | Humans | Arteritis - metabolism | Endothelium, Vascular - drug effects | Male | Receptors, G-Protein-Coupled - agonists | Quinolines - pharmacology | Carotid Artery, Common - immunology | Thromboxane A2 - antagonists & inhibitors | Vascular Resistance - drug effects | Female | Arteritis - immunology | Estrogens - metabolism | Endothelium, Vascular - immunology | Vasoconstrictor Agents - pharmacology | Ovariectomy | Carotid Artery, Common - metabolism | Mice, Inbred C57BL | Receptors, Estrogen - antagonists & inhibitors | Down-Regulation - drug effects | Vasoconstriction - drug effects | Mice, Knockout | Thromboxane A2 - metabolism | Animals | Endothelium, Vascular - metabolism | Carotid Artery, Common - drug effects | 15-Hydroxy-11 alpha,9 alpha-(epoxymethano)prosta-5,13-dienoic Acid - pharmacology | Receptors, G-Protein-Coupled - antagonists & inhibitors | Receptors, G-Protein-Coupled - genetics | In Vitro Techniques | Benzodioxoles - pharmacology | Cell Line, Transformed
Journal Article
Journal Article
Marine Drugs, ISSN 1660-3397, 04/2015, Volume 13, Issue 4, pp. 2390 - 2406
Dihydroaustrasulfone alcohol is the synthetic precursor of austrasulfone, which is a marine natural product, isolated from the Taiwanese soft coral Cladiella... 
Restenosis | Phenotypic modulation | Migration | Cell cycle | Vascular smooth muscle cell | Proliferation | Dihydroaustrasulfone alcohol | ARTERIAL INJURY | CHEMISTRY, MEDICINAL | SOFT CORAL | GENE-EXPRESSION | ATHEROSCLEROSIS | BALLOON ANGIOPLASTY | GROWTH-FACTOR | VASCULAR PROLIFERATION | PROTEIN-KINASES | RAT MODEL | MITOGEN | Sulfones - therapeutic use | Muscle, Smooth, Vascular - metabolism | Butanones - therapeutic use | Humans | Carotid Artery Injuries - drug therapy | Muscle, Smooth, Vascular - immunology | Male | Antineoplastic Agents - therapeutic use | Antineoplastic Agents - administration & dosage | Sulfones - pharmacology | Butanones - pharmacology | Anti-Inflammatory Agents, Non-Steroidal - pharmacology | Cell Cycle Proteins - antagonists & inhibitors | Carotid Artery, Common - immunology | Cell Cycle Proteins - genetics | Female | Antineoplastic Agents - pharmacology | Carotid Artery Injuries - metabolism | Carotid Artery Injuries - pathology | Muscle, Smooth, Vascular - drug effects | Cardiovascular Agents - administration & dosage | Carotid Artery, Common - metabolism | Cell Cycle Proteins - metabolism | Cells, Cultured | Injections, Intraperitoneal | Rats, Sprague-Dawley | Cardiovascular Agents - pharmacology | Butanones - administration & dosage | Gene Expression Regulation - drug effects | Cardiovascular Agents - therapeutic use | Cell Movement - drug effects | Muscle, Smooth, Vascular - pathology | Platelet-Derived Growth Factor - metabolism | Animals | MAP Kinase Signaling System - drug effects | Anti-Inflammatory Agents, Non-Steroidal - therapeutic use | Carotid Artery, Common - drug effects | Anti-Inflammatory Agents, Non-Steroidal - administration & dosage | Carotid Artery, Common - pathology | Carotid Artery Injuries - immunology | Cell Proliferation - drug effects | Aorta - cytology | Cell Cycle - drug effects | Platelet-Derived Growth Factor - antagonists & inhibitors | Sulfones - administration & dosage | restenosis | proliferation | cell cycle | migration | vascular smooth muscle cell | phenotypic modulation | dihydroaustrasulfone alcohol
Journal Article
Journal Article