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ARTHRITIS & RHEUMATOLOGY, ISSN 2326-5191, 12/2015, Volume 67, Issue 12, pp. 3174 - 3183
Objective. To evaluate the extent to which the current designs of clinical trials in knee osteoarthritis (OA) permit detection of a therapeutic effect of... 
VITAMIN-D SUPPLEMENTATION | CARTILAGE VOLUME | JOINT REPLACEMENT | OSTEO-ARTHRITIS | ARTHROPLASTY | DOUBLE-BLIND | PREVALENCE | RHEUMATOLOGY | HIP | EPIDEMIOLOGY | STRUCTURAL PROGRESSION
Journal Article
Journal of Biomedical Materials Research Part A, ISSN 1549-3296, 12/2011, Volume 99A, Issue 3, pp. 467 - 478
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 1/2016, Volume 113, Issue 3, pp. E338 - E347
Pathologic extraskeletal bone formation, or heterotopic ossification (HO), occurs following mechanical trauma, burns, orthopedic operations, and in patients... 
Cartilage | Heterotopic ossification | Mesenchymal condensation | HIF1α | Prx | HYPOXIA-INDUCIBLE FACTOR | PX-478 | RISK-FACTORS | MULTIDISCIPLINARY SCIENCES | cartilage | heterotopic ossification | RECEPTOR | mesenchymal condensation | CANCER | HUMAN SKELETAL-MUSCLE | BONE-FORMATION | IN-VIVO | HIF1 alpha | GROWTH-FACTOR | HIF-1-ALPHA | Ossification, Heterotopic - drug therapy | Chondrogenesis - drug effects | Mustard Compounds - pharmacology | Chondrogenesis - genetics | Tendons - drug effects | Humans | Tenotomy | Hypoxia-Inducible Factor 1, alpha Subunit - antagonists & inhibitors | Luminescent Measurements | Gene Regulatory Networks - drug effects | RNA, Messenger - metabolism | X-Ray Microtomography | Adipose Tissue - metabolism | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Tendons - surgery | Integrases - metabolism | Burns - complications | Wound Healing - drug effects | Disease Models, Animal | SOX9 Transcription Factor - metabolism | Mesenchymal Stromal Cells - drug effects | Receptor, Platelet-Derived Growth Factor alpha - metabolism | Wounds and Injuries - pathology | Hypoxia-Inducible Factor 1, alpha Subunit - genetics | RNA, Messenger - genetics | Wounds and Injuries - complications | Ossification, Heterotopic - diagnostic imaging | Activin Receptors, Type I - metabolism | Sirolimus - pharmacology | Mice, Knockout | Up-Regulation - drug effects | Phenylpropionates - pharmacology | Animals | Signal Transduction - drug effects | Models, Biological | Tendons - pathology | Ossification, Heterotopic - prevention & control | Ossification, Heterotopic - genetics | Adipose Tissue - drug effects | Burns - genetics | Biological Sciences | PNAS Plus
Journal Article
Journal of Bone and Mineral Research, ISSN 0884-0431, 10/2017, Volume 32, Issue 10, pp. 2128 - 2141
ABSTRACT Autophagy is activated during nutritionally depleted or hypoxic conditions to facilitate cell survival. Because growth plate is an avascular and... 
ENDOCHONDRAL OSSIFICATION | AUTOPHAGY | GROWTH PLATE CHONDROCYTES | ATG7 | ER STRESS | MATRIX | CHONDROCYTES | ENDOPLASMIC-RETICULUM STRESS | CELL-DEATH | UNFOLDED PROTEIN RESPONSE | PATHWAY | ENDOCRINOLOGY & METABOLISM | GROWTH-PLATE | BONE | DIFFERENTIATION | Chondrogenesis - drug effects | Apoptosis - drug effects | eIF-2 Kinase - metabolism | Autophagy-Related Protein 7 - metabolism | Growth Plate - embryology | Cartilage - drug effects | Autophagy - drug effects | Chondrocytes - drug effects | Gene Deletion | Phenylbutyrates - pharmacology | Tibia - growth & development | Growth Plate - ultrastructure | Chondrocytes - metabolism | Femur - growth & development | Endoplasmic Reticulum Stress - drug effects | Osteogenesis - drug effects | Cells, Cultured | Femur - drug effects | Cartilage - metabolism | Growth Plate - metabolism | Organ Specificity | Tibia - drug effects | Autophagy-Related Protein 7 - genetics | Mice, Knockout | Animals | Activating Transcription Factor 4 - metabolism | Cell Differentiation - drug effects | Chondrocytes - ultrastructure | Cell Proliferation - drug effects | Embryonic Development - drug effects | Transcription Factor CHOP - metabolism | Autophagy-Related Protein 7 - deficiency | Analysis | Stress (Physiology) | Cell proliferation | Cell survival | Growth rate | Homeostasis | Collagenase 3 | Autophagy | Ossification | Cartilage | Growth plate | Bone growth | Rodents | Chondrocytes | Hypoxia | Chondrogenesis | Stress response | Endoplasmic reticulum | Bone (endochondral) | Phenylbutyric acid | Phagocytosis | Apoptosis
Journal Article
Biomaterials, ISSN 0142-9612, 2011, Volume 33, Issue 10, pp. 2848 - 2857
Abstract Adult bone marrow derived mesenchymal stem cells are undifferentiated, multipotential cells and have the potential to differentiate into multiple... 
Advanced Basic Science | Dentistry | Cartilage | Silk fibroin | Chitosan | Tissue engineering | Mesenchymal stem cells | PORE-SIZE | MATERIALS SCIENCE, BIOMATERIALS | BOVINE ARTICULAR CHONDROCYTES | ENGINEERING, BIOMEDICAL | REGENERATION | CHITOSAN SCAFFOLDS | MESENCHYMAL STEM-CELLS | STROMAL CELLS | Chondrogenesis - drug effects | Rats, Wistar | Male | Cartilage - drug effects | Tissue Scaffolds - chemistry | Mesenchymal Stromal Cells - cytology | Flow Cytometry | Mesenchymal Stromal Cells - ultrastructure | Stromal Cells - drug effects | Chitosan - pharmacology | Bone Marrow Cells - drug effects | Extracellular Matrix Proteins - metabolism | Mesenchymal Stromal Cells - drug effects | Bone Marrow Cells - cytology | Cell Separation | Extracellular Matrix Proteins - genetics | Stromal Cells - metabolism | Cells, Cultured | Fibroins - pharmacology | Mesenchymal Stromal Cells - metabolism | Rats | Cartilage - metabolism | Cell Adhesion - drug effects | Cell Shape - drug effects | Gene Expression Regulation - drug effects | Microscopy, Confocal | Animals | Cell Differentiation - drug effects | Fluorescent Antibody Technique | Cell Proliferation - drug effects | Porosity | Bone Marrow Cells - metabolism | Microscopy, Fluorescence | Stromal Cells - cytology | Silk | Glycosaminoglycans | Biological products | Collagen | Stem cells | Polyelectrolytes | Histochemistry
Journal Article
Biomaterials, ISSN 0142-9612, 2016, Volume 83, pp. 156 - 168
Abstract Conventional 3D printing may not readily incorporate bioactive ingredients for controlled release because the process often involves the use of heat,... 
Advanced Basic Science | Dentistry | Cartilage regeneration | Scaffold | MSC | Customized tissue engineering | 3D printing | HYDROGELS | MATERIALS SCIENCE, BIOMATERIALS | ENGINEERING, BIOMEDICAL | CHONDROGENIC DIFFERENTIATION | ACRYLATE | MESENCHYMAL STEM-CELLS | ELASTOMERS | SPHEROIDS | FABRICATION | LAYER | EXPRESSION | Chondrogenesis - drug effects | Chondrogenesis - genetics | Humans | Ink | Cell Aggregation - drug effects | Extracellular Matrix - metabolism | Hyaluronic Acid - pharmacology | Male | Polyethylene Glycols - chemistry | Cartilage - drug effects | Tissue Scaffolds - chemistry | Polyurethanes - pharmacology | Mesenchymal Stromal Cells - cytology | Solutions | Water - chemistry | Cartilage - physiology | Polyethylene Glycols - pharmacology | Amides - pharmacology | Biomarkers - metabolism | Transforming Growth Factor beta3 - pharmacology | Mesenchymal Stromal Cells - drug effects | Rabbits | Tissue Engineering - methods | Implants, Experimental | Polyurethanes - chemistry | Extracellular Matrix - drug effects | Gene Expression Regulation - drug effects | Regeneration - drug effects | Animals | Cell Proliferation - drug effects | Pyridines - pharmacology | Printing, Three-Dimensional | Delayed-Action Preparations | Crosslinked polymers | Tissue engineering | Stem cells | Hyaluronic acid | Transforming growth factors | Polyurethanes | Polyurethane resins | Cartilage | Ingredients | Biochemistry | Scaffolds | Controlled release
Journal Article
JAMA, ISSN 0098-7484, 05/2017, Volume 317, Issue 19, pp. 1967 - 1975
IMPORTANCE: Synovitis is common and is associated with progression of structural characteristics of knee osteoarthritis. Intra-articular corticosteroids could... 
UNITED-STATES | MEDICINE, GENERAL & INTERNAL | METAANALYSIS | ULTRASOUND | DISEASE | CORTICOSTEROID INJECTIONS | ARTHRITIS | JOINT | PROGRESSION | LESIONS | SYNOVITIS | Sodium Chloride - adverse effects | Glucocorticoids - administration & dosage | Outcome Assessment (Health Care) | Cartilage - pathology | Synovitis - complications | Arthralgia - blood | Glycated Hemoglobin A - metabolism | Humans | Middle Aged | Arthralgia - diagnostic imaging | Male | Cartilage - drug effects | Synovitis - diagnostic imaging | Anti-Inflammatory Agents - adverse effects | Osteoarthritis, Knee - blood | Anti-Inflammatory Agents - administration & dosage | Female | Glucocorticoids - adverse effects | Triamcinolone Acetonide - administration & dosage | Synovitis - drug therapy | Double-Blind Method | Drug Administration Schedule | Linear Models | Sodium Chloride - administration & dosage | Triamcinolone Acetonide - adverse effects | Osteoarthritis, Knee - diagnostic imaging | Magnetic Resonance Imaging | Sample Size | Injections, Intra-Articular | Arthralgia - drug therapy | Cartilage - diagnostic imaging | Osteoarthritis, Knee - drug therapy | Knee | Clinical trials | Arthritis | Knee (anatomy) | Patients | Cartilage | Randomization | Pain | Hemoglobin | Biocompatibility | Osteoarthritis | Triamcinolone acetonide | Cartilage diseases | Original Investigation | Research
Journal Article
Biomaterials, ISSN 0142-9612, 2015, Volume 50, Issue 1, pp. 75 - 86
Abstract Successful bone tissue engineering generally requires an osteoconductive scaffold that consists of extracellular matrix (ECM) to mimic the natural... 
Advanced Basic Science | Dentistry | Extracellular matrix (ECM) | Bone morphogenic protein (BMP)-2 | Bone tissue engineering | Cell-derived matrix | Microenvironment | Polymer mesh scaffold | MIGRATION | MATERIALS SCIENCE, BIOMATERIALS | ENGINEERING, BIOMEDICAL | CONSTRUCTS | STRENGTH | MESENCHYMAL STEM-CELLS | CARTILAGE | MULTI-LINEAGE DIFFERENTIATION | MORPHOGENESIS | VASCULARIZATION | EXTRACELLULAR-MATRIX | Bone Morphogenetic Protein 2 - pharmacology | Bone and Bones - pathology | Calcium - metabolism | Humans | Extracellular Matrix - metabolism | Fibroblasts - ultrastructure | Bone and Bones - drug effects | Tissue Scaffolds - chemistry | Mesenchymal Stromal Cells - cytology | Placenta - cytology | Skull - pathology | Female | Skull - drug effects | Wound Healing - drug effects | Disease Models, Animal | Osseointegration - drug effects | Bone Regeneration - drug effects | Mesenchymal Stromal Cells - drug effects | Extracellular Matrix - drug effects | Osteogenesis - drug effects | Biocompatible Materials - pharmacology | Rats, Sprague-Dawley | Pregnancy | Extracellular Matrix - ultrastructure | Animals | Cell Differentiation - drug effects | Mice, Nude | Fibroblasts - drug effects | Immobilized Proteins - pharmacology | Polymers - pharmacology | Polymers - chemistry | Fibroblasts - cytology | Fibronectins | Biological products | Glycosaminoglycans | Tissue engineering | Analysis | Stem cells | Anticoagulants (Medicine) | Polymer industry | Polymers | Finite element method | Biomedical materials | Biocompatibility | Bones | Coating | Scaffolds | Heparins | Defects
Journal Article
Biomaterials, ISSN 0142-9612, 2015, Volume 51, pp. 238 - 249
Abstract Glycopolypeptides are an emerging class of bioinspired polymers that mimic naturally occurring glycopeptides or glycoproteins, and therefore are... 
Advanced Basic Science | Dentistry | Cartilage tissue engineering | Glycopolypeptide hydrogel | Biomimic scaffold | Enzymatic crosslinking | Injectable hydrogel | MATERIALS SCIENCE, BIOMATERIALS | DRUG-DELIVERY | ENGINEERING, BIOMEDICAL | MESENCHYMAL STEM-CELLS | POLYPEPTIDES | MICHAEL ADDITION | SITU | REPAIR | BIOMEDICAL APPLICATIONS | REGENERATIVE MEDICINE | BONE | CLICK GLYCOSYLATION | Chondrocytes - cytology | Amides - chemical synthesis | Extracellular Matrix - metabolism | Glycopeptides - chemistry | Cartilage - drug effects | Hydrogels - chemical synthesis | Tissue Scaffolds - chemistry | Chondrocytes - drug effects | Polyglutamic Acid - pharmacology | Proton Magnetic Resonance Spectroscopy | Cartilage - physiology | Cell Death - drug effects | Mannose - chemistry | Amides - pharmacology | Cell Line | Cell Survival - drug effects | Rabbits | Tissue Engineering - methods | Glycosaminoglycans - metabolism | Extracellular Matrix - drug effects | Cells, Cultured | Polyglutamic Acid - chemical synthesis | Rats, Sprague-Dawley | Polyglutamic Acid - chemistry | Injections | Animals | Mice, Nude | Amides - chemistry | Cell Proliferation - drug effects | Mannose - pharmacology | Hydrogels - pharmacology | Glycopeptides - pharmacology | Hydrogels - chemistry | Subcutaneous Tissue - drug effects | Proteins | Crosslinked polymers | Glycosaminoglycans | Hydrogen peroxide | Glutamate | Tissue engineering | Surgical implants | Biomedical materials | Biomimetics | Hydrogels | Biocompatibility | In vitro testing | Scaffolds
Journal Article
Development, ISSN 0950-1991, 02/2008, Volume 135, Issue 3, pp. 579 - 588
Overexpression of zinc finger E-box binding homeobox transcription factor 1 (Zeb1) in cancer leads to epithelial-to-mesenchymal transition (EMT) and increased... 
Zeb1 | Epithelial-mesenchymal transition | Senescence | Transcription | STEM-CELLS | TGF-BETA | transcription | COREPRESSOR CTBP | PROTEIN DELTA-EF1 | ZINC-FINGER | DEVELOPMENTAL BIOLOGY | E-CADHERIN | CANCER | senescence | epithelial-mesenchymal transition | GENE ACTIVATION | TRANSCRIPTION FACTOR | EXPRESSION | Embryo, Mammalian - drug effects | Brain - embryology | Cadherins - metabolism | Vimentin - metabolism | Homeodomain Proteins - metabolism | Mesoderm - drug effects | Cellular Senescence - drug effects | Cartilage - drug effects | Stem Cells - cytology | Mesoderm - cytology | Epithelium - embryology | Eye - embryology | Kruppel-Like Transcription Factors - metabolism | Mice, Mutant Strains | Vimentin - genetics | Cartilage - cytology | Eye - drug effects | Cadherins - genetics | Palate - cytology | Palate - drug effects | Repressor Proteins - metabolism | Epithelium - drug effects | Brain - cytology | Cartilage - embryology | Mesoderm - embryology | Gene Expression Regulation, Developmental - drug effects | Eye - cytology | Gene Dosage | Mutation - genetics | Homeodomain Proteins - genetics | Palate - embryology | Brain - drug effects | Transforming Growth Factor beta - pharmacology | Animals | Embryo, Mammalian - embryology | Embryo, Mammalian - cytology | Fibroblasts - drug effects | Cyclin-Dependent Kinase Inhibitor p16 - metabolism | Stem Cells - drug effects | Cell Proliferation - drug effects | Fibroblasts - cytology | Mice | Cyclin-Dependent Kinase Inhibitor p15 - metabolism | Kruppel-Like Transcription Factors - genetics | Zinc Finger E-box-Binding Homeobox 1
Journal Article
Osteoarthritis and Cartilage, ISSN 1063-4584, 2015, Volume 24, Issue 2, pp. 315 - 324
Summary Objective The aetiology of OA is not fully understood although several adipokines such as leptin are known mediators of disease progression. Since... 
Rheumatology | Smad1 | Human chondrocytes | Hip osteoarthritis | β-Catenin | Dedifferentiation | Leptin | COLLAGEN | RHEUMATOLOGY | DIFFERENTIATED PHENOTYPE | HYPERTROPHY | IN-VITRO | SYNOVIAL FIBROBLASTS | HUMAN OSTEOARTHRITIC CARTILAGE | SIGNALING PATHWAY | beta-Catenin | ARTICULAR CHONDROCYTES | ORTHOPEDICS | KNEE OSTEOARTHRITIS | EXPRESSION | Receptors, Transforming Growth Factor beta - genetics | Humans | Leptin - metabolism | Middle Aged | Male | Lymphotoxin-alpha - metabolism | RNA, Messenger - metabolism | Core Binding Factor Alpha 1 Subunit - metabolism | Lymphotoxin-alpha - genetics | Cartilage, Articular - metabolism | Smad5 Protein - metabolism | Smad2 Protein - genetics | Aged, 80 and over | Smad2 Protein - drug effects | Protein-Serine-Threonine Kinases - metabolism | Collagen Type X - drug effects | SOX9 Transcription Factor - metabolism | Matrix Metalloproteinase 13 - genetics | Smad2 Protein - metabolism | Prednisolone - pharmacology | beta Catenin - metabolism | Matrix Metalloproteinase 13 - metabolism | Glycogen Synthase Kinase 3 - genetics | Receptors, Transforming Growth Factor beta - drug effects | Activin Receptors, Type II - genetics | Protein-Serine-Threonine Kinases - drug effects | Activin Receptors, Type II - metabolism | Osteoarthritis, Hip - metabolism | Glycogen Synthase Kinase 3 - drug effects | Activin Receptors, Type II - drug effects | Chondrocytes - drug effects | Collagen Type X - metabolism | beta Catenin - drug effects | Smad1 Protein - genetics | Adult | Female | Smad5 Protein - genetics | Chondrocytes - metabolism | RNA, Messenger - drug effects | Cell Dedifferentiation - drug effects | Cartilage, Articular - cytology | Lymphotoxin-alpha - drug effects | Protein-Serine-Threonine Kinases - genetics | Collagen Type X - genetics | SOX9 Transcription Factor - drug effects | Matrix Metalloproteinase 13 - drug effects | Glycogen Synthase Kinase 3 - metabolism | beta Catenin - genetics | Osteoarthritis, Hip - genetics | Smad5 Protein - drug effects | Core Binding Factor Alpha 1 Subunit - drug effects | Wnt Signaling Pathway - drug effects | Receptors, Transforming Growth Factor beta - metabolism | Wnt Signaling Pathway - genetics | Glucocorticoids - pharmacology | Smad1 Protein - metabolism | Aged | Smad1 Protein - drug effects | Cartilage, Articular - drug effects | In Vitro Techniques | Cell Dedifferentiation - genetics | Core Binding Factor Alpha 1 Subunit - genetics
Journal Article
Biomaterials, ISSN 0142-9612, 2013, Volume 34, Issue 22, pp. 5571 - 5580
Abstract Electrospinning has recently gained much interest due to its ability to form scaffolds that mimic the nanofibrous nature of the extracellular matrix,... 
Advanced Basic Science | Dentistry