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Journal of Leukocyte Biology, ISSN 0741-5400, 02/2015, Volume 97, Issue 2, pp. 425 - 433
ER stress induced autophagy and decreased GRP78 expression of T lymphocytes are involved in the pathogenesis of SLE and potentially important therapeutic... 
T cells | apoptosis | SLE | PATHWAYS | SURVIVAL | ENDOPLASMIC-RETICULUM STRESS | MACROAUTOPHAGY | IMMUNOLOGY | CELL-DEATH | CELL BIOLOGY | UNFOLDED PROTEIN RESPONSE | DISEASE | HEMATOLOGY | PROMOTES | EXPRESSION | RNA, Small Interfering - genetics | Transcription Factor CHOP - genetics | Endoribonucleases - genetics | Activating Transcription Factor 6 - immunology | Humans | bcl-X Protein - genetics | eIF-2 Kinase - immunology | Male | Endoplasmic Reticulum Stress - genetics | Activating Transcription Factor 6 - genetics | Endoplasmic Reticulum Stress - immunology | Autophagy - immunology | Heat-Shock Proteins - genetics | RNA, Small Interfering - immunology | Lupus Erythematosus, Systemic - immunology | Transcription Factor CHOP - immunology | bcl-X Protein - immunology | Apoptosis Regulatory Proteins - genetics | Female | Autophagy - genetics | T-Lymphocytes - pathology | bcl-2-Associated X Protein - genetics | eIF-2 Kinase - genetics | Beclin-1 | Apoptosis Regulatory Proteins - immunology | Gene Expression Regulation - genetics | Gene Expression Regulation - immunology | Membrane Proteins - genetics | Protein-Serine-Threonine Kinases - genetics | Membrane Proteins - immunology | Heat-Shock Proteins - immunology | bcl-2-Associated X Protein - immunology | Caspase 6 - genetics | Eukaryotic Initiation Factor-2 - genetics | Lupus Erythematosus, Systemic - genetics | Protein-Serine-Threonine Kinases - immunology | T-Lymphocytes - immunology | Caspase 6 - immunology | Eukaryotic Initiation Factor-2 - immunology | Lupus Erythematosus, Systemic - pathology | Endoribonucleases - immunology | Index Medicus
Journal Article
Journal Article
The FEBS Journal, ISSN 1742-464X, 07/2012, Volume 279, Issue 13, pp. 2399 - 2411
Nelfinavir induces apoptosis in liposarcoma by inhibiting site‐2 protease (S2P) activity, which leads to suppression of regulated intramembrane proteolysis. We... 
prostate cancer | sterol regulatory element binding protein‐1 (SREBP‐1) | nelfinavir | activating transcription factor 6 (ATF6) | Activating transcription factor 6 (ATF6) | Sterol regulatory element binding protein-1 (SREBP-1) | Prostate cancer | Nelfinavir | APOPTOSIS | CELLS | BIOCHEMISTRY & MOLECULAR BIOLOGY | ANDROGEN INDEPENDENCE | ENDOPLASMIC-RETICULUM STRESS | CLEAVAGE | SITE-2 PROTEASE | IN-VITRO | sterol regulatory element binding protein-1 (SREBP-1) | SREBPS | FATTY-ACID SYNTHASE | HIV PROTEASE INHIBITOR | Fatty Acid Synthase, Type I - genetics | Metalloendopeptidases - genetics | Proprotein Convertases - antagonists & inhibitors | Prostatic Neoplasms - metabolism | RNA, Small Interfering - genetics | Apoptosis - drug effects | Humans | Receptors, Androgen - metabolism | Male | Metalloendopeptidases - metabolism | Green Fluorescent Proteins - genetics | Activating Transcription Factor 6 - genetics | Autophagy | Proprotein Convertases - genetics | Recombinant Fusion Proteins - metabolism | Immunoenzyme Techniques | HIV Protease Inhibitors - pharmacology | Caspases - metabolism | Cell Nucleus - metabolism | Prostatic Neoplasms - genetics | Castration | Proteolysis | Serine Endopeptidases - genetics | Transcription, Genetic | Tumor Cells, Cultured | Real-Time Polymerase Chain Reaction | Sterol Regulatory Element Binding Protein 1 - metabolism | Metalloendopeptidases - antagonists & inhibitors | Green Fluorescent Proteins - metabolism | Prostatic Neoplasms - pathology | Endoplasmic Reticulum Stress - drug effects | Proprotein Convertases - metabolism | RNA, Messenger - genetics | Reverse Transcriptase Polymerase Chain Reaction | Fatty Acid Synthase, Type I - metabolism | Blotting, Western | Nelfinavir - pharmacology | Protein Transport | Activating Transcription Factor 6 - metabolism | Receptors, Androgen - genetics | Sterol Regulatory Element Binding Protein 1 - genetics | Sterol Regulatory Element Binding Protein 1 - antagonists & inhibitors | Recombinant Fusion Proteins - genetics | Activating Transcription Factor 6 - antagonists & inhibitors | Cell Proliferation - drug effects | Serine Endopeptidases - metabolism | Cell Nucleus - drug effects | Proteins | Binding sites | Apoptosis
Journal Article
Clinical Cancer Research, ISSN 1078-0432, 06/2017, Volume 23, Issue 11, pp. 2856 - 2868
Purpose: Anaplastic lymphoma kinase (ALK) is the most frequently mutated oncogene in the pediatric cancer neuroblastoma. We performed an in vitro screen for... 
CELL LUNG-CANCER | GROWTH-FACTOR RECEPTOR | MELANOMA | MEK INHIBITION | ONCOLOGY | KINASE | CRIZOTINIB | RESISTANCE | HIGH-RISK NEUROBLASTOMA | COMBINED BRAF | ACTIVATING MUTATIONS | Cyclin-Dependent Kinase 6 - antagonists & inhibitors | Small Molecule Libraries - administration & dosage | Cyclin-Dependent Kinase 4 - genetics | Humans | Purines - administration & dosage | Receptor Protein-Tyrosine Kinases - antagonists & inhibitors | Cyclin-Dependent Kinase 4 - antagonists & inhibitors | Neuroblastoma - pathology | Aminopyridines - administration & dosage | Pyrimidines - administration & dosage | Neuroblastoma - genetics | Cyclin-Dependent Kinase 6 - genetics | Drug Synergism | Xenograft Model Antitumor Assays | Animals | Receptor Protein-Tyrosine Kinases - genetics | Signal Transduction - drug effects | Retinoblastoma Protein - genetics | Neuroblastoma - drug therapy | Cell Line, Tumor | Cell Proliferation - drug effects | Mice | Mutation | Cyclin D - genetics | Sulfones - administration & dosage | Therapy | Pediatrics | Biotechnology | Animal models | Toxicity | Clinical trials | Cytotoxicity | Neuroblastoma | Drug development | Drug screening | Cyclin-dependent kinase 4 | Anticancer properties | Cyclin-dependent kinase | Cell activation | Cell cycle | Xenografts | Biocompatibility | Cyclin D | Inhibition | Children | Protein-tyrosine kinase | Cyclin-dependent kinases | Caspase | Pharmacology | Lymphoma | Signaling | Inhibitors | Experimental design | Cell death | Cell lines | Antitumor activity | Pharmacokinetics | Viability | Cancer
Journal Article
Apoptosis, ISSN 1360-8185, 11/2017, Volume 22, Issue 11, pp. 1362 - 1379
Cancer cells often have faulty apoptotic pathways resulting in sustenance of survivability, tumour metastasis and resistance to anticancer drugs. Alternate... 
Biochemistry, general | Adenylate cyclase | Biomedicine | RIP1-K63-polyubiquitination | CathepsinB | Rb phosphorylation | Cancer Research | Oncology | Cell Biology | Virology | tAIF | THERAPEUTICS | APOPTOSIS | ACTIVATION | PHOSPHORYLATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | CASPASE-INDEPENDENT NECROPTOSIS | DEATH | CELL BIOLOGY | PATHWAY | LIVER-TUMORS | Cysteine - analogs & derivatives | Leukocytes, Mononuclear - metabolism | RNA-Binding Proteins - genetics | Apoptosis - drug effects | Humans | Caspase 8 - metabolism | NF-kappa B - metabolism | BH3 Interacting Domain Death Agonist Protein - genetics | MAP Kinase Kinase 3 - genetics | Tumor Suppressor Protein p53 - genetics | Caspase 8 - genetics | Nuclear Pore Complex Proteins - genetics | MAP Kinase Kinase 6 - metabolism | Cyclic AMP-Dependent Protein Kinases - genetics | MAP Kinase Kinase 6 - genetics | Cysteine - pharmacology | DNA Fragmentation - drug effects | CASP8 and FADD-Like Apoptosis Regulating Protein - metabolism | Cyclic AMP - metabolism | BH3 Interacting Domain Death Agonist Protein - metabolism | Apoptosis Inducing Factor - metabolism | Cyclic AMP-Dependent Protein Kinases - metabolism | Cell Line | Leukocytes, Mononuclear - drug effects | Signal Transduction | CASP8 and FADD-Like Apoptosis Regulating Protein - genetics | Nuclear Pore Complex Proteins - metabolism | Gene Expression Regulation | Tumor Suppressor Protein p53 - metabolism | Necrosis - metabolism | Adenylyl Cyclases - metabolism | Apoptosis Inducing Factor - genetics | Hep G2 Cells | MAP Kinase Kinase 3 - metabolism | Necrosis - chemically induced | Protein Transport | Berberine - pharmacology | NF-kappa B - genetics | Leukocytes, Mononuclear - cytology | Necrosis - genetics | Adenylyl Cyclases - genetics | Drug Combinations | RNA-Binding Proteins - metabolism | Cysteine | Liver | Zoology | Cyclic adenylic acid | Metastasis | Homeopathy | Materia medica and therapeutics | Liver cancer | Proteases | Analysis | Therapeutics | Molecular biology | Tumor proteins | Cell proliferation | Drugs | Phosphorylation | p53 Protein | Drug resistance | Kinases | Metastases | Impact analysis | E2F protein | Cell growth | Antitumor agents | FLIP protein | DNA fragmentation | Inhibition | Protein transport | Deoxyribonucleic acid--DNA | Translocation | Berberine | Apoptosis-inducing factor | Caspase | c-FLIP protein | Nuclear transport | Survivability | Cancer | Tumors | Apoptosis
Journal Article
PLoS Pathogens, ISSN 1553-7366, 04/2012, Volume 8, Issue 4, p. e1002597
LPS-activated neutrophils secrete IL-1 beta by activation of TLR-4. Based on studies in macrophages, it is likely that ROS and lysosomal destabilization... 
B-CELL RECEPTOR | CYTOPLASMIC DOMAIN | APOPTOSIS | SIGNALING PATHWAYS | MICROBIOLOGY | NLRP3 INFLAMMASOME | NALP3 INFLAMMASOME | ADHESION | INHIBITION | VIROLOGY | SYK TYROSINE KINASE | PARASITOLOGY | LUMEN FORMATION | Neutrophil Activation - genetics | Inflammation - pathology | Inflammasomes - metabolism | Reactive Oxygen Species - metabolism | Protein-Tyrosine Kinases - metabolism | Lysosomes - genetics | Carcinoembryonic Antigen - genetics | Caspase 1 - metabolism | Multiprotein Complexes - genetics | Intracellular Signaling Peptides and Proteins - metabolism | Interleukin-1beta - genetics | Carcinoembryonic Antigen - metabolism | Multiprotein Complexes - metabolism | Inflammation - metabolism | Phosphorylation - genetics | Toll-Like Receptor 4 - agonists | Phosphorylation - immunology | Phosphorylation - drug effects | Intracellular Signaling Peptides and Proteins - genetics | Neutrophils - metabolism | Syk Kinase | Neutrophil Activation - immunology | Neisseria - immunology | Macrophages - pathology | Toll-Like Receptor 4 - genetics | Toll-Like Receptor 4 - immunology | Toll-Like Receptor 4 - metabolism | Mice, Knockout | Macrophages - metabolism | Protein Tyrosine Phosphatase, Non-Receptor Type 6 - metabolism | Lipopolysaccharides - pharmacology | Mice | Enzyme Activation - genetics | Lysosomes - immunology | Multiprotein Complexes - immunology | Protein Tyrosine Phosphatase, Non-Receptor Type 6 - immunology | Neisseriaceae Infections - genetics | Neutrophil Activation - drug effects | Intracellular Signaling Peptides and Proteins - immunology | Caspase 1 - immunology | Enzyme Activation - immunology | Protein-Tyrosine Kinases - immunology | Protein-Tyrosine Kinases - genetics | Lysosomes - metabolism | Reactive Oxygen Species - immunology | Lysosomes - pathology | Interleukin-1beta - biosynthesis | Neisseriaceae Infections - pathology | Macrophages - immunology | Neutrophils - pathology | Neisseriaceae Infections - metabolism | Carcinoembryonic Antigen - immunology | Neutrophils - immunology | Interleukin-1beta - immunology | Inflammation - immunology | Enzyme Activation - drug effects | Protein Tyrosine Phosphatase, Non-Receptor Type 6 - genetics | Inflammasomes - genetics | Animals | Neisseria - metabolism | Caspase 1 - genetics | Neisseriaceae Infections - immunology | Inflammasomes - immunology | Inflammation - genetics | Phosphorylation | Reactive oxygen species | SHP-1 protein | Leukocytes | Macrophages | Caspase-1 | Lipopolysaccharides | Cell activation | CEACAM1 protein | Interleukin 1 | Bone marrow | Toll-like receptors | Meningitis | Gram-negative bacteria | Immune system | Pathogens | RNA-mediated interference | Inflammation | Leukocytes (neutrophilic) | Gonorrhea | TLR4 protein | Infection | Stem cells | CD66 antigen | Syk protein
Journal Article
PLoS ONE, ISSN 1932-6203, 03/2013, Volume 8, Issue 3, p. e59330
Endoplasmic reticulum (ER) stress-mediated cell apoptosis has been implicated in various cell types, including fibroblasts. Previous studies have shown that... 
5-FLUOROURACIL | TRANSLOCATION | OXIDATIVE STRESS | UNFOLDED PROTEIN RESPONSE | LAMINECTOMY | C INDUCES APOPTOSIS | ER STRESS | MULTIDISCIPLINARY SCIENCES | MITOCHONDRIA | CHOP | CELL-DEATH | Transcription Factor CHOP - genetics | Endoribonucleases - genetics | Apoptosis - drug effects | eIF-2 Kinase - metabolism | Humans | Cell Survival - genetics | Apoptosis - genetics | Endoplasmic Reticulum Stress - genetics | Activating Transcription Factor 6 - genetics | Heat-Shock Proteins - genetics | Bcl-2-Like Protein 11 | Lipid Peroxidation - drug effects | Apoptosis Regulatory Proteins - genetics | Membrane Proteins - metabolism | Protein-Serine-Threonine Kinases - metabolism | Fibroblasts - metabolism | eIF-2 Kinase - genetics | In Situ Nick-End Labeling | Proto-Oncogene Proteins - metabolism | Cell Survival - drug effects | Endoplasmic Reticulum Stress - drug effects | Endoribonucleases - metabolism | Membrane Proteins - genetics | Heat-Shock Proteins - metabolism | Cells, Cultured | Protein-Serine-Threonine Kinases - genetics | Proto-Oncogene Proteins - genetics | Signal Transduction - genetics | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Apoptosis Regulatory Proteins - metabolism | Mitomycin - pharmacology | Activating Transcription Factor 6 - metabolism | Signal Transduction - drug effects | Fibroblasts - drug effects | Fibroblasts - cytology | Transcription Factor CHOP - metabolism | Analysis | Mitomycin | Apoptosis | Cell proliferation | Oxidative stress | Cell culture | Reactive oxygen species | Bax protein | Bcl-2 protein | Staining | Homology | Propidium iodide | CCAAT/enhancer-binding protein | Kinases | ADP | Caspase-3 | Stomach cancer | Western blotting | Proteins | Signal transduction | Ribose | Protein folding | Rodents | Surgery | Cell cycle | Fibroblasts | Mitomycin C | Stresses | Inositol | Iodides | Activating transcription factor 6 | Poly(ADP-ribose) polymerase | Caspase | Esophagus | Polymerase | DNA nucleotidylexotransferase | Signaling | Scars | Orthopedics | Annexin V | Endoplasmic reticulum | Viability | BIM protein
Journal Article
Journal of Molecular and Cellular Cardiology, ISSN 0022-2828, 2014, Volume 74, pp. 76 - 87
Abstract Lipopolysaccharide (LPS), an essential component of the outer membrane of Gram-negative bacteria, plays a pivotal role in myocardial anomalies in... 
Cardiovascular | Lipopolysaccharide | Ubiquitination | Cardiac function | TRAF6 | Apoptosis | Tumor Necrosis Factor-alpha - metabolism | RNA, Small Interfering - genetics | Aldehyde Dehydrogenase, Mitochondrial | Proto-Oncogene Proteins c-akt - deficiency | Tumor Necrosis Factor-alpha - genetics | Ubiquitin - metabolism | Glycogen Synthase Kinase 3 beta | TNF Receptor-Associated Factor 6 - antagonists & inhibitors | Proto-Oncogene Proteins c-akt - genetics | Lipopolysaccharides | Cardiomyopathies - genetics | Caspases - metabolism | Myocardium - metabolism | Membrane Potential, Mitochondrial | p38 Mitogen-Activated Protein Kinases - metabolism | TNF Receptor-Associated Factor 6 - genetics | Aldehyde Dehydrogenase - metabolism | Myocardial Contraction | Signal Transduction | Caspases - genetics | Gene Expression Regulation | Toll-Like Receptor 4 - genetics | p38 Mitogen-Activated Protein Kinases - genetics | Aldehyde Dehydrogenase - genetics | Cardiomyopathies - pathology | Myocardium - pathology | Ubiquitin - genetics | Glycogen Synthase Kinase 3 - metabolism | Toll-Like Receptor 4 - metabolism | Mice, Knockout | Myocytes, Cardiac - pathology | Animals | Glycogen Synthase Kinase 3 - genetics | Cardiomyopathies - metabolism | TNF Receptor-Associated Factor 6 - metabolism | Myocytes, Cardiac - metabolism | Mice | Cardiomyopathies - chemically induced | RNA, Small Interfering - metabolism | Ubiquitin | Ligases | Mitogens | apoptosis | cardiac function | lipopolysaccharide | ubiquitination
Journal Article
Nature, ISSN 0028-0836, 03/2012, Volume 483, Issue 7391, pp. 623 - 626
Many bacterial pathogens can enter various host cells and then survive intracellularly, transiently evade humoral immunity, and further disseminate to other... 
PASTEURELLA-MULTOCIDA TOXIN | PROTEIN-KINASE-C | ACTIVATION | CATALYTIC TRIAD | MOLECULAR-GRAPHICS | TRAF6 | MULTIDISCIPLINARY SCIENCES | CRYSTAL-STRUCTURES | HOST | NF-KAPPA-B | PATHOGENS | Shigella flexneri - genetics | Humans | Shigella flexneri - pathogenicity | Glutamine - metabolism | Molecular Sequence Data | Crystallography, X-Ray | NF-kappa B - metabolism | Histidine - metabolism | Neoplasm Proteins - metabolism | Ubiquitin-Conjugating Enzymes - chemistry | Inflammation - metabolism | Caspases - metabolism | Shigella flexneri - immunology | Diglycerides - metabolism | DNA Mutational Analysis | HEK293 Cells | Cysteine - metabolism | Shigella flexneri - enzymology | TNF Receptor-Associated Factor 6 - genetics | Dysentery, Bacillary - microbiology | Amidohydrolases - metabolism | Amino Acid Sequence | Biocatalysis | Amidohydrolases - genetics | Catalytic Domain - genetics | Models, Molecular | Ubiquitin-Conjugating Enzymes - genetics | Inflammation - immunology | Amidohydrolases - chemistry | Immunity, Innate | TNF Receptor-Associated Factor 6 - deficiency | Animals | Diglycerides - antagonists & inhibitors | Ubiquitin-Conjugating Enzymes - metabolism | B-Cell CLL-Lymphoma 10 Protein | TNF Receptor-Associated Factor 6 - metabolism | Aspartic Acid - metabolism | Virulence Factors - metabolism | Glutamic Acid - metabolism | Mice | Mucosa-Associated Lymphoid Tissue Lymphoma Translocation 1 Protein | HeLa Cells | Adaptor Proteins, Signal Transducing - metabolism | Inflammation - enzymology | Physiological aspects | Development and progression | Shigella flexneri | Cellular signal transduction | Genetic aspects | Inflammation | Risk factors | Proteins | Enzymes | Phosphorylation | Bacterial infections | Plasmids | Bacteria | Ligands | Infections | Kinases | Chemokines
Journal Article
Oncogene, ISSN 0950-9232, 11/2017, Volume 36, Issue 47, pp. 6617 - 6626
Dysregulation of microRNAs (miRNAs) plays an important role in the pathogenesis of chronic lymphocytic leukemia (CLL). The E mu-TCL1 transgenic mouse develops... 
SURVIVAL | BIOCHEMISTRY & MOLECULAR BIOLOGY | CANCER |