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Journal Article
AGING-US, ISSN 1945-4589, 06/2019, Volume 11, Issue 12, pp. 3891 - 3892
Journal Article
by Vellingiri, B and V, A and I, A and B, V and V, D and C, S
Annals of the Rheumatic Diseases, ISSN 0003-4967, 06/2018, Volume 77, Issue Suppl 2, p. 1288
BackgroundRheumatoid Arthritis (RA) is a multifactorial complex and chronic inflammatory disease associated with progressive joint destruction, disabling and... 
Polymerase chain reaction | Caspase-5 | Restriction fragment length polymorphism | Genotyping | Rheumatoid arthritis | Transcription activation | Interleukin 1 | Caspase | Population | Genetic factors | Gene expression | Enzyme-linked immunosorbent assay
Journal Article
Nature Immunology, ISSN 1529-2908, 03/2019, Volume 20, Issue 3, pp. 276 - 287
Inflammatory caspases (caspase-1, caspase-4, caspase-5 and caspase-11 (caspase-1/-4/-5/-11)) mediate host defense against microbial infections, processing... 
DEFENSE | ASC | SERPINS | EVOLUTION | ENZYME | MECHANISM | TOXINS | NLRP3 INFLAMMASOME | IMMUNOLOGY | INHIBITOR | Oligomers | Cytokines | Protease inhibitors | Proteases | Inflammation | Health aspects | Mitogens | Research | Serine | Immunological research | Oligomerization | Caspase-11 | Neutrophils | Caspase | Activation | Caspase-4 | Housing | IL-1β | Caspase-1 | Lipopolysaccharides | Microorganisms | Cell activation | Caspase-5 | Clonal deletion | Deletion | Sensitivity enhancement | Endotoxemia | Hygiene
Journal Article
Journal of Cellular Physiology, ISSN 0021-9541, 08/2019, Volume 234, Issue 8, pp. 13571 - 13581
In our previous studies, programmed cell death (PCD) was induced in human periodontal ligament (PDL) cells, through activation of caspase‐3 and upregulation of... 
human periodontal ligament cells | caspase‐5 | cyclic stretch | programmed cell death | caspase‐3 | caspase-5 | caspase-3 | APOPTOSIS | MATRIX | PHYSIOLOGY | INFLAMMASOME ACTIVATION | PYROPTOSIS | AUTOPHAGY | MODEL | CLEAVAGE | FIBROBLASTS | CELL BIOLOGY | GENE-EXPRESSION | Biochemistry | Apoptosis | Proteins | Periodontal ligament | Inhibitors | Cell death | Homeostasis | Caspase | Activation | Inflammation | Inhibition | Stretching
Journal Article
F1000Research, ISSN 2046-1402, 2018, Volume 7
In humans and other mammals, recognition of endotoxins-abundant surface lipopolysaccharides (LPS) of Gram-negative bacteria-provides a potent stimulus for... 
Journal Article
Immunology and Cell Biology, ISSN 0818-9641, 11/2018, Volume 96, Issue 10, pp. 1120 - 1130
Outer membrane vesicles (OMVs) are constitutively produced by Gram‐negative bacteria both in vivo and in vitro. These lipid‐bound structures carry a range of... 
Bacterial membrane vesicles | caspases‐11/‐4/‐5 | NOD‐like receptors | inflammasome | caspases-11/-4/-5 | NOD-like receptors | PATHWAYS | CELLS | RECOGNITION | DEATH | NLRP3 INFLAMMASOME | AUTOPHAGY | IMMUNOLOGY | NALP3 INFLAMMASOME | CELL BIOLOGY | PEPTIDOGLYCAN | INTRACELLULAR LPS | RECEPTORS
Journal Article
Immunology, ISSN 0019-2805, 10/2017, Volume 152, Issue 2, pp. 207 - 217
Summary Inflammatory responses mediated by macrophages are part of the innate immune system, whose role is to protect against invading pathogens.... 
Gram‐negative bacteria | macrophage | inflammation | intracellular lipopolysaccharide | non‐canonical inflammasome | Gram-negative bacteria | non-canonical inflammasome | NLRP3 INFLAMMASOME | IMMUNOLOGY | CUTTING EDGE | CELL-DEATH | BACTERIAL PATHOGENS | OUTER-MEMBRANE VESICLES | INNATE IMMUNE RECOGNITION | PATTERN-RECOGNITION | III SECRETION APPARATUS | GASDERMIN-D | MOLECULAR-MECHANISMS | Inflammasomes - metabolism | Caspases - immunology | Humans | Lipopolysaccharides - metabolism | Gram-Negative Bacteria - metabolism | Gram-Negative Bacteria - immunology | Lipopolysaccharides - immunology | Caspases - metabolism | Inflammation Mediators - metabolism | Macrophages - immunology | Macrophages - microbiology | Cytokines - immunology | Inflammation Mediators - immunology | Inflammation - microbiology | Cytokines - metabolism | Signal Transduction | Inflammation - immunology | Toll-Like Receptor 4 - immunology | Immunity, Innate | Toll-Like Receptor 4 - metabolism | Macrophage Activation | Macrophages - enzymology | Host-Pathogen Interactions | Animals | Inflammasomes - immunology | Inflammation - enzymology | Inflammation | Macrophages | Mitogens | Protein binding | Cysteine | Caspase-11 | Interleukin | Inflammatory response | Activation | Caspase-4 | Lipopolysaccharides | Proteins | Signal transduction | Receptors | Cell activation | Coding | Rodents | Aspartic endopeptidase | Interleukin 1 | Toll-like receptors | Bacteria | Immune system | Oligomerization | Immune response | Cytokines | Secretion | Shock resistance | Caspase | Septic shock | Pattern recognition | Chromosome 3 | Interleukin 18 | Signaling | Caspase-5 | Lysis | In vivo methods and tests | Interferon | Intracellular | Vacuoles | Cytoplasm | Review
Journal Article