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Nature Cell Biology, ISSN 1465-7392, 09/2017, Volume 19, Issue 9, pp. 1116 - 1129
Journal Article
The New England Journal of Medicine, ISSN 0028-4793, 10/2013, Volume 369, Issue 18, pp. 1704 - 1714
Journal Article
Immunology and Cell Biology, ISSN 0818-9641, 07/2019, Volume 97, Issue 6, pp. 552 - 562
The NOD‐like receptor (NLR) family plays an important role in innate immunity. Class II transactivator and NOD‐like receptor caspase activation and recruitment... 
Adaptive immunity | NLRC5, rotavirus | small intestine | dendritic cells | MHC‐I | MHC-I | CLEARANCE | ACTIVATION | NLRC5 | FAMILY-MEMBER NLRC5 | RIG-I | INDUCTION | IMMUNOLOGY | LAMBDA | CELL BIOLOGY | TRANSACTIVATOR | rotavirus | CLASS-I EXPRESSION
Journal Article
FEBS journal, ISSN 1742-464X, 2015, Volume 282, Issue 12, pp. 2327 - 2338
Journal Article
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2015, Volume 137, Issue 4, pp. 1178 - 1188.e7
Background Caspase recruitment domain-containing protein 9 (CARD9) deficiency is an autosomal recessive primary immunodeficiency conferring human... 
Allergy and Immunology | spontaneous central nervous system candidiasis | extracellular signal-regulated kinase | CSF2 | GM-CSF | Caspase recruitment domain-containing protein 9 | Ras protein–specific guanine nucleotide-releasing factor 1 | candidiasis | Ras protein-specific guanine nucleotide-releasing factor 1 | ACTIVATION | COLONY-STIMULATING FACTOR | RAS | KINASE | IMMUNOLOGY | FAMILY | CANDIDA-ALBICANS | COMBINED IMMUNODEFICIENCY | ALLERGY | PATHWAY | GRF | MUTATIONS | ras-GRF1 - immunology | Granulocyte-Macrophage Colony-Stimulating Factor - metabolism | Humans | CARD Signaling Adaptor Proteins - deficiency | Male | Candidiasis, Invasive - genetics | Extracellular Signal-Regulated MAP Kinases - metabolism | Central Nervous System Fungal Infections - genetics | Extracellular Signal-Regulated MAP Kinases - immunology | CARD Signaling Adaptor Proteins - genetics | Candidiasis, Invasive - diagnosis | Adult | Female | Immunologic Deficiency Syndromes - diagnosis | Real-Time Polymerase Chain Reaction | Biomarkers - metabolism | Quebec | Genetic Markers | Immunologic Deficiency Syndromes - microbiology | Point Mutation | Granulocyte-Macrophage Colony-Stimulating Factor - immunology | Candidiasis, Invasive - immunology | ras-GRF1 - metabolism | Central Nervous System Fungal Infections - diagnosis | Immunologic Deficiency Syndromes - genetics | Central Nervous System Fungal Infections - immunology | Cohort Studies | Allergy | Research institutes | Allergic reaction | Immunodeficiency | Medical genetics | Diseases | Mycoses | Analysis | Disease susceptibility | Proteins | Disease | Nervous system | Lymphomas | Mutation | Kinases | Gene expression
Journal Article
PLoS ONE, ISSN 1932-6203, 05/2012, Volume 7, Issue 5, p. e33747
To evaluate the relevance of C-C chemokine receptor type 5 (CCR5) expression and tumor development, we compared melanoma growth in CCR5 knockout (CCR5(-/-))... 
INTERLEUKIN-1 | ANTAGONIST | INHIBITION | ADENOCARCINOMA | IN-VIVO | BIOLOGY | SKIN CARCINOGENESIS | HUMAN BREAST-CANCER | CELL GROWTH | EXPRESSION | CARCINOMA | Immunohistochemistry | Melanoma - metabolism | Receptors, CCR5 - deficiency | Receptors, CCR5 - genetics | Apoptosis - genetics | Interleukin 1 Receptor Antagonist Protein - genetics | NF-kappa B - metabolism | Melanoma - pathology | Blotting, Western | Mice, Knockout | Animals | NF-kappa B - genetics | Melanoma - genetics | Interleukin 1 Receptor Antagonist Protein - metabolism | Cell Line, Tumor | Mice | Apoptosis - physiology | CD57 antigen | Transcription factors | Bcl-2 protein | CD8 antigen | Cytotoxicity | Metastasis | Inactivation | Caspase-3 | Metastases | Skin cancer | Signal transduction | Cell growth | Animal tissues | Inhibition | Natural killer cells | Deoxyribonucleic acid--DNA | Carbon-carbon composites | Spleen | Medical research | NF-κB protein | CCR5 protein | Cytokines | Melanoma | Gene expression | Survival | Studies | Chemotherapy | Pharmacy | Weight reduction | Ligands | Infiltration | Lymphomas | Binding sites | Phosphorylation | Bax protein | Gene regulation | Lymphocytes T | Interleukin 1 receptors | Kinases | Killer cells | Rodents | Localization | Translocation | Deactivation | Interleukin 1 receptor antagonist | Poly(ADP-ribose) polymerase | Caspase | Breast cancer | Cell death | Adenoviruses | Prostate cancer | Chemokines | Apoptosis | Tumors | CC chemokine receptors | Animal models | Transcription | Nuclear transport | NF- Kappa B protein | DNA | Deoxyribonucleic acid
Journal Article
Drug Resistance Updates, ISSN 1368-7646, 2007, Volume 10, Issue 6, pp. 218 - 234
Abstract The increased incidence of malignant melanoma in the last decades, its high mortality and pronounced therapy resistance pose an enormous challenge.... 
Infectious Disease | Hematology, Oncology and Palliative Medicine | NF-κB | CD95 | TRAIL | PKB/Akt | PI3 kinase | Death ligands | Sorafenib | Melanoma | Bcl-2 proteins | B-Raf | MAPK | Apoptosis | CUTANEOUS MALIGNANT-MELANOMA | bcl-2 proteins | ACTIVATED PROTEIN-KINASE | sorafenib | melanoma | apoptosis | FACTOR-KAPPA-B | CHEMOSENSITIZES HUMAN-MELANOMA | BH3 MIMETIC ABT-737 | PHASE-II TRIAL | NF-kappa B | B-raf | MANTLE CELL LYMPHOMA | death ligands | PHARMACOLOGY & PHARMACY | TRAIL-INDUCED APOPTOSIS | CYTOCHROME-C RELEASE | BCL-2 FAMILY-MEMBERS | Apoptosis - drug effects | Humans | Gene Expression Regulation, Neoplastic | Melanoma - enzymology | Apoptosis - genetics | Drug Resistance, Neoplasm | Antineoplastic Agents - therapeutic use | Intracellular Signaling Peptides and Proteins - metabolism | Antineoplastic Combined Chemotherapy Protocols - pharmacology | Receptors, Death Domain - metabolism | Proto-Oncogene Proteins c-bcl-2 - metabolism | Caspases - metabolism | Melanoma - genetics | Antineoplastic Agents - pharmacology | Melanoma - metabolism | Cell Survival - drug effects | Cysteine Proteinase Inhibitors - therapeutic use | Receptors, Death Domain - agonists | Tumor Suppressor Protein p53 - metabolism | Signal Transduction - genetics | Melanoma - pathology | Animals | Signal Transduction - drug effects | Antineoplastic Combined Chemotherapy Protocols - therapeutic use | Protein Kinase Inhibitors - therapeutic use | Melanoma - drug therapy | Proto-Oncogene Proteins c-bcl-2 - antagonists & inhibitors | Mutation | Development and progression | Care and treatment | Health aspects | Cancer
Journal Article
Gastroenterology, ISSN 0016-5085, 12/2016, Volume 151, Issue 6, pp. 1100 - 1104
Interleukin 10 receptor (IL10R)−deficient mice develop spontaneous colitis and, similarly, patients with loss-of-function mutations in develop severe... 
VEOIBD | Ubiquitination | Treg Cell | Caspase-1 | CELLS | ACTIVATION | MACROPHAGES | SPONTANEOUS COLITIS | GASTROENTEROLOGY & HEPATOLOGY | BOWEL-DISEASE | Tumor Necrosis Factor-alpha - metabolism | Inflammasomes - metabolism | Colitis - genetics | Humans | Caspase 8 - metabolism | Child, Preschool | Interferon-gamma - metabolism | Interleukin-1beta - genetics | Macrophages | Inflammatory Bowel Diseases - metabolism | Adenosine Triphosphate - pharmacology | NLR Family, Pyrin Domain-Containing 3 Protein - genetics | Inflammasomes - drug effects | Interleukin-1beta - metabolism | Inflammatory Bowel Diseases - pathology | Inflammatory Bowel Diseases - genetics | Interleukin-10 - metabolism | Adult | CD4-Positive T-Lymphocytes | Colitis - immunology | Antirheumatic Agents - therapeutic use | Inflammatory Bowel Diseases - drug therapy | Signal Transduction | NLR Family, Pyrin Domain-Containing 3 Protein - metabolism | Cells, Cultured | Immunity, Innate | Homeodomain Proteins - genetics | Mice, Knockout | Gene Expression Regulation - drug effects | Interleukin-17 - metabolism | Animals | Interleukin 1 Receptor Antagonist Protein - therapeutic use | Interleukin-10 Receptor alpha Subunit - genetics | Colitis - metabolism | Lipopolysaccharides - pharmacology | Protein Biosynthesis - drug effects | Mice | Mutation | Receptors, Interleukin-10 - deficiency | Interleukin-10 - pharmacology | Receptors, Interleukin-10 - genetics
Journal Article
Journal Article