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Human Molecular Genetics, ISSN 0964-6906, 2/2008, Volume 17, Issue 4, pp. 469 - 477
...  Human Molecular Genetics, 2008, Vol. 17, No. 4 469477 doi:10.1093/hmg/ddm324Advance Access published on November 7, 2007 Hui Wei1, Sung-Jo Kim1, Zhongjian... 
Biochemistry & Molecular Biology | Genetics & Heredity | Life Sciences & Biomedicine | Science & Technology | Gangliosidosis, GM1 - metabolism | Superoxide Dismutase - genetics | Apoptosis - drug effects | Humans | Endoplasmic Reticulum - metabolism | RNA, Messenger - metabolism | Lysosomal Storage Diseases, Nervous System - pathology | Neuronal Ceroid-Lipofuscinoses - pathology | Calnexin - genetics | Mitochondrial Proton-Translocating ATPases - genetics | Taurochenodeoxycholic Acid - pharmacology | Heat-Shock Proteins - genetics | Gangliosidosis, GM1 - genetics | Lysosomes - metabolism | Neuronal Ceroid-Lipofuscinoses - genetics | Lysosomal Storage Diseases - drug therapy | Endoplasmic Reticulum - drug effects | Glutaredoxins - genetics | Nuclear Proteins - genetics | Methylamines - pharmacology | Lysosomal Storage Diseases, Nervous System - drug therapy | Neuronal Ceroid-Lipofuscinoses - drug therapy | Catalase - genetics | RNA, Messenger - genetics | Cells, Cultured | Molecular Chaperones - genetics | Oxidative Stress - genetics | Lysosomal Storage Diseases - genetics | Neuronal Ceroid-Lipofuscinoses - metabolism | Gangliosidosis, GM1 - pathology | Lysosomal Storage Diseases, Nervous System - metabolism | Genetic Markers | DNA-Binding Proteins - genetics | Lysosomal Storage Diseases, Nervous System - genetics | Regulatory Factor X Transcription Factors | Lysosomal Storage Diseases - pathology | Protein Folding | Lysosomal Storage Diseases - metabolism | Transcription Factors | Apoptosis - physiology | Index Medicus
Journal Article
Aging cell, ISSN 1474-9718, 08/2017, Volume 16, Issue 4, pp. 837 - 846
Summary Kallistatin, an endogenous protein, protects against vascular injury by inhibiting oxidative stress and inflammation in hypertensive rats and enhancing... 
sirtuin 1 | kallistatin | vascular senescence | aging | microRNA‐34a | oxidative stress | microRNA-34a | Life Sciences & Biomedicine | Geriatrics & Gerontology | Science & Technology | Cell Biology | Sirtuin 1 - metabolism | Diabetes Mellitus, Experimental - drug therapy | Endothelial Progenitor Cells - cytology | MicroRNAs - antagonists & inhibitors | Streptozocin | Humans | Diabetes Mellitus, Experimental - genetics | NADPH Oxidases - metabolism | Caenorhabditis elegans Proteins - metabolism | Cellular Senescence - drug effects | Male | MicroRNAs - metabolism | Sirtuin 1 - genetics | Telomerase - genetics | Plasminogen Activator Inhibitor 1 - metabolism | Superoxides - metabolism | beta-Galactosidase - metabolism | NADPH Oxidases - genetics | Telomerase - metabolism | Diabetes Mellitus, Experimental - chemically induced | Superoxides - antagonists & inhibitors | Nitric Oxide Synthase Type III - metabolism | Plasminogen Activator Inhibitor 1 - genetics | Diabetes Mellitus, Experimental - metabolism | Sirtuins - genetics | Endothelial Progenitor Cells - metabolism | Cellular Senescence - genetics | Caenorhabditis elegans - metabolism | Caenorhabditis elegans - genetics | Catalase - genetics | Endothelial Progenitor Cells - drug effects | Cells, Cultured | Cyclin-Dependent Kinase Inhibitor p16 - genetics | Nitric Oxide Synthase Type III - genetics | Serpins - pharmacology | Catalase - metabolism | Tumor Necrosis Factor-alpha - pharmacology | Animals | Caenorhabditis elegans - drug effects | Genistein - pharmacology | Cyclin-Dependent Kinase Inhibitor p16 - metabolism | Mice | MicroRNAs - genetics | beta-Galactosidase - genetics | Caenorhabditis elegans Proteins - genetics | Sirtuins - metabolism | Tumor Necrosis Factor-alpha - antagonists & inhibitors | Gene expression | MicroRNA | Oxidative stress | Senescence | Genistein | INK4a protein | Kinases | NAD(P)H oxidase | Catalase | Rodents | Aging | Aorta | miRNA | Telomerase | Protein-tyrosine kinase | Tyrosine | Enzymes | Diabetes mellitus | p16 Protein | Superoxide | SIRT1 protein | Life span | Tumor necrosis factor | MicroRNAs | Stem cells | Nematodes | Diabetes | Index Medicus | Original
Journal Article
Journal Article
The Journal of biological chemistry, ISSN 1083-351X, 10/2014, Volume 289, Issue 41, pp. 28660 - 28675
The cadmium-transformed human lung bronchial epithelial BEAS-2B cells exhibit a property of apoptosis resistance as compared with normal non-transformed... 
Life Sciences & Biomedicine | Biochemistry & Molecular Biology | Science & Technology | Neoplasm Transplantation | RNA, Small Interfering - genetics | RNA-Binding Proteins - genetics | NF-E2-Related Factor 2 - antagonists & inhibitors | Superoxide Dismutase - genetics | Epithelial Cells - metabolism | Reactive Oxygen Species - metabolism | Microtubule-Associated Proteins - genetics | Microtubule-Associated Proteins - metabolism | Epithelial Cells - drug effects | Humans | Lung Neoplasms - metabolism | Gene Expression Regulation, Neoplastic | bcl-X Protein - genetics | Apoptosis - genetics | Lung Neoplasms - pathology | Male | Cell Transformation, Neoplastic - chemically induced | Green Fluorescent Proteins - genetics | Bronchi - drug effects | Proto-Oncogene Proteins c-bcl-2 - metabolism | Cell Transformation, Neoplastic - genetics | Bronchi - metabolism | NF-E2-Related Factor 2 - genetics | Autophagy - genetics | Bronchi - pathology | Genes, Reporter | Superoxide Dismutase - metabolism | RNA-Binding Proteins - antagonists & inhibitors | Lung Neoplasms - genetics | Cell Line | Green Fluorescent Proteins - metabolism | Cadmium - toxicity | Signal Transduction | Catalase - genetics | Epithelial Cells - pathology | Cell Transformation, Neoplastic - metabolism | Catalase - metabolism | Animals | Mice, Nude | NF-E2-Related Factor 2 - metabolism | Mice | Cell Transformation, Neoplastic - pathology | bcl-X Protein - metabolism | Proto-Oncogene Proteins c-bcl-2 - genetics | RNA-Binding Proteins - metabolism | RNA, Small Interfering - metabolism | Index Medicus | Cadmium | Reactive Oxygen Species (ROS) | Nuclear Factor 2 (Erythroid-derived 2-like Factor) (NFE2L2) (Nrf2) | Autophagy | Carcinogenesis | p62 | Cell Biology | Apoptosis
Journal Article
PloS one, ISSN 1932-6203, 01/2014, Volume 9, Issue 1, pp. e86819 - e86819
Journal Article
International journal of molecular sciences, ISSN 1422-0067, 08/2019, Volume 20, Issue 15, p. 3796
Journal Article
PLoS genetics, ISSN 1553-7404, 07/2018, Volume 14, Issue 7, pp. e1007465 - e1007465
Journal Article
The Journal of biological chemistry, ISSN 1083-351X, 12/2009, Volume 284, Issue 51, pp. 35403 - 35411
Inflammatory stimulants such as bacterial endotoxin (lipopolysaccharide (LPS)) are known to induce tissue damage and injury partly through the induction of... 
Life Sciences & Biomedicine | Biochemistry & Molecular Biology | Science & Technology | Gene Expression Regulation, Enzymologic - drug effects | Interleukin-1 Receptor-Associated Kinases - metabolism | Transcription, Genetic - drug effects | Interleukin-1 Receptor-Associated Kinases - genetics | Reactive Oxygen Species - metabolism | Antioxidants - metabolism | Multienzyme Complexes - metabolism | rac GTP-Binding Proteins - metabolism | rac GTP-Binding Proteins - genetics | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha | Lipid Peroxidation - drug effects | Trans-Activators - genetics | Neuropeptides - genetics | NADH, NADPH Oxidoreductases - metabolism | NADH, NADPH Oxidoreductases - genetics | Catalase - genetics | Neuropeptides - metabolism | PPAR alpha - genetics | Multienzyme Complexes - genetics | Transcription Factors - genetics | Enzyme Activation - drug effects | Glutathione Peroxidase - biosynthesis | Glutathione Peroxidase - genetics | NADPH Oxidase 1 | Macrophages - enzymology | Mice, Knockout | Transcription Factors - metabolism | Animals | Lipopolysaccharides - pharmacology | Trans-Activators - metabolism | Mice | PPAR alpha - metabolism | Transcription, Genetic - genetics | rac1 GTP-Binding Protein | Amino Acid Motifs - genetics | Catalase - biosynthesis | Index Medicus | Mechanisms of Signal Transduction
Journal Article
The Journal of physiology, ISSN 0022-3751, 10/2014, Volume 592, Issue 20, pp. 4575 - 4589
Key points Oxidative stress is widely considered a major cause of muscle loss not only in disuse but also in most chronic diseases, triggering carbonylation of... 
Neurosciences | Physiology | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Up-Regulation | Dynamins - metabolism | Superoxide Dismutase - genetics | Oxidative Stress | SKP Cullin F-Box Protein Ligases - genetics | Mitochondria, Muscle - metabolism | Male | Muscle Fibers, Skeletal - drug effects | Muscle Fibers, Skeletal - metabolism | RNA, Messenger - metabolism | Autophagy | Tripartite Motif Proteins | Chromans - pharmacology | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha | Muscle Proteins - metabolism | Apoptosis Regulatory Proteins - genetics | NF-E2-Related Factor 2 - genetics | Muscular Atrophy - prevention & control | Muscle Fibers, Skeletal - physiology | Superoxide Dismutase - metabolism | Beclin-1 | Muscular Atrophy - metabolism | Catalase - genetics | Mice, Inbred C57BL | RNA, Messenger - genetics | Ubiquitin-Protein Ligases - metabolism | Dynamins - genetics | SKP Cullin F-Box Protein Ligases - metabolism | Antioxidants - pharmacology | Transcription Factors - genetics | Apoptosis Regulatory Proteins - metabolism | Catalase - metabolism | Muscle Proteins - genetics | Transcription Factors - metabolism | Antioxidants - therapeutic use | Animals | Chromans - therapeutic use | Hindlimb Suspension - adverse effects | NF-E2-Related Factor 2 - metabolism | Mice | Mitochondria, Muscle - drug effects | Superoxide Dismutase-1 | Ubiquitin-Protein Ligases - genetics | Index Medicus | Muscle
Journal Article