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PLoS ONE, ISSN 1932-6203, 03/2012, Volume 7, Issue 3, pp. e33210 - e33210
Brassinosteroids (BRs) and polyamines (PAs) are well-established growth regulators playing key roles in stress management among plants. In the present study,... 
SEEDLINGS | TOLERANCE | LIGHT | BIOLOGY | PLANTS | ARABIDOPSIS | ABSCISIC-ACID | SUPEROXIDE-DISMUTASE | SPERMIDINE | CELL-DEATH | WATER | Trace Elements - metabolism | Glutathione Reductase - metabolism | Antioxidants - metabolism | NADPH Oxidases - metabolism | Spermidine - metabolism | Seedlings - growth & development | Brassinosteroids - metabolism | Chromium - toxicity | Raphanus - genetics | Abscisic Acid - metabolism | Plant Proteins - metabolism | Stress, Physiological - drug effects | Raphanus - metabolism | Superoxide Dismutase - metabolism | Brassinosteroids - pharmacology | Indoleacetic Acids - metabolism | Seedlings - drug effects | Chromium - metabolism | Enzyme Activation - drug effects | Reverse Transcriptase Polymerase Chain Reaction | Photosystem II Protein Complex - metabolism | Polyamines - metabolism | Catalase - metabolism | Plant Proteins - genetics | Gene Expression Regulation, Plant - drug effects | Polyamines - pharmacology | Raphanus - drug effects | Spermidine - pharmacology | Plant Growth Regulators - metabolism | Plant Growth Regulators - pharmacology | Peroxidase - metabolism | Seedlings - metabolism | Trace Elements - toxicity | Antioxidants | Enzymes | Physiological aspects | Superoxide | Photosynthesis | Polyamines | Metallothionein | Steroids | Regulators | Ascorbic acid | Genomics | Plants (organisms) | Glutathione reductase | Betaine | Pigments | Abscisic acid | Physiology | Glycine betaine | Stresses | Growth regulators | Brassinosteroids | Metabolism | Photosynthetic pigments | Gene expression | Malondialdehyde | Studies | Mitigation | Environmental science | Methods | Reductase | Oxidative stress | Reactive oxygen species | Hydrogen peroxide | Seeds | Horticulture | Proline | Superoxide dismutase | Physiological responses | Catalase | Peroxidase | Plant sciences | Sugars | Glutathione | Soil contamination | Abiotic stress | Data processing | Phytochelatins | Stress | Seedlings | Salinity | Plant growth | Phenols | Chromium | Index Medicus
Journal Article
Ecotoxicology and Environmental Safety, ISSN 0147-6513, 01/2018, Volume 147, pp. 990 - 1001
Journal Article
Amino Acids, ISSN 0939-4451, 1/2017, Volume 49, Issue 1, pp. 129 - 138
High levels of methionine (Met) and methionine sulfoxide (MetO) are found in several genetic abnormalities. Oxidative stress is involved in the pathophysiology... 
Life Sciences | Biochemistry, general | Oxidative stress | Analytical Chemistry | Life Sciences, general | Methionine sulfoxide | Biochemical Engineering | Proteomics | Neurobiology | Methionine | Delta aminolevulinic dehydratase | LIPID-PEROXIDATION | ANTIOXIDANTS | BIOCHEMISTRY & MOLECULAR BIOLOGY | DELTA-AMINOLEVULINATE DEHYDRATASE | DAMAGE | HYPERMETHIONINEMIA | METABOLISM | FAT | ADENOSYLTRANSFERASE I/III DEFICIENCY | PROTEINS | RESIDUES | Glycine N-Methyltransferase - metabolism | Liver - pathology | Reactive Oxygen Species - metabolism | Kidney - pathology | Rats, Wistar | Glycine N-Methyltransferase - deficiency | Male | Methionine - pharmacology | Porphobilinogen Synthase - metabolism | Amino Acid Metabolism, Inborn Errors - metabolism | Kidney - metabolism | Sulfhydryl Compounds - metabolism | Liver - drug effects | Methionine - analogs & derivatives | Injections, Subcutaneous | Female | Urea - metabolism | Amino Acid Metabolism, Inborn Errors - pathology | Superoxide Dismutase - metabolism | Glutathione Peroxidase - metabolism | Amino Acid Metabolism, Inborn Errors - chemically induced | Kidney - drug effects | Methionine - metabolism | Liver - metabolism | Rats | Thiobarbituric Acid Reactive Substances - metabolism | Cholesterol - metabolism | Enzyme Activation - drug effects | Triglycerides - metabolism | Catalase - metabolism | Animals | Glucose - metabolism | Oxidative Stress - drug effects | Lipid Peroxidation | Enzymes | Urea | Liver | Physiological aspects | Amino acids | Triglycerides | Superoxide | Index Medicus
Journal Article
American Journal of Physiology - Cell Physiology, ISSN 0363-6143, 09/2011, Volume 301, Issue 3, pp. C695 - C704
Wang Y, Zang QS, Liu Z, Wu Q, Maass D, Dulan G, Shaul PW, Melito L, Frantz DE, Kilgore JA, Williams NS, Terada LS, Nwariaku FE. Regulation of VEGF-induced... 
Antioxidants | Mitochondria | Reactive oxygen species | Oxidant signaling | Endothelial cells | NADPH OXIDASE | endothelial cells | SUPEROXIDE | ACTIVATION | PHYSIOLOGY | OXIDATIVE DAMAGE | antioxidants | mitochondria | TERMINAL KINASE | CELL BIOLOGY | oxidant signaling | reactive oxygen species | IN-VIVO | TARGETED ANTIOXIDANTS | GROWTH-FACTOR | DYSFUNCTION | STRESS | DNA Polymerase gamma | RNA, Small Interfering - genetics | Reactive Oxygen Species - metabolism | Human Umbilical Vein Endothelial Cells - metabolism | Endothelium, Vascular - injuries | Humans | Cytoplasm - metabolism | Endothelium, Vascular - drug effects | Male | Extracellular Signal-Regulated MAP Kinases - metabolism | Cell Movement - physiology | DNA-Directed DNA Polymerase - genetics | Electron Transport Complex IV - metabolism | Endothelium, Vascular - physiology | Human Umbilical Vein Endothelial Cells - cytology | Superoxides - metabolism | p38 Mitogen-Activated Protein Kinases - metabolism | Phosphorylation - drug effects | Proto-Oncogene Proteins c-akt - metabolism | Vascular Endothelial Growth Factor A - pharmacology | Carotid Artery Injuries - pathology | von Willebrand Factor - metabolism | Cell Line | Human Umbilical Vein Endothelial Cells - drug effects | Transduction, Genetic | Catalase - genetics | Mice, Inbred C57BL | Cells, Cultured | Mitochondria - metabolism | Mitochondria - drug effects | Regeneration - physiology | p21-Activated Kinases - metabolism | Hydrogen Peroxide - metabolism | Catalase - metabolism | Cell Movement - drug effects | Regeneration - drug effects | Animals | Cyclooxygenase 2 - metabolism | Endothelium, Vascular - pathology | Cell Proliferation - drug effects | Mice | DNA-Directed DNA Polymerase - metabolism | Cytoplasm - drug effects | Hydrogen Peroxide - antagonists & inhibitors | rac1 GTP-Binding Protein - metabolism | Vitamin E - pharmacology | rac1 GTP-Binding Protein - genetics | Care and treatment | Atherosclerosis | Physiological aspects | Diagnosis | Research | Vascular endothelial growth factor | Cell migration | Molecules | Metabolism | Cell adhesion & migration | Index Medicus | Vascular Biology
Journal Article
Atherosclerosis, ISSN 0021-9150, 2012, Volume 221, Issue 2, pp. 375 - 382
Highlights ► Liraglutide can normalize TNF-α-induced pro-oxidant production in HUVEC probably through reduced expression of NADPH oxidase subunit gp91phox and... 
Cardiovascular | Oxidative stress | NF-κB | Endothelial cell | Inflammation | Apoptosis | CHRONIC HEART-FAILURE | TRANSLOCATION | PROTEIN-KINASE-C | PENTRAXIN 3 | ACTIVATION | CARDIAC & CARDIOVASCULAR SYSTEMS | IN-VITRO | NF-kappa B | ACUTE MYOCARDIAL-INFARCTION | DEGRADATION | PERIPHERAL VASCULAR DISEASE | DYSFUNCTION | VASCULAR NAD(P)H OXIDASE | Tumor Necrosis Factor-alpha - metabolism | Phosphorylation | Reactive Oxygen Species - metabolism | Human Umbilical Vein Endothelial Cells - metabolism | Membrane Glycoproteins - metabolism | Protein Kinase C-alpha - metabolism | Apoptosis - drug effects | Humans | NADPH Oxidases - metabolism | Human Umbilical Vein Endothelial Cells - immunology | NF-kappa B - metabolism | I-kappa B Proteins - metabolism | Dose-Response Relationship, Drug | C-Reactive Protein - metabolism | Transfection | I-kappa B Kinase - metabolism | Time Factors | Inflammation Mediators - metabolism | Superoxide Dismutase - metabolism | Recombinant Proteins - metabolism | Glutathione Peroxidase - metabolism | Human Umbilical Vein Endothelial Cells - drug effects | Anti-Inflammatory Agents - pharmacology | Glucagon-Like Peptide 1 - analogs & derivatives | Cells, Cultured | Glucagon-Like Peptide 1 - pharmacology | Antioxidants - pharmacology | NADPH Oxidase 2 | Protein Transport | Catalase - metabolism | NF-kappa B - genetics | Signal Transduction - drug effects | Serum Amyloid P-Component - metabolism | Oxidative Stress - drug effects | Liraglutide | Index Medicus
Journal Article
Journal of Hazardous Materials, ISSN 0304-3894, 10/2012, Volume 235-236, pp. 343 - 351
Journal Article
Journal of Molecular and Cellular Cardiology, ISSN 0022-2828, 2014, Volume 79, pp. 275 - 283
Abstract Background Obesity leads to metabolic heart disease (MHD) that is associated with a pathologic increase in myocardial fatty acid (FA) uptake and... 
Cardiovascular | Obesity | Metabolic heart disease | Mitochondria | Lipid excess | OXIDATIVE STRESS | CARDIAC & CARDIOVASCULAR SYSTEMS | PROLIFERATOR-ACTIVATED RECEPTOR | KINASE-C | FATTY-ACIDS | CELL BIOLOGY | REDUCES DIABETIC CARDIOMYOPATHY | SKELETAL-MUSCLE | LIPOTOXIC CARDIOMYOPATHY | FAILING HEART | COACTIVATOR PGC-1 | TRANSCRIPTIONAL CONTROL | Mitochondria, Heart - ultrastructure | Phosphorylation | Mitochondria, Heart - metabolism | Carnitine - metabolism | RNA, Messenger - metabolism | Fatty Acid Transport Proteins - metabolism | Diglycerides - metabolism | Protein Kinase C - metabolism | Adenosine Triphosphate - metabolism | Myocardium - metabolism | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha | Carnitine - analogs & derivatives | Proto-Oncogene Proteins c-akt - metabolism | Myocardium - ultrastructure | Ceramides - metabolism | Sphingomyelins - metabolism | RNA, Messenger - genetics | Gene Expression Regulation | Oxygen Consumption | Lipids - adverse effects | Myocardium - pathology | Organ Specificity | Hydrogen Peroxide - metabolism | Catalase - metabolism | Transcription Factors - metabolism | Animals | Electron Transport Complex II - metabolism | Models, Biological | Cyclic AMP Response Element-Binding Protein - metabolism | Myocytes, Cardiac - metabolism | Mice | PPAR alpha - metabolism | Fatty acids | Analysis | Mitochondrial DNA | Index Medicus | Metabolic Heart Disease | Lipid Excess
Journal Article
Protoplasma, ISSN 0033-183X, 11/2014, Volume 251, Issue 6, pp. 1373 - 1386
Journal Article
PLoS ONE, ISSN 1932-6203, 04/2014, Volume 9, Issue 4, pp. e95319 - e95319
Oxidative stress and the antioxidant response induced by high environmental ammonia (HEA) were investigated in the liver and gills of three freshwater teleosts... 
SUBLETHAL AMMONIA | RAINBOW-TROUT | LIPID-PEROXIDATION | MULTIDISCIPLINARY SCIENCES | HIGH ENVIRONMENTAL AMMONIA | TOXICITY | PRIMARY CULTURES | SEA BASS | TROUT ONCORHYNCHUS-MYKISS | SALMO-SALAR L | FREE-RADICALS | Adaptation, Physiological | Superoxide Dismutase - genetics | Oxidative Stress | Species Specificity | Glutathione - metabolism | Glutathione Reductase - metabolism | Glutathione Reductase - genetics | Ascorbate Peroxidases - metabolism | Gills - drug effects | Goldfish - genetics | Liver - drug effects | Ascorbate Peroxidases - genetics | Xanthine Oxidase - genetics | Superoxide Dismutase - metabolism | Malondialdehyde - metabolism | Oncorhynchus mykiss - metabolism | Carps - metabolism | Glutathione Peroxidase - metabolism | Xanthine Oxidase - metabolism | Goldfish - metabolism | Catalase - genetics | Liver - metabolism | Ammonia - toxicity | Gills - metabolism | Oncorhynchus mykiss - genetics | Glutathione Peroxidase - genetics | Hydrogen Peroxide - metabolism | Catalase - metabolism | Gene Expression Regulation - drug effects | Animals | Carps - genetics | Physiological aspects | Oxidative stress | Genetic aspects | Molybdenum compounds | Trout | Analysis | Hydrogen peroxide | Ascorbic acid | Liver | L-Ascorbate peroxidase | Superoxide dismutase | Accumulation | Antioxidants | Catalase | Glutathione reductase | Rodents | Fish | Peroxidase | Species | Glutathione | Glutathione peroxidase | Stresses | Carp | Salmon | Gills | Exposure | Xanthine | Malondialdehyde | Stress | Ammonia | Xanthine oxidase | Reductase | System effectiveness | Index Medicus
Journal Article