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1. The Ocular lens
: structure, function, and pathology
by Maisel, Harry, 1932
1985, ISBN 9780824772970, x, 479
Cataract | Lens, Crystaline | Crystalline lens
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2. Intraocular lenses
: evolution, designs, complications, and pathology
by Apple, David J
1989, ISBN 9780683002409, xvi, 533
Intraocular lenses -- Complications | Cataract -- Surgery -- Complications
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3. Full Text TUBB4A mutations result in specific neuronal and oligodendrocytic defects that closely match clinically distinct phenotypes
by Curiel, Julian and Bey, Guillermo Rodríguez and Takanohashi, Asako and Bugiani, Marianna and Fu, Xiaoqin and Wolf, Nicole I and Nmezi, Bruce and Schiffmann, Raphael and Bugaighis, Mona and Pierson, Tyler and Helman, Guy and Simons, Cas and van der Knaap, Marjo S and Liu, Judy and Padiath, Quasar and Vanderver, Adeline
Human Molecular Genetics, ISSN 0964-6906, 2017, Volume 26, Issue 22, pp. 4506 - 4518
Hypomyelinating leukodystrophies are heritable disorders defined by lack of development of brain myelin, but the cellular mechanisms of hypomyelination are... 
BETA-TUBULIN | MYELIN PROTEIN | WHITE-MATTER | GENE | SPASTIC PARAPLEGIA | ATROPHY | BASAL GANGLIA | BIOCHEMISTRY & MOLECULAR BIOLOGY | GENETICS & HEREDITY | CEREBELLUM | HYPOMYELINATING LEUKODYSTROPHY | H-ABC SYNDROME | Neurons - pathology | Humans | Middle Aged | Atrophy - pathology | Cataract - pathology | Child, Preschool | Male | Tubulin - genetics | Myelin Sheath - metabolism | Brain - metabolism | Young Adult | Leukodystrophy, Metachromatic - metabolism | Tubulin - metabolism | Adult | Female | Child | Leukodystrophy, Metachromatic - pathology | Hereditary Central Nervous System Demyelinating Diseases - genetics | Basal Ganglia - pathology | Cataract - metabolism | Microtubules - pathology | Cerebellum - pathology | Hereditary Central Nervous System Demyelinating Diseases - metabolism | Oligodendroglia - pathology | Leukodystrophy, Metachromatic - genetics | Magnetic Resonance Imaging | Hereditary Central Nervous System Demyelinating Diseases - pathology | Phenotype | Cataract - congenital | Myelin Sheath - genetics | Adolescent | Brain - pathology | Cataract - genetics | HeLa Cells | Mutation | Index Medicus
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4. Full Text Rapamycin slows aging in mice
by Wilkinson, John E and Burmeister, Lisa and Brooks, Susan V and Chan, Chi‐Chao and Friedline, Sabrina and Harrison, David E and Hejtmancik, James F and Nadon, Nancy and Strong, Randy and Wood, Lauren K and Woodward, Maria A and Miller, Richard A
Aging Cell, ISSN 1474-9718, 08/2012, Volume 11, Issue 4, pp. 675 - 682
Rapamycin increases lifespan in mice, but whether this represents merely inhibition of lethal neoplastic diseases, or an overall slowing in multiple aspects of... 
TOR | interventions | longevity pathology | Interventions | Longevity pathology | TARGET | RESTRICTION | CANCER | FERTILITY | CELL BIOLOGY | GERIATRICS & GERONTOLOGY | LIFE-SPAN EXTENSION | METABOLISM | PATHWAY | AMES DWARF MICE | CELL-CYCLE | Liver - pathology | Cataract - chemically induced | Longevity - drug effects | Tendons - drug effects | Aging - drug effects | Endometrium - drug effects | Motor Activity - drug effects | Male | Mice, 129 Strain | Adrenal Gland Neoplasms - chemically induced | Sirolimus - toxicity | Testis - drug effects | TOR Serine-Threonine Kinases - antagonists & inhibitors | Liver - drug effects | Female | Mice, Inbred DBA | Mice, Inbred C57BL | Sirolimus - blood | Organ Specificity | Sirolimus - pharmacology | Mice, Inbred C3H | Aging - pathology | Myocytes, Cardiac - pathology | Animals | Myocytes, Cardiac - drug effects | Testis - pathology | Aging - physiology | Tendons - pathology | Mice | Endometrium - pathology | Longevity - physiology | Analysis | Rapamycin | Ophthalmology | Aging | Medical research | Rodents | Index Medicus
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5. Full Text Optic disc -Fovea distance, axial length and parapapillary Zones. The Beijing eye study 2011
by Jonas, Rahul Arvo and Wang, Ya Xing and Yang, Hua and Li, Jian Jun and Xu, Liang and Panda-Jonas, Songhomitra and Jonas, Jost Bruno
PLoS ONE, ISSN 1932-6203, 09/2015, Volume 10, Issue 9, pp. e0138701 - e0138701
Purpose To measure the distance between the optic disc center and the fovea (DFD) and to assess its associations. Methods The population-based cross-sectional... 
DIAMETER RATIO | RETINAL FEATURES | ATROPHY | FUNDUS | CHINA | MULTIDISCIPLINARY SCIENCES | SUBFOVEAL CHOROIDAL THICKNESS | SIZE | GLAUCOMA | MACULA DISTANCE | DEGENERATION | Multivariate Analysis | Prevalence | Axial Length, Eye - pathology | Cross-Sectional Studies | Bruch Membrane - pathology | Humans | Middle Aged | Cataract - pathology | Fovea Centralis - pathology | Male | Myopia - pathology | Cataract - epidemiology | Optic Atrophy - pathology | Aged, 80 and over | Macular Degeneration - epidemiology | Beijing - epidemiology | Female | Aged | Glaucoma - epidemiology | Glaucoma - pathology | Optic Disk - pathology | Fluorescein Angiography | Macular Degeneration - pathology | Myopia - epidemiology | Macular degeneration | Demographic aspects | Physiological aspects | Optic disc | Personality | Research | Risk factors | Glaucoma | Correlation coefficient | Diabetic retinopathy | Cornea | Correlation coefficients | Cortex | Fovea | Myopia | Science | Optics | Retina | Systematic review | Multivariate analysis | Eye | Mathematical analysis | Anterior chamber | Photographs | Degeneration | Age | Elongation | Enlargement | Eye lens | Index Medicus
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6. Full Text Alzheimer's disease amyloid-β links lens and brain pathology in down syndrome
by Moncaster, Juliet A and Pineda, Roberto and Moir, Robert D and Lu, Suqian and Burton, Mark A and Ghosh, Joy G and Ericsson, Maria and Soscia, Stephanie J and Mocofanescu, Anca and Folkerth, Rebecca D and Robb, Richard M and Kuszak, Jer R and Clark, John I and Tanzi, Rudolph E and Hunter, David G and Goldstein, Lee E
PLoS ONE, ISSN 1932-6203, 2010, Volume 5, Issue 5, pp. e10659 - e10659
Down syndrome (DS, trisomy 21) is the most common chromosomal disorder and the leading genetic cause of intellectual disability in humans. In DS, triplication... 
DEMENTIA | CATARACTS | DEPOSITION | MULTIDISCIPLINARY SCIENCES | HYPOTHESIS | PEOPLE | PROTEIN-PRECURSOR | CRYSTALLIN | CDNA | EXPRESSION | LOCUS | Down Syndrome - metabolism | Humans | Middle Aged | Cataract - pathology | Child, Preschool | Eye Proteins - chemistry | Molecular Sequence Data | Lens, Crystalline - ultrastructure | Male | Lens, Crystalline - pathology | Alzheimer Disease - pathology | Young Adult | Light | Amyloid beta-Peptides - metabolism | Brain - ultrastructure | Protein Structure, Quaternary | Aged, 80 and over | Adult | Female | Scattering, Radiation | Child | Amino Acid Sequence | Down Syndrome - pathology | Aging - pathology | Eye Proteins - metabolism | Adolescent | Alzheimer Disease - metabolism | Brain - pathology | Aged | Amyloid beta-Peptides - chemistry | Aging - metabolism | Cataracts | Brain | Neurosciences | Trisomy | Peptides | Laboratories | Pathogenesis | Cognitive ability | Accumulation | Medical schools | Proteins | Genotype & phenotype | Down's syndrome | Etiology | Surgery | Aging | Genetics | Alzheimer's disease | Age | Enzyme-linked immunosorbent assay | Enzymes | Phenotypes | Incubation | Neurodegenerative diseases | Complications | Light scattering | Down syndrome | Gene expression | Amyloid precursor protein | Children & youth | Pathology | Neurology | Hypotheses | Womens health | Chromosome 21 | Alzheimers disease | Protein interaction | Lenses | Dementia | Index Medicus
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7. Full Text CLPB Mutations Cause 3-Methylglutaconic Aciduria, Progressive Brain Atrophy, Intellectual Disability, Congenital Neutropenia, Cataracts, Movement Disorder
by Wortmann, Saskia B and Ziętkiewicz, Szymon and Kousi, Maria and Szklarczyk, Radek and Haack, Tobias B and Gersting, Søren W and Muntau, Ania C and Rakovic, Aleksandar and Renkema, G. Herma and Rodenburg, Richard J and Strom, Tim M and Meitinger, Thomas and Rubio-Gozalbo, M. Estela and Chrusciel, Elzbieta and Distelmaier, Felix and Golzio, Christelle and Jansen, Joop H and van Karnebeek, Clara and Lillquist, Yolanda and Lücke, Thomas and Õunap, Katrin and Zordania, Riina and Yaplito-Lee, Joy and van Bokhoven, Hans and Spelbrink, Johannes N and Vaz, Frédéric M and Pras-Raves, Mia and Ploski, Rafal and Pronicka, Ewa and Klein, Christine and Willemsen, Michel A.A.P and de Brouwer, Arjan P.M and Prokisch, Holger and Katsanis, Nicholas and Wevers, Ron A
The American Journal of Human Genetics, ISSN 0002-9297, 02/2015, Volume 96, Issue 2, pp. 245 - 257
We studied a group of individuals with elevated urinary excretion of 3-methylglutaconic acid, neutropenia that can develop into leukemia, a neurological... 
ANKYRIN REPEAT | PATIENT | PROTEINS | GENETICS & HEREDITY | Abnormalities, Multiple - pathology | Humans | Atrophy - pathology | Cataract - pathology | Molecular Sequence Data | Intellectual Disability - genetics | Base Sequence | Atrophy - genetics | Neutropenia - pathology | Neutropenia - genetics | Abnormalities, Multiple - genetics | Sarcoplasmic Reticulum Calcium-Transporting ATPases - metabolism | Movement Disorders - pathology | Intellectual Disability - pathology | Adenosine Triphosphatases - metabolism | Endopeptidase Clp - genetics | Zebrafish | Endopeptidase Clp - metabolism | Metabolism, Inborn Errors - genetics | Sequence Analysis, DNA | Exome - genetics | Animals | Brain - pathology | Polymorphism, Single Nucleotide - genetics | Cataract - genetics | Movement Disorders - genetics | Metabolism, Inborn Errors - pathology | Gene mutations | Causes of | Genetic research | Genetic aspects | Research | Mental retardation | Movement disorders | Brain diseases | Genotype & phenotype | Neurology | Congenital diseases | Pathogenesis | Leukemia | Adenosine triphosphatase | Index Medicus
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8. Full Text CLPB Variants Associated with Autosomal-Recessive Mitochondrial Disorder with Cataract, Neutropenia, Epilepsy, and Methylglutaconic Aciduria
by Saunders, Carol and Smith, Laurie and Wibrand, Flemming and Ravn, Kirstine and Bross, Peter and Thiffault, Isabelle and Christensen, Mette and Atherton, Andrea and Farrow, Emily and Miller, Neil and Kingsmore, Stephen F and Ostergaard, Elsebet
The American Journal of Human Genetics, ISSN 0002-9297, 02/2015, Volume 96, Issue 2, pp. 258 - 265
3-methylglutaconic aciduria (3-MGA-uria) is a nonspecific finding associated with mitochondrial dysfunction, including defects of oxidative phosphorylation.... 
RAT-LIVER | PROTEIN | CONGENITAL CATARACT | MUTATION | GENETICS & HEREDITY | MEVALONATE METABOLISM | BARTH-SYNDROME | 3-METHYLGLUTACONIC ACIDURIA | SHUNT PATHWAY | HOMOLOG | HSP78 | Abnormalities, Multiple - pathology | Mitochondrial Diseases - pathology | Genes, Recessive - genetics | Humans | Atrophy - pathology | Cataract - pathology | Child, Preschool | Molecular Sequence Data | Infant | Male | Mutation, Missense - genetics | Base Sequence | Atrophy - genetics | Fatal Outcome | Neutropenia - pathology | Epilepsy - genetics | Female | Neutropenia - genetics | Abnormalities, Multiple - genetics | Infant, Newborn | Fibroblasts - metabolism | Mitochondrial Diseases - genetics | Movement Disorders - pathology | Liver - metabolism | Endopeptidase Clp - genetics | Endopeptidase Clp - metabolism | Metabolism, Inborn Errors - genetics | Sequence Analysis, DNA | Exome - genetics | Brain - pathology | Cataract - genetics | Movement Disorders - genetics | Metabolism, Inborn Errors - pathology | Epilepsy - pathology | Codon, Nonsense - genetics | Greenland | Genetic variation | Physiological aspects | Genetic research | Mitochondrial diseases | Genetic aspects | Research | Neutropenia | Proteins | Mitochondria | Phosphorylation | Genetic disorders | Proteases | Epilepsy | Genomics | Index Medicus | Report
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9. Full Text Vitreo‐retinal pathology and multifocal IOLs
by ASCASO, F.J
Acta Ophthalmologica, ISSN 1755-375X, 09/2017, Volume 95, Issue S259, p. n/a
Cataract surgery is the most common and successful surgical procedure performed today. Multifocal intraocular lenses (m‐ IOL s) offer good uncorrected distance... 
Eye | Surgery | Cataracts | Medical personnel | Complications | Implantation | Retina | Visual acuity | Intraocular lenses | Visual perception | Acuity | Pathology | Eye (anatomy) | Lenses
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10. Full Text Zinc Exacerbates Tau Pathology in a Tau Mouse Model
by Craven, Kristen M and Kochen, William R and Hernandez, Carlos M and Flinn, Jane M
Journal of Alzheimer's Disease, ISSN 1387-2877, 2018, Volume 64, Issue 2, pp. 617 - 630
Hyperphosphorylated tau protein is a key pathology in Alzheimer's disease (AD), frontotemporal dementia, chronic traumatic encephalopathy, and Parkinson's... 
tauopathies | tau proteins | Circadian rhythm | western blotting | zinc | spatial memory | PROTEIN | PHOSPHORYLATION | ABNORMALITIES | ALZHEIMERS-DISEASE | NEUROSCIENCES | SUPPLEMENTATION | COGNITIVE DECLINE | ENHANCED ZINC | MICE | DEFICITS | Cataracts | Elderly people | Phosphorylation | Nesting behavior | Biochemistry | Proteins | Macular degeneration | Nest building | Rodents | Encephalopathy | Dementia disorders | Degeneration | Behavior | Supplementation | Sequestering | Alzheimer's disease | Immune system | Nesting | Circadian rhythms | Neurodegenerative diseases | Drosophila | Transgenic mice | Rhythm | Zinc | Fear conditioning | Pathology | Tau protein | Trace elements | Mice | Frontotemporal dementia | Elderly | Geriatrics | Index Medicus
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11. Full Text Mutational analysis and genotype-phenotype correlations in southern Indian patients with sporadic and familial aniridia
by Dubey, Sushil Kumar and Mahalaxmi, Nagasubramanian and Vijayalakshmi, Perumalsamy and Sundaresan, Periasamy
Molecular Vision, ISSN 1090-0535, 01/2015, Volume 21, pp. 88 - 97
Purpose: Aniridia is a rare panocular disorder characterized by iris hypoplasia and other associated eye anomalies. Heterozygous null mutations in paired box... 
DEFECTS | NONSENSE | DNA | BIOCHEMISTRY & MOLECULAR BIOLOGY | OPHTHALMOLOGY | MESSENGER-RNA DECAY | PAX6 GENE | DELETIONS | MISSENSE MUTATIONS | PREDICTION | REGION | Iris - metabolism | Humans | Middle Aged | Cataract - pathology | Child, Preschool | Fovea Centralis - pathology | Male | Nystagmus, Congenital - genetics | Aniridia - genetics | Aniridia - pathology | DNA Mutational Analysis | Nystagmus, Pathologic - complications | Base Sequence | Child | Amino Acid Sequence | Eye Diseases, Hereditary - pathology | Eye Diseases, Hereditary - complications | Repressor Proteins - genetics | Aniridia - complications | Genetic Heterogeneity | India | Cataract - complications | Adolescent | Cataract - genetics | Mutation | Glaucoma - genetics | Paired Box Transcription Factors - genetics | Retinal Diseases - genetics | Nystagmus, Congenital - complications | Nystagmus, Pathologic - genetics | Open Reading Frames | Molecular Sequence Data | Infant | Retinal Diseases - congenital | Case-Control Studies | Retinal Diseases - pathology | Adult | Female | Eye Proteins - genetics | Nystagmus, Pathologic - pathology | Fovea Centralis - abnormalities | Genetic Association Studies | Introns | Retinal Diseases - complications | PAX6 Transcription Factor | Glaucoma - complications | Iris - pathology | Homeodomain Proteins - genetics | Eye Diseases, Hereditary - genetics | Haploinsufficiency | Nystagmus, Congenital - pathology | Aged | Glaucoma - pathology | Index Medicus
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12. Full Text Ocular phenotypic consequences of a single copy deletion of the Yap1 gene ( Yap1 +/- ) in mice
by Kim, Soohyun and Thomasy, Sara M and Raghunathan, Vijay Krishna and Teixeira, Leandro B C and Moshiri, Ala and FitzGerald, Paul and Murphy, Christopher J
Molecular vision, ISSN 1090-0535, 2019, Volume 25, Issue 1, pp. 129 - 142
To identify the effects of a single copy deletion of ( ) in the mouse eye, the ocular phenotypic consequences of were determined in detail. Complete ophthalmic... 
Retina - metabolism | Eye Abnormalities - diagnostic imaging | Iris - metabolism | Descemet Membrane - metabolism | Microphthalmos - genetics | Cataract - diagnostic imaging | Cataract - pathology | Male | Microphthalmos - pathology | Microphthalmos - metabolism | Epithelium, Corneal - pathology | Iris - diagnostic imaging | Tonometry, Ocular | Corneal Stroma - diagnostic imaging | Female | Epithelium, Corneal - diagnostic imaging | Eye Abnormalities - metabolism | Gene Expression | Microphthalmos - diagnostic imaging | Corneal Stroma - metabolism | Tomography, Optical Coherence | Intraocular Pressure - physiology | Descemet Membrane - diagnostic imaging | Phosphoproteins - genetics | Cataract - metabolism | Eye Abnormalities - genetics | Iris - pathology | Descemet Membrane - pathology | Mice, Knockout | Retina - diagnostic imaging | Phenotype | Animals | Eye Abnormalities - pathology | Adaptor Proteins, Signal Transducing - deficiency | Phosphoproteins - deficiency | Adaptor Proteins, Signal Transducing - genetics | Corneal Stroma - pathology | Fibrosis | Epithelium, Corneal - metabolism | Cataract - genetics | Heterozygote | Mice | Retina - pathology | single copy deletion | microphthalmia | cataract | corneal fibrosis
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13. Full Text Tumor parameters predict the risk of side effects after ruthenium-106 plaque brachytherapy of uveal melanomas
by Tarmann, Lisa and Wackernagel, Werner and Ivastinovic, Domagoj and Schneider, Mona and Winkler, Peter and Langmann, Gerald
PLoS ONE, ISSN 1932-6203, 08/2017, Volume 12, Issue 8, pp. e0183833 - e0183833
To report on radiation-related side effects and complications after ruthenium-106 plaque brachytherapy of uveal melanomas. Medical records of 143 eyes with... 
Retinal Detachment - etiology | Eye - pathology | Melanoma - complications | Retinal Detachment - pathology | Visual Acuity - radiation effects | Humans | Middle Aged | Cataract - pathology | Male | Uveal Neoplasms - complications | Vitreous Hemorrhage - etiology | Optic Nerve Diseases - pathology | Eye - radiation effects | Vitreous Hemorrhage - pathology | Aged, 80 and over | Adult | Female | Ruthenium Compounds - adverse effects | Uveal Neoplasms - pathology | Cataract - etiology | Melanoma - radiotherapy | Melanoma - pathology | Aged | Uveal Neoplasms - radiotherapy | Optic Nerve Diseases - etiology | Brachytherapy - adverse effects | Physiological aspects | Complications and side effects | Care and treatment | Radioisotope brachytherapy | Uvea | Melanoma | Glaucoma | Cataracts | Edema | Retinopathy | Ruthenium | Complications | Radiation | Medical records | Risk | Retina | Family medical history | Neuropathy | Radiation therapy | Patients | Hemorrhage | Eye | Side effects | Detachment | Vascular diseases | Eye (anatomy) | Brachytherapy | Background radiation | Index Medicus
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14.