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PLoS ONE, ISSN 1932-6203, 2011, Volume 6, Issue 1, p. e16442
Background & Aims: CCL25/CCR9 is a non-promiscuous chemokine/receptor pair and a key regulator of leukocyte migration to the small intestine. We investigated... 
PLASMACYTOID DENDRITIC CELLS | CROHNS-DISEASE | SULFATE-MEDIATED COLITIS | ULCERATIVE-COLITIS | BIOLOGY | CHEMOKINE RECEPTOR | CCR9 | CD4(+) T-CELLS | MICE | EXPRESSION | BOWEL-DISEASE | T-Lymphocyte Subsets - immunology | Dextran Sulfate | Acute Disease | Cytokines | Receptors, CCR - metabolism | Dendritic Cells - immunology | Protein Binding - immunology | Mice, Knockout | Animals | Receptors, CCR - immunology | Intestinal Mucosa - immunology | Colitis - chemically induced | Chemokines, CC - immunology | Mice | Colitis - immunology | Macrophages - immunology | Chemokines, CC - metabolism | Dextran | Medical research | RNA | Dendritic cells | Gastrointestinal diseases | Medicine, Experimental | Inflammation | Colitis | Sulfates | T cells | Flow cytometry | Pediatrics | Drinking water | Animal models | Inflammatory bowel diseases | Leukocyte migration | Mucosa | Helper cells | Lymphocytes T | mRNA | Macrophages | Small intestine | Recovery | Lymph nodes | Medical schools | Interleukin 6 | Interleukin 2 | Lymphocytes | Rodents | Gastroenterology | Dextran sulfate | Colon | Antigens | Lymphatic system | Immune response | Nutrition | Tumor necrosis factor-α | Gene expression | Children & youth | Crohns disease | Inflammatory bowel disease | White blood cells | Cytometry | Sodium | γ-Interferon | Large intestine | Interleukin 10 | Sulfate | CCR9 protein | Cell migration | Chemokines
Journal Article
Journal Article
MOLECULAR AND CELLULAR NEUROSCIENCE, ISSN 1044-7431, 12/2016, Volume 77, pp. 1 - 10
Autism is a neurodevelopmental disorder categorized by qualitative impairments in social interaction, communication, and repetitive stereotypic behavior.... 
Spleen | Brain | CELLS | REPERTOIRE | BTBR T plus tf/J | CCR and CXCR | C57BL/6J | NEUROSCIENCES | Autism | MULTIPLE-SCLEROSIS | INFLAMMATION | Resveratrol | CENTRAL-NERVOUS-SYSTEM | MICE | CCR5 | CEREBRAL-ISCHEMIA | LYMPHOCYTES
Journal Article
Immunobiology, ISSN 0171-2985, 2012, Volume 217, Issue 8, pp. 795 - 807
Abstract Analysis of the mechanisms underlying the inflammatory response in amoebiasis is important to understand the immunopathology of the disease. Mucosal... 
Allergy and Immunology | Advanced Basic Science | Amoebiasis | Entamoeba histolytica | Chemokine receptor | Inflammation | Colitis | Regulatory T cells | Chemokines | CHEMOKINE RESPONSES | IGA-SECRETING CELLS | NECROSIS-FACTOR-ALPHA | ENTAMOEBA-HISTOLYTICA | IMMUNOLOGY | LAMINA PROPRIA | INTERLEUKIN 6-DEFICIENT MICE | BOWEL-DISEASE | RETINOIC ACID | EPITHELIAL-CELLS | INTRAEPITHELIAL LYMPHOCYTES | Forkhead Transcription Factors - immunology | Tumor Necrosis Factor-alpha - metabolism | Entamoeba histolytica - immunology | Chemokine CCL20 - metabolism | Receptors, CCR - metabolism | Entamoeba histolytica - physiology | Humans | Interleukin-17 - immunology | Interferon-gamma - metabolism | CD4-Positive T-Lymphocytes - immunology | Chemokine CCL20 - immunology | Flow Cytometry | Forkhead Transcription Factors - metabolism | Inflammation Mediators - metabolism | Tumor Necrosis Factor-alpha - immunology | Chemokine CCL20 - genetics | Chemokines, CC - immunology | Interleukin-6 - metabolism | Dysentery, Amebic - metabolism | Dysentery, Amebic - parasitology | Disease Models, Animal | Interleukin-2 Receptor alpha Subunit - immunology | Inflammation Mediators - immunology | Gene Expression | Chemokine CCL11 - metabolism | Interleukin-4 - metabolism | Mice, Inbred C57BL | Receptors, CCR - genetics | CD4-Positive T-Lymphocytes - metabolism | Chemokine CCL11 - genetics | Reverse Transcriptase Polymerase Chain Reaction | Mice, Knockout | Interleukin-17 - metabolism | Interleukin-4 - immunology | Trophozoites - physiology | Animals | Receptors, CCR - immunology | Interleukin-2 Receptor alpha Subunit - metabolism | Interferon-gamma - immunology | Chemokines, CC - genetics | Interleukin-6 - immunology | Trophozoites - immunology | Chemokine CCL11 - immunology | Dysentery, Amebic - immunology | Mice | Chemokines, CC - metabolism
Journal Article
Journal Article
Journal of Gastroenterology and Hepatology, ISSN 0815-9319, 01/2013, Volume 28, Issue 1, pp. 188 - 201
Background and Aim Hepatic fibrosis is a worldwide healthy burden associated with significant morbidity and mortality. It is caused by a variety of chronic... 
thioacetamide | hepatic stellate cells | fibrosis | imidazolium salts | matrix metalloproteinases | CCR‐2 | CCR-2 | Thioacetamide | Hepatic stellate cells | Imidazolium salts | Matrix metalloproteinases | Fibrosis | ACTIVATION | RAT | LIVER FIBROSIS | STELLATE CELLS | FIBROGENESIS | MECHANISMS | CIRRHOSIS | INFLAMMATION | MICE | GASTROENTEROLOGY & HEPATOLOGY | EXPRESSION | Receptors, CCR2 - genetics | Actins - metabolism | Male | Alanine Transaminase - blood | Phosphatidylinositol 3-Kinases - metabolism | Liver Cirrhosis, Experimental - drug therapy | RNA, Messenger - metabolism | Actins - genetics | MAP Kinase Signaling System | Liver Cirrhosis, Experimental - prevention & control | Imidazoles - therapeutic use | Hepatic Stellate Cells - drug effects | Gene Expression | Cells, Cultured | Hepatic Stellate Cells - enzymology | Matrix Metalloproteinase 3 - metabolism | Imidazoles - pharmacology | Collagen - metabolism | Receptors, CCR2 - metabolism | Animals | Transforming Growth Factor beta - genetics | Analysis of Variance | Aspartate Aminotransferases - blood | Liver Cirrhosis, Experimental - chemically induced | Mice | Mice, Inbred BALB C | Protein Kinase Inhibitors - pharmacology | Matrix Metalloproteinase 3 - genetics | Liver Cirrhosis, Experimental - metabolism | Mitogen-Activated Protein Kinases - metabolism | Messenger RNA | Liver diseases | Peptides | Analysis | Collagen | Genes | Fluorides | Transforming growth factors | Muscle proteins | Gene expression | Cells
Journal Article
European Journal of Operational Research, ISSN 0377-2217, 2011, Volume 209, Issue 2, pp. 129 - 140
This paper aims at integrating data envelopment analysis (DEA) and analytic hierarchy process (AHP) to evaluate the economic development achieved by local... 
BCC model | Economic performance | CCR model | Analytic hierarchy process | Data envelopment analysis | DECISION-MAKING UNITS | OPERATIONS RESEARCH & MANAGEMENT SCIENCE | AHP | DEA MODELS | SELECTION | EFFICIENCY | Data envelopment analysis Analytic hierarchy process CCR model BCC model Economic performance
Journal Article
PLoS ONE, ISSN 1932-6203, 03/2015, Volume 10, Issue 3, p. e0120711
Hepatic steatosis is the accumulation of excess fat in the liver. Recently, hepatic steatosis has become more important because it occurs in the patients with... 
RECRUITMENT | ACTIVATION | DB/DB MICE | FATTY LIVER-DISEASE | INSULIN-RESISTANCE | MACROPHAGES | MULTIDISCIPLINARY SCIENCES | NONALCOHOLIC STEATOHEPATITIS | ENDOPLASMIC-RETICULUM STRESS | DYSFUNCTION | CCR2 | Humans | Diet, High-Fat - adverse effects | Male | Immunoenzyme Techniques | Receptors, CCR2 - antagonists & inhibitors | Inflammation - metabolism | Piperidines - pharmacology | Diabetes Mellitus, Experimental - complications | Real-Time Polymerase Chain Reaction | Diabetes Mellitus, Type 2 - complications | Diabetes Mellitus, Experimental - physiopathology | Disease Models, Animal | Glucose Tolerance Test | Fatty Liver - metabolism | Obesity - complications | Mice, Inbred C57BL | RNA, Messenger - genetics | Cells, Cultured | Fatty Liver - prevention & control | Insulin Resistance | Reverse Transcriptase Polymerase Chain Reaction | Inflammation - etiology | Blotting, Western | Animals | Diabetes Mellitus, Type 2 - physiopathology | Endoplasmic Reticulum Stress | Benzoxazines - pharmacology | Inflammation - prevention & control | Mice | Fatty Liver - etiology | Type 2 diabetes | Obesity | Liver | Insulin resistance | Triglycerides | Inflammation | Glucose | Dextrose | CC chemokine receptors | Kinases | Experiments | Interleukin 6 | Proteins | Mitochondria | Fatty liver | CCR2 protein | Rodents | Physiology | Diabetes mellitus (non-insulin dependent) | Carbon-carbon composites | Stresses | Cytokines | Hyperlipidemia | Diabetes mellitus | Alcoholism | Markers | Glycerol | Tumor necrosis factor-α | Metabolism | Insulin | Fatty acids | Stress | Steatosis | Medicine | Glucose tolerance | Intolerance | Inhibitors | Pharmacy | Diabetes | Endoplasmic reticulum | Endocrinology | Chemokines | Monocyte chemoattractant protein 1
Journal Article
Nature Biotechnology, ISSN 1087-0156, 06/2015, Volume 33, Issue 6, pp. 656 - 660
Journal Article