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PLoS genetics, ISSN 1553-7390, 12/2011, Volume 7, Issue 12, pp. e1002367 - e1002367
Journal Article
Nature genetics, ISSN 1546-1718, 03/2008, Volume 40, Issue 4, pp. 395 - 402
Journal Article
PLoS genetics, ISSN 1553-7404, 02/2017, Volume 13, Issue 2, pp. e1006587 - e1006587
.... It is considered that these genetic variants contribute to disease by altering the binding of regulatory proteins and thus gene expression, but whether these variants alter the binding of lineage... 
SYSTEMIC-LUPUS-ERYTHEMATOSUS | TRANSCRIPTION FACTOR-BINDING | DISTAL REGULATORY ELEMENTS | IMMUNE-SYSTEM | CROHNS-DISEASE | ULCERATIVE-COLITIS | SUSCEPTIBILITY LOCI | INNATE LYMPHOID-CELLS | GENOME-WIDE ASSOCIATION | CELIAC-DISEASE | Life Sciences & Biomedicine | Genetics & Heredity | Science & Technology | Crohn Disease - genetics | Gene Expression | Genetic Predisposition to Disease - genetics | Protein Binding - genetics | Celiac Disease - genetics | Colitis, Ulcerative - metabolism | Humans | CD4-Positive T-Lymphocytes - metabolism | Cells, Cultured | Colitis, Ulcerative - genetics | Crohn Disease - metabolism | Binding Sites - genetics | T-Box Domain Proteins - genetics | Blotting, Western | Regulatory Sequences, Nucleic Acid - genetics | T-Box Domain Proteins - metabolism | Mice, Knockout | Th1 Cells - metabolism | Interleukin-18 Receptor beta Subunit - genetics | Animals | Polymorphism, Single Nucleotide | Genome-Wide Association Study - methods | Interleukin-18 Receptor beta Subunit - metabolism | Celiac Disease - metabolism | Analysis | Inflammation | Genetic aspects | Genetic transcription | Single nucleotide polymorphisms | Research | Health aspects | Risk factors | Medical research | Transcription factors | Biomedical research | Funding | Genomics | Genomes | Grants | Gene expression | Inflammatory diseases | Crohns disease | Celiac disease | Hospitals | Genetics | Autoimmune diseases | Deoxyribonucleic acid--DNA | Cancer | University colleges | Index Medicus | Deoxyribonucleic acid | DNA
Journal Article
The New England journal of medicine, ISSN 0028-4793, 07/2014, Volume 371, Issue 1, pp. 42 - 49
In this cohort study involving 6403 children at increased risk for celiac disease on the basis of HLA haplotype, the risks of celiac disease autoimmunity and celiac disease by the age of 5 years were 26% and 12... 
Medicine, General & Internal | Life Sciences & Biomedicine | General & Internal Medicine | Science & Technology | Autoimmune Diseases - epidemiology | United States - epidemiology | Genetic Predisposition to Disease | Prospective Studies | Celiac Disease - genetics | Humans | Kaplan-Meier Estimate | Child, Preschool | Europe - epidemiology | Infant | Male | Risk | Transglutaminases - immunology | Autoimmune Diseases - genetics | Homozygote | HLA-DR4 Antigen - genetics | HLA-DR3 Antigen - genetics | HLA-DQ Antigens - genetics | Antibodies - blood | Female | Celiac Disease - epidemiology | Infant, Newborn | Haplotypes | Viral antibodies | Celiac disease | Histocompatibility antigens | Analysis | HLA histocompatibility antigens | Antibodies | Children | Health aspects | Risk factors | Diabetes mellitus | Heterozygotes | Environmental factors | Small intestine | Transglutaminase 2 | Children & youth | Homozygotes | Genotype & phenotype | Biopsy | Histocompatibility antigen HLA | Autoimmune diseases | Age | Genotypes | Immune system | Index Medicus | Abridged Index Medicus | Autoimmune Diseases: epidemiology | Celiac Disease: genetics | Medical and Health Sciences | Medicin och hälsovetenskap | Europe: epidemiology | Antibodies: blood | HLA-DQ Antigens: genetics | HLA-DR3 Antigen: genetics | Klinisk medicin | Transglutaminases: immunology | United States: epidemiology | HLA-DR4 Antigen: genetics | Clinical Medicine | Endokrinologi och diabetes | Autoimmune Diseases: genetics | Celiac Disease: epidemiology | Endocrinology and Diabetes
Journal Article
Nature (London), ISSN 1476-4687, 07/2010, Volume 466, Issue 7302, pp. 113 - 117
Alopecia areata (AA) is among the most highly prevalent human autoimmune diseases, leading to disfiguring hair loss due to the collapse of immune privilege of the hair follicle and subsequent autoimmune attack... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Humans | Middle Aged | GPI-Linked Proteins | Immunity, Innate - genetics | NK Cell Lectin-Like Receptor Subfamily K - immunology | Male | Alopecia Areata - genetics | Antigens, CD - genetics | Case-Control Studies | Adaptive Immunity - genetics | T-Lymphocytes, Regulatory - immunology | Autoimmune Diseases - genetics | Ikaros Transcription Factor - genetics | Intercellular Signaling Peptides and Proteins - metabolism | Interleukin-2 Receptor alpha Subunit - genetics | Peroxiredoxins - genetics | T-Lymphocytes, Regulatory - cytology | Qa-SNARE Proteins - genetics | Adult | Female | Hair Follicle - immunology | Adaptive Immunity - immunology | Alopecia Areata - immunology | Genetic Predisposition to Disease | Genome-Wide Association Study | Autoimmune Diseases - immunology | Intercellular Signaling Peptides and Proteins - genetics | CTLA-4 Antigen | Immunity, Innate - immunology | Hair Follicle - cytology | Hair Follicle - metabolism | Alleles | Polymorphism, Single Nucleotide - genetics | Aged | Alopecia | Immune response | Baldness | Genetic aspects | Research | Health aspects | Risk factors | Haplotypes | Review boards | Principal components analysis | Celiac disease | Rheumatoid arthritis | Genetics | Autoimmune diseases | Health risk assessment | Chromosomes | Immune system | Validation studies | Hair | Antigens | Receptors | Pathogenesis | Genes | Zinc | Index Medicus
Journal Article
The journal of clinical endocrinology and metabolism, ISSN 1945-7197, 05/2017, Volume 102, Issue 5, pp. 1468 - 1477
...) subjects and patients with celiac disease (CD) as gut inflammatory disease controls. Design/Setting/Participants... 
Life Sciences & Biomedicine | Endocrinology & Metabolism | Science & Technology | Receptors, CCR2 - genetics | Serum Amyloid P-Component - immunology | Receptors, CCR2 - immunology | Humans | Middle Aged | Tumor Necrosis Factor-alpha - genetics | Transcriptome | Child, Preschool | Male | Monocyte Chemoattractant Proteins - immunology | Vascular Endothelial Growth Factor A - genetics | Antigens, CD - genetics | Interleukin-4 Receptor alpha Subunit - genetics | Young Adult | Celiac Disease - microbiology | Cyclooxygenase 2 - genetics | Intestinal Mucosa - immunology | Diabetes Mellitus, Type 1 - microbiology | Tumor Necrosis Factor-alpha - immunology | Child | Real-Time Polymerase Chain Reaction | Duodenum - immunology | Vascular Endothelial Growth Factor A - immunology | C-Reactive Protein - genetics | Intestinal Mucosa - microbiology | Reverse Transcriptase Polymerase Chain Reaction | Monocyte Chemoattractant Proteins - genetics | Chemokine CCL22 - genetics | Chemokine CCL19 - genetics | Adolescent | RNA, Ribosomal, 16S - genetics | C-Reactive Protein - immunology | Antigens, CD - immunology | Infant | Case-Control Studies | Duodenum - microbiology | Chemokine CCL22 - immunology | Celiac Disease - immunology | Adult | Female | Diabetes Mellitus, Type 1 - immunology | Chemokine CCL19 - immunology | Cyclooxygenase 2 - immunology | Antigens, Differentiation, Myelomonocytic - immunology | Diabetes Mellitus, Type 1 - genetics | Gastrointestinal Microbiome - genetics | Interleukin-4 Receptor alpha Subunit - immunology | Antigens, Differentiation, Myelomonocytic - genetics | Serum Amyloid P-Component - genetics | Aged | Immunohistochemistry | CC chemokine receptors | rRNA 16S | CCL19 protein | Pathogenesis | Mucosa | Microbiomes | Diabetes mellitus (insulin dependent) | Macrophages | Small intestine | Gene sequencing | CCL22 protein | Microbiota | CCR2 protein | Intestine | Interleukin 4 receptors | Digestive tract | Duodenum | Digestive system | Abnormalities | Diabetes mellitus | Inflammation | Tumor necrosis factor-α | Gene expression | Ribonucleic acid--RNA | Disease control | Patients | Celiac disease | Biopsy | Infiltration | Diabetes | Cyclooxygenase-2 | Autoimmune diseases | Monocyte chemoattractant protein 1 | Index Medicus | Abridged Index Medicus
Journal Article