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1. Full Text Review article: anti‐adhesion therapies for inflammatory bowel disease
by Lobatón, T and Vermeire, S and Assche, G and Rutgeerts, P
Alimentary Pharmacology & Therapeutics, ISSN 0269-2813, 03/2014, Volume 39, Issue 6, pp. 579 - 594
Background A high proportion of patients with inflammatory bowel disease (IBD) do not achieve clinical remission with the current therapies including... 
CELL-ADHESION MOLECULE-1 | RELAPSING MULTIPLE-SCLEROSIS | PROGRESSIVE MULTIFOCAL LEUKOENCEPHALOPATHY | EVIDENCE-BASED CONSENSUS | CROHNS-DISEASE | LAMINA PROPRIA LYMPHOCYTES | HUMANIZED MONOCLONAL-ANTIBODY | CD4(+) T-CELLS | PHARMACOLOGY & PHARMACY | ACTIVE ULCERATIVE-COLITIS | PLACEBO-CONTROLLED TRIAL | GASTROENTEROLOGY & HEPATOLOGY | Inflammatory Bowel Diseases - drug therapy | Animals | Humans | Cell Adhesion Molecules - antagonists & inhibitors | Inflammatory Bowel Diseases - physiopathology | T-Lymphocytes - immunology | Colitis, Ulcerative - drug therapy | Tumor Necrosis Factor-alpha - antagonists & inhibitors | Colitis, Ulcerative - physiopathology | Mesalamine | Care and treatment | Ulcerative colitis | Integrins
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2. Full Text Targeting cell adhesion molecules with nanoparticles using in vivo and flow-based in vitro models of atherosclerosis
by Khodabandehlou, Khosrow and Masehi-Lano, Jacqueline J and Poon, Christopher and Wang, Jonathan and Chung, Eun Ji
Experimental Biology and Medicine, ISSN 1535-3702, 4/2017, Volume 242, Issue 8, pp. 799 - 812
Atherosclerosis is a leading cause of death worldwide; in addition to lipid dysfunction, chronic arterial wall inflammation is a key component of... 
atherosclerosis | endothelial cells | targeting | nanoparticles | flow-based models | Cell adhesion molecules | MEDICINE, RESEARCH & EXPERIMENTAL | DRUG-DELIVERY | NANOCARRIERS | VCAM-1 | MULTIFUNCTIONAL NANOPARTICLES | INFLAMMATION | ENDOTHELIAL-CELLS | SHEAR-STRESS | MAGNETIC-RESONANCE | SMOOTH-MUSCLE-CELLS | ACTIVATED ENDOTHELIUM | Atherosclerosis - pathology | Animals | Cell Adhesion - physiology | Endothelial Cells - metabolism | Nanoparticles - therapeutic use | Cell Adhesion Molecules - antagonists & inhibitors | Mice | Cell Adhesion Molecules - metabolism | Bionanoscience
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3. Full Text Anti-inflammatory therapies for cardiovascular disease
by Ridker, Paul M and Lüscher, Thomas F
European Heart Journal, ISSN 0195-668X, 07/2014, Volume 35, Issue 27, pp. 1782 - 1791
Atherothrombosis is no longer considered solely a disorder of lipoprotein accumulation in the arterial wall. Rather, the initiation and progression of... 
Darapladib | Atherosclerosis | Inflammasome | Inflammation | Colchicine | Salsalate | C reactive protein | Canakinumab | Methotrexate | Interleukin-6 | C-REACTIVE PROTEIN | FUTURE MYOCARDIAL-INFARCTION | CARDIAC & CARDIOVASCULAR SYSTEMS | INTERLEUKIN-1 RECEPTOR ANTAGONIST | RANDOMIZED CLINICAL-TRIAL | SECRETORY PHOSPHOLIPASE A | ACUTE CORONARY SYNDROME | APPARENTLY HEALTHY-MEN | REVERSE CHOLESTEROL TRANSPORT | PLACEBO-CONTROLLED TRIAL | TYPE-2 DIABETES-MELLITUS | Hydroxychloroquine - therapeutic use | Interleukin-6 - antagonists & inhibitors | NLR Family, Pyrin Domain-Containing 3 Protein | Colchicine - therapeutic use | Humans | Cell Adhesion Molecules - antagonists & inhibitors | Methotrexate - therapeutic use | Sirtuins - therapeutic use | Vaccination - methods | Lipoproteins, HDL - drug effects | Interleukin-1 - physiology | Interleukin-6 - physiology | Anti-Inflammatory Agents - therapeutic use | Interleukin-1 - antagonists & inhibitors | Leukotriene Antagonists - therapeutic use | Carrier Proteins - physiology | Adaptive Immunity - physiology | Phospholipase A2 Inhibitors - therapeutic use | Atherosclerosis - drug therapy | Serpins - therapeutic use | C-Reactive Protein - physiology | Antioxidants - therapeutic use | Tumor Necrosis Factor-alpha - drug effects | Signal Transduction - drug effects | Tumor Necrosis Factor-alpha - physiology | Thrombosis - drug therapy | Receptors, Interleukin-1 - antagonists & inhibitors | Review
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4. Full Text Anti-inflammatory protein TSG-6 secreted by activated MSCs attenuates zymosan-induced mouse peritonitis by decreasing TLR2/NF-κB signaling in resident macrophages
by Choi, Hosoon and Lee, Ryang Hwa and Bazhanov, Nikolay and Oh, Joo Youn and Prockop, Darwin J
Blood, ISSN 0006-4971, 07/2011, Volume 118, Issue 2, pp. 330 - 338
Human mesenchymal stem/progenitor cells (hMSCs) repair tissues and modulate immune systems but the mechanisms are not fully understood. We demonstrated that... 
RHEUMATOID-ARTHRITIS | COLLAGEN-INDUCED ARTHRITIS | INTER-ALPHA-INHIBITOR | INFLAMMATION | STEM/PROGENITOR CELLS | HYALURONAN | STROMAL CELLS | DBA/1J MICE | HEMATOLOGY | MESENCHYMAL STEM-CELLS | CD44 | Humans | Cell Adhesion Molecules - antagonists & inhibitors | Male | NF-kappa B - metabolism | Anti-Inflammatory Agents - metabolism | Peritonitis - prevention & control | Anti-Inflammatory Agents - therapeutic use | Cell Adhesion Molecules - pharmacology | Drug Evaluation, Preclinical | Macrophages, Peritoneal - drug effects | Mesenchymal Stromal Cells - physiology | Zymosan | Anti-Inflammatory Agents - pharmacology | Mice, Inbred C57BL | RNA, Small Interfering - pharmacology | Cells, Cultured | Mesenchymal Stromal Cells - metabolism | Mice, Transgenic | Peritonitis - chemically induced | Toll-Like Receptor 2 - metabolism | Down-Regulation - drug effects | Animals | Signal Transduction - drug effects | Mice | Cell Adhesion Molecules - therapeutic use | Macrophages, Peritoneal - metabolism | Cell Adhesion Molecules - secretion | Immunobiology | Hematopoiesis and Stem Cells
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5. Full Text Cell adhesion molecules and ischemic stroke
by Yilmaz, Gokhan and Granger, D. Neil
Neurological Research, ISSN 0161-6412, 10/2008, Volume 30, Issue 8, pp. 783 - 793
Objective: To describe the role of adhesion molecules in ischemic stroke. Methods: A PubMed search of literature pertaining to this study was conducted in... 
STROKE | CEREBRAL ISCHEMIA | ADHESION | LEUKOCYTES | PLATELETS | INFLAMMATION | Cerebral ischemia | Stroke | Inflammation | Leukocytes | Platelets | Adhesion | MESSENGER-RNA EXPRESSION | BRAIN-INJURY | THROMBOEMBOLIC STROKE | MONOCLONAL-ANTIBODY | stroke | NEUROSCIENCES | platelets | ANTI-ICAM-1 ANTIBODY | CLINICAL NEUROLOGY | NEUTROPHIL INHIBITORY FACTOR | inflammation | LEUKOCYTE-ENDOTHELIAL ADHERENCE | cerebral ischemia | CEREBRAL-ARTERY OCCLUSION | P-SELECTIN | leukocytes | RAT-BRAIN | Cell Adhesion Molecules - physiology | Humans | Cell Adhesion Molecules - antagonists & inhibitors | Brain Ischemia - metabolism | Brain Ischemia - physiopathology | Stroke - drug therapy | Cell Adhesion Molecules - metabolism | Stroke - physiopathology | Stroke - metabolism | Animals | Signal Transduction - drug effects | Models, Biological | Brain Ischemia - drug therapy | Inflammation Mediators - metabolism | Reperfusion Injury - physiopathology | Anti-Inflammatory Agents - therapeutic use | Signal Transduction - physiology | Inflammation Mediators - antagonists & inhibitors | Inflammation Mediators - physiology | Reperfusion Injury - metabolism
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6. Full Text Heparan Sulfate Organizes Neuronal Synapses through Neurexin Partnerships
by Zhang, Peng and Lu, Hong and Peixoto, Rui T and Pines, Mary K and Ge, Yuan and Oku, Shinichiro and Siddiqui, Tabrez J and Xie, Yicheng and Wu, Wenlan and Archer-Hartmann, Stephanie and Yoshida, Keitaro and Tanaka, Kenji F and Aricescu, A. Radu and Azadi, Parastoo and Gordon, Michael D and Sabatini, Bernardo L and Wong, Rachel O.L and Craig, Ann Marie
Cell, ISSN 0092-8674, 09/2018, Volume 174, Issue 6, pp. 1450 - 1464.e23
Synapses are fundamental units of communication in the brain. The prototypical synapse-organizing complex neurexin-neuroligin mediates synapse development and... 
LRRTM | heparan sulphate | synaptic transmission | thorny excrescence | mossy fiber | synaptogenesis | synaptic adhesion protein | proteoglycan | neurexin | neuroligin | HIPPOCAMPAL SLICES | PTP-SIGMA | BIOCHEMISTRY & MOLECULAR BIOLOGY | SKELETAL DYSPLASIA | BRAIN-DEVELOPMENT | LNS-DOMAIN | DEVELOPMENTAL DELAY | NEURAL CIRCUITS | NEUROLIGINS | AUTISM SPECTRUM DISORDERS | EXPRESSION | CELL BIOLOGY | Humans | Neurons - cytology | Drosophila Proteins - metabolism | Heparitin Sulfate - metabolism | Glycopeptides - analysis | Synapses - metabolism | RNA Interference | Female | Cell Adhesion Molecules, Neuronal - metabolism | Neurons - metabolism | Drosophila Proteins - antagonists & inhibitors | Amino Acid Sequence | Cell Adhesion Molecules, Neuronal - antagonists & inhibitors | Drosophila | Mice, Inbred C57BL | Rats | Neural Cell Adhesion Molecules - genetics | Cell Adhesion Molecules, Neuronal - genetics | Sequence Alignment | Animals | Heparitin Sulfate - chemistry | Neural Cell Adhesion Molecules - metabolism | Protein Binding | Neural Cell Adhesion Molecules - antagonists & inhibitors | Mice | Drosophila Proteins - genetics | RNA, Small Interfering - metabolism | Autism | Yuan (China) | Partnership | Neurons | Zoology | Schizophrenia | Binding proteins | Sulfates | Neurophysiology | Protein binding
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7. Full Text Spatial regulation of VEGF receptor endocytosis in angiogenesis
by Nakayama, Masanori and Nakayama, Akiko and Van Lessen, Max and Yamamoto, Hiroyuki and Hoffmann, Sarah and Drexler, Hannes C. A and Itoh, Norimichi and Hirose, Tomonori and Breier, Georg and Vestweber, Dietmar and Cooper, Jonathan A and Ohno, Shigeo and Kaibuchi, Kozo and Adams, Ralf H
Nature Cell Biology, ISSN 1465-7392, 03/2013, Volume 15, Issue 3, pp. 249 - 260
Activities as diverse as migration, proliferation and patterning occur simultaneously and in a coordinated fashion during tissue morphogenesis. In the growing... 
PAR COMPLEX | MORPHOGENESIS | TIP CELLS | CELL-MIGRATION | VASCULATURE | ENDOTHELIAL-CELLS | NOTCH | TRAFFICKING | POLARITY PROTEINS | POLARIZATION | CELL BIOLOGY | Endothelium, Vascular - cytology | Protein Kinase C - genetics | RNA, Small Interfering - genetics | Cell Adhesion Molecules - genetics | Phosphorylation | Retina - metabolism | Cell Proliferation | Humans | Adaptor Proteins, Vesicular Transport - genetics | Cell Adhesion Molecules - antagonists & inhibitors | Male | Chromatography, High Pressure Liquid | Adaptor Proteins, Vesicular Transport - metabolism | Immunoenzyme Techniques | Morphogenesis | Ephrin-B2 - antagonists & inhibitors | Receptors, Vascular Endothelial Growth Factor - metabolism | Retina - cytology | Protein Kinase C - metabolism | Receptors, Vascular Endothelial Growth Factor - antagonists & inhibitors | Female | Spectrometry, Mass, Matrix-Assisted Laser Desorption-Ionization | Receptors, Vascular Endothelial Growth Factor - genetics | Real-Time Polymerase Chain Reaction | Signal Transduction | Adaptor Proteins, Vesicular Transport - antagonists & inhibitors | RNA, Messenger - genetics | Cells, Cultured | Rats | Mice, Transgenic | Reverse Transcriptase Polymerase Chain Reaction | Cell Adhesion Molecules - metabolism | Ephrin-B2 - metabolism | Blotting, Western | Endocytosis - physiology | Ephrin-B2 - genetics | Animals | Endothelium, Vascular - metabolism | Mice | Neovascularization, Physiologic | Cell Movement | Cellular control mechanisms | Research | Properties | Observations | Vascular endothelial growth factor | Endothelium | Testing
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8. Full Text Blocking adhesion molecules as therapy for multiple sclerosis: natalizumab
by Steinman, Lawrence
Nature Reviews Drug Discovery, ISSN 1474-1776, 06/2005, Volume 4, Issue 6, pp. 510 - 518
Immunologists have long hypothesized that particular 'molecular addresses' govern lymphocyte entry to a given organ. In 1992, α 4β 1 integrin was identified as... 
MYELIN BASIC-PROTEIN | ALPHA-4-BETA-1 INTEGRIN | EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS | L-SELECTIN | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | NONOBESE DIABETIC MICE | EXPERIMENTAL ALLERGIC ENCEPHALOMYELITIS | PREVENTION | PHARMACOLOGY & PHARMACY | CENTRAL-NERVOUS-SYSTEM | SUPPRESSES | I-A ANTIBODY | Antibodies, Monoclonal, Humanized | Animals | Natalizumab | Antibodies, Monoclonal - pharmacology | Humans | Cell Adhesion Molecules - antagonists & inhibitors | Antibodies, Monoclonal - therapeutic use | Cell Adhesion Molecules - metabolism | Multiple Sclerosis - drug therapy | Multiple Sclerosis - metabolism | Drug therapy | Multiple sclerosis
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