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Molecular Cell, ISSN 1097-2765, 05/2016, Volume 62, Issue 3, pp. 462 - 471
Journal Article
Developmental Cell, ISSN 1534-5807, 03/2012, Volume 22, Issue 3, pp. 501 - 514
Gradients of vascular endothelial growth factor (VEGF) induce single endothelial cells to become leading tip cells of emerging angiogenic sprouts. Tip cells... 
DEFECTS | TIP CELLS | ANGIOGENESIS | VEGF | ENDOTHELIAL-CELLS | ID PROTEINS | HES1 | DEVELOPMENTAL BIOLOGY | DIFFERENTIATION | EXPRESSION | INHIBITS TUMOR-GROWTH | CELL BIOLOGY | Transcription Factor HES-1 | Humans | Intercellular Signaling Peptides and Proteins - biosynthesis | Intracellular Signaling Peptides and Proteins - metabolism | Inhibitor of Differentiation Proteins - biosynthesis | Inhibitor of Differentiation Protein 1 - biosynthesis | Serrate-Jagged Proteins | Smad5 Protein - metabolism | Basic Helix-Loop-Helix Transcription Factors - biosynthesis | Smad1 Protein - genetics | Membrane Proteins - metabolism | Smad5 Protein - genetics | Intracellular Signaling Peptides and Proteins - genetics | Jagged-1 Protein | Calcium-Binding Proteins - biosynthesis | Homeodomain Proteins - biosynthesis | Signal Transduction | Inhibitor of Differentiation Protein 2 - biosynthesis | Membrane Proteins - genetics | Down-Regulation | Cells, Cultured | Mice, Transgenic | Cell Cycle Proteins - biosynthesis | Vascular Endothelial Growth Factor Receptor-1 - biosynthesis | Mice, Knockout | Membrane Proteins - biosynthesis | Phenotype | Animals | Smad1 Protein - metabolism | Mice | Neovascularization, Physiologic | Proteins | Endothelial growth factors | Genes | Genetic aspects | Transforming growth factors | Vascular endothelial growth factor | Endothelium | Dll4 | tip cell | lateral inhibition | sprouting angiogenesis | BMP | directed migration | Notch | stalk cell | mouse embryo | Smad | polarity
Journal Article
Cancer Cell, ISSN 1535-6108, 2010, Volume 18, Issue 3, pp. 244 - 257
Journal Article
Journal Article
细胞研究:英文版, ISSN 1001-0602, 2015, Volume 25, Issue 4, pp. 412 - 428
Journal Article
Circulation, ISSN 0009-7322, 03/2016, Volume 133, Issue 11, pp. 1081 - 1092
BACKGROUND—Adult mammalian cardiomyocytes (CMs) have the potential to proliferate, but this is not sufficient to generate adequate CMs after myocardial... 
Myocardial infarction | Regeneration | Myocytes, cardiac | Molecular biology | Cell cycle | molecular biology | RENEWAL | CARDIAC & CARDIOVASCULAR SYSTEMS | cell cycle | regeneration | STAGE | myocardial infarction | GENES | YAP | PERIPHERAL VASCULAR DISEASE | myocytes | cardiac | Single-Blind Method | T-Box Domain Proteins - physiology | RNA, Small Interfering - genetics | T-Box Domain Proteins - biosynthesis | Muscle Proteins - biosynthesis | RNA, Messenger - biosynthesis | RNA Interference | Cell Division | Myocardial Infarction - pathology | Tumor Suppressor Proteins - genetics | Cell Cycle Proteins - genetics | Electrocardiography | Female | Myocardial Infarction - physiopathology | Genes, Reporter | Heart - physiopathology | Immediate-Early Proteins - physiology | Cell Size - drug effects | Immediate-Early Proteins - biosynthesis | RNA, Messenger - genetics | Genes, cdc - drug effects | Mice, Transgenic | Myocardium - pathology | Cell Cycle Proteins - biosynthesis | Myocardial Infarction - metabolism | Random Allocation | Fetal Proteins - biosynthesis | T-Box Domain Proteins - genetics | Gene Expression Regulation - drug effects | Muscle Proteins - genetics | Myocytes, Cardiac - pathology | Tumor Suppressor Proteins - physiology | Organ Size - drug effects | Animals | Immediate-Early Proteins - genetics | Signal Transduction - drug effects | Tamoxifen - pharmacology | Myocytes, Cardiac - metabolism | Fetal Proteins - genetics | Mice | Tumor Suppressor Proteins - biosynthesis | Cell proliferation | Care and treatment | Transcription factors | Research | Heart attack | Heart cells | remodeling
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 02/2015, Volume 125, Issue 2, pp. 636 - 651
Cornelia de Lange syndrome (CdLS) is a genetically heterogeneous disorder that presents with extensive phenotypic variability, including facial dysmorphism,... 
SISTER-CHROMATID COHESION | INDIVIDUALS | MEDICINE, RESEARCH & EXPERIMENTAL | HYPERTRICHOSIS-CUBITI | VARIANTS | GENETIC-HETEROGENEITY | ACTIVATOR PROTEIN | MUTATIONS | IDENTIFICATION | HUMAN HOMOLOG | NIPBL REARRANGEMENTS | Myeloid-Lymphoid Leukemia Protein - biosynthesis | Histone Deacetylases - biosynthesis | Chondroitin Sulfate Proteoglycans - genetics | Humans | Transcriptome | Child, Preschool | Exonucleases | Infant | Male | Gene Expression Profiling | Exome | De Lange Syndrome - metabolism | Chromosomal Proteins, Non-Histone - biosynthesis | Cell Cycle Proteins - genetics | Adult | Chondroitin Sulfate Proteoglycans - biosynthesis | Child | De Lange Syndrome - genetics | Genome-Wide Association Study | Histone Deacetylases - genetics | Histone-Lysine N-Methyltransferase | Gene Expression Regulation | Repressor Proteins - genetics | Cell Cycle Proteins - biosynthesis | Codon, Nonsense | Chromosomal Proteins, Non-Histone - genetics | Proteins - genetics | Phenotype | Proteins - metabolism | Repressor Proteins - biosynthesis | Myeloid-Lymphoid Leukemia Protein - genetics | Adolescent | Heterozygote | De Lange Syndrome - pathology | De Lange syndrome | Gene mutations | Exome sequencing | Genetic research | Development and progression | Genetic transcription | Research | Diagnosis | Identification and classification | Methods | Proteins | Studies | Genotype & phenotype | Genomics | Zebrafish | Genomes | Mutation | Chromosomes
Journal Article
Cancer Research, ISSN 0008-5472, 02/2007, Volume 67, Issue 4, pp. 1626 - 1635
Cervical cancer is a leading cause of death due to cancer among women worldwide. Using transgenic mice to dissect the contributions of the human papillomavirus... 
UTERINE CERVIX | DIFFERENTIAL EXPRESSION | EPITHELIAL-CELLS | ONCOLOGY | UBIQUITIN-PROTEIN LIGASE | HUMAN SCRIBBLE | TYPE-16 E7 ONCOGENE | TUMOR-SUPPRESSOR PROTEIN | MEDIATED DEGRADATION | TRANSGENIC MOUSE MODEL | HUMAN HOMOLOG | Cell Cycle - genetics | Up-Regulation | Tumor Suppressor Protein p53 - biosynthesis | Skin - metabolism | Humans | Uterine Cervical Neoplasms - pathology | Minichromosome Maintenance Complex Component 7 | Oncogenes - physiology | Cyclin E - genetics | Tumor Suppressor Protein p53 - genetics | Retinoblastoma Protein - biosynthesis | Nuclear Proteins - biosynthesis | Uterine Cervical Neoplasms - chemically induced | Cell Cycle Proteins - genetics | Cyclin-Dependent Kinase Inhibitor p16 - biosynthesis | Female | Oncogene Proteins, Viral - genetics | Nuclear Proteins - genetics | Skin - virology | Estradiol - pharmacology | Repressor Proteins - metabolism | Oncogene Proteins, Viral - metabolism | Cyclin-Dependent Kinase Inhibitor p16 - genetics | Repressor Proteins - genetics | Mice, Transgenic | Cell Cycle Proteins - biosynthesis | DNA-Binding Proteins - genetics | Oncogene Proteins, Viral - biosynthesis | Cocarcinogenesis | Animals | Repressor Proteins - biosynthesis | Retinoblastoma Protein - genetics | Cyclin E - biosynthesis | Uterine Cervical Neoplasms - virology | Mice | DNA-Binding Proteins - biosynthesis
Journal Article
Carcinogenesis, ISSN 0143-3334, 03/2010, Volume 31, Issue 3, pp. 496 - 503
The polycomb group (PcG) proteins are epigenetic regulators of gene expression that enhance cell survival. This regulation is achieved via action of two... 
HISTONE METHYLTRANSFERASE ACTIVITY | SIGNALING PATHWAYS | INCREASED EXPRESSION | DNA METHYLATION | ONCOLOGY | GROUP GENE | PROSTATE-CANCER | DEPENDENT KINASE INHIBITORS | HUMAN KERATINOCYTES | GREEN TEA POLYPHENOL | MAMMARY EPITHELIAL-CELLS | Apoptosis - drug effects | DNA Replication - drug effects | Carcinoma, Squamous Cell - pathology | Cell Count | Humans | Neoplasm Proteins - physiology | Recombinant Fusion Proteins - physiology | Anticarcinogenic Agents - antagonists & inhibitors | Culture Media, Serum-Free | Cell Line, Tumor - drug effects | Cell Line, Transformed - drug effects | Proto-Oncogene Proteins - biosynthesis | Gene Knockdown Techniques | Repressor Proteins - physiology | Nuclear Proteins - biosynthesis | Cell Cycle Proteins - genetics | Epigenesis, Genetic - drug effects | Catechin - pharmacology | Neoplasm Proteins - genetics | Nuclear Proteins - genetics | Anticarcinogenic Agents - pharmacology | Cell Line, Transformed - cytology | Skin Neoplasms - pathology | DNA-Binding Proteins - physiology | Transcription Factors - physiology | Neoplasm Proteins - biosynthesis | Catechin - antagonists & inhibitors | Repressor Proteins - genetics | Proto-Oncogene Proteins - genetics | Transcription Factors - biosynthesis | Cell Cycle Proteins - biosynthesis | Keratinocytes - cytology | Polycomb-Group Proteins | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Enhancer of Zeste Homolog 2 Protein | Polycomb Repressive Complex 2 | Polycomb Repressive Complex 1 | Repressor Proteins - biosynthesis | Genetic Vectors - pharmacology | Keratinocytes - drug effects | Cell Line, Tumor - cytology | Proto-Oncogene Proteins - physiology | Catechin - analogs & derivatives | Nuclear Proteins - physiology | Cell Cycle Proteins - physiology | DNA-Binding Proteins - biosynthesis | Index Medicus | Cancer Prevention
Journal Article