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Journal of Clinical Investigation, ISSN 0021-9738, 03/2012, Volume 122, Issue 3, pp. 821 - 832
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Nature, ISSN 0028-0836, 04/2017, Volume 544, Issue 7651, pp. 503 - 507
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Nature Medicine, ISSN 1078-8956, 05/2012, Volume 18, Issue 5, pp. 783 - 790
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Nature, ISSN 0028-0836, 10/2017, Volume 550, Issue 7675, pp. 260 - 264
Mammalian organs vary widely in regenerative capacity. Poorly regenerative organs, such as the heart are particularly vulnerable to organ failure. Once... 
SURVIVAL | APOPTOSIS | MYOCARDIAL-INFARCTION | REGENERATION | TAMOXIFEN | MULTIDISCIPLINARY SCIENCES | INJURY | DYNAMICS | GENERATION | CARDIAC FIBROSIS | TRANSGENIC MICE | Genetic Therapy | Myocardial Infarction - genetics | Protein-Serine-Threonine Kinases - deficiency | Cell Proliferation | Humans | RNA, Messenger - metabolism | Heart Failure, Systolic - pathology | Myocardial Infarction - pathology | Cell Cycle Proteins - genetics | Protein-Serine-Threonine Kinases - metabolism | Quality Control | Heart Failure, Systolic - etiology | Myocytes, Cardiac - cytology | Heart Failure, Systolic - therapy | RNA, Messenger - genetics | Cell Cycle Proteins - metabolism | Protein-Serine-Threonine Kinases - genetics | Cell Cycle Proteins - deficiency | Signal Transduction - genetics | Mice, Knockout | Heart Failure, Systolic - metabolism | Myocardial Infarction - complications | Myocytes, Cardiac - pathology | Animals | Myocytes, Cardiac - metabolism | Mice | Ubiquitin-Protein Ligases - genetics | Heart failure | Complications and side effects | Infarction | Cellular signal transduction | Health aspects | Risk factors | Myocardial infarction | Heart | Purification | Heart attacks | Genes | Organs | Viruses | Cardiomyocytes | mRNA | Heart function | Kinases | Gene expression | Mitochondria | Clonal deletion | Ribonucleic acids | Quality control | Fibrosis | Myocardium | Fibroblasts | Gene therapy | Heart diseases | Growth factors
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Nature, ISSN 0028-0836, 11/2011, Volume 479, Issue 7372, pp. 237 - 240
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Nature, ISSN 0028-0836, 03/2013, Volume 495, Issue 7442, pp. 524 - 528
Macrophages consist of at least two subgroups, M1 and M2 (refs 1-3). Whereas M1 macrophages are proinflammatory and have a central role in host defence against... 
GLUCOSE-HOMEOSTASIS | OBESITY | TRIBBLES HOMOLOG | INSULIN-RESISTANCE | INFLAMMATION | MULTIDISCIPLINARY SCIENCES | MYELOID LEUKEMOGENESIS | POLARIZATION | ASSOCIATION | ALTERNATIVE ACTIVATION | ADIPOSE-TISSUE | Neutrophils - cytology | Protein-Serine-Threonine Kinases - deficiency | CCAAT-Enhancer-Binding Protein-alpha - metabolism | Eosinophils - metabolism | Hypertriglyceridemia - genetics | Cell Count | Ubiquitin - metabolism | Adipose Tissue - cytology | Diet, High-Fat - adverse effects | Male | Intracellular Signaling Peptides and Proteins - metabolism | Lung - cytology | Intracellular Signaling Peptides and Proteins - deficiency | Adipose Tissue - metabolism | Inflammation Mediators - metabolism | Protein-Serine-Threonine Kinases - antagonists & inhibitors | Cell Cycle Proteins - genetics | Female | Cell Differentiation | Cytokines - genetics | Intracellular Signaling Peptides and Proteins - genetics | Neutrophils - metabolism | Protein-Serine-Threonine Kinases - metabolism | Protein Structure, Tertiary | Macrophages - classification | Bone Marrow Cells - cytology | Adipose Tissue - pathology | Cell Cycle Proteins - metabolism | Protein-Serine-Threonine Kinases - genetics | Cell Cycle Proteins - deficiency | Lipodystrophy - metabolism | Spleen - cytology | Macrophages - cytology | Organ Specificity | Lipolysis | Hypertriglyceridemia - chemically induced | Macrophages - metabolism | Animals | Intracellular Signaling Peptides and Proteins - chemistry | Insulin Resistance - genetics | Lipodystrophy - pathology | Lipodystrophy - chemically induced | Mice | Protein-Serine-Threonine Kinases - chemistry | Eosinophils - cytology | Bone Marrow Cells - metabolism | Proteins | Care and treatment | Metabolic diseases | Cellular signal transduction | Research | Macrophages | Properties | Identification and classification | Testing | Studies | Spleen | Heart attacks | Transplants & implants | Bone marrow | Binding sites | Metabolic disorders | Defects
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The EMBO Journal, ISSN 0261-4189, 05/2012, Volume 31, Issue 9, pp. 2076 - 2089
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Journal of Experimental Medicine, ISSN 0022-1007, 2014, Volume 211, Issue 6, pp. 1027 - 1036
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