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Cell reports (Cambridge), ISSN 2211-1247, 2016, Volume 16, Issue 10, pp. 2576 - 2592
..., radial glial cells (RGCs), causing disrupted mitoses, supernumerary centrosomes, structural disorganization, and cell death... 
NEURAL PROGENITORS | LONG-TERM | HUMAN BRAIN | ADAPTER | CENTRAL-NERVOUS-SYSTEM | INFECTION | MICE | BINDING KINASE 1 | ORGANOIDS | INNATE IMMUNITY | CELL BIOLOGY | Neocortex - pathology | Neurons - pathology | Transcription, Genetic - drug effects | Brain - embryology | Neuroglia - ultrastructure | Neuroglia - pathology | Humans | Brain - virology | Centrosome - drug effects | Gene Expression Profiling | Microcephaly - virology | Neural Stem Cells - ultrastructure | Zika Virus Infection - virology | Neural Stem Cells - immunology | Neuroepithelial Cells - immunology | Neuroprotective Agents - pharmacology | Spinal Cord - pathology | Microcephaly - pathology | Neuroepithelial Cells - virology | Nucleosides - pharmacology | Fetus - virology | Cell Death - drug effects | Phosphorylation - drug effects | Neurons - drug effects | Protein-Serine-Threonine Kinases - metabolism | Zika Virus - pathogenicity | Proto-Oncogene Proteins - metabolism | Zika Virus - ultrastructure | Neurons - virology | Virus Replication - drug effects | Neuroepithelial Cells - ultrastructure | Immunity, Innate - drug effects | Zika Virus Infection - pathology | Neural Stem Cells - virology | Zika Virus - physiology | Zika Virus - drug effects | Mitochondria - metabolism | Mitochondria - drug effects | Receptor Protein-Tyrosine Kinases - metabolism | Neural Stem Cells - enzymology | Centrosome - metabolism | Mitosis - drug effects | Brain - pathology | Protein Kinase Inhibitors - pharmacology | Neuroglia - virology | Neuroepithelial Cells - drug effects | Neurons/pathology | Zika Virus/pathogenicity | Mitochondria/metabolism | Virus Replication/drug effects | Microcephaly/pathology | Neurons/drug effects | Protein-Serine-Threonine Kinases/metabolism | Neural Stem Cells/immunology | Neuroglia/ultrastructure | Neuroepithelial Cells/drug effects | Neuroepithelial Cells/virology | Life Sciences | Brain/pathology | Zika Virus/drug effects | Brain/embryology | Mitochondria/drug effects | Fetus/virology | Neocortex/pathology | Neuroglia/pathology | Cell Death/drug effects | Mitosis/drug effects | Transcription, Genetic/drug effects | Nucleosides/pharmacology | Neural Stem Cells/enzymology | Neural Stem Cells/ultrastructure | Neuroepithelial Cells/ultrastructure | Receptor Protein-Tyrosine Kinases/metabolism | Microcephaly/virology | Proto-Oncogene Proteins/metabolism | Neuroprotective Agents/pharmacology | Zika Virus/ultrastructure | Neuroepithelial Cells/immunology | Brain/virology | Immunity, Innate/drug effects | Spinal Cord/pathology | Zika Virus/physiology | Neuroglia/virology | Microbiology and Parasitology | Zika Virus Infection/pathology | Neurons/virology | Zika Virus Infection/virology | Neural Stem Cells/virology | Centrosome/drug effects | Protein Kinase Inhibitors/pharmacology | Centrosome/metabolism | Phosphorylation/drug effects
Journal Article
Drug resistance updates, ISSN 1368-7646, 2011, Volume 14, Issue 3, pp. 191 - 201
.... Three basic categories underlie most cases of chemotherapy failure: Inadequate pharmacokinetic properties of the drug, tumor cell intrinsic factors such as the... 
Infectious Disease | Hematology, Oncology and Palliative Medicine | HIF-2α | Hypoxia | HIF-1α | Microenvironment | Drug resistance | Autophagy | Cancer | p53 | CANCER-CELLS | TRANSCRIPTIONAL ACTIVITY | HIF-1 alpha | HIF-2 alpha | INDUCIBLE FACTOR 1-ALPHA | ENDOTHELIAL-GROWTH-FACTOR | DNA-DAMAGE | BREAST-CANCER | FACTOR-I-ALPHA | PHARMACOLOGY & PHARMACY | FACTORS HIF-1-ALPHA | MULTIDRUG-RESISTANCE | FACTOR-1 HIF-1 | Neoplasms - metabolism | Apoptosis - drug effects | Humans | Gene Expression Regulation, Neoplastic | Hypoxia-Inducible Factor 1, alpha Subunit - antagonists & inhibitors | Autophagy - drug effects | Tumor Suppressor Protein p53 - genetics | Hypoxia - metabolism | Basic Helix-Loop-Helix Transcription Factors - metabolism | Neoplasms - genetics | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Antineoplastic Agents - pharmacology | Tumor Microenvironment - drug effects | Cell Hypoxia - drug effects | Basic Helix-Loop-Helix Transcription Factors - genetics | Hypoxia-Inducible Factor 1, alpha Subunit - genetics | Tumor Suppressor Protein p53 - metabolism | Basic Helix-Loop-Helix Transcription Factors - antagonists & inhibitors | Mitochondria - metabolism | Mitochondria - drug effects | Neoplasms - drug therapy | Cell Cycle | Signal Transduction - drug effects | Cell Line, Tumor | Cell Proliferation - drug effects | Cell Hypoxia - genetics | Neoplasms - pathology | Drug Resistance, Neoplasm - drug effects | Chemotherapy | Cell death | Drug interactions | Biochemistry | DNA binding proteins | Tumor proteins | Tumors
Journal Article
Nature medicine, ISSN 1546-170X, 2016, Volume 22, Issue 10, pp. 1101 - 1107
Journal Article
Journal
Autophagy, ISSN 1554-8627, 09/2012, Volume 8, Issue 9, pp. 1333 - 1341
...B AS ic Re S e AR ch P AP e R B AS ic Re S e AR ch P AP e R A TG7 deficiency suppresses apoptosis and cell death induced by lysosomal photodamage David h... 
apoptosis | lysosomes | autophagy | subcellular localization | photodynamic therapy | Binding | Proteins | Landes | Calcium | Bioscience | Biology | Cell | Cycle | Cancer | Organogenesis | Lysosomes | Photodynamic therapy | Autophagy | Subcellular localization | Apoptosis | CANCER-CELLS | ACTIVATION | AUTOPHAGIC RESPONSES | WST11 | CELL BIOLOGY | CYTOCHROME-C | BCL-2 | PHOTOSENSITIZER NPE6 | REACTIVE OXYGEN | MEMBRANE PERMEABILIZATION | Apoptosis - drug effects | Apoptosis - radiation effects | Microtubule-Associated Proteins - metabolism | Fluorescence | Phagosomes - radiation effects | Protein Transport - drug effects | Recombinant Fusion Proteins - metabolism | Vacuoles - drug effects | Cell Shape - radiation effects | Lysosomes - radiation effects | Photosensitizing Agents - pharmacology | Cell Nucleus - metabolism | Lysosomes - metabolism | Cell Nucleus - radiation effects | Light | Phagosomes - ultrastructure | Microtubule-Associated Proteins - deficiency | Phagosomes - drug effects | Lysosomes - drug effects | Cell Survival - drug effects | Green Fluorescent Proteins - metabolism | Permeability - drug effects | Subcellular Fractions - drug effects | Bacteriochlorophylls - pharmacology | Porphyrins - pharmacology | Cell Nucleus - ultrastructure | Amines - metabolism | Cell Survival - radiation effects | Subcellular Fractions - metabolism | Cell Shape - drug effects | Vacuoles - radiation effects | Autophagy-Related Protein 7 | Animals | Permeability - radiation effects | Protein Transport - radiation effects | Subcellular Fractions - radiation effects | Bacteriochlorophylls - chemistry | Cell Line, Tumor | Vacuoles - metabolism | Mice | Cell Nucleus - drug effects | Basic Research Paper
Journal Article
Journal Article
The Journal of neuroscience, ISSN 0270-6474, 02/2010, Volume 30, Issue 8, pp. 2967 - 2978
Journal Article
Journal of the American College of Cardiology, ISSN 0735-1097, 2010, Volume 56, Issue 25, pp. 2115 - 2125
Objectives In this study, we have investigated the effects of cannabidiol (CBD) on myocardial dysfunction, inflammation, oxidative/nitrative stress, cell death... 
Cardiovascular | Internal Medicine | diabetic complications | cannabinoids | inflammation | oxidative stress | CONSTITUENT | CARDIAC & CARDIOVASCULAR SYSTEMS | METALLOTHIONEIN | MITOCHONDRIAL | ENDOCANNABINOID SYSTEM | ANTAGONISM | PROTECTS | INHIBITION | DISEASE | NF-KAPPA-B | Diabetic Cardiomyopathies - metabolism | Reactive Oxygen Species - metabolism | Diabetic Cardiomyopathies - drug therapy | Apoptosis - drug effects | Humans | Body Weight - drug effects | Male | NF-kappa B - metabolism | Glucose | Cannabidiol - therapeutic use | Drug Evaluation, Preclinical | Disease Models, Animal | Mice, Inbred C57BL | Cells, Cultured | Pancreas - drug effects | Cannabidiol - pharmacology | Myocardium - pathology | Pancreas - metabolism | Blood Glucose - drug effects | Insulin - metabolism | Animals | MAP Kinase Signaling System - drug effects | Myocytes, Cardiac - drug effects | Fibrosis | Diabetic Cardiomyopathies - pathology | Hemodynamics - drug effects | Mice | Oxidative Stress - drug effects | Phosphates | Oxidative stress | Medical colleges | Niacinamide | Cardiomyopathy | Alcoholism | Radiation | Superoxide | Biochemistry | Dextrose | Purines | Cannabinoids | Cell death | Analysis | Nitric oxide | Protein kinases | Heart diseases | Adenosine triphosphatase | Jewish schools | Mortality | Cardiovascular disease | Cardiomyocytes | Kinases | Experiments | Variance analysis | Antioxidants | Proteins | Polymerase chain reaction | Rodents | Atherosclerosis | Diabetes | Pancreas | Apoptosis
Journal Article
Journal Article