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Neuron, ISSN 0896-6273, 12/2014, Volume 84, Issue 6, pp. 1213 - 1225
Journal Article
Circulation, ISSN 0009-7322, 06/2012, Volume 125, Issue 25, pp. 3170 - 3181
Journal Article
Nature Cell Biology, ISSN 1465-7392, 02/2009, Volume 11, Issue 2, pp. 211 - 218
The phosphatidylinositol-3-kinase-like kinase ATM (ataxia-telangiectasia mutated) has a central role in coordinating DNA damage responses, including cell-cycle... 
INDUCED CELL-DEATH | NERVOUS-SYSTEM | APOPTOSIS | ACTIVATION | TRANSCRIPTION | ATAXIA-TELANGIECTASIA | AUTOPHOSPHORYLATION | CYCLIN-DEPENDENT KINASE-5 | P53 | REQUIREMENT | CELL BIOLOGY | Cell Cycle - genetics | Phosphorylation | Rats, Long-Evans | Nerve Degeneration - genetics | Amino Acid Sequence - genetics | Nerve Degeneration - metabolism | Tumor Suppressor Protein p53 - genetics | Cell Cycle Proteins - chemistry | Cyclin-Dependent Kinase 5 - genetics | DNA-Binding Proteins - metabolism | Cell Death - genetics | Tumor Suppressor Proteins - chemistry | Tumor Suppressor Proteins - genetics | Apoptosis Regulatory Proteins - genetics | Cell Cycle Proteins - genetics | Protein-Serine-Threonine Kinases - metabolism | bcl-2-Associated X Protein - genetics | Animals, Newborn | Tumor Suppressor Proteins - metabolism | Cell Cycle Proteins - metabolism | Cells, Cultured | Protein-Serine-Threonine Kinases - genetics | Tumor Suppressor Protein p53 - metabolism | bcl-2-Associated X Protein - metabolism | Rats | Signal Transduction - genetics | Ataxia Telangiectasia Mutated Proteins | DNA-Binding Proteins - genetics | DNA-Binding Proteins - chemistry | Serine - metabolism | Apoptosis Regulatory Proteins - metabolism | Cyclin-Dependent Kinase 5 - metabolism | Animals | Histones - genetics | Protein-Serine-Threonine Kinases - chemistry | DNA Damage | Histones - metabolism | Enzyme Activation - genetics | DNA damage | Physiological aspects | Ataxia telangiectasia | Genetic aspects | Research | Risk factors | Index Medicus
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 11/2013, Volume 288, Issue 47, pp. 33927 - 33938
Pancreatic cancer, the fourth most prevalent cancer-related cause of death in the United States, is a disease with a dismal survival rate of 5% 5 years after... 
PATHWAYS | APOPTOSIS | ACTIVATION | ADENOCARCINOMA | BIOCHEMISTRY & MOLECULAR BIOLOGY | Pancreatic Cancer | KAPPA-B | O-GlcNAc | N-ACETYLGLUCOSAMINE TRANSFERASE | Heat Shock Protein | Minnelide | IN-VITRO | INHIBITION | Sp1 | Triptolide | Pancreas | TUMOR-GROWTH | EXPRESSION | Pancreatic Neoplasms - metabolism | Humans | Cell Survival - genetics | N-Acetylglucosaminyltransferases - genetics | Antineoplastic Agents, Alkylating - pharmacology | NF-kappa B - metabolism | Sp1 Transcription Factor - metabolism | Glycosylation - drug effects | Neoplasm Proteins - metabolism | Acetylglucosamine - metabolism | Heat Shock Transcription Factors | DNA-Binding Proteins - metabolism | Pancreatic Neoplasms - drug therapy | Cell Nucleus - metabolism | Epoxy Compounds - pharmacology | Cell Death - genetics | Cell Nucleus - pathology | Cell Death - drug effects | Gene Expression Regulation, Neoplastic - drug effects | Neoplasm Proteins - genetics | Active Transport, Cell Nucleus - genetics | Gene Expression Regulation, Neoplastic - genetics | HSP70 Heat-Shock Proteins - biosynthesis | Diterpenes - pharmacology | Cell Survival - drug effects | Pancreatic Neoplasms - pathology | HSP70 Heat-Shock Proteins - genetics | Pancreatic Neoplasms - genetics | Signal Transduction - genetics | Transcription Factors - genetics | Acetylglucosamine - genetics | DNA-Binding Proteins - genetics | Down-Regulation - drug effects | Down-Regulation - genetics | N-Acetylglucosaminyltransferases - metabolism | Transcription Factors - metabolism | Phenanthrenes - pharmacology | Cell Nucleus - genetics | NF-kappa B - genetics | Signal Transduction - drug effects | Sp1 Transcription Factor - genetics | Active Transport, Cell Nucleus - drug effects | Cell Line, Tumor | N-Acetylglucosaminyltransferases - antagonists & inhibitors | Index Medicus | Molecular Bases of Disease
Journal Article
PLoS Pathogens, ISSN 1553-7366, 10/2010, Volume 6, Issue 10, pp. e1001137 - e1001137
Certain pathogens deliver effectors into plant cells to modify host protein targets and thereby suppress immunity. These target modifications can be detected... 
SIGNALING PATHWAYS | DEFENSE RESPONSES | DISEASE-RESISTANCE GENES | MICROBIOLOGY | NUCLEOTIDE-BINDING SITE | SYSTEMIC ACQUIRED-RESISTANCE | PROGRAMMED CELL-DEATH | MOSAIC-VIRUS RESISTANCE | VIROLOGY | THALIANA | HISTONE MODIFICATIONS | TRANSCRIPTION FACTOR | PARASITOLOGY | Arabidopsis - physiology | Receptors, Immunologic - physiology | Cell Death - immunology | Immunity, Innate - genetics | Arabidopsis - immunology | Chromatin Assembly and Disassembly - genetics | Membrane Transport Proteins - physiology | Epigenesis, Genetic - physiology | Cell Death - genetics | Membrane Transport Proteins - genetics | Apoptosis Regulatory Proteins - genetics | Arabidopsis Proteins - genetics | Arabidopsis Proteins - physiology | Histone-Lysine N-Methyltransferase - genetics | Arabidopsis - metabolism | Chromatin Assembly and Disassembly - immunology | Arabidopsis - genetics | Autoimmunity - physiology | Histone-Lysine N-Methyltransferase - metabolism | Epigenesis, Genetic - immunology | Gene Expression Regulation, Plant - physiology | Apoptosis Regulatory Proteins - physiology | Receptors, Immunologic - genetics | Histone-Lysine N-Methyltransferase - physiology | Chromatin Assembly and Disassembly - physiology | Autoimmunity | Physiological aspects | Chromatin | Genetic aspects | Immune response | Arabidopsis | Index Medicus | Enzymes | Pathogens | Immunoprecipitation | Transcription | Methyltransferase | Immunity | epigenetics | Chromatin remodeling | Plant cells | DNA microarrays | histone methyltransferase | Cell death | Lysine | DNA | Transcription activation | Histones | double prime R protein | Flowering | Perception | Mutation | Methylation | Proteins | Epigenetics | Seeds | Experiments | Apoptosis
Journal Article
Neuron, ISSN 0896-6273, 08/2012, Volume 75, Issue 4, pp. 618 - 632
Mitochondrial abnormalities have been documented in Alzheimer’s disease and related neurodegenerative disorders, but the causal relationship between... 
ALZHEIMERS-DISEASE BRAIN | DOMINANT OPTIC ATROPHY | MITOCHONDRIAL-FUNCTION | MOUSE MODEL | LIGHT-CHAIN | FRONTOTEMPORAL DEMENTIA | AXONAL-TRANSPORT | NEUROSCIENCES | DYNAMIN-RELATED PROTEIN | PHOSPHORYLATION SITES | TRANSGENIC MICE | Neurons - pathology | Microtubule-Associated Proteins - genetics | Tauopathies - genetics | Cytoskeletal Proteins - genetics | Gelsolin - metabolism | Microtubule-Associated Proteins - metabolism | Humans | Actins - metabolism | Tauopathies - pathology | Cytoplasm - metabolism | MicroRNAs - metabolism | Green Fluorescent Proteins - genetics | Mitochondrial Proteins - genetics | Drosophila Proteins - metabolism | GTP-Binding Proteins - genetics | Nerve Degeneration - metabolism | Neurons - ultrastructure | tau Proteins - genetics | Cell Death - genetics | Mitochondria - genetics | Mitochondrial Proteins - metabolism | ATP Synthetase Complexes - metabolism | Cell Cycle Proteins - genetics | Tauopathies - complications | Cytoskeletal Proteins - metabolism | Myosins - metabolism | Cytoplasm - genetics | RNA Interference - physiology | Disease Models, Animal | In Situ Nick-End Labeling | Green Fluorescent Proteins - metabolism | Animals, Genetically Modified | Gene Expression Regulation - genetics | Drosophila | Cell Cycle Proteins - metabolism | Mitochondria - metabolism | Mitochondria - pathology | Mutation - genetics | Animals | GTP Phosphohydrolases - metabolism | Analysis of Variance | GTP Phosphohydrolases - genetics | Gelsolin - genetics | Mice | Drosophila Proteins - genetics | Nerve Degeneration - etiology | Voltage-Dependent Anion Channels - metabolism | GTP-Binding Proteins - metabolism | Nervous system diseases | Actin | Neurons | Utrophin | Myosin | Mitochondrial DNA | Alzheimer's disease | Proteins | Phosphorylation | Mitochondria | Neurotoxicity | Insects | Microscopy | Neurodegeneration | Pathogenesis | Morphology | Mutation | Defects | Index Medicus | Neurodegenerative diseases | Tau protein | Cell death | Elongation
Journal Article
PLoS ONE, ISSN 1932-6203, 09/2011, Volume 6, Issue 9, pp. e24248 - e24248
Glioblastoma is a highly aggressive malignant tumor involving glial cells in the human brain. We used high-throughput sequencing to comprehensively profile the... 
MALIGNANT GLIOMA | STEM-CELLS | THERAPY | HUMAN CYTOMEGALOVIRUS | BIOLOGY | RECEPTOR | PROLIFERATION | DIFFERENTIATION | CANCER | TUMORS | SIGNATURE | Genetic Therapy | Neurons - pathology | RNA Editing | Humans | MicroRNAs - metabolism | DNA Primers - genetics | Brain - metabolism | Cell Death - genetics | Glioblastoma - genetics | Base Sequence | Neurons - metabolism | Reproducibility of Results | Genes, Transgenic, Suicide - genetics | Thymidine Kinase - genetics | Transcriptome - genetics | Organ Specificity | Down-Regulation - genetics | Genetic Vectors - genetics | Glioblastoma - therapy | Herpesvirus 1, Human - enzymology | Glioblastoma - pathology | Brain - pathology | Cell Line, Tumor | Genetic Variation - genetics | High-Throughput Nucleotide Sequencing | MicroRNAs - genetics | MicroRNA | Gliomas | Cell death | Analysis | Genes | Brain tumors | Biochemistry | Genetic aspects | Gene therapy | Cancer | Enrichment | Cluster analysis | Brain | Post-transcription | Brain cancer | Glioblastoma | Herpes simplex | Viruses | Nervous system | Genomes | Thymidine | Glial cells | Kinases | Suicide | Tissues | Cancer therapies | Gene sequencing | Ganciclovir | Next-generation sequencing | Epidermal growth factor | Cell cycle | Suicide genes | Killing | Thymidine kinase | Medical research | MiRNA | Gene expression | Virology | Brain research | Insects | MicroRNAs | Medical prognosis | Stem cells | Photoreceptors | Glioblastoma cells | Cocaine | Mutation | Apoptosis | Tumors | Transcription | Gene regulation | Data processing | miRNA | glioblastoma cells | Expression vectors | suicide genes | Index Medicus
Journal Article
Oncogene, ISSN 0950-9232, 2018, Volume 38, Issue 15, pp. 2750 - 2766
Journal Article
PLoS Genetics, ISSN 1553-7390, 09/2016, Volume 12, Issue 9, pp. e1006311 - e1006311
Previous studies have shown that multivesicular bodies (MVBs)/endosomes-mediated vesicular trafficking may play key roles in plant immunity and cell death.... 
LESION MIMIC MUTANTS | DEFENSE RESPONSES | SALICYLIC-ACID | GENE | BIOSYNTHESIS | GENETICS & HEREDITY | RESISTANCE | MEMBRANE TRAFFICKING | REPEAT PROTEIN | EXPRESSION | INNATE IMMUNITY | Arabidopsis - growth & development | Plant Diseases - immunology | Endosomal Sorting Complexes Required for Transport - genetics | rab GTP-Binding Proteins - genetics | Disease Resistance - immunology | Endosomes - metabolism | Arabidopsis Proteins - metabolism | Oryza - growth & development | Cell Death - genetics | Oryza - genetics | Adenosine Triphosphatases - biosynthesis | Gene Expression Regulation, Plant | Plant Diseases - genetics | Disease Resistance - genetics | Multivesicular Bodies - immunology | Arabidopsis Proteins - genetics | Immunity, Cellular - genetics | Endosomes - genetics | Plants, Genetically Modified - genetics | Plants, Genetically Modified - immunology | Endosomal Sorting Complexes Required for Transport - metabolism | Protein Transport - genetics | Plant Leaves - immunology | Arabidopsis - genetics | Plant Proteins - genetics | Plant Leaves - genetics | Multivesicular Bodies - genetics | Adenosine Triphosphatases - genetics | Mutation | Physiological aspects | Observations | Cell death | Adenosine triphosphatase | Yeast | Developmental biology | Laboratories | Genomics | Genes | Cloning | River basins | Proteins | Collaboration | Genetics | Crop diseases | Research centers | Apoptosis | Rice | Index Medicus
Journal Article