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Neuromolecular medicine, ISSN 1535-1084, 2002
Journal
Journal Article
PLoS genetics, ISSN 1553-7404, 2016, Volume 12, Issue 9, p. e1006311
Previous studies have shown that multivesicular bodies (MVBs)/endosomes-mediated vesicular trafficking may play key roles in plant immunity and cell death... 
LESION MIMIC MUTANTS | DEFENSE RESPONSES | SALICYLIC-ACID | GENE | BIOSYNTHESIS | GENETICS & HEREDITY | RESISTANCE | MEMBRANE TRAFFICKING | REPEAT PROTEIN | EXPRESSION | INNATE IMMUNITY | Arabidopsis - growth & development | Plant Diseases - immunology | Endosomal Sorting Complexes Required for Transport - genetics | rab GTP-Binding Proteins - genetics | Disease Resistance - immunology | Endosomes - metabolism | Arabidopsis Proteins - metabolism | Oryza - growth & development | Cell Death - genetics | Oryza - genetics | Adenosine Triphosphatases - biosynthesis | Gene Expression Regulation, Plant | Plant Diseases - genetics | Disease Resistance - genetics | Multivesicular Bodies - immunology | Arabidopsis Proteins - genetics | Immunity, Cellular - genetics | Endosomes - genetics | Plants, Genetically Modified - genetics | Plants, Genetically Modified - immunology | Endosomal Sorting Complexes Required for Transport - metabolism | Protein Transport - genetics | Plant Leaves - immunology | Arabidopsis - genetics | Plant Proteins - genetics | Plant Leaves - genetics | Multivesicular Bodies - genetics | Adenosine Triphosphatases - genetics | Mutation | Physiological aspects | Observations | Cell death | Adenosine triphosphatase | Yeast | Developmental biology | Laboratories | Genomics | Genes | Cloning | River basins | Proteins | Collaboration | Genetics | Crop diseases | Apoptosis | Rice
Journal Article
Plant physiology (Bethesda), ISSN 1532-2548, 2016, Volume 170, Issue 3, pp. 1757 - 1771
... that ¹O₂-induced changes in gene expression can lead to either programmed cell death (PCD) or acclimation. A transcriptomic analysis of the mutant has allowed the identification of genes whose expression is specifically affected by each phenomenon... 
SIGNALING AND RESPONSE | AGC KINASES | MUTANT | SIGNALING PATHWAYS | OXIDATIVE STRESS | SALICYLIC-ACID | PLANT-RESPONSES | PROTEIN-KINASE | GROWTH | RESISTANCE | FLU | PLANT SCIENCES | Oligonucleotide Array Sequence Analysis | Apoptosis - genetics | Arabidopsis Proteins - metabolism | Singlet Oxygen - metabolism | Light | Photosystem II Protein Complex - genetics | Signal Transduction - radiation effects | Protein-Serine-Threonine Kinases - metabolism | Arabidopsis Proteins - genetics | Gene Expression Regulation, Plant - radiation effects | Protein-Serine-Threonine Kinases - genetics | Gene Expression Regulation, Plant - genetics | Gene Expression Profiling - methods | Signal Transduction - genetics | Cyclopentanes - pharmacology | Reverse Transcriptase Polymerase Chain Reaction | Arabidopsis - metabolism | Photosystem II Protein Complex - metabolism | Arabidopsis - genetics | Gene Expression Regulation, Plant - drug effects | Signal Transduction - drug effects | Oxylipins - pharmacology | Models, Genetic | Mutation | Plant Growth Regulators - pharmacology | Arabidopsis thaliana | Botanical research | Cell death | Physiological aspects | Plant cellular control mechanisms | Genetic aspects | Stress (Physiology) | Research | Phosphotransferases | Life Sciences
Journal Article
PLoS genetics, ISSN 1553-7404, 2013, Volume 9, Issue 5, p. e1003532
Journal Article
Nature cell biology, ISSN 1476-4679, 2015, Volume 17, Issue 10, pp. 1270 - 1281
.... Here, we present biochemical and genetic evidence that NOXA is a bona fide activator BH3... 
CYTOCHROME-C | MITOCHONDRIAL APOPTOSIS | ACTIVATION | CONFORMATIONAL-CHANGE | MEMBERS | PROAPOPTOTIC BAX | BH3 DOMAINS | PUMA | BH3-ONLY PROTEINS | MEMBRANE PERMEABILIZATION | CELL BIOLOGY | bcl-2 Homologous Antagonist-Killer Protein - genetics | Immunoblotting | BH3 Interacting Domain Death Agonist Protein - genetics | Cytochromes c - genetics | bcl-2 Homologous Antagonist-Killer Protein - metabolism | Embryo, Mammalian - metabolism | Proto-Oncogene Proteins c-bcl-2 - metabolism | Bcl-2-Like Protein 11 | Mitochondria - genetics | RNA Interference | Tumor Suppressor Proteins - genetics | Apoptosis Regulatory Proteins - genetics | Membrane Proteins - metabolism | BH3 Interacting Domain Death Agonist Protein - metabolism | bcl-2-Associated X Protein - genetics | Fibroblasts - metabolism | Proto-Oncogene Proteins - metabolism | Tumor Suppressor Proteins - metabolism | Membrane Proteins - genetics | Cytochromes c - metabolism | Mice, Inbred C57BL | Cells, Cultured | bcl-2-Associated X Protein - metabolism | Proto-Oncogene Proteins - genetics | Mitochondria - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Apoptosis Regulatory Proteins - metabolism | Mice, Knockout | Animals | Intestine, Small - cytology | Embryo, Mammalian - cytology | Models, Biological | Fibroblasts - cytology | Intestine, Small - metabolism | Proto-Oncogene Proteins c-bcl-2 - genetics | Apoptosis | Genotype | Genetic aspects | Properties | Cell death | Cellular control mechanisms
Journal Article
PloS one, ISSN 1932-6203, 09/2011, Volume 6, Issue 9, p. e24248
Glioblastoma is a highly aggressive malignant tumor involving glial cells in the human brain. We used high-throughput sequencing to comprehensively profile the... 
MALIGNANT GLIOMA | STEM-CELLS | THERAPY | HUMAN CYTOMEGALOVIRUS | BIOLOGY | RECEPTOR | PROLIFERATION | DIFFERENTIATION | CANCER | TUMORS | SIGNATURE | Genetic Therapy | Neurons - pathology | RNA Editing | Humans | MicroRNAs - metabolism | DNA Primers - genetics | Brain - metabolism | Cell Death - genetics | Glioblastoma - genetics | Base Sequence | Neurons - metabolism | Reproducibility of Results | Genes, Transgenic, Suicide - genetics | Thymidine Kinase - genetics | Transcriptome - genetics | Organ Specificity | Down-Regulation - genetics | Genetic Vectors - genetics | Glioblastoma - therapy | Herpesvirus 1, Human - enzymology | Glioblastoma - pathology | Brain - pathology | Cell Line, Tumor | Genetic Variation - genetics | High-Throughput Nucleotide Sequencing | MicroRNAs - genetics | MicroRNA | Gliomas | Cell death | Analysis | Genes | Brain tumors | Biochemistry | Genetic aspects | Gene therapy | Cancer | Enrichment | Cluster analysis | Brain | Post-transcription | Brain cancer | Glioblastoma | Herpes simplex | Viruses | Nervous system | Genomes | Thymidine | Glial cells | Kinases | Suicide | Tissues | Cancer therapies | Gene sequencing | Ganciclovir | Next-generation sequencing | Epidermal growth factor | Cell cycle | miRNA | Suicide genes | Killing | Thymidine kinase | Medical research | Gene expression | Virology | Brain research | Insects | MicroRNAs | Medical prognosis | Stem cells | Photoreceptors | Glioblastoma cells | Cocaine | Mutation | Apoptosis | Tumors | Transcription | Gene regulation | Data processing | glioblastoma cells | Expression vectors | suicide genes
Journal Article
PLoS biology, ISSN 1545-7885, 2012, Volume 10, Issue 3, p. e1001281
In eukaryotes, the classical form of programmed cell death (PCD) is apoptosis, which has as its specific characteristics DNA fragmentation and membrane depolarization... 
MESSENGER-RNAS | MAINTENANCE | MUTAGENESIS | PROTEIN | BIOCHEMISTRY & MOLECULAR BIOLOGY | FACTOR EDF | BIOLOGY | DNA GYRASE | SOS RESPONSE | STRESS | TRANSLATION | TOXIN-ANTITOXIN SYSTEM | Cell Polarity | Endoribonucleases - genetics | Cell Membrane - genetics | RNA, Messenger - metabolism | DNA-Binding Proteins - metabolism | Flow Cytometry | Escherichia coli - metabolism | Plasmids - genetics | Apoptosis Regulatory Proteins - genetics | Transcription, Genetic | Cell Membrane - metabolism | Escherichia coli - physiology | Rec A Recombinases - genetics | In Situ Nick-End Labeling | Genes, Bacterial | Rec A Recombinases - metabolism | Quorum Sensing | Endoribonucleases - metabolism | RNA, Messenger - genetics | Escherichia coli Proteins - metabolism | Microbial Viability | DNA-Binding Proteins - genetics | Plasmids - metabolism | Apoptosis Regulatory Proteins - metabolism | Microscopy, Confocal | Escherichia coli Proteins - genetics | DNA Damage | Mutation | Apoptosis | Cell death | Escherichia coli | Physiological aspects | Genetic aspects | Research | Bacterial genetics | Health aspects | Microbiology | E coli | Microscopy | DNA damage | Bacteria | Addictions | Experiments | Chromosomes | Flow cytometry | quorum sensing | pentapeptides | Confocal microscopy | Cell membranes | SOS response | mRNA | DNA repair | Depolarization | Molecular modelling | RecA protein | Cell cycle | DNA fragmentation | Antitoxins | Toxins | Membrane potential
Journal Article
by Shungin, Dmitry and Winkler, Thomas W and Croteau-Chonka, Damien C and Ferreira, Teresa and Locke, Adam E and Mägi, Reedik and Strawbridge, Rona J and Pers, Tune H and Fischer, Krista and Justice, Anne E and Workalemahu, Tsegaselassie and Wu, Joseph M. W and Buchkovich, Martin L and Heard-Costa, Nancy L and Roman, Tamara S and Drong, Alexander W and Song, Ci and Gustafsson, Stefan and Day, Felix R and Esko, Tonu and Fall, Tove and Kutalik, Zoltán and Luan, Jian’an and Randall, Joshua C and Scherag, André and Vedantam, Sailaja and Wood, Andrew R and Chen, Jin and Fehrmann, Rudolf and Karjalainen, Juha and Kahali, Bratati and Liu, Ching-Ti and Schmidt, Ellen M and Absher, Devin and Amin, Najaf and Anderson, Denise and Beekman, Marian and Bragg-Gresham, Jennifer L and Buyske, Steven and Demirkan, Ayse and Ehret, Georg B and Feitosa, Mary F and Goel, Anuj and Jackson, Anne U and Johnson, Toby and Kleber, Marcus E and Kristiansson, Kati and Mangino, Massimo and Mateo Leach, Irene and Medina-Gomez, Carolina and Palmer, Cameron D and Pasko, Dorota and Pechlivanis, Sonali and Peters, Marjolein J and Prokopenko, Inga and Stančáková, Alena and Ju Sung, Yun and Tanaka, Toshiko and Teumer, Alexander and Van Vliet-Ostaptchouk, Jana V and Yengo, Loïc and Zhang, Weihua and Albrecht, Eva and Ärnlöv, Johan and Arscott, Gillian M and Bandinelli, Stefania and Barrett, Amy and Bellis, Claire and Bennett, Amanda J and Berne, Christian and Blüher, Matthias and Böhringer, Stefan and Bonnet, Fabrice and Böttcher, Yvonne and Bruinenberg, Marcel and Carba, Delia B and Caspersen, Ida H and Clarke, Robert and Warwick Daw, E and Deelen, Joris and Deelman, Ewa and Delgado, Graciela and Doney, Alex S. F and Eklund, Niina and Erdos, Michael R and Estrada, Karol and Eury, Elodie and Friedrich, Nele and Garcia, Melissa E and Giedraitis, Vilmantas and Gigante, Bruna and Go, Alan S and Golay, Alain and Grallert, Harald and Grammer, Tanja B and Gräßler, Jürgen and Grewal, Jagvir and Groves, Christopher J and