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Journal Article
The Journal of Cell Biology, ISSN 0021-9525, 1/2007, Volume 176, Issue 2, pp. 231 - 241
Neutrophil extracellular traps (NETs) are extracellular structures composed of chromatin and granule proteins that bind and kill microorganisms. We show that... 
Oxidases | Chromatin | Cell death | Antimicrobials | Cell nucleus | Neutrophils | Nuclear membrane | Cell membranes | Neuroendocrine tumors | Apoptosis | STREPTOCOCCUS | APOPTOSIS | ACTIVATION | MYELOPEROXIDASE | REACTIVE OXYGEN | LEUKOCYTES | CHRONIC GRANULOMATOUS-DISEASE | AUTOTOXICITY | STAPHYLOCOCCUS-AUREUS | HYDROGEN-PEROXIDE | CELL BIOLOGY | Catalase - pharmacology | Leukocyte Elastase - metabolism | Chromatin - metabolism | Neutrophils - cytology | Vacuoles - ultrastructure | Reactive Oxygen Species - metabolism | Tetradecanoylphorbol Acetate - pharmacology | Apoptosis - drug effects | Staphylococcus aureus - physiology | Catalase - antagonists & inhibitors | Humans | Cell Death - immunology | Granulomatous Disease, Chronic - metabolism | Neutrophil Activation - drug effects | Nuclear Envelope - ultrastructure | fas Receptor - immunology | Immunity, Innate - physiology | Cytoplasmic Granules - metabolism | Cell Death - drug effects | Neutrophils - microbiology | Granulomatous Disease, Chronic - pathology | Cell Survival - physiology | Cell Survival - drug effects | NADPH Oxidases - antagonists & inhibitors | Enzyme Inhibitors - pharmacology | Hydrogen Peroxide - pharmacology | Neutrophils - physiology | Phagocytosis - immunology | Onium Compounds - pharmacology | Cell Survival - immunology | Microscopy, Electron | Neutrophil Activation - physiology | Antibodies - pharmacology | Nuclear Envelope - metabolism | Apoptosis - immunology | Cell Death - physiology | Cytoplasmic Granules - ultrastructure | Vacuoles - metabolism | Apoptosis - physiology | Heterochromatin | Index Medicus
Journal Article
Neuron, ISSN 0896-6273, 03/2014, Volume 81, Issue 5, pp. 1001 - 1008
Journal Article
Journal Article
Cell, ISSN 0092-8674, 2007, Volume 129, Issue 7, pp. 1287 - 1298
and are considered to be prototypical intracellular pathogens that have evolved strategies to enable growth in the intracellular phagosomes. In contrast, we... 
CELLBIO | HUMDISEASE | MICROBIO | HOST-CELLS | II MOLECULES | DENDRITIC CELLS | BOVIS BCG | PHAGOSOMAL MEMBRANES | BIOCHEMISTRY & MOLECULAR BIOLOGY | ALVEOLAR MACROPHAGES | INFECTION | VIRULENCE | CALMETTE-GUERIN | CROSS-PRESENTATION | CELL BIOLOGY | Immunohistochemistry | Cell Proliferation | Intracellular Membranes - physiology | Humans | Myeloid Cells - physiology | Phagosomes - physiology | Myeloid Cells - ultrastructure | Lysosomes - physiology | Cell Movement - physiology | Intracellular Membranes - ultrastructure | Mycobacterium tuberculosis - physiology | Mycobacterium - ultrastructure | Phagosomes - ultrastructure | Membrane Proteins - metabolism | Mycobacterium leprae - physiology | Biomarkers - metabolism | Mycobacterium leprae - ultrastructure | Microscopy, Electron, Transmission | Host-Parasite Interactions - physiology | Mycobacterium - physiology | Cells, Cultured | Cytosol - ultrastructure | Mycobacterium tuberculosis - ultrastructure | Mycobacterium leprae - genetics | Gene Expression Regulation, Bacterial - physiology | Mycobacterium - genetics | Cell Compartmentation - physiology | Cell Division - physiology | Lysosomes - ultrastructure | Cell Death - physiology | Cytosol - physiology | Antigen Presentation - physiology | Myeloid Cells - microbiology | Mycobacterium tuberculosis - genetics | Tuberculosis | Index Medicus
Journal Article
Journal Article
Cell Reports, ISSN 2211-1247, 06/2014, Volume 7, Issue 6, pp. 1796 - 1808
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 05/2009, Volume 119, Issue 5, pp. 1109 - 1123
Imatinib mesylate (IM), a potent inhibitor of the BCR/ABL tyrosine kinase, has become standard first-line therapy for patients with chronic myeloid leukemia... 
CHRONIC MYELOGENOUS LEUKEMIA | MEDICINE, RESEARCH & EXPERIMENTAL | MALIGNANT GLIOMA-CELLS | BLAST CRISIS | CLINICAL RESISTANCE | BCR-ABL MUTATIONS | ENDOPLASMIC-RETICULUM | CYTOCHROME-C RELEASE | CASPASE ACTIVATION | IMATINIB RESISTANCE | CHRONIC MYELOID-LEUKEMIA | Transcription Factor CHOP - genetics | Neoplastic Stem Cells - cytology | Gene Expression - drug effects | Calcium - metabolism | Gene Expression - genetics | Microtubule-Associated Proteins - metabolism | Neoplastic Stem Cells - drug effects | Humans | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - drug therapy | Endoplasmic Reticulum - metabolism | Antineoplastic Agents - therapeutic use | Autophagy - physiology | Thiazoles - therapeutic use | Autophagy - drug effects | Chloroquine - pharmacology | Neoplastic Stem Cells - metabolism | RNA Interference | Endoplasmic Reticulum - drug effects | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - pathology | Macrolides - pharmacology | Antineoplastic Agents - pharmacology | Cell Death - drug effects | Dasatinib | Chloroquine - therapeutic use | Piperazines - therapeutic use | Pyrimidines - pharmacology | Imatinib Mesylate | Piperazines - pharmacology | Mice, Inbred C3H | Xenograft Model Antitumor Assays | Fusion Proteins, bcr-abl - genetics | Animals | Cell Death - physiology | Protein Kinase Inhibitors - therapeutic use | Pyrimidines - therapeutic use | Fusion Proteins, bcr-abl - antagonists & inhibitors | Cell Line, Tumor | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - metabolism | Mice | Protein Kinase Inhibitors - pharmacology | Thiazoles - pharmacology | Benzamides | Macrolides - therapeutic use | Protein-Tyrosine Kinases - antagonists & inhibitors | Causes of | Physiological aspects | Genetic aspects | Chronic myeloid leukemia | Research | Drug therapy | Phagocytosis | Index Medicus |