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Journal Article
British Journal of Cancer, ISSN 0007-0920, 05/2014, Volume 110, Issue 10, pp. 2593 - 2603
Journal Article
Nature Medicine, ISSN 1078-8956, 11/2007, Volume 13, Issue 11, pp. 1349 - 1358
Data providing direct evidence for a causative link between endothelial dysfunction, microvascular disease and diabetic end-organ damage are scarce. Here we... 
MEDICINE, RESEARCH & EXPERIMENTAL | POLY(ADP-RIBOSE) POLYMERASE INHIBITORS | ANTICOAGULANT PATHWAY | BIOCHEMISTRY & MOLECULAR BIOLOGY | MOUSE | ALPHA | CELL BIOLOGY | THROMBOMODULIN | INFLAMMATION | DYSFUNCTION | CELL | RECEPTOR-1 | SEVERE SEPSIS | Kidney Glomerulus - blood supply | Diabetic Nephropathies - enzymology | Protein C - physiology | Diabetes Mellitus, Experimental - enzymology | Humans | Diabetes Mellitus, Experimental - genetics | Podocytes - enzymology | Apoptosis - genetics | Endothelium, Vascular - enzymology | Thrombomodulin - physiology | Diabetes Mellitus, Experimental - prevention & control | Mice, Mutant Strains | Diabetic Nephropathies - prevention & control | Kidney Glomerulus - enzymology | Diabetic Nephropathies - pathology | Mice, Inbred C57BL | Cells, Cultured | Diabetic Nephropathies - genetics | Mice, Transgenic | Signal Transduction - genetics | Kidney Glomerulus - pathology | Podocytes - pathology | Protein C - biosynthesis | Animals | Microcirculation - pathology | Microcirculation - enzymology | Diabetes Mellitus, Experimental - pathology | Amino Acid Substitution - genetics | Cytoprotection - genetics | Endothelium, Vascular - pathology | Mice | Enzyme Activation - genetics | Cell Line, Transformed | Protein C - genetics | Proteins | Diabetes | Kidney diseases | Cellular biology | Rodents | Apoptosis
Journal Article
Journal of the American Society of Nephrology, ISSN 1046-6673, 06/2014, Volume 25, Issue 6, pp. 1237 - 1254
Journal Article
Molecular Cell, ISSN 1097-2765, 12/2018, Volume 72, Issue 5, pp. 823 - 835.e5
High-risk human papilloma viruses (HPVs) cause cervical, anal, and oropharyngeal cancers, unlike the low-risk HPVs, which cause benign lesions. E6 oncoproteins... 
USP46 | Cdt2 | E6 | oncogenic virus | cervical cancer | deubiquitinase | HPV | CRL4(CDT2) UBIQUITIN LIGASE | MATRIX-ASSOCIATED PROTEIN | HUMAN-PAPILLOMAVIRUS TYPE-16 | BIOCHEMISTRY & MOLECULAR BIOLOGY | UBIQUITYLATION | DEGRADATION | CELL-CYCLE | REGULATED NUCLEAR | EXPRESSION | PROMOTES | CELL BIOLOGY | RNA, Small Interfering - genetics | Humans | Uterine Cervical Neoplasms - pathology | Recombinant Fusion Proteins - metabolism | DNA-Binding Proteins - metabolism | Papillomavirus Infections - pathology | Injections, Intralesional | Human papillomavirus 18 - metabolism | Female | Oncogene Proteins, Viral - genetics | Papillomavirus Infections - virology | Human papillomavirus 18 - genetics | Nuclear Proteins - genetics | Repressor Proteins - metabolism | Oncogene Proteins, Viral - metabolism | Endopeptidases - metabolism | Histone-Lysine N-Methyltransferase - genetics | Human papillomavirus 16 - pathogenicity | Uterine Cervical Neoplasms - enzymology | Signal Transduction | Papillomavirus Infections - enzymology | Gene Expression Regulation | Ubiquitin-Protein Ligases - metabolism | Repressor Proteins - genetics | Nuclear Proteins - metabolism | DNA-Binding Proteins - genetics | Uterine Cervical Neoplasms - genetics | Human papillomavirus 16 - metabolism | Papillomavirus Infections - genetics | Xenograft Model Antitumor Assays | Animals | Endopeptidases - genetics | Cell Cycle | Histone-Lysine N-Methyltransferase - metabolism | Host-Pathogen Interactions - genetics | Human papillomavirus 18 - pathogenicity | Cell Line, Tumor | Recombinant Fusion Proteins - genetics | Uterine Cervical Neoplasms - virology | Mice | HeLa Cells | Ubiquitin-Protein Ligases - genetics | Human papillomavirus 16 - genetics | RNA, Small Interfering - metabolism | Ubiquitin | Cellular proteins | Epigenetic inheritance | Growth | Ligases | Health aspects | Papillomavirus infections | Cervical cancer | Cancer
Journal Article
Circulation Research, ISSN 0009-7330, 11/2016, Volume 119, Issue 11, pp. 1183 - 1189
Journal Article
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