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PLoS Genetics, ISSN 1553-7390, 2015, Volume 11, Issue 12, p. e1005634
...), mediates these functions of EGFR signaling. Depleting Cic in ISCs activated them for division, whereas overexpressed Cic inhibited ISC proliferation and midgut regeneration... 
ACTIVATION | CANCER GENOMICS | PATHWAY | MIDGUT | GROWTH | TORSO | GENETICS & HEREDITY | PROTEINS | EXPRESSION | RECEPTOR TYROSINE KINASE | TRANSCRIPTIONAL RESPONSE | Proto-Oncogene Proteins c-ets - genetics | ErbB Receptors - genetics | Stem Cells - cytology | HMGB Proteins - biosynthesis | Proto-Oncogene Proteins - biosynthesis | Drosophila Proteins - biosynthesis | RNA, Messenger - biosynthesis | Receptors, Invertebrate Peptide - genetics | Gene Expression Regulation, Developmental | HMGB Proteins - genetics | Nerve Tissue Proteins - biosynthesis | Drosophila - genetics | Cell Proliferation - genetics | Intestines - growth & development | Repressor Proteins - genetics | Proto-Oncogene Proteins - genetics | Transcription Factors - biosynthesis | Signal Transduction - genetics | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Nerve Tissue Proteins - genetics | Animals | Repressor Proteins - biosynthesis | Drosophila Proteins - genetics | DNA-Binding Proteins - biosynthesis | Intestines - cytology | Cell proliferation | Cellular signal transduction | Genetic aspects | Observations | Drosophila | Stem cells | Medical research | Transcription factors | Genes | Cell division | Homeostasis | Infections | Proteins | Studies | Cell growth | Epidermal growth factor | Insects | Ligands | Mutation
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