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Molecular and cellular biology, ISSN 0270-7306, 2008, Volume 28, Issue 7, pp. 2426 - 2436
Article Usage Stats Services MCB Citing Articles Google Scholar PubMed Related Content Social Bookmarking CiteULike Delicious Digg Facebook Google+ Mendeley... 
INACTIVATION | APOPTOSIS | PROTEIN | SIGNALING PATHWAY | BIOCHEMISTRY & MOLECULAR BIOLOGY | TRANSCRIPTION | YAP | SIZE-CONTROL | DIFFERENTIATION | DROSOPHILA | TUMOR-SUPPRESSOR PATHWAY | CELL BIOLOGY | 14-3-3 Proteins - physiology | Phosphorylation | Transferases (Other Substituted Phosphate Groups) - genetics | Humans | Receptor Protein-Tyrosine Kinases - physiology | Recombinant Fusion Proteins - physiology | Transferases (Other Substituted Phosphate Groups) - physiology | Mesoderm - cytology | Drosophila Proteins - physiology | Cell Transdifferentiation - physiology | Membrane Proteins - physiology | Cell Division | c-Mer Tyrosine Kinase | Cell Cycle Proteins - genetics | Conserved Sequence | Transcription, Genetic | Epithelial Cells - cytology | Proteins - physiology | Nerve Tissue Proteins - physiology | Transcription Factors - physiology | Membrane Proteins - genetics | Protein-Serine-Threonine Kinases - physiology | Protein-Serine-Threonine Kinases - genetics | Proto-Oncogene Proteins - genetics | Transcription Factors - genetics | Nerve Tissue Proteins - genetics | Protein Processing, Post-Translational - physiology | Amino Acid Motifs | Proteins - genetics | Receptor Protein-Tyrosine Kinases - genetics | Cell Transformation, Neoplastic | Proto-Oncogene Proteins - physiology | Cell Line, Tumor | Nuclear Proteins - physiology | Drosophila Proteins - genetics | Cell Cycle Proteins - physiology | Cell Movement
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The EMBO Journal, ISSN 0261-4189, 04/2018, Volume 37, Issue 7, p. n/a
Journal Article
Science (American Association for the Advancement of Science), ISSN 1095-9203, 2018, Volume 362, Issue 6417, pp. eaar2971 - 909
...). Multiple mouse models and transplantation assays demonstrate that proliferation of APs but not other myofibroblasts is activated by CD301b-expressing macrophages through insulin-like growth factor 1... 
FIBROBLAST HETEROGENEITY | FIBROSIS | IGF-I | GROWTH-FACTOR-C | REGENERATION | MULTIDISCIPLINARY SCIENCES | PHENOTYPE | ALPHA | PDGF-C | EXPRESSION | CELL-LINEAGE-ABLATION | Cell Proliferation | Myofibroblasts - physiology | Mesenchymal Stem Cells - physiology | Transcriptome | Extracellular Matrix - metabolism | Adipocytes - physiology | Skin - injuries | Re-Epithelialization - physiology | Lectins, C-Type - metabolism | Lectins, C-Type - analysis | Lymphokines - metabolism | Skin Aging - physiology | Wound Healing - genetics | Skin - pathology | Skin - immunology | Macrophages - physiology | Keloid - pathology | Mice, Inbred C57BL | Platelet-Derived Growth Factor - metabolism | Animals | Re-Epithelialization - genetics | Fibrosis | Mice | Wound Healing - physiology | Integrin beta1 - genetics | Fibroblasts | Physiological aspects | Skin | Macrophages | Flow cytometry | Animal models | Populations | CD26 antigen | Platelet-derived growth factor | Transcription | Fluorescence | Smooth muscle | Transplantation | Insulin-like growth factors | Adipocytes | CD29 antigen | Cell surface | Heterogeneity | Signal transduction | Bleomycin | Precursors | Actin | Aging | Extracellular matrix | Repair | Growth factors | Injuries | Deposition | Subpopulations | Composition | Myeloid cells | Wound healing | Muscles | Gene expression | Insulin | Ablation | Resilience | Scars | Molecular modelling | Collagen | Galactose | Surface markers
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